ライフサイエンスコーパス: rapid eye movement

1 wed significantly higher V(E) throughout non-rapid eye movement (20.1 vs. -27.7 mL/min respectively,

2 increases in theta activity during both non-rapid eye movement and rapid eye movement sleep and a re

3 s throughout the night, including during non-rapid eye movement and rapid eye movement sleep, to repo

4 gnificant effects of pattern A human IgGs on rapid eye movement and slow-wave sleep time parameters i

5 ally similar to that of wake, accompanied by rapid eye movements and muscle atonia.

6 nkeys and humans, one such CD keeps track of rapid eye movements, and in monkeys, a circuit carrying

7 phalogram (EEG), hippocampal theta activity, rapid eye movements, autonomic activation and loss of po

8 During head restraint, mice make rapid eye movements coupled between the eyes, similar to

9 These metrics included rapid eye movement duration, features of the electroence

10 associated with increased gamma activity and rapid eye movements (EMs), and upon source modeling disp

11 the suppression of visual perception during rapid eye movements in primates, demonstrating common fu

12 NT Patients with strabismus are able to make rapid eye movements, known as saccades, toward visual ta

13 There was an inverse correlation between the rapid eye movement latency and the peak of the pupillary

14 Aergic VTA neurons elicited long-lasting non-rapid-eye-movement-like sleep resembling sedation.

15 mised if the sensor moves rapidly, as during rapid eye movements, making the period immediately after

16 t one of the prominent EEG signatures of non-rapid eye movement (non-REM) sleep and are thought to pl

17 responses at powers 3-20 mW compared to non-rapid eye movement (non-REM) sleep and wakefulness (each

18 tory electrical events that occur during non-rapid-eye-movement (non-REM) sleep(1-8) and whose disrup

19 mal sleep architecture (undifferentiated non-rapid-eye-movement [non-REM] sleep or poorly structured

20 omote the transition to wakefulness from non-rapid eye movement (NREM) and rapid eye movement (REM) s

21 ion of POA Tac1 neurons obliterates both non-rapid eye movement (NREM) and rapid eye movement (REM) s

22 tituted of two global behavioral states, non-rapid eye movement (NREM) and rapid eye movement (REM),

23 ntrained region in early visual areas in non-rapid eye movement (NREM) and REM sleep.

24 p cycles: delta (0-3 Hz) activity during non-rapid eye movement (NREM) associated stages was greater

25 dic junctions may be instantiated during non-rapid eye movement (NREM) sleep after hippocampal associ

26 reduced slow wave activity (SWA) during non-rapid eye movement (NREM) sleep and impaired long-term r

27 hosphorylation site, S551, showed longer non-rapid eye movement (NREM) sleep and increased NREMS delt

28 g/kg) at 21:00 decreased the latency to non-rapid eye movement (NREM) sleep and increased the durati

29 using HD-EEG source-localization, during non-rapid eye movement (NREM) sleep and rapid eye movement (

30 replay of task-related ensembles during non-rapid eye movement (NREM) sleep and temporal shifts that

31 ess, rapid eye movement (REM) sleep, and non-rapid eye movement (NREM) sleep are characterized by dis

32 ed rapid transitions to wakefulness from non-rapid eye movement (NREM) sleep but did not affect REM-w

33 ociates beta-amyloid pathology with both non-rapid eye movement (NREM) sleep disruption and memory im

34 ity associated with consciousness during non-rapid eye movement (NREM) sleep following parietal TMS.

35 Non-rapid eye movement (NREM) sleep is supposed to play a ke

36 clear whether learning is facilitated by non-rapid eye movement (NREM) sleep or by REM sleep, whether

37 rolateral PAG (vlPAG) powerfully promote non-rapid eye movement (NREM) sleep partly through their pro

38 42) and OSA (n = 129) groups during the non-rapid eye movement (NREM) sleep period, after controllin

39 The slow waves of non-rapid eye movement (NREM) sleep reflect experience-depen

40 thesis that initial baseline measures of non-rapid eye movement (NREM) sleep slow-wave activity (SWA)

41 larify the synergistic role of different non-rapid eye movement (NREM) sleep stages (stages 2 and 3-4

42 ighttime), XX males had more spontaneous non-rapid eye movement (NREM) sleep than XX females.

