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1 ciation from the receptor, or elimination of receptor reserve.
2 d for TPalpha-Galpha13 and in the context of receptor reserve.
3 tor apparently depends on the degree of P2Y1 receptor reserve.
4 g the SH interval indicated a small (10-20%) receptor reserve.
5 AMP accumulation revealed a relatively large receptor reserve (64%) for the maximal response.
6                               Modulating the receptor reserve also revealed acetylcholine's greater a
7  least two effectors with distinct levels of receptor reserve and that differentially reflect recepto
8 portant cases including (a) the presence of "receptor reserve" and/or partial agonism, (b) multiple r
9              Therefore, our data demonstrate receptor reserve as a novel principle of T cell activati
10 HC variant weak agonists require significant receptor reserve, because decreasing the level of T cell
11 uggesting little difference in the levels of receptor reserve between the two assays.
12 ed in IP assays performed after reduction of receptor reserve by the alkylating agent, phenoxybenzami
13 knockdown of MORs revealed that depletion of receptor reserve does not account for presynaptic resist
14 ffects on CCL3L1 affinity, although possible receptor reserve effects obscure complete interpretation
15 c and postsynaptic responses suggests that a receptor reserve exists for presynaptic inhibition, but
16                  In these cells, there is no receptor reserve for 5-HT and 5-HT2C inverse agonists di
17                                          The receptor reserve for A2A-AdoRs to cause an increase of c
18 n in female rats such that there is a larger receptor reserve for morphine-mediated antinociception.
19 gonist and Furchgott analysis revealed a low receptor reserve for the activation of GIRK channels but
20 e to the existence of a greater A1 adenosine receptor reserve for the inhibition of beta-ICa,L than f
21 r the activation of GIRK channels but a >90% receptor reserve for the inhibition of Ca(2+) events.
22 t, these data suggest a substantial level of receptor reserve for the PI response in mouse hippocampu
23                                              Receptor reserves for 3 A2A-AdoR agonists were large.
24 e of assay incubation time and the degree of receptor reserve in applying the analytical model.
25                                 Changing the receptor reserve is a key determinant of the rate of coc
26                          An implication of a receptor reserve is the expansion of the number of ligan
27  assess whether differences in the degree of receptor reserve might explain this discrepancy of resul
28 acellular receptors, which could represent a receptor reserve, near the postsynaptic density.
29 that the IP response exhibited a much larger receptor reserve than the AA response, and reduction of
30          To determine whether T cells follow receptor reserve, we have characterized the effect of re
31                                If there is a receptor reserve, when the number of receptors is altere
32          Some ligand-receptor systems have a receptor reserve where a maximal response can be achieve
33 tivity to low-potency agonists by decreasing receptor reserve without significantly altering receptor