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1 ccurring within dimers comprised of the same receptor type.
2 cell firing, and identification of dopamine receptor type.
3 ansmitter release, cell firing, and dopamine-receptor type.
4 nnervation to this sense organ employs other receptor types.
5 tors and also as versatile ligands for other receptor types.
6 y tracts, with similar distributions of both receptor types.
7 ting that GABA actions can be local for both receptor types.
9 dentified one important gene, angiotensin II receptor type 1 (AGTR1), in the Ca2+/AT-IIR/alpha-AR sig
10 2 downregulation enhances the angiotensin II receptor type 1 (AT(1) R) axis associated with oxidative
12 l evidence indicates that the angiotensin II receptor type 1 (AT1 R) is inherently mechanosensitive a
14 rize a GPCR-TRP channel pair, angiotensin II receptor type 1 (AT1R), and transient receptor potential
15 estin-1-biased agonist of the angiotensin II receptor type 1 (AT1R), stimulates acute catecholamine s
17 or signalling through BMEC AT2R, HSCP Ang-II receptor type 1 (AT1R)/AT2R or HSC/P RhoA, but not by in
19 ligomerization between the GPCRs cannabinoid receptor type 1 (CB(1)R) and 5-hydroxytryptamine 2A (5-H
20 otoxemic mice were reversed by a cannabinoid receptor type 1 (CB(1)R) inverse agonist (SR141716), and
21 d neuroprotective efficacies via cannabinoid receptor type 1 (CB1) or type 2 (CB2) or via peroxisome
22 al maturation, downregulation of cannabinoid receptor type 1 (CB1) receptors, and impaired neurite ou
23 nist/inverse agonist activity at cannabinoid receptor type 1 (CB1R) and inhibitory effect on inducibl
26 models, peripherally restricted cannabinoid receptor type 1 (CB1R) inhibitors, which are devoid of t
27 oseful movements was reverted by cannabinoid receptor type 1 (CB1r) manipulations directly into the S
30 ha2A-adrenergic receptor, GABAB, cannabinoid receptor type 1 (CB1R), and dopamine receptor type 2.
31 aptic transmission in the PL via cannabinoid receptor type 1 (CB1R)- and 2-arachidonoylglycerol-depen
32 we aimed to further evaluate the cannabinoid receptor type 1 (CB1R)-mediated mechanisms in a postnata
33 ne receptor type 4 (CXCR4) and C-C chemokine receptor type 1 (CCR1), which are the receptors of SDF-1
34 ide driven by corticotropin-releasing factor receptor type 1 (CRF1) potentiation of the anandamide ca
35 f the class B corticotropin-releasing factor receptor type 1 (CRF1R) with two peptide agonists and th
36 eurons (MSN), expressing either the dopamine receptor type 1 (D(1) -R MSN) and forming the direct, mo
37 edium spiny neurons expressing dopamine (DA) receptor type 1 (D1-MSNs) and type 2 (D2-MSNs) of the NA
39 rojection neurons (SPNs) expressing dopamine receptor type 1 (D1-SPNs) or 2 (D2-SPNs) in mice, we dem
41 ablation of this receptor-type, but not FGF receptor type 1 (FGFR1), resulted in attenuation of myel
42 ta knockout (KO) mice and perioperative IL-1 receptor type 1 (IL-1R1) blockade with the drug anakinra
43 rs, IL-1 receptor accessory protein and IL-1 receptor type 1 (IL-1R1), and enhanced sensitivity to IL
44 pertussis toxin administration leads to IL-1 receptor type 1 (IL-1R1)-dependent IL-1beta expression b
45 ticulum protein inositol 1,4,5-trisphosphate receptor type 1 (IP3R1), which triggers proteasomal degr
47 f a dimeric GPCR, the metabotropic glutamate receptor type 1 (mGluR1), by two entirely different meth
48 ar learning induced a metabotropic glutamate receptor type 1 (mGluR1)-mediated late long-term potenti
50 X-A)-expressing inputs and express both OX-A receptor type 1 (OX-1R) and cannabinoid receptor type 1
52 microscopy (cryo-EM) snapshots of ryanodine receptor type 1 (RyR1), a calcium-activated calcium chan