43 nNOS/NK1 neurons is directly related to non-rapid eye movement (NREM) sleep time, NREM bout duration

44 nstance, compared to wakefulness, during non-rapid eye movement (NREM) sleep TMS elicits a larger pos

45 c methods elicits rapid transitions from non-rapid eye movement (NREM) sleep to wakefulness and produ

46 examined how neural reactivations during non-rapid eye movement (NREM) sleep were causally linked to

47 During non-rapid eye movement (NREM) sleep, a global decrease in sy

48 ne in CMR(glc(ox)) during anesthesia and non-rapid eye movement (NREM) sleep, and another, the invers

49 During non-rapid eye movement (NREM) sleep, cortical neurons altern

50 z) in LS, with strongest coupling during non-rapid eye movement (NREM) sleep, followed by waking immo

51 reased EEG delta (0.5-4 Hz) power during non-rapid eye movement (NREM) sleep, increased time spent in

52 During non-rapid eye movement (NREM) sleep, neuronal populations in

53 dles are thalamocortical oscillations in non-rapid eye movement (NREM) sleep, that play an important

54 , odors were presented to one nostril in non-rapid eye movement (NREM) sleep.

55 cally-dominant consolidated trace during non-rapid eye movement (NREM) sleep.

56 ontent of stimuli was conserved in light non-rapid eye movement (NREM) sleep.

57 nal neuronal oscillations characterizing non-rapid eye movement (NREM) sleep.

58 during memory consolidation processes in non-rapid eye movement (NREM) sleep.

59 thalamus, the brainstem is essential for non-rapid eye movement (NREM) sleep.

60 G) and the arousal threshold (AT) during non-rapid eye movement (NREM) sleep.

61 ple-mediated information transfer during non-rapid eye movement (NREM) sleep.

62 we identified pIII neurons that promote non-rapid eye movement (NREM) sleep.

63 G) and the arousal threshold (AT) during non-rapid eye movement (NREM) sleep.

64 In contrast, non-rapid eye movement (NREM) slow-wave oscillations offer a

65 In non-rapid eye movement (NREM) stage 3 sleep (N3), phase-lock

66 sence of PTSD in the awake state, during non-rapid eye movement (NREM) stage N2 sleep, and in a hybri

67 cal slow waves are a defining feature of non-rapid eye-movement (NREM) sleep and are thought to be im

68 During non-rapid eye-movement (NREM) sleep, cortical and thalamic n

69 iations and plausible mechanisms linking non-rapid-eye-movement (NREM) sleep disruption, Abeta, and A

70 e oscillations and sleep spindles during non-rapid-eye-movement (NREM) sleep has been proposed to sup

71 e interaction of the cardinal rhythms of non-rapid-eye-movement (NREM) sleep-the thalamo-cortical spi

72 ted from hippocampus to neocortex during non-rapid-eye-movement (NREM) sleep.

73 in intensive care units [17], induces a non-rapid-eye-movement (NREM)-like sleep but with undesirabl

74 nd network junction instantiation during non-rapid eye movement [NREM] periods) brings greater specif

75 Slow-wave sleep and rapid eye movement (or paradoxical) sleep have been foun

76 ime, sleep fragmentation, abnormal short non-rapid eye movement - rapid eye movement (REM) cycling, R

77 y, MK-1064 promotes sleep and increases both rapid eye movement (REM) and non-REM (NREM) sleep in rat

78 in nocturnal activity (siesta), a period of rapid eye movement (REM) and non-REM sleep, was absent i

79 We propose that rapid eye movement (REM) and slow-wave sleep contribute

80 ulsive disorders, blood pressure, urate, and rapid eye movement (REM) behaviour disorder scores.

