ライフサイエンスコーパス: quadriceps

1 art, diaphragm, lung, fat, gastrocnemius and quadriceps.

2 ts in motoneurons innervating muscles of the quadriceps.

3 ning, and increased muscle fiber loss in the quadriceps.

4 maximum (1RM) per exercise) emphasizing the quadriceps.

5 ex I defect, with 85% A3243G mutation in his quadriceps.

6 ith the oxidative capacity and volume of the quadriceps.

7 atrix genes than did those from the mdx(5cv) quadriceps.

8 igher in mdx(5cv) diaphragm than in mdx(5cv) quadriceps.

9 emities' muscles, with marked sparing of the quadriceps.

10 Specifically, in the quadriceps, a muscle almost exclusively composed of type

11 knee may reveal a positive posterior drawer, quadriceps active test, or Godfrey test.

12 eive surface coil was placed under the right quadriceps, allowing determination of intramuscular [PCr

13 yofibres in 76% of FSHD muscle biopsies from quadriceps and 91% from tibialis anterior.

14 icantly increase in the weight ratios of the quadriceps and biceps muscles to the whole body.

15 excitation was produced by afferents of the quadriceps and deep peroneal muscle nerves (which discha

16 CCR2 deficiency reduced quadriceps and diaphragm muscle damage and fibrosis at 1

17 of wild-type (WT) and Ccr2(-/-) mice, and in quadriceps and diaphragm muscles of mdx(5cv) mice, a mou

18 d in acutely injured muscles and in mdx(5cv) quadriceps and diaphragm muscles.

19 rmance on the balance board (P < 0.001), and quadriceps and hamstring strength also increased signifi

20 ance was assessed using a balance board, and quadriceps and hamstring strength were measured by an is

21 aneously recording surface electromyography (quadriceps and hamstrings).

22 s also attenuated atrophy in the nonoperated quadriceps and in the hamstring and adductor muscles of

23 In vivo analyses of quadriceps and in vitro analyses of vastus lateralis mus

24 was reduced during postural tasks involving quadriceps and soleus co-contraction (standing and late-

25 task, which also involved co-contraction of quadriceps and soleus, was greater than during matched v

26 ty, delayed degeneration of skeletal muscle (quadriceps), and shifted the profiles of the serum metab

27 ear cells from human vastus lateralis, mouse quadriceps, and mouse diaphragm.

28 ion, no tissue disease in the foot/ankle and quadriceps, and no evidence of viral persistence in foot

29 For boys, a quadriceps angle of >or=15 degrees and a history of mult

30 nd for girls, Cox regression revealed that a quadriceps angle of >or=20 degrees and an injury during

31 de various skeletal abnormalities, increased quadriceps angle, generalized ligamentous laxity, and fa

32 SUVs in gluteal and quadriceps areas were 0.56+/-0.09 and 0.64+/-0.07.

33 Quadriceps atrophy resolved in 27% patients at 6 months.

34 Of the 42 patients, 23 (55%) developed quadriceps atrophy.

35 ng protein-delta, in 3-month-old A/J(dys-/-) quadriceps (before marked histopathology is evident), in

36 on of [1,2-(1)(3)C]leucine with intermittent quadriceps biopsies for determination of MPS and anaboli

37 Finally, analysis of the quadriceps biopsies suggested increased CI-MPR at wk 12

38 ng freshly isolated CD31(+) cells from human quadriceps biopsies, we found that the down-regulation o

39 ean proportion of regenerating myofibres per quadriceps biopsy was 0.48%, significantly less than 1.7

40 torque in WT (39 +/- 6%) and mdx (76 +/- 8%) quadriceps, but significant changes in NMJ morphology, n

41 In myofibers from desmin -/- quadriceps, by contrast, most costameric structures are

42 Quadriceps contractile fatigue was detected by a fall in

43 sessed at a given (weaker than 30%) level of quadriceps contraction was reduced during postural tasks

44 ely matching target forces during submaximal quadriceps contractions and in the same tasks, OA patien

45 mass (dual-energy X-ray absorptiometry) and quadriceps cross-sectional area (CSA; computed tomograph

46 Quadriceps cross-sectional area was measured by magnetic

47 Strength and quadriceps cross-sectional areas were decreased 7 days a

48 an tissue loss and a 3.2 +/- 0.9% decline in quadriceps CSA (both P < 0.01).

