ライフサイエンスコーパス: receptor-interacting protein

1 HLA-B-associated transcript 3) as a TGF-beta receptor-interacting protein.

2 The Cannabinoid Receptor Interacting Protein 1 (Cnrip1) was discovered a

3 A SNP within the glutamate receptor interacting protein 1 (GRIP1) gene presented a

4 subunit of the AMPAR and requires glutamate receptor interacting protein 1 (GRIP1) interaction with

5 Glutamate receptor interacting protein 1 (GRIP1) is a neuronal sca

6 P90/DLG/ZO-1 (PDZ) domain 2 of the glutamate receptor interacting protein 1 (GRIP1) through its intra

7 ssary for synaptic accumulation of glutamate receptor interacting protein 1 (GRIP1), a crucial scaffo

8 Here we show that glutamate receptor interacting protein 1 (GRIP1), an AMPAR-binding

9 ch repeat and Ig-like domain-containing Nogo receptor interacting protein 1 (LINGO-1) is a negative r

10 ch repeat and Ig-like domain-containing Nogo receptor interacting protein 1 (LINGO-1) is a negative r

11 The recent discovery of the role of receptor interacting protein 1 (RIP1) kinase in tumor ne

12 The death domain kinase receptor interacting protein 1 (RIP1) plays a key role i

13 2 is necessary for the polyubiquitination of receptor interacting protein 1 (RIP1) that then serves a

14 In this study, we show that the receptor interacting protein 1 (RIP1), a central compone

15 d both K48- and K63-linked ubiquitination of receptor interacting protein 1 (RIP1).

16 We found that receptor interacting protein 1 and 3 (RIP1 and RIP3) kin

17 singly, TRADD is required for recruitment of receptor interacting protein 1 and TNFR-associated facto

18 ation (c.279delG, p.Trp93fs*) of the nuclear receptor interacting protein 1 gene (NRIP1) in all seven

19 oxicity was also abolished by necrostatin or receptor interacting protein 1 knock down.

20 uit fly, the PDZ7 domain of GRIP1 (glutamate receptor interacting protein 1) from rat and the PDZ2 do

21 naling complex and for the ubiquitination of receptor interacting protein 1.

22 Glutamate receptor-interacting protein 1 (GRIP1) and GRIP2 are clo

23 r recruitment to the receptor/Glucocorticoid Receptor-Interacting Protein 1 (GRIP1) complex that bind

24 Overexpression of glutamate receptor-interacting protein 1 (GRIP1) rescues ephrinB3

25 lating the formation of a complex containing receptor-interacting protein 1 (RIP1) and receptor-inter

26 f Casp8 prevents proteolytic cleavage of the receptor-interacting protein 1 (RIP1) in hepatocytes and

27 ily promote K11-linked polyubiquitination of receptor-interacting protein 1 (RIP1) in vitro and in vi

28 f necroptosis, which could be blocked by the receptor-interacting protein 1 (RIP1) inhibitor, necrost

29 Although receptor-interacting protein 1 (RIP1) is well known as a

30 retroviral expression of TAK1, inhibition of receptor-interacting protein 1 (RIP1) kinase activity wi

31 tatin-1 (Nec-1), a specific inhibitor of the receptor-interacting protein 1 (RIP1) kinase domain, pre

32 Finally, the receptor-interacting protein 1 (RIP1) may be involved in

33 Ubiquitination and deubiquitination of receptor-interacting protein 1 (RIP1) play an important

34 NF) receptor-associated factor 2 (TRAF2) and receptor-interacting protein 1 (RIP1) play critical role

35 has been attributed to ubiquitin editing of receptor-interacting protein 1 (RIP1) to influence activ

36 rongly reduced in murine fibroblasts lacking receptor-interacting protein 1 (RIP1), a known M45-inter

37 mbly of a death-signaling complex containing receptor-interacting protein 1 (RIP1), FADD, and caspase

38 Here we report that receptor-interacting protein 1 (RIP1)-regulated interleu

39 romises the formation of the downstream TRIF/receptor-interacting protein 1 (RIP1)/TBK1 complex.