53 ed the effects of BPA and TBBPA on ryanodine receptor type 1 (RyR1), dihydropyridine receptor (DHPR),
54 red the binding of the capsid to transferrin receptor type 1 (TfR), particularly during virus dissoci
55 (VEGFR), vascular endothelial growth factor receptor type 1 (VEGFR1), vascular endothelial growth fa
56 rarenal angiotensinogen production and ANGII receptor type 1 activation that are associated with incr
57 pocampal slices, and hippocampal cannabinoid receptor type 1 and brain-derived neurotrophic factor (B
58 RAP; IL1R3) is a coreceptor of interleukin-1 receptor type 1 and has been found upregulated on CML st
59 diated in part through IL-1, because an IL-1 receptor type 1 antagonist ameliorated the effects of LT
61 nverting enzyme inhibitors or angiotensin-II receptor type 1 blockers may be less effective and suppo
64 complex with transforming growth factor beta receptor type 1 kinase domain were determined by X-ray c
65 eased placental HIF-1alpha in an angiotensin receptor type 1 receptor agonistic autoantibody (AT(1) -
67 at central cannabinoid receptor (cannabinoid receptor type 1) agonists decrease the power of neural o
68 a interacts with its receptor (interleukin 1 receptor type 1) on hematopoietic stem and progenitor ce
69 oexcitation is independent of angiotensin II receptor type 1, oxytocin, ionotropic glutamate and GABA
72 angiotensin II, which activates angiotensin receptor types 1 and 2, and angiotensin 1-7 and alamandi
73 ated genes included the innate interleukin 1 receptor, type 1 and the antimicrobial peptide(AMP) adre
74 incipal neurons were dependent on muscarinic receptors type 1, engaging different intracellular mecha
75 therapeutic targets include the cannabinoid receptors, type 1 and 2, as well as biosynthetic and cat
80 CRH expression, and blocking hippocampal CRH receptor type-1 (CRHR1) immediately following early-life
82 pathogenic protein tyrosine phosphatase, non-receptor type 11 (PTPN11) variant and variants of uncert
83 of PTPN14 (protein tyrosine phosphatase, non-receptor type 14) which, in turn, regulated the Hippo pa
84 al ligand of the growth hormone secretagogue receptor type 1a (GHS-R1a), is mainly secreted from the
85 receptor type 1L, bone morphogenetic protein receptor type 1A, and bone morphogenetic protein recepto
86 tivating two high-affinity G-protein-coupled receptors, type 1A (MT(1)) and type 1B (MT(2))(3,6).
87 of SMAD2 via the type 1 receptors activin A receptor type 1B (ALK4), TGFBR1, and activin A receptor
89 ptor type 1A, and bone morphogenetic protein receptor type 1B and phosphorylation of SMAD2 by ALK4, T
90 se-related microRNAs targeting the activin A receptor type 1C (ACVR1C), a component of the transformi
92 diated phosphorylation of SMAD1 by activin A receptor type 1L, bone morphogenetic protein receptor ty
93 growth factor beta (47%) and angiotensin II receptor type 2 (132%), 27% less elastin as well as conc
95 Expression of bone morphogenetic protein receptor type 2 (BMPR2) and its target, collagen IV, sig
96 gene encoding the bone morphogenetic protein receptor type 2 (BMPR2) are the commonest genetic cause
97 mutations in the bone morphogenetic protein receptor type 2 (BMPR2) gene and patients with idiopathi
98 e mutation in the bone morphogenetic protein receptor type 2 (BMPR2) gene, right ventricle (RV) dysfu
99 MO, T cells, and expression of C-C chemokine receptor type 2 (Ccr2) and C-C chemokine receptor type 5
100 stream to Fyn, MCP1 stimulated C-C chemokine receptor type 2 (CCR2) and Gi/o and inhibition of either
102 tients with a special focus on C-C chemokine receptor type 2 (CCR2) expressions on classical Mo.
