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1 d levels of EEA1, a protein involved in AMPA receptor endocytosis.
2 wever, CIE treatment did not reduce the NMDA receptor endocytosis.
3 it promotes receptor insertion and decreases receptor endocytosis.
4 s can act as dominant negative inhibitors of receptor endocytosis.
5 receptor dimerization increases TRH-induced receptor endocytosis.
6 io-ligand binding and recovery after maximal receptor endocytosis.
7 rin/activator protein 2 (AP2)-mediated GABAA receptor endocytosis.
8 d specific isoforms of endophilin to enhance receptor endocytosis.
9 ults in complete abolition of ligand-induced receptor endocytosis.
10 from the plasma membrane without concomitant receptor endocytosis.
11 ptor subunit NR2B at Tyr1472 correlated with receptor endocytosis.
12 n-2, a signal transducing GTPase involved in receptor endocytosis.
13 EGFR is essential and sufficient to support receptor endocytosis.
14 gesting that stargazin does not inhibit AMPA receptor endocytosis.
15 regulated kinases 1 and 2 (ERK1/2) and rapid receptor endocytosis.
16 zed, presumptively through clathrin-mediated receptor endocytosis.
17 H1R) results in several signaling events and receptor endocytosis.
18 not necessary for ERK1/2 phosphorylation or receptor endocytosis.
19 ptors, is mediated in part by agonist-driven receptor endocytosis.
20 he tyrosine kinase in the activity-dependent receptor endocytosis.
21 ating classic complement pathway and Fcgamma receptor endocytosis.
22 -95 ubiquitination prevent NMDA-induced AMPA receptor endocytosis.
23 E3 ligase that participates in cell surface receptor endocytosis.
24 teraction did not interfere with transferrin receptor endocytosis.
25 ithout any significant effect on transferrin receptor endocytosis.
26 lerance does not necessarily seem to involve receptor endocytosis.
27 nucleotide exchange factor, ARNO, to enhance receptor endocytosis.
28 time course of ligand-stimulated muscarinic receptor endocytosis.
29 ester but had no detectable effect on basal receptor endocytosis.
30 ing this lipase to participate in muscarinic receptor endocytosis.
31 esicle fraction may have a role in muscarine receptor endocytosis.
32 mobilization, chemotaxis, and ligand-induced receptor endocytosis.
33 a role for PLD in signaling that facilitates receptor endocytosis.
34 ), a dominant negative mutant known to block receptor endocytosis.
35 r FKBP12 as a negative regulator of TGF-beta receptor endocytosis.
36 tion is correlated with an increased rate of receptor endocytosis.
37 ling activities and cessation of others upon receptor endocytosis.
38 y to allow the normal rate of ligand-induced receptor endocytosis.
39 inhibits capping of both receptors, but not receptor endocytosis.
40 t Tyr992, reduces the rate of ligand-induced receptor endocytosis.
41 activation, we postulated a role for CAM in receptor endocytosis.
42 stin as the agonist-driven switch initiating receptor endocytosis.
43 ction in clathrin mediated G protein-coupled receptor endocytosis.
44 e formation and trafficking, cytokinesis and receptor endocytosis.
45 evels of P. carinii phagocytosis and mannose receptor endocytosis.
46 ARF proteins in regulating beta2-adrenergic receptor endocytosis.
47 de or be needed indirectly in events such as receptor endocytosis.
48 ly contain NPxY motifs that are required for receptor endocytosis.
49 suppressor mutant dynamin K44A, which blocks receptor endocytosis.
50 ere weaker in strains that were defective in receptor endocytosis.
51 bits Fz7 signaling, probably by blocking Fz7 receptor endocytosis.
52 ction for monoubiquitination in alpha-factor receptor endocytosis.
53 inal tail is crucial for glucagon-stimulated receptor endocytosis.
54 ated signal transduction properties, and for receptor endocytosis.
55 nhances signaling duration with retention of receptor endocytosis.
56 ssenger pathways, and induce ligand-mediated receptor endocytosis.
57 in-coated pits (CCPs), eventually leading to receptor endocytosis.
58 ced nuclear cAMP signaling without affecting receptor endocytosis.
59 bility of TLR2 and TLR4 through promotion of receptor endocytosis.
60 s independently of B-arrestin engagement and receptor endocytosis.
61 plex, leading to inhibition of autophagy and receptor endocytosis.
62 K3 phosphorylation at Tyr980/Tyr981 followed receptor endocytosis.
63 cAMP production, and St-Ht31 increased basal receptor endocytosis.
64 ition of cAMP production and increased basal receptor endocytosis.