40 e of necrostatin-1, a selective inhibitor of receptor-interacting protein 1 (RIP1, also known as RIPK

41 additional cell death pathways, necroptosis (receptor-interacting protein 1 [RIP1] and RIP3 mRNAs) an

42 sis, a form of programmed necrosis involving receptor-interacting protein 1 and the mixed lineage kin

43 king the inhibition of polyubiquitination of receptor-interacting protein 1 in TNF receptor 1 complex

44 mice and THP-1 macrophages in vitro with the receptor-interacting protein 1 kinase (RIP1) inhibitor n

45 id receptor coactivator-1 and glucocorticoid receptor-interacting protein 1 to inhibit hCAR activity.

46 Although TNFalpha-induced receptor-interacting protein 1 ubiquitination is indeed

47 he absence of TRAF2 and TRAF5 expression and receptor-interacting protein 1 ubiquitination.

48 ges leads to reduced ubiquitination of RIP1 (receptor-interacting protein 1), suggesting a role for L

49 formation of 2 cell death complexes, RIP 1 (receptor-interacting protein 1)-FADD (Fas-associated pro

50 ys, such as hypoxia-inducing factor-1-alpha, receptor-interacting protein 1, and apoptosis-inducing f

51 factor 6, TNF receptor-associated factor 2, receptor-interacting protein 1, Hemo-oxidized iron regul

52 ule, interferon regulatory factor 3 (IRF-3), receptor-interacting protein 1, IFNbeta-1, and nucleotid

53 PMN lysis was dependent on receptor-interacting protein 1, suggesting that PMN-SA u

54 receptor coactivator 1 or the glucocorticoid receptor-interacting protein 1.

55 nd hepatocyte death in vitro, independent of receptor-interacting protein 1.

56 teractions with TNFR-associated factor 6 and receptor-interacting protein 1.

57 riments show that WNV-E acts at the level of receptor-interacting protein 1.

58 ation is dependent upon its interaction with receptor-interacting protein 1.

59 itochondria, the role of necroptosis through receptor-interacting proteins 1 and 3 (RIPK1 and RIPK3)

60 Mechanistically, Traf2 critically regulates receptor-interacting proteins 1 and 3 and mixed lineage

61 nd resulted in the downregulation of nuclear receptor interacting protein-1 (NRIP1), which influences

62 We identified receptor interacting protein-1 (RIP1) as a novel API2-MA

63 how that the AMPAR-binding protein glutamate receptor-interacting protein-1 (GRIP1) is essential for

64 NF-alpha/Fas-induced cell death is a type of receptor-interacting protein-1 (RIP-1)-dependent program

65 -associated death domain protein (TRADD) and receptor-interacting protein-1 (RIP1) in TRAIL signaling

66 We identified TRIP12 (thyroid hormone receptor-interacting protein 12), an E3 ubiquitin-protei

67 In this study, we demonstrated that thyroid receptor interacting protein 13 (TRIP13) is a critical m

68 0-Mad2 subcomplex and identify it as Thyroid Receptor Interacting Protein 13 (TRIP13), an AAA-ATPase

69 The AAA-ATPase thyroid hormone receptor interacting protein 13 (TRIP13), jointly with t

70 In this study, we found receptor interacting protein 140 (RIP140) can regulate t

71 Receptor interacting protein 140 (RIP140) is a nuclear r

72 T3 repression of Crabp1 requires receptor interacting protein 140 (RIP140).

73 ment of histone deacetylase-containing/GRIP1/receptor-interacting protein 140 (RIP140) complex in dif

74 onal carcinoma cell line P19, which involves receptor-interacting protein 140 (RIP140) for heterochro

75 urons induces rapid translocation of nuclear receptor-interacting protein 140 (RIP140) to the cytopla

76 coactivator 1alpha or 1beta and repressed by receptor-interacting protein 140.

77 Cannabinoid receptor interacting protein 1a (CRIP1a) is a CB1 recept

78 Cannabinoid receptor-interacting protein 1a (CRIP1a) binds to the CB

79 icated in the breakdown of 2-AG; cannabinoid receptor-interacting protein 1a (CRIP1a), a protein that

80 it a higher expression of IRAK1, IRAK-M, and receptor interacting protein 2 (Rip2).

