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1 ptor activation, suggesting the existence of receptor cross talk.
2 -Me-NB1 enables imaging of GluN1/GluN2B NMDA receptor cross talk.
3  to study events such as desensitization and receptor cross-talk.
4 rc homology phosphatase-1 in Ly49A/chemokine receptor cross-talk.
5 y tool to study the underlying mechanisms of receptor cross-talk.
6 molecular mechanism responsible for cytokine receptor cross-talk.
7 eta-arrestins and reveal their novel role in receptor cross-talk.
8 a novel mechanism of signal transduction and receptor cross-talk.
9 lso discuss recent advances in understanding receptor cross talk and how the activities of guidance c
10 mental data, we construct a network model of receptor cross-talk and analyze its dynamics.
11 asing interest for potentially circumventing receptor cross-talk and c-MET-mediated acquired resistan
12 s what we believe to be a novel mechanism of receptor cross-talk and highlights the potential importa
13 s provides a novel mechanism that may govern receptor cross-talk and the hierarchy of xenobiotic and
14 tolerance, we propose that G-protein coupled receptor cross-talk and the level of protein kinase C ac
15 ylinositol-3 kinase and Akt, perhaps through receptor cross talk, and that ligand-induced internaliza
16          To assess the possibility of ligand-receptor cross-talk as a developmental mechanism, we hav
17 s to the tumor and facilitating CSF1 and EGF receptor cross-talk between cancer cells and macrophages
18     These results identify a novel system of receptor cross-talk between CD40 and BCR and indicate th
19 findings suggest that Src mediates the inter-receptor cross-talk between Na(+)/K(+)-ATPase and the EG
20 tain tolerance through a mechanism involving receptor cross-talk between the BCR, TLR4, and the IL-6R
21 d heterotrimeric GTP-binding protein-coupled receptor cross talk by which D1 receptors can shift effe
22 n monomer may promote receptor clustering or receptor cross-talking for regulation of the cytoskeleto
23 n of multiple receptors has been ascribed to receptor cross talk; however, the specific molecules tha
24  been reported to trigger EGFR signaling via receptor cross-talk in cancer cells.
25                  Here we examined LH and EGF receptor cross-talk in testicular steroidogenesis using
26 eserved, indicating that membrane-to-nuclear receptor cross-talk in vivo is modest in the uterus.
27 tial endothelial-to-perivascular cell ligand-receptor cross-talk, including immune-related and angiog
28 mic analysis and subsequent investigation of receptor cross-talk indicate that growth signaling betwe
29          These results suggest that GPCR-EGF receptor cross-talk is a conserved regulator of gonadotr
30 ation, and neutrophil reactivation through a receptor cross-talk mechanism.
31 imeric receptor complex, and uncovered broad receptor cross-talk of IL-22Ralpha1 and IL-20R2 with gp1
32 was also able to engage post-transcriptional receptor cross-talk pathways since, like anti-IgM, DC-SI
33                       Integrin-growth factor receptor cross-talk plays a role in growth factor signal
34 receptor-mediated gene transcription through receptor cross-talk, possibly involving competition for
35                     The details of GPCR-iGlu receptor cross-talk should inform a better understanding
36 and an enhancement of integrin-growth factor receptor cross-talk, specifically in unanchored cell sta
37                 In this study, we describe a receptor cross-talk system that addresses this issue.
38 ignaling specifically phosphorylates TF in a receptor cross-talk that distinguishes upstream from dow
39 n cytoskeleton is a point of integration for receptor cross talk through modulation of protein dynami
40  in the signaling cascade of FPR and how two receptors cross-talk to exacerbate tumor growth.
41                The observation that collagen receptor cross-talk underlies alpha-SMA-dependent collag
42 resses chemokine receptor function, and such receptor cross-talk was based on the simple mechanism of
43       To identify a molecular basis for this receptor cross-talk, we examined ERK activation and NF-k
44 it between LAMA and LABA as a consequence of receptor cross-talk, which in turn could modify beta-2 r
45 sical association of CD4 and CCR5 results in receptor cross-talk with allosteric CD4-dependent regula
46                                 The androgen receptor cross-talks with transforming growth factor-bet
47 s increasing evidence that G-protein-coupled receptors cross-talk with growth factor receptor-mediate