65 eptor levels, and enhanced postsynaptic AMPA receptor endocytosis.
66 ons, which result in altered calcium-sensing receptor endocytosis.
67 n gephyrin is only reduced after the initial receptor endocytosis.
68 ng corticosterone-induced inhibition of NMDA receptor endocytosis.
69 s (5-HT, DA, DOI, and clozapine) bring about receptor endocytosis.
70 neuronal plasma membrane, and regulation of receptor endocytosis.
71 that GPCR/14-3-3 interaction occurred after receptor endocytosis.
72 hape synaptic connectivity through localized receptor endocytosis.
73 adaptin protein-2 complex (AP2) and ensuing receptor endocytosis.
74 ell surface mGluR1a expression via decreased receptor endocytosis.
75 face heparan sulfate or a reduction in TRAIL receptor endocytosis.
76 D-95 may regulate NMDA receptor-induced AMPA receptor endocytosis.
77 een implicated in NMDA receptor-induced AMPA receptor endocytosis.
78 through the RAS-RAF-MAPK/ERK1/2 pathway and receptor endocytosis.
79 e molecular level, at least antibody induced receptor endocytosis.
80 orting protein Disabled-2 (Dab2) in TGF-beta receptor endocytosis.
81 estin SUMOylation mediates G protein-coupled receptor endocytosis.
82 and reversed by blocking ARF6 activation or receptor endocytosis.
83 partial agonism for betaarr recruitment and receptor endocytosis.
84 and Dynamin regulate cell migration by PDGF receptor endocytosis.
85 by which cell migration is regulated by PDGF receptor endocytosis.
86 portant role in activity-dependent glutamate receptor endocytosis.
87 ), a protein involved in AMPA-type glutamate receptor endocytosis.
88 losely related isoform PLIC-1 did not affect receptor endocytosis.
89 cluding DNA repair, signal transduction, and receptor endocytosis.
91 n growth factor-regulated actin assembly and receptor endocytosis, although the underlying mechanisms
92 ion stemmed from the known interplay between receptor endocytosis and actin filament formation, becau
93 tions of these molecules to include roles in receptor endocytosis and activation of MAP kinase signal
94 ptor cytoplasmic loop blocked PMA-stimulated receptor endocytosis and also prevented PMA inhibition o
95 f insulin are largely independent of insulin receptor endocytosis and are initiated by activation of
98 plicated Ruk in the regulation of apoptosis, receptor endocytosis and cytoskeletal rearrangements.
99 he channel through a mechanism that promotes receptor endocytosis and degradation and lend support to
101 and Ihog activity are mutually required for receptor endocytosis and degradation, triggered by Hedge
104 in surface AMPARs is accompanied by enhanced receptor endocytosis and dependent on proteasomal activi
105 for Vav family GEFs as regulators of ligand-receptor endocytosis and determinants of repulsive signa
109 diated by DOPr signaling and is dependent on receptor endocytosis and downstream protein kinase C sig
110 ut report here that CD14 is not required for receptor endocytosis and downstream signaling mediated b
111 e PMM (phorbol 12-monomyristate), stimulated receptor endocytosis and inhibited glycine-gated chlorid
112 motes ectopic Notch signalling by increasing receptor endocytosis and inhibiting Sanpodo trafficking
113 (EGFR) to block ligand binding and initiates receptor endocytosis and intracellular trafficking.
114 e endosomes that is involved in cell-surface receptor endocytosis and it also directly interacts with
116 dence that this form of memory requires AMPA receptor endocytosis and long-term depression of excitat
117 exposure of TRPV1 to agonists induces rapid receptor endocytosis and lysosomal degradation in both s
119 nternalization motif, resulting in defective receptor endocytosis and markedly increased TfR1 express
121 also suggest, conversely, that mechanisms of receptor endocytosis and molecular sorting may themselve
123 f spinophilin with Group I mGluRs attenuates receptor endocytosis and phosphorylation of ERK1/2, an e
124 F-dependent cell migration by promoting PDGF receptor endocytosis and Rac1 activation at the cell mem
125 myeloid lineage, regulates dynamin-dependent receptor endocytosis and recycling and is a necessary co
126 of neuronal proteins implicated in synaptic receptor endocytosis and recycling, as well as in membra
130 R4 in rvs161 cells reinitiates Ste3 a-factor receptor endocytosis and requires Cdc55 function to do s
131 UDP-GlcNAc, a sugar-nucleotide that inhibits receptor endocytosis and signaling by promoting N-acetyl
133 ecursor protein modulates alpha2A-adrenergic receptor endocytosis and signaling through disrupting ar
134 gnaling pathway and demonstrated its role in receptor endocytosis and termination of the ERK1/2 signa
135 se results suggest a role for PIP5K-Ibeta in receptor endocytosis and that the truncated enzyme compe
138 actor receptor (EGFR) is mediated in part by receptor endocytosis and trafficking to the lysosomal de
139 he MET receptor tyrosine kinase by hastening receptor endocytosis and transport to the lysosomal comp
140 mannose 6-phosphate receptors, we found that receptor endocytosis and transport to the trans-Golgi ne
143 sory deprivation most likely results in AMPA receptor endocytosis and/or lateral diffusion to the ext
144 In vivo Prk1p inhibition blocked pheromone receptor endocytosis, and caused cortical actin patches
148 of G proteins, while concurrently mediating receptor endocytosis, and some aspects of receptor signa
149 nd provide an in vivo link between arrestin, receptor endocytosis, and temporal recovery from adaptat
150 rrestin-stabilized receptor phosphorylation, receptor endocytosis, and the acceleration of mitogen-ac
151 lar signal-regulated kinase phosphorylation, receptor endocytosis, and trafficking into lysosomes.