81 nd their common downstream adaptor molecule, receptor interacting protein 2 (RIP2; also known as RIPK

82 now show that MDP induces ubiquitylation of receptor- interacting protein 2 (RIP2) in primary macrop

83 Receptor-interacting protein 2 (RIP2) is a kinase that m

84 Receptor-interacting protein 2 (Rip2) is a transcription

85 1 (NOD1) interacts with its adaptor protein receptor-interacting protein 2 (RIP2) to propagate immun

86 pensable for recruiting a downstream kinase, receptor-interacting protein 2 (RIP2), that activates nu

87 ient in the gene coding for the NLR adaptor, receptor-interacting protein 2 (RIP2).

88 8 as well as NOD1 and its signaling molecule receptor-interacting protein 2 (RIP2).

89 on of MAPKs, and deficient ubiquitination of receptor-interacting protein 2 in response to MDP.

90 reduced in the absence of NOD proteins, but receptor-interacting protein 2 is not involved in CD8 si

91 protein 2) binds to the protein kinase RIP2 (receptor-interacting protein 2) to coordinate NF-kappaB

92 njury was seen with a deficiency of Nod2 and receptor-interacting protein 2, and the simultaneous def

93 of IkappaB kinase (IKKbeta) and its upstream receptor-interacting protein 2, whereas IKKbeta inhibito

94 , and in their downstream signaling molecule receptor-interacting protein 2.

95 ulation and NF-kappaB activation, indicating receptor-interacting protein 2/IKKbeta signaling plays c

96 Responses were mediated through receptor interacting protein-2 (RIP2)- and tumor necrosi

97 ifferentiation primary response gene 88, and receptor-interacting protein-2 but reduced the expressio

98 rated that a super-low dose of LPS activates receptor interacting protein 3 kinase (RIP3), a key mole

99 IF) and led to persistent phosphorylation of receptor-interacting protein 3 (Rip3) kinase, which resu

100 was correlated with increased expression of receptor-interacting protein 3 (RIP3), a master regulato

101 ne products due to mu1 knockdown potentiates receptor-interacting protein 3 (RIP3)-dependent cell dea

102 ia and that Gpx4 is essential for preventing receptor-interacting protein 3 (RIP3)-dependent necropto

103 ng receptor-interacting protein 1 (RIP1) and receptor-interacting protein 3 (RIP3).

104 lly, inhibition of STIM1 signaling prevented receptor-interacting protein 3-dependent (RIP3-dependent

105 lpha and CD95 ligand, but was independent of receptor-interacting protein 3.

106 +) exchange regulatory factor 2, and thyroid receptor interacting protein 6, which has been shown pre

107 of two proteins in the zyxin family, thyroid receptor-interacting protein 6 (TRIP6) and lipoma-prefer

108 In this study, a role for thyroid hormone receptor-interacting protein 6 (TRIP6) in sealing zone f

109 LIM domain-containing TRIP6 (Thyroid Hormone Receptor-interacting Protein 6) is a focal adhesion mole

110 Fisetin also inhibited TNF-induced TAK1 and receptor-interacting protein activation, events that lie

111 e by the "death" adaptor protein caspase and receptor interacting protein adaptor with death domain (

112 cular mechanisms leading to aryl hydrocarbon receptor interacting protein (AIP) mutation-induced aggr

113 partners, we found that the aryl hydrocarbon receptor-interacting protein (AIP) directly associates w

114 Aryl hydrocarbon receptor-interacting protein (AIP) mutations lead to som

115 RISPR screen identified the aryl hydrocarbon receptor-interacting protein (AIP), a co-chaperone prote

116 homozygous deletion of the aryl hydrocarbon receptor-interacting protein (AIP), a survivin-associate

117 cluding Hsp90, p23, and the aryl hydrocarbon receptor-interacting protein (AIP), also known as ARA9 a

118 These G protein-coupled receptor-interacting proteins also facilitate fine-tunin

119 re, we have identified AIP (aryl hydrocarbon receptor interacting protein) as a new binding partner o

120 ng protein adaptor with death domain (CRADD)/receptor interacting protein-associated ICH-1/CED-3 homo

121 -induced protein with a death domain (PIDD), receptor-interacting protein-associated ICH-1/CED-3 homo

122 r show that RIP1 (the Ser/Thr protein kinase receptor-interacting protein) associates with the GAP do

123 primary keratinocytes (KCs) as the vitamin D receptor-interacting protein complex.