105 or pharmacologic inhibition of CC chemokine receptor type 2 (CCR2) reduced macrophage (MP) infiltrat
106 40-B, a selective inhibitor of C-C chemokine receptor type 2 (CCR2), could further reduce albuminuria
107 generated MDSC were expressed C-C chemokine receptor type 2 (CCR2), which was enhanced by exposure t
111 ivated C3b, which are ligands for complement receptor type 2 (CR2/CD21), the aim of the current study
112 expression of corticotropin-releasing factor receptor type 2 (CRFR2) to be associated with post-traum
113 ect, movement-promoting pathway, or dopamine receptor type 2 (D(2) -R MSN), forming the indirect move
116 xplored role of the hydroxyl carboxylic acid receptor type 2 (HCAR2) in 2 models of neuropathic pain.
117 patients) and human epidermal growth factor receptor type 2 (HER2) (in 15,418 patients in trials wit
118 lar imaging of human epidermal growth factor receptor type 2 (HER2) expression may help to stratify b
121 effective for human epidermal growth factor receptor type 2 (HER2)-positive breast cancer but is ass
122 rated that the human epidermal growth factor receptor type 2 (HER2)-targeting ADAPT6 labeled with rad
123 31)I-GMIB-anti-human epidermal growth factor receptor type 2 (HER2)-VHH1 is a targeted radionuclide t
125 coupling, using the muscarinic acetylcholine receptor type 2 (M2R) as the primary Gi/o-coupling recep
129 erent forms of AF, have implicated ryanodine receptor type 2 (RyR2) dysfunction and enhanced spontane
130 l stimulation and nearly abolished ryanodine receptor type 2 (RyR2)-dependent sarcoplasmic Ca(2+) lea
133 by Tregs was fully abolished by blocking TNF receptor type 2 (TNFR2) or by using TNF-deficient donor
135 d role of vascular endothelial growth factor receptor type 2 (VEGFR2) in the regulation of gingival v
136 ffects on vascular endothelial growth factor receptor type 2 (VEGFR2) phosphorylation, tumor volume,
137 (VEGFR1), vascular endothelial growth factor receptor type 2 (VEGFR2), and vascular endothelial growt
139 onformational dynamics of the neuropeptide Y receptor type 2 (Y2R) during activation was investigated
140 1 receptor blockade diminished C-C chemokine receptor type 2 [CCR2(neg) (Ly6C(lo))] monocytes, attenu
141 of immunoprecipitated JMC proteins ryanodine receptor type 2 and junctophilin-2 (JPH2) followed by ma
142 of immunoprecipitated JMC proteins ryanodine receptor type 2 and junctophilin-2 (JPH2) followed by ma
143 lt of increased activity of mutant ryanodine receptor type 2 channels, sarcoplasmic reticulum Ca(2+)
145 unexplored role of hydroxyl carboxylic acid receptor type 2 in the pathophysiology of neuropathic pa
146 lar similarity to bone morphogenetic protein receptor type 2 mutation-associated heritable pulmonary
147 ing miRYR2-U10 in correcting RyR2 (Ryanodine Receptor type 2 protein) function after in vivo delivery
148 ha2A-adrenergic receptor, GABAB, or dopamine receptor type 2 receptors did not reveal any interaction
149 ortantly, pharmacological inhibition of VEGF receptor Type 2 signaling in rats causes analgesia and b
152 compatibility complex II/C-C motif chemokine receptor type 2) macrophages expressed higher levels of
156 g PIDs are mediated by inositol triphosphate receptor type 2-dependent (IP3R2-dependent) release from
157 tages of Huntington's disease, when dopamine receptor type 2-expressing striatal medium spiny neurons
161 mone receptor, human epidermal growth factor receptor type 2/neu, Ki-67) were extracted and compared
163 anogenic effect of ATP to binding purinergic receptors type 2 X7 (P2X7), which are expressed on human
164 mRNA for sweet receptor subunits T1R (Taste receptor type) 2 and 3, as well as other markers associa
165 oV-2) receptor angiotensin-converting enzyme receptor type-2 (ACE2) and can be infected with virus.