81 ion, abnormal short non-rapid eye movement - rapid eye movement (REM) cycling, REM sleep reduction or

82 Rapid eye movement (REM) dreaming results in "emotionall

83 llyn has written a fascinating article about rapid eye movement (REM) dreams and how they promote the

84 Unlike mneomotechnically encoded material, rapid eye movement (REM) dreams are inherently difficult

85 was translated into sustained inhibition of rapid eye movement (REM) in vivo.

86 of memories, and features of sleep, such as rapid eye movement (REM) or sleep spindles, have been sh

87 ons, were more active during wakefulness and rapid eye movement (REM) sleep (wake/REM active) than du

88 lso outline key models for the regulation of rapid eye movement (REM) sleep and non-REM sleep, how mu

89 , oral administration of glycine-induced non-rapid eye movement (REM) sleep and shortened NREM sleep

90 It also induced a reduction in time spent in rapid eye movement (REM) sleep and slow-wave sleep and a

91 hat these ADRB1(+) neurons are active during rapid eye movement (REM) sleep and wakefulness.

92 Waking and rapid eye movement (REM) sleep are characterized by ongo

93 Although quiet wake and rapid eye movement (REM) sleep are characterized by simi

94 Rapid eye movement (REM) sleep behavior disorder (RBD) i

95 ze the timeline, prevalence, and survival of rapid eye movement (REM) sleep behavior disorder (RBD) i

96 The presence of probable rapid eye movement (REM) sleep behavior disorder was str

97 Rapid eye movement (REM) sleep behaviour disorder (RBD)

98 ory aversive conditioning during stage 2 and rapid eye movement (REM) sleep but not following aversiv

99 ta oscillations in the waking rat and during rapid eye movement (REM) sleep by simultaneously recordi

100 Rapid eye movement (REM) sleep constitutes a distinct "t

101 The hypothesis that rapid eye movement (REM) sleep disturbances are the hall

102 tral not only to our waking life but also to rapid eye movement (REM) sleep dreams.

103 his response enhancement was proportional to rapid eye movement (REM) sleep duration.

104 Although wake and rapid eye movement (REM) sleep exhibit long timescales,

105 induce an immediate transition to waking or rapid eye movement (REM) sleep from slow-wave sleep (SWS

106 Initial theories of rapid eye movement (REM) sleep generation posited that i

107 o wakefulness, and the developmental role of rapid eye movement (REM) sleep in children.

108 ough rodent models suggest the importance of rapid eye movement (REM) sleep in spatial navigational m

109 Rapid eye movement (REM) sleep is a distinct brain state

110 Rapid eye movement (REM) sleep is a recurring part of th

111 Although rapid eye movement (REM) sleep is also associated with d

112 Rapid eye movement (REM) sleep is an important component

113 -wave sleep, whereas during the second half, rapid eye movement (REM) sleep is more predominant.

114 results of this study suggest that baseline rapid eye movement (REM) sleep may serve a protective fu

115 oneutral zone (TNZ) preferentially increases rapid eye movement (REM) sleep over non-REM (NREM) sleep

116 The occurrence of dreaming during rapid eye movement (REM) sleep prompts interest in the r

117 Notably, rapid eye movement (REM) sleep regulates emotional memor

118 The functions and underlying mechanisms of rapid eye movement (REM) sleep remain unclear.

119 Rapid eye movement (REM) sleep serves an important funct

120 ysiological, and neurobiological features of rapid eye movement (REM) sleep suggest more functions th

121 Reward provided during training enhanced rapid eye movement (REM) sleep time, increased oscillato

122 While rapid eye movement (REM) sleep was marked by decreased h

123 uated the diagnostic utility of quantitative rapid eye movement (REM) sleep without atonia analysis i

124 Wakefulness, rapid eye movement (REM) sleep, and non-rapid eye moveme

125 hing about equally during quiet wake and non-rapid eye movement (REM) sleep, and to a lesser degree d

126 During rapid eye movement (REM) sleep, behavioral unresponsiven

127 as long been implicated in the generation of rapid eye movement (REM) sleep, but the underlying circu

128 ring non-rapid eye movement (NREM) sleep and rapid eye movement (REM) sleep, in six medication-refrac

129 Narcolepsy, a disorder of rapid eye movement (REM) sleep, is characterized by exce

130 ond half of the night, which is dominated by rapid eye movement (REM) sleep, led to better discrimina

131 rates both non-rapid eye movement (NREM) and rapid eye movement (REM) sleep, strongly consolidating t

132 When dreaming during rapid eye movement (REM) sleep, we can perform complex m

133 amatergic/NOS1 neurons, which were wake- and rapid eye movement (REM) sleep-active, produced wakefuln

134 halamus actively contribute to forgetting in rapid eye movement (REM) sleep.