124 of two structurally related CB1 cannabinoid receptor interacting proteins (CRIP1a and CRIP1b) that b

125 factor receptor (TNFR) superfamily, through receptor-interacting protein-dependent K63-linked ubiqui

126 Using VDR affinity beads, the vitamin D receptor interacting protein (DRIP)/mediator complex was

127 kout mice and chimeras revealed that Eph-Eph receptor-interacting proteins (ephrins) are expressed bo

128 We identified a novel NMDA receptor-interacting protein, GIPC (GAIP-interacting pro

129 with one of the PDZ domains of the Glutamate receptor interacting protein (Grip), another factor requ

130 -associated protein (GABARAP), and glutamate receptor interacting protein (GRIP).

131 on, two AMPAR-interacting proteins-glutamate receptor-interacting protein (GRIP) and protein interact

132 Glutamate receptor-interacting protein (GRIP) is a neuronal scaffo

133 brane through its interaction with glutamate receptor-interacting protein (GRIP), a PDZ domain protei

134 g domains for left-handed helix (Z-form) and receptor-interacting protein homotypic interaction motif

135 of RAMPs highlight the need to consider all receptor-interacting proteins in future drug development

136 PRIP (peroxisome proliferator-activator receptor interacting protein) is a nuclear receptor coac

137 ty of NEMO to bind to K63-linked polyUb RIP (receptor-interacting protein) is necessary for efficient

138 Our findings suggest that BAT3, a TGF-beta receptor-interacting protein, is capable of modulating T

139 Programmed necrosis mediated by receptor interacting protein kinase (RIP)3 (also called

140 d cIAP2 (cIAP1/2), leading to the release of receptor interacting protein kinase (RIPK1) from the act

141 Degradation of cIAPs triggers the release of receptor interacting protein kinase (RIPK1) from TNF rec

142 he necroptosis inhibitor Nec-1, which blocks receptor interacting protein kinase 1 (RIP kinase 1), al

143 ding tumor necrosis factor receptor (TNFR1), receptor interacting protein kinase 1 (RIP1), RIP3, phos

144 D and caspase 8, and the necroptosis kinases receptor interacting protein kinase 1 (RIPK1) and RIPK3.

145 emonstrated that the scaffolding function of receptor interacting protein kinase 1 (RIPK1) and tumor

146 In contrast, inhibition and knockdown of receptor interacting protein kinase 1 (RIPK1) had no eff

147 Receptor interacting protein kinase 1 (RIPK1) has an ess

148 Receptor interacting protein kinase 1 (RIPK1) is a criti

149 by tumor necrosis factor (TNF) signaling via receptor interacting protein kinase 1 (RIPK1)- and caspa

150 iven by two serine threonine kinases, RIPK1 (Receptor Interacting Protein Kinase 1) and RIPK3, and a

151 ction model, such variant procaspase-1 binds receptor interacting protein kinase 2 (RIP2) via Caspase

152 Receptor interacting protein kinase 2 (RIPK2) is critica

153 itment of additional proteins (such as RIP2, receptor interacting protein kinase 2), which is further

154 Recent findings suggest that the receptor interacting protein kinase 3 (RIP3) acts as a s

155 orm of nonapoptotic cell death driven by the receptor interacting protein kinase 3 (RIPK3) and its su

156 sis is a cell death pathway regulated by the receptor interacting protein kinase 3 (RIPK3) and the mi

157 ptosis, functions as a negative regulator of receptor interacting protein kinase 3 (RIPK3), an essent

158 l survival, caspase-8-mediated apoptosis, or receptor interacting protein kinase 3 (RIPK3)-dependent

159 pases, while necroptosis is dependent on the receptor interacting protein kinase 3 (RIPK3).

160 ll death defined by activation of the kinase receptor interacting protein kinase 3 and its downstream

161 ed phosphorylation and decreased cleavage of receptor interacting protein kinase-1 (Rip1), leading to

162 8, revealing a new regulatory axis affecting receptor interacting protein kinase-1 (RIPK1)>CASPASE-8

163 Receptor Interacting Protein Kinase-1 (RIPK1), a key pla

164 osis is mediated by the kinase activities of receptor interacting protein kinase-1 and receptor inter

165 ch is transduced by the kinase activities of receptor interacting protein kinase-1 and receptor inter

166 Receptor interacting protein kinase-3 (RIP3) is an essen

167 ath pathway downstream of the protein kinase Receptor Interacting Protein Kinase-3 (RIPK3).