168 the present study, we show that vasopressin receptor type-2 (V2R) is localized to cilia in kidney ep
169 n an astrocyte-specific inositol 3-phosphate receptor type-2 knockout mouse, near-infrared light-indu
170 ncreased, and PKA sites Ser2808 in ryanodine receptor type-2, Ser16 in phospholamban, and Ser23/24 in
171 blance to physiological inositol 3-phosphate receptor type-2-independent Ca(2+) signals, whereas soma
172 variant in protein tyrosine phosphatase non-receptor type 22 (PTPN22) is associated with reduced ris
174 e (SS) and HCV, presence of anti- muscarinic receptor type 3 (M3R) antibodies in SS, the role that M3
177 lude C17orf104, inositol 1,4,5-trisphosphate receptor, type 3 (ITPR3), and discoidin domain receptor
178 mokine receptor type 5 and chemokine-related receptor type 4 (CCR5 and CXCR4) on peripheral blood mon
179 in-coupled receptors (GPCRs) C-X-C chemokine receptor type 4 (CXCR4) and atypical chemokine receptor
180 mpaired by the inhibitors of C-X-C chemokine receptor type 4 (CXCR4) and C-C chemokine receptor type
182 including overexpression of C-X-C chemokine receptor type 4 (CXCR4) and upregulation of plasminogen
183 onstrated that antagonism of C-X-C chemokine receptor type 4 (CXCR4) by plerixafor (AMD3100) can decr
184 The human protein tyrosine phosphatase non-receptor type 4 (PTPN4) prevents cell death induction in
185 stromal cell-derived 1 alpha receptor (C-X-C receptor type 4 or CXCR4) using AMD3100 prevented the po
186 ated with the number of dermal C-C chemokine receptor type 4(+) T helper type 2 cells, IL-17(+) cells
187 , were antagonized by CXCR4 (C-X-C chemokine receptor type 4) and regulated the outer topology of neu
188 olesterol to the G protein coupled chemokine receptor type 4, and the identification of permeation pa
189 en and elastin staining, and C-X-C chemokine receptor type 4, nuclear factor kappa beta, and tartrate
191 (Fuzeon, enfuvirtide) and the C-C chemokine receptor type 5 (CCR5) blocker maraviroc (Selzentry).
192 ine receptor type 2 (Ccr2) and C-C chemokine receptor type 5 (Ccr5) in the livers of patients with AL
194 nstrated that ORM1 can bind to C-C chemokine receptor type 5 (CCR5) on muscle cells and deletion of t
196 he G-protein-coupled receptor, C-C chemokine receptor type 5 (CCR5), in human disease since ancient t
197 opulation is enriched with a C-X-C chemokine receptor type 5 (CXCR5)(+)CD4(+) TFH precursor phenotype
198 global reductions in metabotropic glutamate receptor type 5 (mGluR5) binding in smokers and recent e
199 through activation of metabotropic glutamate receptor type 5 (mGluR5) signaling and that this astrocy
202 rentiation 4 (CD4) and coreceptors chemokine receptor type 5 and chemokine-related receptor type 4 (C
203 c CD8 T cells express CXCR5 (C-X-C chemokine receptor type 5, a chemokine receptor required for homin
204 s in the activation muscarinic acetylcholine receptors type 5, which mediate potentiation of dopamine
205 fied in the protein tyrosine phosphatase non-receptor type 6 (PTPN6) gene, also known as SHP-1, an im
206 (ACKR3), previously known as C-X-C chemokine receptor type 7 (CXCR7), has emerged as a key player in
208 iated with increased glomerular endothelin-1 receptor type A (Ednra) expression and increased circula
209 two G protein-coupled receptors - endothelin receptor type A (ET(A)) and endothelin receptor type B (
210 BMSCs was attenuated by blocking endothelin receptor type A (ETAR) and/or endothelin receptor type B
212 d cells, we further identified the scavenger receptor type A member I (SR-AI) to be a macrophage-spec
213 the pore of the alpha(2)beta(3)gamma(2) GABA receptor type A receptor at the so-called T6' ring, wher
214 the signaling receptor, natriuretic peptide receptor type A, to the clearance receptor, nprc, was in
215 ants influencing expression of EPHA2 (Ephrin-receptor Type-A2), a tyrosine kinase receptor that has b
216 spite the little structural similarity, both receptor types activate similar responses with different
217 S/heparin and NRP-1 may dictate the specific receptor type activated by VEGF and ultimately determine
220 cally important to identify the specific FGF receptor type and its downstream signaling molecules in
224 helin receptor type A (ET(A)) and endothelin receptor type B (ET(B)) - with equal affinity, whereas E
227 helin 3 (EDN3), its receptor (the endothelin receptor type B [EDNRB]), and the transcription factors
228 lar pathway whereby membrane-bound scavenger receptor type B-1 (SR-B1) in parent cells becomes incorp
230 tor usage is highly heterogeneous, with some receptor types being orders of magnitude more abundant t
232 ogical manipulation to reveal the individual receptor type contributions, we find that m2 muscarinic
233 efore, blocking detrimental activity of both receptor types could be therapeutically beneficial.
