ライフサイエンスコーパス: red blood cell

1 which indicated a low binding of [(3)H]23 to red blood cells.

2 ron to the bone marrow for the production of red blood cells.

3 total protein level, white blood cells, and red blood cells.

4 lative nanoimaging on malaria-infected human red blood cells.

5 enhances the production of mature enucleated red blood cells.

6 apidly frozen Plasmodium falciparum-infected red blood cells.

7 e decidual stromal cells and ability to lyse red blood cells.

8 en-specific Ab after immunization with sheep red blood cells.

9 ed to receive either fresh or standard-issue red blood cells.

10 ding mortality) compared with standard-issue red blood cells.

11 s that lack functional haemoglobin genes and red blood cells.

12 n of, asexual growth within, and egress from red blood cells.

13 increased the risk for transfusion of packed red blood cells.

14 volumes to be computed for motor neurons and red blood cells.

15 regulated process that generates all mature red blood cells.

16 in expression in the majority of circulating red blood cells.

17 ed by the presence of NPs in endothelial and red blood cells.

18 arities to a protein crucial for invasion of red blood cells.

19 ins the haemoglobin profile of the patient's red blood cells.

20 dens by rounds of asexual replication within red blood cells.

21 resistance and enhanced cytotoxicity towards red blood cells.

22 perienced loss of leukocytes, platelets, and red blood cells.

23 als in the tails of a quantitative trait for red blood cells.

24 essential for egress of parasites from host red blood cells.

25 enation, respectively were (median +/- IQR): Red blood cell: 6.0 +/- 0.5 (10(6)/muL) and 6.5 +/- 0.4

26 ted invasion efficiency while accounting for red blood cell accessibility to parasites.

27 ional antibodies, based on the inhibition of red blood cell agglutination by ETEC.

28 cular release of fibrinogen correlating with red blood cell aggregation and microvascular plugging.

29 s red blood cell structure, for example, and red blood cells also undergo dramatic changes in morphol

30 se), (2) enhanced phagocytic activity toward red blood cells (an in vitro model of hematoma clearance

31 ite blood cell differential, 2) quantitative red blood cell and hemoglobin characterization, 3) clear

32 O(2) flux occurs predominantly via elevating red blood cell and plasma flux in already flowing capill

33 r trial demonstrated that prehospital packed red blood cell and plasma had the greatest reduction in

34 d products when available, preferably packed red blood cell and plasma.

35 tically influenced telomere length increased red blood cell and white blood cell counts, decreased me

36 by clearing hemoglobin that has leaked from red blood cells and also restricts the availability of e

37 er is characterized by reduced production of red blood cells and an increase in myelopoiesis, which c

38 eir role in hemolytic activity against human red blood cells and antimicrobial activity against seven

39 rface of Plasmodium falciparum-infected host red blood cells and binds to specific chondroitin-4-sulf

40 between native VAR2CSA expressed on infected red blood cells and chondroitin sulfate A in an in vitro

41 gastrulating embryo, resulting in a loss of red blood cells and downregulation of erythropoietic gen

42 eceptor preference as measured with modified red blood cells and glycan arrays.

43 b(th3/+) mice, accompanied by an increase in red blood cells and hemoglobin and a decrease in reticul

44 tration and that hemoglobin diffusion in the red blood cells and in solutions at similar concentratio

45 associated with sequestration of parasitized red blood cells and increased gastrointestinal permeabil

46 helial damage accompanied by high numbers of red blood cells and inflammatory cells (macrophage surfa

47 nction formation during parasite invasion of red blood cells and is a potential vaccine candidate for

48 insufficiency that limits the production of red blood cells and leads to tissue hypoxia and intracel

49 (12 microm-50 microm) used to first deplete red blood cells and platelets.

50 for understanding cytoadherence of infected red blood cells and potentially provides a starting poin

51 ently placed on EVNP using oxygenated packed red blood cells and supplemental nutrition for a period

52 ignificantly impaired in its ability to lyse red blood cells and survive in defibrinated blood.