135 lness from non-rapid eye movement (NREM) and rapid eye movement (REM) sleep.

136 ed by high apnea and hypopnea indices during rapid eye movement (REM) sleep.

137 rtical motor activity was related to time in rapid eye movement (REM) sleep.

138 nspatial behaviors such as wheel running and rapid eye movement (REM) sleep.

139 egions and are integral to the regulation of rapid eye movement (REM) sleep.

140 latter structure shows decreased activity in rapid eye movement (REM) sleep.

141 the scalp electroencephalogram (EEG) during rapid eye movement (REM) sleep.

142 ave sought to determine which species 'have' rapid eye movement (REM) sleep.

143 to be active only during sleep, particularly rapid eye movement (REM) sleep.

144 al states, non-rapid eye movement (NREM) and rapid eye movement (REM), characterized by quiescence an

145 neurons indicate that they are both wake and rapid eye movement (REM)-sleep active.

146 hat Llewellyn's hypothesis about the lack of rapid eye movement (REM)-sleep dreaming leading to loss

147 during NREM states was lower than Awake and rapid eye movement (REM).

148 itionally, dreaming has been identified with rapid eye-movement (REM) sleep, characterized by wake-li

149 al activity and behavioral states, including rapid eye-movement (REM) sleep.

150 IEDs also induce spindles during rapid-eye movement (REM) sleep and wakefulness-behaviora

151 sm also increases wakefulness and suppresses rapid-eye movement (REM) sleep in mice and rats and redu

152 Interestingly, IIS primarily occurred during rapid-eye movement (REM) sleep, which is notable because

153 Recently, restless rapid-eye-movement (REM) sleep emerged as a robust signa

154 rstanding of the mechanisms and functions of rapid-eye-movement (REM) sleep have occurred over the pa

155 eduction in nonrapid-eye-movement (NREM) and rapid-eye-movement (REM) sleep, as well as increased sle

156 e when they are asleep, in particular during rapid-eye-movement (REM) sleep.

157 role for acetylcholine in the regulation of rapid-eye-movement (REM) sleep.

158 g active association between memories during rapid eye movement [REM] dreams followed by indexation a

159 leep time, sleep efficiency, sleep onset and rapid eye movement [REM] sleep latencies, non-REM and RE

160 Are rapid eye movements (REMs) in sleep associated with visu

161 are also described, including impairment of rapid eye movements (saccades) and the fixations intersp

162 However, this encoding of action for rapid eye movements (saccades) has remained unclear: Pur

163 -to-grasp movements are often accompanied by rapid eye movements (saccades) that displace the desired

164 explore static visual scenes by alternating rapid eye movements (saccades) with periods of slow and

165 isions are naturally served by attention and rapid eye movements (saccades), but little is known abou

166 na and visual cortex into motor commands for rapid eye movements (saccades).

167 Human observers make large rapid eye movements-saccades-to bring behaviorally relev

168 vs. -27.7 mL/min respectively, P < 0.05) and rapid eye movement sleep (16.5 vs 23.4 mL/min, P < 0.05)

169 time, and the duration of stages 1 and 2 and rapid eye movement sleep (all P < 0.001), whereas slow-w

170 nd during sleep-deprived wakefulness and non-rapid eye movement sleep (experiment 2, n = 37).

171 olute and relative total sleep time, and non-rapid eye movement sleep (N1, N2, and N3).