168 nt mice is completely rescued by ablation of receptor interacting protein kinase-3 (RIPK3).

169 of receptor interacting protein kinase-1 and receptor interacting protein kinase-3, eventually leadin

170 of receptor interacting protein kinase-1 and receptor interacting protein kinase-3, which eventually

171 Receptor-interacting protein kinase (RIP) 3 (also called

172 Because receptor-interacting protein kinase (RIP) 3-mediated nec

173 inase (FAK) which then interact with AKT and receptor-interacting protein kinase (RIP), respectively.

174 Inhibition of receptor-interacting protein kinase (RIP)1 by necrostati

175 NF-kappaB is shown to depend on the adaptor receptor-interacting protein kinase (RIP)1, acting via a

176 Receptor-interacting protein kinase (RIPK) 1 functions a

177 es neutrophil extracellular traps (NETs) via receptor-interacting protein kinase (RIPK) 1/3- and mixe

178 l of peritonitis, we show in this study that receptor-interacting protein kinase (RIPK) 3 impacts pyr

179 Receptor-interacting protein kinase (RIPK)-1 is involved

180 ochondrial permeability transition (MPT) and receptor-interacting protein kinase (RIPK)1-mediated nec

181 on by APAP was p53 independent, and required receptor-interacting protein kinase 1 (RIP1) and c-Jun N

182 cells by inhibiting the interaction between receptor-interacting protein kinase 1 (RIP1) and RIP3, a

183 ck the Fas-associated death domain (FADD) or receptor-interacting protein kinase 1 (RIP1) cell death

184 noic acid receptor gamma (RARgamma) controls receptor-interacting protein kinase 1 (RIP1)-initiated c

185 Receptor-interacting protein kinase 1 (RIPK1) and RIPK3

186 Receptor-interacting protein kinase 1 (RIPK1) induces ap

187 Receptor-interacting protein kinase 1 (RIPK1) is a serin

188 Receptor-interacting protein kinase 1 (RIPK1) is an impo

189 Inhibition of receptor-interacting protein kinase 1 (RIPK1) kinase act

190 Knock-in expression of kinase inactive receptor-interacting protein kinase 1 (RIPK1) prevented

191 Receptor-interacting protein kinase 1 (RIPK1) promotes c

192 Receptor-interacting protein kinase 1 (RIPK1) regulates

193 s acidic neuronal necroptosis via recruiting receptor-interacting protein kinase 1 (RIPK1) to its C t

194 ed apoptosis requires the kinase activity of receptor-interacting protein kinase 1 (RIPK1), a key reg

195 ath known as necroptosis that is mediated by receptor-interacting protein kinase 1 (RIPK1), RIPK3 and

196 programmed cell death and innate immunity is receptor-interacting protein kinase 1 (RIPK1), which has

197 Rather, receptor-interacting protein kinase 1, a kinase also lin

198 Activation of macrophages required DR5 and receptor-interacting protein kinase 1.

199 n to directly binding its downstream targets receptor-interacting protein kinase 2 (RIP2) and autopha

200 reporting conflicting requirements for RIP2 (receptor-interacting protein kinase 2) in autophagy indu

201 Ly6/PLAUR domain-containing protein 6, in a receptor-interacting protein kinase 2-TGF-beta-activated

202 Receptor-interacting protein kinase 3 (RIP3 or RIPK3) ha

203 Receptor-interacting protein kinase 3 (RIP3) and its sub

204 We further demonstrate that receptor-interacting protein kinase 3 (RIP3), which is h

205 shed by caspase-8 compromise and mediated by receptor-interacting protein kinase 3 (RIP3).

206 The receptor-interacting protein kinase 3 (RIP3/RIPK3) has e

207 tein expression of the necroptosis mediators receptor-interacting protein kinase 3 (RIPK3) and mixed

208 otein with death domain (FADD) combined with receptor-interacting protein kinase 3 (RIPK3) deficiency

209 Besides causing necroptotic cell death, receptor-interacting protein kinase 3 (RIPK3) has an alt

210 Receptor-interacting protein kinase 3 (RIPK3) is a serin

211 Receptor-interacting protein kinase 3 (RIPK3) mediates n

212 RNA interference for receptor-interacting protein kinase 3 (RIPK3) or mixed l

213 The receptor-interacting protein kinase 3 (RIPK3) plays cruc

214 Receptor-interacting protein kinase 3 (RIPK3)-mediated n

215 c approach, we demonstrate the presence of a receptor-interacting protein kinase 3 (RIPK3)-mixed line

216 m of cell death that critically requires the receptor-interacting protein kinase 3 (RIPK3).