153 sphorylation, beta-arrestin recruitment, and receptor endocytosis are all mediated primarily by GRK2/
154 The functional consequences of signaling receptor endocytosis are determined by the endosomal sor
155 determine whether beta-arrestin binding and receptor endocytosis are required for receptor dephospho
156 taxis assays, CCL22 showed dominance in both receptor endocytosis assays and heterologous competitive
157 The kinase activity of Ypk1 is required for receptor endocytosis because mutations in two residues i
158 ells results in an inhibition of transferrin receptor endocytosis because of a competition between AC
161 if accelerated CXCL12-induced wild-type (WT) receptor endocytosis but enabled CXCL12-mediated endocyt
162 3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor endocytosis but is reliably blocked by an endoc
163 hat is required for its clathrin binding and receptor endocytosis but not for its receptor binding an
164 necessary for 5-HT-mediated and DOI-mediated receptor endocytosis, but DA-mediated and clozapine-medi
165 II, WT treatment did not impair the rate of receptor endocytosis, but significantly reduced the init
166 differential regulation of G protein-coupled receptor endocytosis by different ligands, but also a di
168 ether phosphorylation is required for opioid receptor endocytosis by examining a functional, truncate
171 derived factor-1 (SDF-1), as demonstrated by receptor endocytosis, calcium mobilization, and actin po
174 ivity at endosomes was found to rely on both receptor endocytosis-dependent and -independent mechanis
175 ribe protein interactions that link zones of receptor endocytosis directly to the postsynaptic scaffo
176 2), consistent with its inability to promote receptor endocytosis, elastase did stimulate GRK6 recrui
177 xin (Bgt), facilitating the visualization of receptor endocytosis, exocytosis and delivery to synapti
178 ysosomal permeabilization and assess whether receptor endocytosis followed by trafficking to lysosome
181 ed cell extracts were performed to determine receptor endocytosis, HER surface and internalized prote
182 that decreases the basal rate of IgM antigen receptor endocytosis in altering the threshold of B-cell
183 ized and quantified basal and regulated AMPA receptor endocytosis in cultured hippocampal neurons, in
186 f transforming growth factor-beta (TGF-beta) receptor endocytosis in signaling have been investigated
187 iency induces immunoglobulin M (IgM) antigen receptor endocytosis in the absence of immune stimulatio
188 AIS master organizer, ankyrinG, antagonizes receptor endocytosis in the AIS, causes receptor accumul
189 n the amygdaloid complex, induction of NK(1) receptor endocytosis in the amygdala following immobilis
190 mber of dendritic processes undergoing NK(1) receptor endocytosis in the basolateral amygdala that wa
192 revision of our understanding of the role of receptor endocytosis in the biology of opiate drug actio
193 These data demonstrate a critical role for receptor endocytosis in the development of adverse side
197 RK1/2) activation, calcium mobilization, and receptor endocytosis] in the same cell background (human
198 ity binding of CCR5 chemokines but preserves receptor endocytosis, indicating that chemokines prefere
199 Morphine, a mu agonist triggering little mu receptor endocytosis, induced neither cross-desensitizat
200 tagonist (ifenprodil) or infusion of an AMPA receptor endocytosis inhibitor (GluA23Y) before rapamyci
201 us, our study demonstrated that Nef-mediated receptor endocytosis involves the ubiquitination motif a
204 igands that require PKC activation to effect receptor endocytosis is dependent on receptor dephosphor
207 urthermore, we find that mGluR-mediated AMPA receptor endocytosis is enhanced by CaM binding to group
209 our data indicate that the ligand-stimulated receptor endocytosis is required for CXCR2-mediated chem
211 an unresolved question in the field-whether receptor endocytosis is required for Wnt signal transduc
213 evious work uncovered that clathrin, but not receptor endocytosis, is required for EGF-stimulated Akt
214 amin mutant (Dyn/K44A) inhibited transferrin receptor endocytosis, it had no effect on phogringreen f