53 yrin accumulation progressively decreased in red blood cells and urine, and skin photosensitivity in

54 Candida species and major blood cells (i.e., red blood cells and white blood cells) have a size distr

55 parasitemias in Plasmodium-infected cultured red blood cells, and discrimination between healthy indi

56 IMVs-MSCs reduced serum levels of anti-sheep red blood cell antibody and have limited effects on neut

57 odium falciparum-infected human erythrocytes/red blood cells are hallmarks of severe pathogenesis con

58 Mice transfused with these red blood cells are resistant to highly lethal doses of

59 an environmentally friendly method by using red blood cells as the raw material and hemoglobin (Hb)

60 el segmentation approach that uses images of red blood cells as training data.

61 Furthermore, tracking fluorescent-labeled red blood cells at the endocrine-exocrine interface reve

62 Red blood cell-based solutions, artificial hemoglobin so

63 The red blood cell binding segment of EBA-140 is comprised o

64 scopy (QPM), is a powerful tool for studying red blood cell biomechanics.

65 l thrombi and emboli contained few biconcave red blood cells but many polyhedrocytes or related forms

66 heme play central roles in the production of red blood cells, but the underlying mechanisms remain in

67 ected with 1.1-1.5 x 108 B. microti-infected red blood cells by intraperitoneal injection.

68 , animal cells (neuroblastoma N115 and sheep red blood cells), cancer cells (MCF-7, MDA-435 and CD34(

69 Mice whose red blood cells carry the chimeric proteins exhibit resi

70 implicated in antiphospholipid syndrome, or red blood cells coated with anti-(alpha)-Rh(D) antibodie

71 aoperative transfusion of >6 units of packed red blood cell concentrates and recipients who were olde

72 eived fresh frozen plasma and eight received red blood cell concentrates), and 21 patients received p

73 mpathetic nerve activity, and (iv) increased red blood cell concentration and mass leading to elevate

74 9)/L (normal range, [4.0-11.0] x 10(9)/L), a red blood cell count of 3.39 x 10(12)/L (normal range, [

75 en (delta(15)N) and carbon (delta(13)C) from red blood cells declined over time, with a steeper trend

76 meter, and oxygen level-dependent changes in red blood cell deformability.

77 olyhedrocytes or related forms of compressed red blood cells, demonstrating that these structures are

78 Flu-SEC was validated using red blood cell derived EVs (REVs), which provide an idea

79 that specialized phagocytes that internalize red blood cells develop in Toll-like receptor 7 (TLR7)-d

80 lly ill pediatric patients, the use of fresh red blood cells did not reduce the incidence of new or p

81 Overall, red blood cells displayed high iron intensities, whereas

82 FC decreased red blood cell distribution width (RDW) compared with co

83 nd explore the oxygenation and flow of human red blood cells during tidal ventilation and distension

84 nge imaging showed reduced (129)Xe uptake by red blood cells early in the progression of the disease,

85 SPCs, a significant reduction in sickling of red blood cells, engraftment of gene-edited SCD HSPCs in

86 tical hyperelastic constitutive model of the red blood cell (erythrocyte) membrane based on recently

87 d T-cell leukemia/lymphomas (T-ALL) and pure red blood cell erythroleukemias (EL).

88 ained throughout parasite development within red blood cells, even during a period coincident with ex

89 Transfusion of red blood cells expressing self-antigen epitopes can all

90 uently sampled intravenous glucose test, and red blood cell fatty acid profiles were measured by gas

91 Red blood cell flux (laser Doppler flowmetry) was measur

92 Red blood cell flux was measured by laser Doppler flowme

93 ements in layers I-V show that the capillary red-blood-cell flux and oxygenation heterogeneity, and t

94 ere receiving at least 150 mL/kg per year of red blood cells for the past 2 years at the time of enro

95 otes erythroid precursor cell maturation and red blood cell formation in contexts of homeostasis and

96 cular mechanism by which TH functions during red blood cell formation, results that are potentially u

97 y contribute to the early loss of uninfected red blood cells found in malarial anemia from both speci

98 f RBC ratios, a decrease was observed in the red blood cell-free layer and platelet margination due t

99 is an important cyclic nucleotide exporter, red blood cells from ABCC4null/PEL-negative individuals

100 beads from small molecules and surface bound red blood cells from dimethyl sulfoxide for antigen typi

101 umanized NOD/SCID mouse model engrafted with red blood cells from G6PD deficient donors.

