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1 etion of the ubiquitin-like domain causes UV radiation sensitivity.
2  within the tumours without increasing their radiation sensitivity.
3 n increases in C16 ceramide accumulation and radiation sensitivity.
4 ely target such cells for potential enhanced radiation sensitivity.
5  cell cycle checkpoint defects, and ionizing radiation sensitivity.
6  methyl methanesulfonate, but only slight UV radiation sensitivity.
7 ilized as an adjuvant therapeutic to enhance radiation sensitivity.
8  determine if PI 3-kinase activity regulates radiation sensitivity.
9 ules from tumor sites and inadequate tumoral radiation sensitivity.
10 because refractory disease typically retains radiation sensitivity.
11  a novel therapeutic option to enhance tumor radiation sensitivity.
12 nalized blood-based metabolic biomarkers for radiation sensitivity.
13 features to identify parameters that predict radiation sensitivity.
14 n therapy, we studied MSI1 in the context of radiation sensitivity.
15 d in male non-small cell lung cancer (NSCLC) radiation sensitivity.
16 mitotic catastrophe and a modest increase in radiation sensitivity.
17 t lead to perceptible alterations in drug or radiation sensitivity.
18 therapy, to identify parameters that predict radiation sensitivity.
19 e and adaptive immunity as key correlates of radiation sensitivity.
20  the miR-99 family of miRNAs correlates with radiation sensitivity.
21 NAs with the doubling-time of cells or their radiation sensitivity.
22 ination of H2AX profoundly enhances ionizing radiation sensitivity.
23 esistant to many anticancer agents, enhances radiation sensitivity.
24 cing had a minimal effect on NL20 growth and radiation sensitivity.
25  predisposition, chromosomal instability and radiation sensitivity.
26 omosome instability, DNA repair defects, and radiation sensitivity.
27 identification of novel molecular markers of radiation sensitivity.
28 IP76 via a liposomal delivery system rescued radiation sensitivity.
29  which expression values are correlated with radiation sensitivity.
30 on of DNA-PKcs at these sites show increased radiation sensitivity.
31               Y240F-Pten knockin mice showed radiation sensitivity.
32 n+/-mice were equivalent to p53 null mice in radiation sensitivity.
33  independent pathway leading to induction of radiation sensitivity.
34 and SKMG-3 cells, rapamycin had no impact on radiation sensitivity.
35 hairpin ends and increased cellular ionizing radiation sensitivity.
36 terogeneous mix of cell types with differing radiation sensitivities.
37                        Differential ionizing radiation sensitivities among the hypomorphic mre11 alle
38 nt cells demonstrate an increase in ionizing radiation sensitivity and a decrease in DNA DSB repair a
39 rmore, depletion of ubc-9 and tac-1 leads to radiation sensitivity and a high incidence of males, res
40 strand break detection resulting in cellular radiation sensitivity and a predisposition to cancer.
41 dant, hydroxyethyldisulfide, caused enhanced radiation sensitivity and an inability to repair DNA dou
42 2 expression have phenotypic consequences on radiation sensitivity and cancer predisposition.
43  into why BRCA2 C-terminal deletions lead to radiation sensitivity and cancer predisposition.
44 pect to other checkpoint functions, ionizing radiation sensitivity and chromosome stability.
45 scopy is extremely challenging, due to their radiation sensitivity and complex structure.
46  exhibiting these interactions had increased radiation sensitivity and decreased ability to repair do
47 normalities such as chromosomal instability, radiation sensitivity and defects in cell-cycle checkpoi
48 zygous for null alleles of atm reproduce the radiation sensitivity and high-tumor incidence of the hu
49          Mutation of Thr2609 to Ala leads to radiation sensitivity and impaired DSB rejoining.
50 ential clinical significance with respect to radiation sensitivity and local control will be highligh
51 AD50, and XRS2 are characterized by ionizing radiation sensitivity and mitotic interhomologue hyperre
52 s and suggest new target options to increase radiation sensitivity and prevent relapse.
53 ome instability, PARP inhibitor and ionizing radiation sensitivity and reduced HR repair.
54 d organ-on-chips capture key determinants of radiation sensitivity and resistance, improving translat
55 work proposes a mechanism for the control of radiation sensitivity and suggests tools to alter the dy
56 k to seven prominent published signatures of radiation sensitivity and test for equivalence to contro
57 s about the generality of the role of ATM in radiation sensitivity and the potential use of ATM inhib
58 patients, including chromosomal instability, radiation sensitivity, and aberrant cell-cycle-checkpoin
59 actate production, PKM1 and PKM2 expression, radiation sensitivity, and cell cycle duration of GSCs a
60 nduced cell-cycle checkpoints, apoptosis and radiation sensitivity, and cellular proliferation.