172 indles, defining oscillations of stage 2 non-rapid eye movement sleep (N2), mediate memory consolidat

173 Deep non-rapid eye movement sleep (NREM) and general anesthesia w

174 MD mice spent significantly less time in non-rapid eye movement sleep (NREMS) during the light phase

175 ced sleep efficiency (P = .008), and reduced rapid eye movement sleep (P = .02).

176 responding to specific behaviors, including rapid eye movement sleep (REM sleep), a sleep phase when

177 Rapid eye movement sleep (REM) and exploratory wake, bot

178 Sleep, and particularly rapid eye movement sleep (REM), has been implicated in t

179 of sleep including slow wave sleep (SWS) and rapid eye movement sleep (REM), raising the question of

180 shown fear conditioning disrupts subsequent rapid eye movement sleep (REM).

181 ular mechanisms that determine the amount of rapid eye movement sleep (REMS) and non-REMS (NREMS) rem

182 phase while spending more time in NREMS and rapid eye movement sleep (REMS) during the dark phase.

183 id eye movement sleep (stages N2 and N3) and rapid eye movement sleep (stage R) were selected from th

184 Sections during non-rapid eye movement sleep (stages N2 and N3) and rapid ey

185 primary brain vigilance states (waking, non-rapid eye movement sleep [NREM] and REM sleep) within an

186 trocytes become less synchronized during non-rapid eye movement sleep after sleep deprivation at the

187 ivity during both non-rapid eye movement and rapid eye movement sleep and a reduction of delta power

188 < 0.05) as a result of greater slow wave and rapid eye movement sleep and lower fragmentation.

189 ll pair and collectively, and across waking, rapid eye movement sleep and non-rapid eye movement slee

190 e relative contribution of two sleep states, rapid eye movement sleep and slow-wave sleep, to offline

191 Among other OSA-related variables, AHI in rapid eye movement sleep and time spent with oxygen satu

192 ty and further highlight the prospect of non-rapid eye movement sleep as a therapeutic target for mea

193 disease (PD), in 15 subjects with idiopathic rapid eye movement sleep behavior disorder (iRBD) and co

194 Isolated (or idiopathic) rapid eye movement sleep behavior disorder (iRBD) is ass

195 sporter (DAT) imaging to identify idiopathic rapid eye movement sleep behavior disorder (IRBD) patien

196 pical Parkinson syndromes (n=11), idiopathic rapid eye movement sleep behavior disorder (n=10), and h

197 Rapid eye movement sleep behavior disorder (RBD) is a pr

198 The dream enactment of rapid eye movement sleep behavior disorder (RBD) is ofte

199 variants was performed in PD (n = 1,575) and rapid eye movement sleep behavior disorder (RBD) patient

200 rior cingulate, and parietal metabolism; and rapid eye movement sleep behavior disorder (RBD) with bi

201 ysical activity, sex, constipation, possible rapid eye movement sleep behavior disorder (RBD), and sm

202 atic hypotension, mild cognitive impairment, rapid eye movement sleep behavior disorder (RBD), depres

203 is downregulated in patients with idiopathic rapid eye movement sleep behavior disorder and antedates

204 Rapid eye movement sleep behavior disorder is distinguis

205 eline, 0.65; mean follow-up, 2.88; P = .01), rapid eye movement sleep behavior disorder scores were s

206 eflected by supranuclear ophtalmoparesis and rapid eye movement sleep behavior disorder with underlyi

207 s such as insomnia, obstructive sleep apnea, rapid eye movement sleep behavior disorder, and circadia

208 Some non-motor symptoms such as hyposmia, rapid eye movement sleep behavior disorder, and constipa

209 ve daytime sleepiness, insomnia, narcolepsy, rapid eye movement sleep behavior disorder, and restless

210 rum samples from 56 patients with idiopathic rapid eye movement sleep behavior disorder, before and a

211 kers using standardized scales for hyposmia, rapid eye movement sleep behavior disorder, depression,

212 History can include prodromal features (eg, rapid eye movement sleep behavior disorder, hyposmia, co

213 inson's disease and patients with idiopathic rapid eye movement sleep behaviour disorder (iRBD) exemp

214 ients (age 65.0+/-5.6 years) with idiopathic rapid eye movement sleep behaviour disorder and 21 age/g