102 a, lymphocyte, macrophage, karyorrhexis, and red blood cells from standard hematoxylin and eosin-stai

103 re were 728 patients randomized to the fresh red blood cell group and 733 to the standard-issue group

104 re randomized with 768 patients in the fresh red blood cell group and 770 in the standard-issue group

105 resh (147 of 728 [20.2%]) and standard-issue red blood cell groups (133 of 732 [18.2%]), with an unad

106 characterized by an excessive production of red blood cells, have markedly reduced endothelial funct

107 ctuations in contrast and pixel intensity of red blood cells in an aqueous vein were calculated to co

108 mpaired the capacity of phagocytes to engulf red blood cells in the ICH brain and in primary cultures

109 We simulate deformable red blood cells in the microcirculation using the immers

110 cclusion by increasing entrapment of sickled red blood cells in the microvasculature.

111 This molecule enhances clearance of CO from red blood cells in vitro and in vivo Herein, we tested w

112 Red blood cell indices significantly declined during acu

113 to measurements performed on intact infected red blood cells (intact infected RBC, 77.3% and 79.2%).

114 Assessment of red blood cell integrity, white blood cell differential

115 e (LD50) of BoNT/A, and transfusion of these red blood cells into naive mice affords protection for u

116 for chemokines, and their capacity to block red blood cell invasion by a transgenic Plasmodium knowl

117 odies that significantly reduce the speed of red blood cell invasion by the merozoite, thereby potent

118 asmodium falciparum (PfMyoA) is critical for red blood cell invasion.

119 a potential regulator of genes important for red blood cell invasion.

120 Plasmodium invasion of red blood cells involves malaria proteins, such as retic

121 Secondary endpoints were changes in red blood cell, iron and mineral, and bone-related param

122 with the underlying cytoskeleton, whereas in red blood cells, it is also known to cause lysis.

123 inducers, HbF is present only in a subset of red blood cells known as F cells.

124 lying the retina act together to acidify the red blood cell leading to O(2) secretion.

125 ilico experiment-simulating an entire mammal red blood cell lipid bilayer and cytoskeleton as modeled

126 a combination of substance P, periostin, and red blood cell lysate (representing hemosiderin).

127 to the analysis of endogenous alpha-syn from red blood cells lysate of healthy controls and PD patien

128 Red blood cells mature within the erythroblastic island

129 emia defined as reduced hemoglobin levels of red blood cells may carry less oxygen to skeletal muscle

130 Thus, the ecology of red blood cells may play a key role in determining the r

131 ade of polymeric cores wrapped with modified red blood cell membrane with two inserted key components

132 ortance to human life, be it gas exchange in red blood cells, metabolite excretion, drug/toxin uptake

133 tion near the reactive surface determined by red-blood-cell migration, the platelet effective reactiv

134 We used "red blood cell mimicry" to achieve long sensor circulati

135 We have fabricated nanoengineered red blood cells (NERBCs) by an environmentally friendly

136 0.01), S-nitrosothiols (P = 0.03) and total red blood cell NO (P = 0.001) were collectively reduced

137 output, blood vessel growth and circulating red blood cell numbers.

138 m relying on distinct invasion pathways into red blood cells of different ages.

139 malaria parasite replicates asexually in the red blood cells of its vertebrate host employing epigene

140 orne intracellular parasites that infect the red blood cells of their mammalian host, leading to seve

141 In this study we examined the effects of red blood cells on smooth muscle cell mineralization and

142 moglobin S polymerization and its effects on red blood cells, only two therapies for SCD - hydroxyure

143 opically confirmed requiring >=2 U of packed red blood cells or resulting in death.

144 total of 367 blood smears and corresponding red blood cell pellets, including 185 smears (50.4%) tha

145 es, focusing on three distinct stages of the red blood cell phase of the Plasmodium life cycle.

146 holesteryl esters, plasma phospholipids, and red blood cell phospholipids were generally not associat

147 Primary outcome was blood components (red blood cells, platelets, plasma) administered during

148 orce throughout human history, selecting for red blood cell polymorphisms that confer innate protecti

149 the proportion of patients receiving packed red blood cell (PRBC) using a liberal trigger hemoglobin

150 Third-party packed red blood cells (pRBCs) are often used as an oxygen carr

151 The benefits of prehospital packed red blood cells (PRBCs), plasma, or transfusion of both

152 rease the iron supply when needed to support red blood cell production and other essential functions.

153 g electron microscopy analysis, SAA mediated red blood cell (RBC) agglutination, platelet activation

154 GLT2 inhibitor dapagliflozin on haematocrit, red blood cell (RBC) counts and reticulocyte levels in h

155 f these progenitors are unneeded to maintain red blood cell (RBC) counts.