49 owing 12 wk of combination therapy in heart, quadriceps, diaphragm, and soleus, compared with vector

50 Quadriceps dysfunction in knee OA includes impaired prop

51 CMD was estimated via quadriceps electromyogram.

52 ive during the time trials was estimated via quadriceps electromyogram.

53 s were observed and accompanied by sustained quadriceps electromyographic activity often lasting > 2s

54 drive during the three TTs was estimated via quadriceps EMG.

55 y males who performed repeated constant-load quadriceps exercise of a moderate intensity in a whole-b

56 for interventions such as aerobic exercise, quadriceps exercises, footwear modification, education,

57 moter E-box mutation decreased expression in quadriceps, extensor digitorum longus, and soleus approx

58 ficant decrease in distal tongue, but not in quadriceps, extensor digitorum longus, soleus, or ventri

59 Quadriceps fat pad is located posterior to the quadricep

60 Quadriceps fat pad oedema detected in MRI examinations s

61 tatistically significant correlation between quadriceps fat pad oedema intensity and its dimensions,

62 Sixty-one knees with quadriceps fat pad oedema out of 457 knee MRI examinatio

63 Quadriceps fat pad signal intensity, dimensions, posteri

64 gnal intensity and morphological features of quadriceps fat pad, as well as various pathologies of th

65 The purpose of this study was to compare quadriceps fatigability in patients with varying severit

66 Furthermore, quadriceps fatigability was evaluated in 12 patients by

67 ion during the control test had the greatest quadriceps fatigue attenuation with hyperoxia (r(2) = 0.

68 O2 delivery and exacerbate exercise-induced quadriceps fatigue in healthy men.

69 H) during exercise decreases the severity of quadriceps fatigue in men.

70 Quadriceps fatigue was assessed by measuring force in re

71 Peripheral quadriceps fatigue was assessed via changes in force out

72 Exercise-induced peripheral quadriceps fatigue was assessed via changes in potentiat

73 Quadriceps fatigue was quantified by pre- to post-exerci

74 Quadriceps fatigue was quantified by the reduction in po

75 /- 6% control, P < 0.05), the attenuation of quadriceps fatigue was similar between the sexes (36 +/-

76 men's work of breathing, the attenuation of quadriceps fatigue was similar between the sexes.

77 f superimposed inspiratory muscle loading on quadriceps fatigue were highly reproducible.

78 formance and its interaction with peripheral quadriceps fatigue.

79 xercise demonstrated the most attenuation of quadriceps fatigue.

80 sting women may be especially susceptible to quadriceps fatigue.

81 nge in work of breathing is needed to reduce quadriceps fatigue.

82 a hip pain associated with abnormalities in quadriceps femoris muscle and ipsilaterally reduced dist

83 elicit motor evoked potentials (MEPs) in the quadriceps femoris muscle and structural magnetic resona

84 Quadriceps femoris muscle fibers from affected cats appe

85 time evoked by a startling stimulus) in the quadriceps femoris muscle in male and females with and w

86 reservation of voluntary motor output in the quadriceps femoris muscle.

87 esponses were recorded in biceps brachii and quadriceps femoris.

88 Quadriceps force accuracy and steadiness were determined

89 Twitch quadriceps force in response to magnetic femoral nerve s

90 sessed via changes in pre- vs. post-exercise quadriceps force in response to supramaximal magnetic fe

91 For both sexes, quadriceps force was equally reduced after the control t

92 Maximal voluntary quadriceps force was measured during eccentric, isometri

93 necrosis was also observed in diaphragms and quadriceps from mdx:mIgf+/+ mice when compared with age-

94 4-fold, caused a reduction in weight of the quadriceps, gastrocnemius, soleus, and even the heart it

95 ith rAAV1-PG9DP or placebo in the deltoid or quadriceps in one of four dose-escalating cohorts (group

96 In conclusion, the quadriceps in patients with severe chronic obstructive p

97 Furthermore, quadriceps integrated EMG, blood lactate, heart rate, an

98 ng in COPD, and low frequency fatigue of the quadriceps is an infrequent feature of incremental walki

99 entilator weaning facility failed to improve quadriceps leg strength in a majority of mechanically ve

100 le young p110beta-mKO mice possessed a lower quadriceps mass and exhibited less strength than control

101 3; P = 0.038); the same pattern was true for quadriceps maximal voluntary force (30.8 +/- 10.4 and 36

102 Quadriceps maximum voluntary contraction force and fat-f

103 Quadriceps maximum voluntary contraction force was 58.3

104 tective effects of testosterone treatment on quadriceps motoneuron morphology.

