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1 D mice were more resistant to infection when reexposed.
2 as significantly shorter (P<0.05) in animals reexposed 1 or 2 days after the first exposure.
3  recorded by electroencephalograph (EEG) and reexposed 1, 2, or 6 days later.
4 ntrol and clearance as those seen in animals reexposed after HCV clearance.
5                                              Reexposed baboons with low worm recovery and oviposition
6 rogressed, compared with both uninfected and reexposed baboons.
7 rol of pulmonary histoplasmosis in naive and reexposed mice.
8       These complexes can either dissociate, reexposing p58 to the degradation pathway, or can bind t
9 drug relapse model, we found previously that reexposing rats to heroin-associated contexts, after ext
10 ats to self-administer heroin and 11 d later reexposed them to heroin-associated cues or novel cues f
11                         Subsequently, fibers reexposed to antigen retained greater than 85% of the in
12 dence, suffer little or no fetal damage when reexposed to CMV.
13               But the few survivors who were reexposed to diseased patients were not attacked a secon
14 current HIT in 20 patients with previous HIT reexposed to heparin 4.4 years (mean) post-HIT; 17 patie
15 17 (47%), whereas none of 3 medical patients reexposed to heparin developed seroconversion.
16 ients with a previous history of HIT who are reexposed to intraoperative (but not postoperative) hepa
17  in strength when visually deprived mice are reexposed to light.
18 behavior when abstinent rats or monkeys were reexposed to nicotine and/or cues that had previously be
19  drug- and training-free period before being reexposed to noncontingent presentations of the light co
20 protection and enhanced immunopathology when reexposed to one of the two viruses.
21 weeks later, sensitized/challenged mice were reexposed to OVA (secondary challenge) and airway respon
22 also were isolated overnight and were either reexposed to pups for 2 hours or kept isolated from pups
23 nal, they were isolated from pups overnight, reexposed to pups for 2 hours, and then killed.
24  brain regions is seen in human addicts when reexposed to the drug.
25                      The next day, rats were reexposed to the FST to assess ketamine-induced antidepr
26 ue-induced heroin seeking in rats previously reexposed to the heroin-associated cues on induction day
27 ed to a footshock show conditional fear when reexposed to the shock context.
28 mmediately shocked (controls) and thereafter reexposed to the shocked context to test for fear memory
29 eproduce the initial attachment pattern when reexposed to the surrogate nipple.
30  was unaffected by whether the rats had been reexposed to the training reward under the antagonists.
31                                When pups are reexposed to the unconditioned stimulus (footshock) befo
32                                Finally, rats reexposed to TMT after a 24-h delay did not demonstrate
33 ted with 1 microM TRH for 10 s or 10 min and reexposed to TRH, there was almost no IP3 or [Ca2+]i inc
34 s and test solution for 20 minutes, and then reexposed to Tyrode's for 20 minutes.
35 s shielded by the alpha subunit, and becomes reexposed upon receptor activation.
36      The potential to renew morphogenesis is reexposed when the field is reduced back to amplitudes b