156 The influence of red blood cell (RBC) deformability in whole blood on pla

157 Red blood cell (RBC) invasion by malaria merozoites invo

158 The biconcave disk shape of the mammalian red blood cell (RBC) is unique to the RBC and is vital f

159 The mature red blood cell (RBC) lacks a nucleus and organelles char

160 translational modifications (PTMs) of Hb and red blood cell (RBC) membrane proteins of transgenic SCD

161 rating lipid asymmetry was first detected in red blood cell (RBC) membranes, but the P4-ATPases respo

162 s part of a 3-component complex that affects red blood cell (RBC) membranes.

163 samples depending on their abundance in the red blood cell (RBC) or plasma; it is essential to prein

164 A red blood cell (RBC) performs its function of adequately

165 Red blood cell (RBC) preparations were used in vitro to

166 es are commonly used for the manipulation of red blood cell (RBC) suspensions and analyses of flow-me

167 port of nitric oxide (NO) bioactivity by the red blood cell (RBC) to mediate hypoxic vasodilation and

168 that many enhancer variants associated with red blood cell (RBC) traits map to enhancers that are co

169 There is no consensus on the benefit of red blood cell (RBC) transfusion after transcatheter aor

170 ed clinical trial findings support decreased red blood cell (RBC) transfusion and short-term toleranc

171 Although non-O blood group and red blood cell (RBC) transfusion are known to be associa

172 Current guidelines advocate to limit red blood cell (RBC) transfusion during surgery, but the

173 iron-supplemented IDA patients required less red blood cell (RBC) transfusion during the postoperativ

174 terplay between these factors on measures of red blood cell (RBC) transfusion efficacy, we conducted

175 tion/Comparison/Outcome (PICO) questions for red blood cell (RBC) transfusion in adult patients in 3

176 Evidence regarding red blood cell (RBC) transfusion practices and their imp

177 One sequela that occurs in a subset of red blood cell (RBC) transfusion recipients is the devel

178 estinal bleeding is a leading indication for red blood cell (RBC) transfusion worldwide, although opt

179 ical studies have linked NEC with antecedent red blood cell (RBC) transfusions, but the underlying me

180 esistant to available treatments, leading to red blood cell (RBC) transfusions, iron overload, shorte

181 boinflammatory complications associated with red blood cell (RBC) transfusions.

182 by changes in O(2) pressure that occur as a red blood cell (RBC) transits between the lungs and tiss

183 In this paper, we investigate the role that red blood cell (RBC) transport plays in establishing oxy

184 using these clinical measurements of single-red blood cell (RBC) volume and hemoglobin.

185 e parameters such as hematocrit, hemoglobin, red blood cell (RBC), white blood cell (WBC), and platel

186 erized the most abundant EVs of human blood, red blood cell (RBC)- and platelet (PLT)-derived EVs and

187 oxygen supply, typically delivered by packed red blood cells (RBC).

188 , polymerization of HbS leads to sickling of red blood cells (RBC).

189 n on red cell production (ie, no increase in red blood cell [RBC] count despite elevated erythropoiet

190 of transferrin receptor 2 [Tfr2] to control red blood cell [RBC] synthesis).

191 n dependent (mean hemoglobin, <10.0 g/dL; <4 red blood cell [RBC] units transfused per 8 weeks), and

192 ve measurement of the Hb content in a single red blood cell, RBC, based on magnetophoretic mobility.

193 xposed to the blood flow, clearing senescent red blood cells (RBCs) and recycling iron from hemoglobi

194 omechanical characteristics of healthy human red blood cells (RBCs) and their membrane mechanical pro

195 Stored red blood cells (RBCs) are needed for life-saving blood

196 results in dramatic morphological changes in red blood cells (RBCs) because of polymerization of the

197 ten affect the shape, number, and content of red blood cells (RBCs) dramatically.

198 Malaria parasites invade healthy red blood cells (RBCs) during the blood stage of the dis

199 I:C]), followed by the transfusion of murine red blood cells (RBCs) expressing the human KEL glycopro

200 killed human labor to distinguish the normal red blood cells (RBCs) from sickled cells.