105 esulted in a decrease in dendritic length of quadriceps motoneurons in untreated animals, and this de

106 erone treatment, the activation of remaining quadriceps motoneurons was assessed using peripheral ner

107 d in decreased dendritic length in remaining quadriceps motoneurons, and this atrophy was attenuated

108 ad no effect on the number or soma volume of quadriceps motoneurons.

109 ronymous recurrent inhibition from soleus to quadriceps motor neurones is under at least two types of

110 ronymous recurrent inhibition from soleus to quadriceps motor neurones of the human lower limb.

111 We investigated the responses of quadriceps MPS to a approximately 1.7-fold increase in p

112 Acceptable M-waves from the quadriceps muscle (not obscured by stimulus artifact) we

113 issues by analyzing covariation patterns in quadriceps muscle activity during locomotion in rats.

114 MRS) transmit-receive surface coil under the quadriceps muscle allowed estimation of [PCr]; .V(O2) wa

115 -receive surface coil placed under the right quadriceps muscle allowed estimation of intramuscular [P

116 18F-FDG PET/CT scans of gluteal and quadriceps muscle area were obtained with arterial input

117 Change in quadriceps muscle atrophy was significantly associated w

118 patients receiving placebo exhibited greater quadriceps muscle atrophy, with a -14.3 +/- 3.6% change

119 Quadriceps muscle biopsies obtained before and after 10

120 see text]o2max) and microcirculation loss on quadriceps muscle biopsy (in CD31(+) immunofluorescence

121 ial occlusion pressures; cardiac output; and quadriceps muscle blood flow and oxygen tension (PMo2).

122 ts in significant contractile fatigue of the quadriceps muscle but not of the diaphragm in healthy, r

123 ed significantly improved motor performance, quadriceps muscle contractility, and sciatic nerve condu

124 Inhibition declined during quadriceps muscle contraction under all conditions exami

125 onymous recurrent inhibition during isolated quadriceps muscle contraction, but to a much lesser exte

126 Quadriceps muscle cross-sectional area increased in pati

127 easured by dual-energy x-ray absorptiometry, quadriceps muscle cross-sectional area measured by magne

128 a phosphorus magnetic resonance spectroscopy quadriceps muscle exercise-recovery study before and aft

129 y muscles (IRL) exacerbated exercise-induced quadriceps muscle fatigue (Q(tw) = -12 +/- 8% IRL-CTRL v

130 ible to work of breathing-related changes in quadriceps muscle fatigue.

131 en fluorescent protein (EGFP) in transfected quadriceps muscle fibers in living mice subjected to con

132 (COPD) develop contractile fatigue of their quadriceps muscle following endurance exercise.

133 ation (NMES), applied to the surgical limb's quadriceps muscle for the first 6 weeks following surger

134 sarcoglycan-positive fibers detected in the quadriceps muscle from all 14 mice analyzed.

135 he IC/MS assay was used to quantitate OAs in quadriceps muscle from sedentary mice compared to fatigu

136 describe the biomechanical properties of the quadriceps muscle in ICU survivors 12 months after ICU d

137 kinase activation) and GLUT4-GFP-transfected quadriceps muscle in living, anesthetized mice either mu

138 ulted in increased fatigue resistance of the quadriceps muscle in patients with COPD.

139 ves at 10 months of age, along with enhanced quadriceps muscle innervation.

140 of archvillin protein was observed in human quadriceps muscle lacking gamma-SG.

141 ty in patients with severe COPD by enhancing quadriceps muscle mass and function.

142 n, exercise training significantly increased quadriceps muscle mitochondrial capacity by 24% and VO2

143 tudy was to determine whether fatigue of the quadriceps muscle occurs after high intensity cycle exer

144 In conclusion, contractile fatigue of the quadriceps muscle occurs after high intensity cycle exer

145 d a large-scale gene expression profiling in quadriceps muscle of arctic ground squirrels, comparing

146 Muscle biopsy samples were obtained from the quadriceps muscle of both the control and exercised legs

147 r 2 kinase than were observed in Mtm1 p.R69C quadriceps muscle or in muscles from wild-type littermat