234 he model also suggests that overly sensitive receptor types could dominate the entire response and ma
235 pithelial neural network of Beroe, with five receptor types, covers the entire body surface and expan
236 ndicates that spiking pattern, regardless of receptor type, determines vibrotactile frequency percept
237 t the first high-resolution structure of the receptor type found in muscle-endplate membrane and in t
240 cient trafficking of a sensory receptor, the receptor-type guanylate cyclase GCY-9, to cilia in chemo
241 rent affinity of striatal D1 and D2 dopamine receptor types has been argued to constrain the D1 and D
242 sized that disrupting the cognate type I IFN receptor (type I IFN alpha/beta receptor [IFNAR]) to int
245 tokine family that signals through the IL-20 receptor type I (IL-20Ralpha:IL-20Rbeta), is a cytokine
247 o an increase in the expression of the AngII receptor type I a and activation of signaling at 100-fol
248 factor kappaB signaling, and increases AngII receptor type I a expression, thus potentiating AngII si
254 orphogenic protein receptor kinase activin A receptor, type I (ACVR1), and the subsequent reduction o
255 for binding to intact, functional ryanodine receptors type I (RyR1) and II (RyR2) from skeletal and
256 Strikingly, we found that IFN-alpha/beta receptor (type-I IFN receptor) was expressed by primary
257 s contain somatic mutations in the activin A receptor type IB (ACVR1B) gene, indicating that ACVR1B c
260 on factor 11 (GDF11) through soluble activin receptor type II (ActRII) ligand traps or neutralizing a
261 rent from AT(1)R, in particular, angiotensin receptor type II (AT(2)R), resulting in biological and p
262 gene encoding the bone morphogenetic protein receptor type II (BMPR2) are the commonest genetic cause
265 at drugs that modulate the two dominant GABA receptor types in the brain, GABA(A) (clobazam) and GABA
266 nd mesogleal) and the structure of different receptor types in the comb jelly Beroe abyssicola-the vo
267 nformation depends strongly on the number of receptor types included in the primacy representation, b
268 conjunction with their cognate PLXNB family receptors, type IV members have been increasingly shown
269 ine phosphatase protein tyrosine phosphatase receptor type J (PTPRJ), a known extracellular signal-re
270 umor suppressor Protein tyrosine phosphatase receptor-type kappa (PTPRK), as a Wnt inhibitor in human
271 ecific receptor states, rather than specific receptor types, may be a viable strategy for future drug
272 quencies > 60 Hz), subserved by two distinct receptor types (Meissner's and Pacinian corpuscle, respe
274 se peptides binds to all three of the opioid receptor types (mu, delta, or kappa), albeit with differ
275 ing networks where information from numerous receptor types passes through a small set of signaling p
282 fear, and anxiety, and the specific cell and receptor types responsible is an active area of investig
283 e transferrin receptor and several glutamate receptor types, resulting in their appearance in the axo
285 e the minimal structural differences between receptor types, selectivity can be achieved in a number
286 itivities of L and M cones were, on average, receptor-type specific, but individual cone thresholds v
287 zebrafish single cell RGCs were treated with receptor-type-specific antagonists and, respectively, wi
289 g, where the odor identity is encoded by the receptor types that respond earliest, might provide a co
290 mined the average quantity of each glutamate receptor type, their nanoscale organization, and their r
291 ll sizes to have a large number of olfactory receptor types, to maintain olfactory precision in their
292 ir C-terminal tail, and are activated by the receptor-type tyrosine phosphatase PTPRJ (CD148, DEP-1),
297 cells express the putative HCO(3)(-)-sensor receptor-type tyrosine-protein phosphatase RPTPgamma, wh
298 inhibition of cell-cell contacts through the receptor-type tyrosine-protein phosphatases kappa led to
299 postsynaptic partners, the neurotransmitter receptor type used to receive input from presynaptic neu