217 rammed necrosis, or "necroptosis", driven by receptor-interacting protein kinase 3 (RIPK3).

218 Receptor-interacting protein kinase 4 (RIPK4) and interf

219 Receptor-interacting protein kinase 4 (RIPK4) is require

220 ted kinase (PKK), which is also known as the receptor-interacting protein kinase 4, as a suppressor o

221 D), Fas-associated death domain protein, and receptor-interacting protein kinase proteins form the si

222 nteract via a CARD-CARD interaction with the receptor-interacting protein kinase RIP2, an inducer of

223 at MLKL-mediated death is independent of the receptor-interacting protein kinase RIPK3.

224 Instead, robust cross-priming required receptor-interacting protein kinase-1 (RIPK1) signaling

225 because the molecules initiating it [such as receptor-interacting protein kinase-1 (RIPK1), RIPK3, or

226 y assays and immunohistochemistry for RIPK1 (receptor-interacting protein kinase-1), MLKL (mixed line

227 Receptor-interacting protein kinase-3 (RIP3 or RIPK3) is

228 nonapoptotic form of cell death initiated by receptor-interacting protein kinase-3 (RIPK3) and mixed

229 Receptor-interacting protein kinase-3 (RIPK3) is an acti

230 Here, we show that modulating the receptor-interacting protein kinase-3 (Ripk3) pathway ma

231 ological cell suicide mechanism initiated by receptor-interacting protein kinase-3 (RIPK3) phosphoryl

232 ation of mixed lineage kinase-like (MLKL) by receptor-interacting protein kinase-3 (RIPK3) results in

233 onents of the necroptotic pathway, including receptor-interacting protein kinase-3 (RIPK3), mixed lin

234 l approach, we demonstrate the presence of a receptor-interacting protein kinase-3 (RIPK3)-a mixed li

235 tosis, in which dying cells are generated by receptor-interacting protein kinase-3 and caspase-8 dime

236 is represented by necroptosis, in which the receptor-interacting protein kinase-3 and its substrate

237 sure and, more recently, also in relation to receptor-interacting protein kinase-driven necroptosis,

238 ting Fas-associated death domain protein and receptor-interacting protein kinase.

239 The receptor interacting protein kinases-1 (RIPK1) and RIPK3

240 Recent studies have demonstrated that receptor-interacting protein kinases (RIPKs) RIPK1 and R

241 of programmed necrosis that is regulated by receptor-interacting protein kinases (RIPs).

242 dence points to unregulated signaling by the receptor-interacting protein kinases-1 (RIPK1) and RIPK3

243 LCA) caused by mutations in Aryl hydrocarbon receptor interacting protein like-1 (Aipl1) is a severe

244 photoreceptor-specific gene aryl hydrocarbon receptor interacting protein-like 1 (Aipl1) are associat

245 the photoreceptor-expressed aryl hydrocarbon receptor interacting protein-like 1 (AIPL1) are associat

246 P351Delta12 human isoform, aryl hydrocarbon receptor interacting protein-like 1 (hAIPL1) mice demons

247 IPL1 and the mouse isoform, aryl hydrocarbon receptor interacting protein-like 1 (mAipl1).

248 ptor-specific gene encoding aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) are clinical

249 Aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) is a distinc

250 Aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) is a photore

251 Aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) is a photore

252 n the chaperone activity of aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1), and mutatio

253 the enzyme itself or AIPL1 (aryl hydrocarbon receptor-interacting protein-like 1), leads to retinal d

254 RICK (receptor-interacting protein-like interacting caspase-li

255 Defects in aryl hydrocarbon receptor interacting protein-like1 (AIPL1) are associate

256 identify LRR and Ig domain-containing, Nogo receptor-interacting protein (LINGO-1) as a potent axona