148 We studied quadriceps muscle samples from 24 patients with genetica

149 Smaller quadriceps muscle size, as measured by ultrasound in the

150 aximal exercise performance, respiratory and quadriceps muscle strength and endurance and quality-of-

151 Deficits in quadriceps muscle strength and physical performance are

152 exercise capacity (6-min walk distance), and quadriceps muscle strength corrected for body weight (QS

153 e measures included demographic information, quadriceps muscle strength, and QA using a burst-superim

154 Quadriceps muscle strengthening is a common goal in the

155 idation in myocytes cultured from human male quadriceps muscle taken from subjects with varied BMI, f

156 Quadriceps muscle weakness is an important contributor t

157 s, CP-640186 lowered hepatic, soleus muscle, quadriceps muscle, and cardiac muscle malonyl-CoA with E

158 and ear), heart (heart base and ventricle), quadriceps muscle, and the tibiotarsal joint at 1, 2, 4,

159 Following surgery to reattach his quadriceps muscle, he experienced an emotional period in

160 ne of B. burgdorferi in the ear, heart base, quadriceps muscle, skin, and tibiotarsal joint tissue of

161 oenzyme A dehydrogenase enzyme activities in quadriceps muscle.

162 maximum voluntary contractions of the right quadriceps muscle.

163 splantation underwent muscle biopsy from the quadriceps muscle.

164 The three monoarticular quadriceps muscles (vastus medialis [VM], vastus lateral

165 y (wild-type, WT) and dystrophic (mdx) mouse quadriceps muscles and evaluated transcript levels of th

166 We analyzed the coordination between quadriceps muscles during locomotion in rats before and

167 Five differences were conserved in quadriceps muscles from dystrophic mice treated with spi

168 The morphology of the soleus and quadriceps muscles from MGSKO mice appeared normal.

169 ulate in obesity, we performed lipidomics on quadriceps muscles from obese mice with impaired glucose

170 be more severely affected by weakness of the quadriceps muscles in individuals with knee OA who have

171 findings, eosinophils were prominent in the quadriceps muscles of 4-wk-old male mdx mice but no prof

172 wed many lipid droplets within the psoas and quadriceps muscles of dysferlin-deficient A/J(dys-/-) mi

173 Levels of total ATP in the quadriceps muscles of the subjects were determined durin

174 quadriceps muscles, and WSCV for gluteal and quadriceps muscles was 2.2% and 3.6%, respectively.

175 rmine the kinetics of IL-6 secretion, intact quadriceps muscles were transfected with enhanced green

176 6 (0.82-0.99), respectively, for gluteal and quadriceps muscles, and WSCV for gluteal and quadriceps

177 nly for maximum standardized uptake value in quadriceps muscles.

178 le group involved in physical activity - the quadriceps muscles.

179 ngs in permitting a large involvement of the quadriceps musculature in closed chain lower limb extens

180 ificant increases in body mass, muscle mass, quadriceps myofiber size, and survival, but other measur

181 dal lengths, conduction velocities in mutant quadriceps nerves were also reduced.

182 e C-X-C motif ligand (cxcl)13 were higher in quadriceps of ATF3-KO mice than in control mice.

183 peroxidase-1, and myosin heavy chain IIa in quadriceps of control mice but not in ATF3-KO mice.

184 3p/5p (miR-542-3p/5p) were quantified in the quadriceps of patients with chronic obstructive pulmonar

185 icroRNAs (miRNAs) that were increased in the quadriceps of patients with muscle wasting and to determ

186 In the queenless ant Dinoponera quadriceps, only the alpha female reproduces.

187 rpose of this study was to determine whether quadriceps or diaphragmatic fatigue or both occur after

188 ertrophy in gastrocnemius muscles but not in quadriceps or triceps.

189 f oxidative capacity and muscle volume - the quadriceps oxidative capacity - was 36 % of the adult va

190 Quadriceps oxidative capacity was linearly correlated wi

191 The decline in quadriceps oxidative capacity with age resulted from red

192 y fitness (CRF, VO(2peak)), muscle strength (quadriceps peak torque), body composition (%fat), and PA

193 ance spectroscopy to measure whole-body VO2, quadriceps phosphate metabolism and pH during continuous

194 in both solei (SOL(R) and SOL(L)) and right quadriceps (QD(R)), and stimulating cuffs on both poster

195 e (OR, 12.4; 95% CI, 4.53-33.77; P < 0.001), quadriceps (rectus femoris) cross-sectional area (Qcsa)

196 subjects with the slope indicating that the quadriceps represented 36 % of the VO(2) increase during

197 : muscle mass using bioelectrical impedance, quadriceps, respiratory muscle and handgrip strength, an

198 After exercise, a microarray analysis in quadriceps revealed ATF3 affects genes modulating chemot

199 were classified into four quartiles based on quadriceps size and compared.