215 glia network dysfunction differentiated both rapid eye movement sleep behaviour disorder and Parkinso

216 nnectivity, and for loss of tracer uptake in rapid eye movement sleep behaviour disorder and Parkinso

217 lso elevated (P<0.0001) in the patients with rapid eye movement sleep behaviour disorder but lower th

218 Rapid eye movement sleep behaviour disorder has been eva

219 depression, excessive daytime sleepiness, or rapid eye movement sleep behaviour disorder in early Par

220 m baseline was observed, as reflected in the Rapid Eye Movement Sleep Behaviour Disorder Questionnair

221 roved by addition of clinical scores (UPSIT, Rapid Eye Movement Sleep Behaviour Disorder Screening Qu

222 c networks may provide markers of idiopathic rapid eye movement sleep behaviour disorder to identify

223 Rapid eye movement sleep behaviour disorder was indistin

224 In addition, eight patients with rapid eye movement sleep behaviour disorder, 10 with Par

225 tients with polysomnographically-established rapid eye movement sleep behaviour disorder, 48 patients

226 h sensitivity (96%) and specificity (74% for rapid eye movement sleep behaviour disorder, 78% for Par

227 changes are present in so-called idiopathic rapid eye movement sleep behaviour disorder, a condition

228 We identified a rapid eye movement sleep behaviour disorder-related meta

229 itive functions and were more likely to have rapid eye movement sleep behaviour disorder.

230 cations for future neuroprotective trials in rapid eye movement sleep behaviour disorder.

231 detect basal ganglia network dysfunction in rapid eye movement sleep behaviour disorder.

232 Traumatic brain injury (TBI) and rapid eye movement sleep behavioural disorder (RBD) are

233 The rapid eye movement sleep behavioural disorder (RBD) popu

234 ical features of Parkinson's disease such as rapid eye movement sleep behavioural disorder and the po

235 escia cohorts consisting of individuals with rapid eye movement sleep behavioural disorder, Parkinson

236 d that astroglial calcium signals during non-rapid eye movement sleep change in proportion to sleep n

237 Hypersomnia, rapid eye movement sleep disorder and/or narcolepsy were

238 symptoms, non-motor manifestations (such as rapid eye movement sleep disorder, anosmia, constipation

239 ctal spikes emerged predominantly during non-rapid eye movement sleep in 24-hour vEEG of Syngap1(+/-)

240 RO5256390 profoundly reduced rapid eye movement sleep in wild-type mice; these effect

241 evidence suggests that the slow waves of non-rapid eye movement sleep may function as markers to trac

242 in abnormalities in the brainstem disinhibit rapid eye movement sleep motor activity, leading to drea

243 aking that are thought to be an intrusion of rapid eye movement sleep muscle atonia into wakefulness.

244 ncreased slow wave activity power during non-rapid eye movement sleep over widespread, bilateral scal

245 res and supported by biomarkers: evidence of rapid eye movement sleep periods soon after sleep onset;

246 trast, frequent interictal spikes during non-rapid eye movement sleep predicted a reduced homeostatic

247 lectroencephalogram (delta power) during non-rapid eye movement sleep reflects homeostatic sleep need

248 p period, sleep efficiency, or slow-wave and rapid eye movement sleep stage duration (P > .30).

249 uish wake, non-rapid eye movement sleep, and rapid eye movement sleep states.

250 d intact, males had more total sleep and non-rapid eye movement sleep than females during the active

251 y clinical features and the demonstration of rapid eye movement sleep without atonia on polysomnograp

252 roencephalographic (EEG) activity during non-rapid eye movement sleep, a highly heritable trait with

253 r task are reactivated during subsequent non-rapid eye movement sleep, and disrupting this neuronal r

254 .e., greater right-sided alpha power) during rapid eye movement sleep, and during evening wakefulness

255 n the spectral profile, observed only during rapid eye movement sleep, and only at the highest dose t

256 recordings are used to distinguish wake, non-rapid eye movement sleep, and rapid eye movement sleep s

257 delta power spectrum, produced low-delta non-rapid eye movement sleep, and slightly increased wakeful

258 akefulness promotes slow-wave sleep, but not rapid eye movement sleep, during a period of low sleep p

259 During non-rapid eye movement sleep, MD firing rate decreased aroun

260 those of slow-wave sleep and paradoxical or rapid eye movement sleep, respectively.