61 tal arrest in Brca2-deficient embryos, their radiation sensitivity, and the association of Brca2 with
62 and composition when evaluating biomaterials radiation sensitivity, and to the development of strateg
63  role of DNA-PKcs in lymphocyte development, radiation sensitivity, and tumorigenesis, we disrupted t
64 netic loss of atm and p21 on growth control, radiation sensitivity, and tumorigenesis.
65                    In our approach, we treat radiation sensitivity as a continuous variable, signific
66                        Consistent with this, radiation sensitivity, as measured by clonogenic assay o
67  transport capacity and stepwise increase in radiation sensitivity associated with heterozygous or ho
68        HPV+ HNCs exhibited greater intrinsic radiation sensitivity (average SF2 HPV-: 0.59 vs. HPV+:
69 s of T2609 cluster phosphorylation increases radiation sensitivity but whether T2609 phosphorylation
70                            We establish that radiation sensitivity can be predicted based on gene exp
71 phenotypes, including cancer predisposition, radiation sensitivity, cell-cycle checkpoint defects, im
72 e syndrome (NBS) is characterized by extreme radiation sensitivity, chromosomal instability and cance
73 karyotic elongation factor-2 (eEF-2) kinase, radiation sensitivity complementing kinase-2 (RCK-2), an
74 osure-both features reported for nonionizing radiation sensitivity, consistent with shared mitochondr
75 er a patient-specific molecular signature of radiation sensitivity could be used to identify the opti
76 SS-depleted cells display increased ionizing radiation sensitivity, defective G2/M checkpoint, and im
77 Nbs1 and Mre11 are responsible for the human radiation sensitivity disorders Nijmegen breakage syndro
78  with radiation resistance (KEAP1/NFE2L2) or radiation sensitivity (DNA damage repair pathway).
79 gue recombination, chromosome loss, ionizing radiation sensitivity, double-strand break repair, and p
80 ediatric imaging due to children's increased radiation sensitivity, especially at younger ages.
81 ited syndrome ataxia telangiectasia, exhibit radiation sensitivity, fertility defects, and are T-cell
82 dentifying effective therapeutics to enhance radiation sensitivity for the treatment of DMG.
83 s a recessive human disease characterized by radiation sensitivity, genetic instability, immunodefici
84            Whether apoptosis is required for radiation sensitivity has been controversial.
85 ne pathways involved in tumor initiation and radiation sensitivity have been identified.
86  and molecularly targeted therapy to enhance radiation sensitivity have contributed equally to the im
87 ses to DSBs, such as cell cycle checkpoints, radiation sensitivity, immune dysfunction, infertility a
88 angiectasia characteristics such as ionizing radiation sensitivity, immunodeficiency, and infertility
89 PD52, and DEPDC1B each significantly altered radiation sensitivity in at least two cancer cell lines.
90 MLH1 produced a further increase in ionizing radiation sensitivity in both SW620 and HCT116 1-2 cells
91 covered ARFRP1 as a new target for enhancing radiation sensitivity in breast cancer.
92 data suggest that GATA TFs are important for radiation sensitivity in C. neoformans by regulating spe
93  inhibition of Akt phosphorylation increases radiation sensitivity in clonogenic assays, suggesting t
94 hd analogue in DNA, there was an increase in radiation sensitivity in HCT116 cells but not in HCT116/
95 e, and a supporting mechanism, for increased radiation sensitivity in HPV+ HNC relative to HPV- HNC.
96       ATR can functionally complement esr1-1 radiation sensitivity in S. cerevisiae.
97                             Despite moderate radiation sensitivity in the DNA-PKcs(PQR/PQR) fibroblas
98 mic lymphomagenesis and an increase in acute radiation sensitivity in vivo (the latter principally be
99 he dynamic behavior of p53 and its impact on radiation sensitivity in vivo.
100                             IC87361 enhanced radiation sensitivity in wild-type C57BL6 endothelial ce
101 onal, because mutations in this motif confer radiation sensitivity in yeast and disrupt binding at th
102 , we identified 5 TFs that are important for radiation sensitivity, including SRE1, BZP2, GAT5, GAT6,
103            We used the gene-expression-based radiation-sensitivity index and the linear quadratic mod
104 e microcrystals with low symmetry and severe radiation sensitivity, interfering with the standard tec
105 n cell-cycle checkpoint mutants and that the radiation sensitivity is a consequence of this defect.