200 We assessed motor functional capacity, quadriceps size, strength, and voluntary contractile cap

201 A low quadriceps slow-twitch (ST), oxidative (relative to fast

202 onfirmed the homogenous pattern of selective quadriceps sparing but noted additional features like as

203 associated with distal muscle weakness with quadriceps sparing.

204 f our microarray data and a qPCR analysis of quadriceps specimens from COPD patients and controls wer

205 nergy balance but had significantly impaired quadriceps strength (-21.9 +/- 6.0 Newton-meters [Nm]/kg

206 , muscle fiber atrophy (-27%), and decreased quadriceps strength (-31%).

207 Deficits in quadriceps strength (n = 27) and physical performance (n

208 95% CI, 1.65 to 4.09), and 1 SD decrease in quadriceps strength (OR, 1.49; 95% CI, 1.23 to 1.82) wer

209 chniques) with appendicular lean mass (ALM), quadriceps strength (QS), and bone mineral density (BMD)

210 Subjects with greater quadriceps strength also had less knee pain and better p

211 There was no association between quadriceps strength and cartilage loss at the tibiofemor

212 indicated that subjects with lower levels of quadriceps strength and higher levels of QAF had lower l

213 Quadriceps strength and I(KOS) score significantly stren

214 Rehabilitation should focus on quadriceps strength and improving joint mobility to impr

215 serves to moderate the relationship between quadriceps strength and physical function.

216 number of muscle deficits (low muscle mass, quadriceps strength and physical performance) and 6-minu

217 The relationship between quadriceps strength and progression of knee osteoarthrit

218 On completion of the program, quadriceps strength and QA were reassessed.

219 Quadriceps strength and QAF were measured in 105 subject

220 While controlling for baseline quadriceps strength and type of exercise therapy, the le

221 ed by a burst superimposition test to assess quadriceps strength and volitional activation 3 and 12 m

222 aseline QA was significantly associated with quadriceps strength at baseline (rho = 0.30, P < 0.01) a

223 Greater quadriceps strength at baseline was associated with incr

224 ise therapy, the level of QA did not predict quadriceps strength at the 2-month followup (beta = -0.0

225 Quadriceps strength deficits and knee flexion impairment

226 Baseline QA did not predict changes in quadriceps strength following exercise therapy.

227 Those with greater quadriceps strength had less knee pain and better physic

228 Greater quadriceps strength had no influence on cartilage loss a

229 is associated with reduced fat-free mass and quadriceps strength in COPD.

230 zyme (ACE) gene is associated with preserved quadriceps strength in COPD.

231 a ventilator weaning facility would increase quadriceps strength in mechanically ventilated patients.

232 rphism is a determinant of fat-free mass and quadriceps strength in patients with COPD.

233 smI polymorphism was associated with greater quadriceps strength in patients-37.0 +/- 13.3, 33.8 +/-

234 e deletion allele is associated with greater quadriceps strength independent of confounding factors.

235 s no significant between-group difference in quadriceps strength or health-related quality of life.

236 nrelated to exercise adherence or changes in quadriceps strength or knee pain.

237 We report for the first time that greater quadriceps strength protected against cartilage loss at

238 d to determine whether baseline QA predicted quadriceps strength scores at the 2-month followup.

239 All participants underwent quadriceps strength testing with a burst superimposition

240 e C allele of the FokI polymorphism had less quadriceps strength than did those with > or =1 T allele

241 The effect of BsmI on quadriceps strength was least apparent in patients with

242 Quadriceps strength was measured at baseline, isokinetic

243 However, greater quadriceps strength was protective against cartilage los

244 rity of knee OA, knee pain, knee motion, and quadriceps strength were also recorded.

245 for the previously identified association of quadriceps strength with ACE genotype.

246 h knee OA who will have difficulty improving quadriceps strength with exercise therapy.