257 eviously, LRR and Ig domain-containing, Nogo receptor-interacting protein (LINGO-1) has been identifi

258 iated factor 2, TGF-beta-activated kinase 1, receptor-interacting protein, NF-kappaB-inducing kinase,

259 Nuclear receptor interacting protein (Nrip1), also known as RIP1

260 We have identified an N receptor-interacting protein, NRIP1, that directly inter

261 the recruitment of PPARalpha and the nuclear receptor-interacting protein, nuclear receptor corepress

262 ct of multiple ligands, association with the receptor-interacting protein receptor activity-modifying

263 f TLR2, TLR3, TLR4, TLR5, or TLR9 results in receptor interacting protein (RIP) 3 kinase-dependent pr

264 Wild-type C57BL/6 and receptor interacting protein (RIP) 3(-/-) mice were rand

265 Here, we show that receptor interacting protein (RIP) kinase-mediated necro

266 It is known that receptor interacting protein (RIP) kinases, RIP1 and RIP

267 Receptor interacting protein (RIP)3 kinase (also called

268 e cytomegalovirus M45 protein interacts with receptor-interacting protein (RIP) 1 and RIP3 via a RIP

269 Receptor-interacting protein (RIP) and cellular Fas-asso

270 The receptor-interacting protein (RIP) family kinase RIP4 in

271 ing motifs, one of which is related to RHIM [receptor-interacting protein (RIP) homotypic interaction

272 of IFN-regulatory factors (DAI) that contain receptor-interacting protein (RIP) homotypic interaction

273 Because this involves receptor-interacting protein (RIP) kinase 1/3, this stud

274 Receptor-interacting protein (RIP) kinase 3 (RIPK3)-depe

275 In this study, we show that receptor-interacting protein (RIP) kinase mediates necro

276 The complex interplay between caspase-8 and receptor-interacting protein (RIP) kinase RIP 3 (RIPK3)

277 Here we provide genetic evidence for a receptor-interacting protein (RIP) kinase-caspase-8-depe

278 , which plays a key role in the execution of receptor-interacting protein (RIP) kinase-dependent necr

279 type I interferon (IFN-I) response to induce receptor-interacting protein (RIP) kinase-mediated necro

280 ase-11, IL-1R-associated kinases (IRAK), and receptor-interacting protein (RIP) kinases contribute to

281 /Fas-mediated signaling pathway regulated by receptor-interacting protein (RIP), a kinase that shuttl

282 ern recognition receptor that assembles with receptor-interacting protein (RIP)-2 kinase in response

283 e-2, or the caspase-2 adaptor protein RAIDD (receptor-interacting protein (RIP)-associated Ich-1/CED

284 ase-8, TNFR-associated factor 2 (TRAF2), and receptor-interacting protein (RIP).

285 t the principal components of the necrosome, receptor-interacting protein (RIP)1 and RIP3, are highly

286 and type II (gamma) IFNs induce precipitous receptor-interacting protein (RIP)1/RIP3 kinase-mediated

287 The pronecrotic kinase, receptor interacting protein (RIP1, also called RIPK1) m

288 Receptor-interacting protein (RIP1) and p38 mitogen-acti

289 Receptor-interacting protein (RIP1) is a serine/threonin

290 The receptor-interacting protein RIP140 interacts with the t

291 ced anti-inflammation engineered by lowering receptor interacting protein (RIP140) expression in macr

292 milar to programmed necrosis that depends on receptor interacting protein (Ripk1).

293 In particular, we focus on the roles of receptor-interacting proteins, scaffold proteins, synapt

294 n ID-related gene encoding the synaptic NMDA-receptor interacting protein synapse-associated protein

295 TO-ETHYLENE SENSITIVITY1 (RTE1), an ethylene receptor interacting protein that regulates the activity

296 In an attempt to discover novel receptor interacting proteins, the C-terminal tail of th

297 Two such proteins, mannose 6-phosphate receptor-interacting protein TIP47 and Arfaptin2, were f

298 tic Wnt-receiving cell to target dGRIP, a Wg-receptor-interacting protein, to postsynaptic sites.

299 y, we cloned the tomato ortholog of TGF-beta Receptor Interacting Protein (TRIP1), which was previous

300 uman PIMT (peroxisome proliferator-activated receptor-interacting protein with methyltransferase) pro