201 IVIg were tested for IgM and IgG binding to red blood cells (RBCs) from wild-type (WT), alpha1,3-gal

202 mortality, among persons who receive stored red blood cells (RBCs) have recently garnered considerab

203 articles (NPs) to glycophorin A receptors on red blood cells (RBCs) improved the blood half-life.

204 The motion of red blood cells (RBCs) in microchannels is important for

205 hemoglobin molecule that polymerizes inside red blood cells (RBCs) in reduced oxygen tension.

206 ze the phospholipids in the outer leaflet of red blood cells (RBCs) is reported.

207 laria parasite, Plasmodium chabaudi, and the red blood cells (RBCs) it targets.

208 es, and had lower baseline levels of CD35 on red blood cells (RBCs) leading to a significant reductio

209 e consequence of the synchronous bursting of red blood cells (RBCs) on completion of the malaria para

210 Red blood cells (RBCs) passing through heart pumps, pros

211 Red blood cells (RBCs) release ATP in response to deoxyg

212 In response to haemoglobin deoxygenation, red blood cells (RBCs) release ATP, which binds to endot

213 inical medicine that a 1-unit transfusion of red blood cells (RBCs) should yield a posttransfusion he

214 s capable of accurately tracking the flow of red blood cells (RBCs) through a no-reaction lateral flo

215 , HbS polymerizes into rigid fibers, causing red blood cells (RBCs) to sickle; leading to numerous ad

216 s initiated by increased adherence of sickle red blood cells (RBCs) to the vascular endothelium.

217 e (G6PD) deficiency decreases the ability of red blood cells (RBCs) to withstand oxidative stress.

218 Red blood cells (RBCs) transport oxygen to tissues and r

219 Transient pore formation on the membrane of red blood cells (RBCs) under high mechanical tensions is

220 y also relate to inefficient gas exchange by red blood cells (RBCs), a process that is poorly charact

221 mprehensive biological investigations on the red blood cells (RBCs), advanced strategies of RBC-media

222 To invade red blood cells (RBCs), apicomplexan parasites have to a

223 Transfusion of blood, or more commonly red blood cells (RBCs), is integral to health care syste

224 Specifically, we discuss how red blood cells (RBCs), platelets, neutrophils, mesenchy

225 C differentiation into platelets rather than red blood cells (RBCs), show a strong heritability enric

226 Upon Plasmodium falciparum infection of the red blood cells (RBCs), the parasite replicates and cons

227 e to each other within a group of individual red blood cells (RBCs), which is crucial for imaging cel

228 ria parasite Plasmodium falciparum to invade red blood cells (RBCs).

229 to the physical properties and behaviors of red blood cells (RBCs).

230 erations in membranes of isolated and intact red blood cells (RBCs).

231 major surface antigen displayed on infected red blood cells (RBCs).

232 rm Infrared (ATR-FTIR) to detect B. bovis in red blood cells (RBCs).

233 f diseases are associated with stiffening of red blood cells (RBCs; e.g., sickle cell anemia or malar

234 ature has evolved from viewing erythrocytes (red blood cells [RBCs]) as passive carriers of oxygen to

235 e their ability to opsonize erythrocytes (or red blood cells, RBCs) and cause anemia.

236 As red blood cells rupture, the overall conductivity of the

237 nclude all the analytical operations needed: red blood cell separation, conditioning, enzymatic recog

238 tal number of Fe atoms and the total mass of red blood cells show very good agreement with previously

239 administration of haptenated syngeneic mouse red blood cells (sMRBC) leads to hapten-specific suppres

240 The clinical consequences of red blood cell storage age for critically ill pediatric

241 Host genetic background affects red blood cell structure, for example, and red blood cel

242 on, it may also reflect the heterogeneity of red blood cell surfaces within and between hosts.

243 The abundance of infected red blood cells that accumulate in the cerebral vasculat

244 if challenged with P. berghei ANKA-infected red blood cells that bypass the liver stage of infection

245 cytes may express proteins on the surface of red blood cells that elicit immune responses in naturall

246 m malaria mediate phagocytosis of uninfected red blood cells that expose PS and have been linked to l

247 roved CD34+ culture system to engineer human red blood cells that express these chimeric proteins.