247 Osteoarthritis Index, had isometric tests of quadriceps strength, and underwent weight-bearing radiog

248 ty, malalignment, proprioceptive inaccuracy, quadriceps strength, hamstring strength), activity level

249 Quadriceps strength, knee laxity and alignment, and oste

250 Quadriceps strength, medial knee laxity, and limb alignm

251 arthritis (OA) to determine the influence of quadriceps strength, medial knee laxity, limb alignment,

252 s predicted knee motion after accounting for quadriceps strength, underscoring the importance of addr

253 adjusted for age, sex, body mass index, and quadriceps strength.

254 ge, painful knee osteoarthritis, and reduced quadriceps strength.

255 he effect of the BK(2)R and ACE genotypes on quadriceps strength.

256 with and without videography, and isometric quadriceps strength.

257 (in kilograms), knee joint pain, and reduced quadriceps strength.

258 ents such as knee pain, range of motion, and quadriceps strength.

259 minimisation to balance age, GOLD stage, and quadriceps strength.

260 allele was associated with greater isometric quadriceps strength; mean (SD) 31.4 (10.8) kg for insert

261 Progressive quadriceps strengthening with or without NMES enhances c

262 y conservative, emphasizing early motion and quadriceps strengthening.

263 t 75 +/- 7 W for 21.9 +/- 1.8 min during the quadriceps studies, and for 17.7 +/- 1.9 min during the

264 adriceps fat pad is located posterior to the quadriceps tendon.

265 la becomes superficially embedded within the quadriceps tendon.

266 asuring unpotentiated and potentiated twitch quadriceps tensions before and 30 minutes after incremen

267 t change in mean pre- and postwalking twitch quadriceps tensions.

268 (Polistes canadensis) and an ant (Dinoponera quadriceps) that live in simple eusocial societies.

269 In contralateral biceps and quadriceps the responses had high thresholds and delayed

270 alized (31)P spectra were collected from the quadriceps throughout using a dual-tuned ((1)H and (31)P

271 le nerves, listed in order of effectiveness: quadriceps, tibialis posterior (throughout L6 and L7), g

272 -superimposition maximum voluntary isometric quadriceps torque test procedure.

273 s of physical therapy were poor; on average, quadriceps twitch fell by -1.02 +/- 0.71 Newtons.

274 igue was assessed via changes in potentiated quadriceps twitch force (DeltaQ(tw,pot)) as measured pre

275 igue was assessed via changes in potentiated quadriceps twitch force (DeltaQ(tw,pot)) from pre- to po

276 On one occasion, quadriceps twitch force (Q(tw)) was measured during magn

277 Quadriceps twitch force (Q(tw)), in response to supramax

278 ed with a figure-of-eight magnetic coil, and quadriceps twitch force (TwQ) was measured before and at

279 a novel, nonvolitional objective technique, quadriceps twitch force generation in response to femora

280 ontractile fatigue was detected by a fall in quadriceps twitch force postexercise.

281 .05).Maximum voluntary force and potentiated quadriceps twitch force were decreased below baseline af

282 pre- to post-exercise changes in potentiated quadriceps twitch torque (DeltaQTsingle ) evoked by elec

283 xercise were identified to positively impact quadriceps twitch.

284 erwent measures of muscle function including quadriceps ultrasound.

285 The quadriceps volume was reduced in the elderly to 67 % of

286 Oxidative capacity per quadriceps volume was reduced to 53 % of the adult value

287 In EL subjects, our results show that (i) quadriceps volume, maximum voluntary contraction isometr

288 itioning of the stretch reflex of biceps and quadriceps was abnormal in both hemizygous males and car

289 Persistent quadriceps weakness and impaired knee kinematics after r

290 There is a relationship between quadriceps weakness and knee OA in all compartments, wit

291 ing an established cohort, we tested whether quadriceps weakness in patients with COPD is influenced

292 Quadriceps weakness is a risk factor for incident knee o

293 Quadriceps weakness is an important complication of chro

294 symptoms were excluded, the relationship of quadriceps weakness to OA was attenuated, with only the

295 In women, quadriceps weakness was associated with tibiofemoral OA

296 Its clinical features (finger flexor and quadriceps weakness) and pathological features (invasion

297 igher degrees of QAF than those who may have quadriceps weakness, but do not have QAF.

298 ated with the presence of knee pain and with quadriceps weakness.

299 nervating the vastus lateralis muscle of the quadriceps were labeled with cholera toxin-conjugated ho

300 ycling - the hamstrings, gluteus maximus and quadriceps - were all lower in the elderly group.