215 Furthermore, in both cases, inhibition of receptor endocytosis led to reduced N- and H-Ras activat
217 ated injury may involve facilitation of NMDA receptor endocytosis likely stimulated by DHPG-induced i
218 g integral to the process of cell migration, receptor endocytosis may be a terminal stop signal when
219 IP2 to EphA2 attenuates a positive signal to receptor endocytosis mediated by phosphatidylinositol 3-
220 the effect of immobilisation stress on NK(1) receptor endocytosis morphology, a marker of local subst
221 e high enough for chemotaxis but too low for receptor endocytosis, neutrophil CXCR1 and CXCR2 could b
222 vious studies have demonstrated that neither receptor endocytosis nor arrestin is required for ERK ac
223 acellular and endosomal O(2)(*-) production, receptor endocytosis, nuclear factor-kB (NF-kB) activati
224 inhibited TNFa-induced O(2)(*-) production, receptor endocytosis, nuclear factor-kB (NF-kB) activati
225 t in hippocampal neurons revealed that GABAA receptor endocytosis occurred exclusively at extrasynapt
226 overexpression did not detectably influence receptor endocytosis or the stability of the receptor pr
227 minant-negative Dyn3 did not seem to inhibit receptor endocytosis, overexpression of a specific Dyn3
229 ssociated with beta-arrestin recruitment and receptor endocytosis, promoting CCR3 internalization and
230 key structural elements important for EphA2 receptor endocytosis provide possible ways for the devel
231 fferent terminals and that agonist dependent receptor endocytosis provides evidence of a spatially an
232 hat the interplay between different modes of receptor endocytosis provides for segregation between di
233 ons and is a key player in postsynaptic AMPA receptor endocytosis, providing multiple ways of negativ
234 induction causes long-term increases in AMPA receptor endocytosis rate and dendritic synthesis of Arc
235 ntracellular domain shows similar effects on receptor endocytosis rate as that of the EpoR, but does
236 was partially explained by a 50% decrease in receptor endocytosis rate; however, at 37 degrees C, PDG
237 min after withdrawal results from increased receptor endocytosis rather than decreased exocytosis.
238 ediated CXCR2 internalization is crucial for receptor endocytosis, resensitization, and chemotaxis.
240 results indicate neither time-averaging nor receptor endocytosis significantly improves the cell's a
241 prototype, we find that myosin VI regulates receptor endocytosis, spatiotemporal localization, and s
242 d the subunit-specific contributions to NMDA receptor endocytosis, specifically defining the endocyti
244 trate that PKC activation stimulates glycine receptor endocytosis, that both constitutive endocytosis
245 ceptors upon activation, and (iii) regulates receptor endocytosis, thereby impacting the fate of acti
246 ion of NMDA receptors alone can trigger AMPA receptor endocytosis through calcium influx and activati
247 ch beta-arrestin orchestrates the process of receptor endocytosis through the activation of ADP-ribos
248 wnstream mechanisms, including autophagy and receptor endocytosis, through SCF (Skp1-Cul1-F-box)-medi
249 ges from healthy individuals reduced mannose receptor endocytosis to 53.2% (P < 0.05) and P. carinii
251 st, mutating only the Thr residues inhibited receptor endocytosis to the same extent as in the full m
252 tion, NMDA receptor activation enhances AMPA receptor endocytosis via a signaling mechanism required
254 ination is the predominant sorting signal in receptor endocytosis, we investigated whether Nef is ubi
255 cts of AFR1 in strains that are defective in receptor endocytosis were probably an indirect consequen
256 eins of the endocytic machinery and promotes receptor endocytosis, whereas C-terminally truncated ARR
258 promotion of random cell migration requires receptor endocytosis, whereas the chemotactic response t
259 arrestin1 functions as a clathrin adaptor in receptor endocytosis which is regulated by dephosphoryla
260 at the synaptic membrane and impairing AMPA receptor endocytosis, while leaving basal synaptic trans
261 aptor protein recruitment to the beta2AR and receptor endocytosis without affecting the internalizati
262 r abolished the effect of pp120 to stimulate receptor endocytosis, without affecting pp120 phosphoryl