261 During rapid eye movement sleep, the spontaneous drift of the a

262 including during non-rapid eye movement and rapid eye movement sleep, to report their thoughts in th

263 oss waking, rapid eye movement sleep and non-rapid eye movement sleep, we found preserved patterns of

264 as well as a theta power (4-7Hz) decrease in rapid eye movement sleep, were associated with disease b

265 ifted progressively from 7 Hz to 6 Hz during rapid eye movement sleep, whereas slow wave activity dec

266 isometry and is invariant across waking and rapid eye movement sleep.

267 ncephalogram (EEG) signatures of stage 2 non-rapid eye movement sleep.

268 derlying mechanism may be the suppression of rapid eye movement sleep.

269 ctive or attentive waking and paradoxical or rapid eye movement sleep.

270 rns characterized by increased percentage of rapid eye movement sleep.

271 with skeletal muscle paralysis characterizes rapid eye movement sleep.

272 sleep and a reduction of delta power in non-rapid eye movement sleep.

273 efulness and had an increased propensity for rapid eye movement sleep.

274 wave activity decreased gradually during non-rapid eye movement sleep.

275 st, the RMTg plays an essential role for non-rapid eye movement sleep.

276 ctive or attentive waking and paradoxical or rapid eye movement sleep.

277 ic, and CSF dynamics that appears during non-rapid eye movement sleep.

278 tionship with the slow wave component of non-rapid eye-movement sleep (NR) arousals.

279 served for the spike index in NR stage 2 and rapid eye-movement sleep.

280 ays were characterized by a larger amount of rapid eyes movement sleep with dominant theta waves with

281 otinic acid elicited robust increases in non-rapid-eye movement sleep (NREMS) and decreases in body t

282 drug, elicited an almost 50% increase in non-rapid-eye movement sleep (NREMS) in mice for 4 hours aft

283 gonist, induced significant increases in non-rapid-eye movement sleep (NREMS) lasting for 4-10 h.

284 e electroencephalography (EEG) traces during rapid-eye movement sleep (REM) has intrigued scientists

285 paring 46 phenotypic variables revealed that rapid-eye-movement sleep (REMS), corticosterone regulati

286 ch was associated with time in slow-wave and rapid-eye-movement sleep after training.

287 ), corticobasal syndrome (n = 1), idiopathic rapid-eye-movement sleep behavior disorder (n = 1), and

288 igue, insomnia, anosmia, hypersalivation and rapid-eye-movement sleep behaviour disorder) in the year

289 e effect of orthostatic hypotension (OH) and rapid-eye-movement sleep behavioural disorder (RBD) on s

290 marked wake-promoting effects with decreased rapid-eye-movement sleep in orexin-B saporin lesioned ra

291 In contrast, during rapid-eye-movement sleep, the neocortical tone is sustai

292 w-frequency (0.5-2.0 Hz) oscillations in non-rapid-eye-movement sleep, was significantly larger in th

293 me functional differences between waking and rapid-eye-movement sleep.

294 mes in Parkinson's Disease (SCOPA-AUT), REM (Rapid Eye Movement) Sleep Behavior Disorder Single-Quest

295 Wave Sleep, but also a disinhibition of REM (rapid eye movement) sleep, demonstrated as a shortening

296 ed between periods of presumed slow-wave and rapid-eye-movement-sleep/active-state, which were charac

297 It has been demonstrated that in sleep onset rapid eye movement (SOREM) periods in BIISS, REM sleep t

298 defining thalamocortical oscillation of non-rapid eye movement stage 2 sleep, correlate with IQ and

299 Saccades are rapid eye movements that orient the visual axis toward o

300 ure were used, including total sleep period, rapid eye movement, wake after sleep onset, absolute and