106       Another factor that critically affects radiation sensitivity is cell-cycle regulation.
107                                          The radiation sensitivity is dependent on the size of the ma
108 erse effects (RadAEs) than controls; whether radiation-sensitivity is tied to reported chemical-sensi
109 ormation to colorectal adenocarcinoma, broad radiation sensitivity occurs in a large subset of patien
110 the RAD3 epistasis group by quantitating the radiation sensitivities of dun1, rad52, rad1, rad9, rad1
111 haracteristic can be utilized to compare the radiation sensitivities of these proteins in the two sta
112                                 The ionizing radiation sensitivity of a mutant defective for extensiv
113           We systematically investigated the radiation sensitivity of all available validated HPV+ HN
114         Although little effect of p53 on the radiation sensitivity of asynchronously growing cultures
115 nuclease, was found to increase the ionizing radiation sensitivity of both mre11Delta and mre11-H125N
116 ate, to examine the cell cycle dependence of radiation sensitivity of Brca2(Tr/Tr)/p53(-/-) compared
117 radation after IR may be responsible for the radiation sensitivity of CD34+ cells compared with tumor
118              TFP did not alter the intrinsic radiation sensitivity of glioma-initiating cells (GICs).
119  expression of p16INK4a dramatically affects radiation sensitivity of HNSCC cells.
120 s study, we investigated the role of Prx1 in radiation sensitivity of human lung cancer cells, with s
121 have examined this question by measuring the radiation sensitivity of human tumor cell lines with onc
122  Martin and colleagues hypothesized that the radiation sensitivity of intestinal organoids could pred
123 ated by virtue of its ability to correct the radiation sensitivity of irs1SF cells.
124                                   Studies of radiation sensitivity of long-term engrafting cells have
125                                     Enhanced radiation sensitivity of MIA PaCa-2/RII cells was associ
126 promote chromosomal instability and increase radiation sensitivity of mouse fibroblasts.
127 Ku70 (yKu70) deficiency reduces the ionizing radiation sensitivity of mre11Delta mutants.
128 to interact with Mre11 and to complement the radiation sensitivity of NBS cells.
129 n vitro, whereas NS-123 did not increase the radiation sensitivity of normal human astrocytes or deve
130 ild-type p16INK4a (Ad/p16) expression on the radiation sensitivity of NSCLC cell lines, all of which
131                    The results show that the radiation sensitivity of proteins depends on the mass of
132 ed by a complete suppression of the ionizing radiation sensitivity of rad55 or rad57 mutants by conco
133 0 fusion protein is also active in restoring radiation sensitivity of rec2 but is hyperactive to an e
134 ive of this study was to compare the in situ radiation sensitivity of recombinant human granulocyte c
135 can complement the slow growth phenotype and radiation sensitivity of S.pombe rad31.
136 aluating molecular markers as they relate to radiation sensitivity of solid tumors.
137                          Results confirm the radiation sensitivity of the breast in girls age 10 to 2
138  of a dominant negative MEK1 does not affect radiation sensitivity of the cell, the G2/M checkpoint o
139  highest activity in suppression of ionizing radiation sensitivity of the rad57 mutant, and Val 328 a
140 raction could be targeted for enhancing drug/radiation sensitivity of tumor cells.
141  laser trapping technique for measurement of radiation sensitivity of untreated and chemo-treated can
142 c response, thus constituting a mechanism of radiation sensitivity or resistance.
143 ic response and constituting a mechanism for radiation sensitivity or resistance.
144           There was no evidence of increased radiation sensitivity or sequelae in breast tissue heter
145 zation, but reduced NPM level does not alter radiation sensitivity per se.
146 treatment of HT-29 cells with U0126 enhanced radiation sensitivity possibly due to the accumulation o
147              The development of a successful radiation sensitivity predictive assay has been a major
148 expressing an undegradable BLM mutant showed radiation sensitivity, probably by triggering end resect
149                      The estimated values of radiation sensitivity (represented as SF2, the survival
150 owing RNF8 depletion, and mitigated ionizing radiation sensitivity resulting from RNF8 deficiency.
151                                              Radiation sensitivity studies also indicate that hyperth
152 s, inhibition of autophagy promotes enhanced radiation sensitivity through a mechanism that requires
153      Importantly, LRF loss restores ionizing radiation sensitivity to p53 null cells, making LRF an a
154        Blocking Y240 phosphorylation confers radiation sensitivity to tumors and extends survival in
155 agement of Polbeta affects DSB formation and radiation sensitivity, underscoring its role in promotin
156                                              Radiation sensitivity varies greatly between tissues.
157                                              Radiation sensitivity was demonstrated in trophectoderm-
158  tonsillar epithelial (HTE) cells, increased radiation sensitivity was seen in cell expressing HPV-16
159 ic for further investigations, we found that radiation sensitivity was significantly associated with
160     The influence of autophagy inhibition on radiation sensitivity was studied in human breast, head
161 influence of the chromosome 17-linked QTL on radiation sensitivity, we conducted studies on congenic
162 on tests showed that Nrf2 deletion increased radiation sensitivity, whereas Nrf2-inducing drugs did n
163 onment components that may lead to increased radiation sensitivity with dual anti-HER2/EGFR therapy i

 
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