248 Splenic sequestration of red blood cells, the appearance of circulating poikilocy

249 eplicate, this parasite must invade immature red blood cells through a process requiring interaction

250 antigen VAR2CSA, which mediates adherence of red blood cells to chondroitin sulfate A (CSA) in the pl

251 = 24) were inoculated with P. vivax-infected red blood cells to initiate infection, and were treated

252 We utilize red blood cells to prolong the circulatory half-life of

253 capillary network and dynamically channeling red blood cells toward the initiating signal.

254 e genes contained in 75 loci associated with red blood cell traits.

255 ial, we compared a restrictive threshold for red blood cell transfusion (transfuse if hemoglobin<7.5

256 Both anemia and red blood cell transfusion are independently associated

257 te no evidence of difference in incidence of red blood cell transfusion for a titration-dose strategy

258 ions of blood donor sex with mortality among red blood cell transfusion recipients is conflicting.

259 Among red blood cell transfusion recipients, transfusions from

260 o liberal (n = 492) or restrictive (n = 521) red blood cell transfusion thresholds based on infants'

261 days and 16 years were eligible if the first red blood cell transfusion was administered within 7 day

262 Sixty-six (43.1%) red blood cell transfusion-dependent and 53 (40.2%) plat

263 ime CD4(+) T cells in response to allogeneic red-blood-cell transfusion.

264 tigens that are present at high frequency on red blood cells, transfusion incompatibility problems, d

265 0.32 transmissions per million (106) packed red blood cell transfusions (95% CI, 0.29-0.65 transmiss

266 otocol (MHP) and received at least 1 unit of red blood cell transfusions (RBC).

267 A patient with asplenia and multiple red blood cell transfusions acquired babesiosis infectio

268 Red blood cell transfusions are commonly administered to

269 o post transplant complications, with packed red blood cell transfusions being the most common interv

270 Safely reducing red blood cell transfusions can prevent transfusion-rela

271 -5 CKD and anemia, we evaluated incidence of red blood cell transfusions for participants randomized

272 The median number of red blood cell transfusions per patient was 3 in the KPN

273 ent for at least 28 days who either required red blood cell transfusions while on ruxolitinib or ruxo

274 ctive transfusion approach resulted in fewer red blood cell transfusions without increasing the risk

275 Among patients who received red blood cell transfusions, receipt of a transfusion fr

276 ive renal replacement therapy, postoperative red blood cell transfusions, time to first extubation, t

277 ulative exposure-while reducing the need for red blood cell transfusions-is unknown.

278 nal replacement therapy (RRT), postoperative red blood cells transfusions, time to extubation, time t

279 of Hb toxicity resulting from physiological red blood cell turnover.

280 y, this work describes the AFM-IR spectra of red blood cells under polyvinyl alcohol hydrogels.

281 ohort who received at least 1 uncrossmatched red blood cell unit in the trauma bay (2013-2016).

282 sk of mortality, adjusting for the number of red blood cell unit transfusions.

283 The number of transfused red blood cell units from female donors, previously preg

284 interval (CI) 0.55-0.68, P < 0.00001], 0.43 red blood cell units per patient (mean difference -0.43,

285 associated with reduced transfusion need of red blood cell units, lower complication and mortality r

286 tility by measuring ATP release from flowing red blood cells using a luciferin/luciferase chemilumine

287 g laser ophthalmoscope to measure changes in red blood cell velocities, vessel diameter, and flow in

288 Tenofovir-diphosphate (TFV-DP) in red blood cells was used to categorize participants into

289 Circulating red blood cells were found to retain transferrin recepto

290 globin-O(2) affinity (~32% fall in P(50)) of red blood cells, when exposed to reciprocal changes in O

291 te life cycle, Plasmodium falciparum invades red blood cells, where it catabolizes hemoglobin and seq

292 cation of Plasmodium falciparum within human red blood cells, which relies on a precisely timed casca

293 le parasites must have access to susceptible red blood cells, which they invade using pairs of parasi

294 in the synchronous release of parasites from red blood cells, which triggers 48-hour fever cycles in

295 tivation of indiscriminate host clearance of red blood cells while increasing the half-life of that r

296 by sorting for droplets containing a single red blood cell with 85% purity.

297 By mapping such infected red blood cells with nondestructive X-ray microscopy, we

298 thology of malaria is caused by infection of red blood cells with unicellular Plasmodium parasites.

299 applicability for biological samples, sheep red blood cells with various melittin peptide contents s

300 plasma iron, microcytosis, and an increased red blood cell zinc-protoporphyrin-to-heme ratio.