ライフサイエンスコーパス: reflex

1 nal sensitivity, presence of a photic sneeze reflex).

2 ice at one minute after the loss of righting reflex.

3 ate with the upper airway or by a vago-vagal reflex.

4 t intermediate stages in the vestibulocollic reflex.

5 tion or ethanol-induced loss of the righting reflex.

6 = 0.007) and an attenuated exercise pressor reflex.

7 and both activated the medial olivocochlear reflex.

8 mulation of the median nerve generating an H-reflex.

9 WKY rats, thus indicating a parasympathetic reflex.

10 isplay sophistication beyond a stereotypical reflex.

11 +) monocytes in the cholinergic inflammatory reflex.

12 fman reflex (H-reflex) and (3) conditioned H-reflex.

13 ility or maladaption of the vestibulo-ocular reflex.

14 train at a rapid speed, as inspired by human reflex.

15 ol on locomotor activity or loss-of-righting reflex.

16 g; they have only unconditioned consummatory reflexes.

17 l fold adduction, swallowing, and expiratory reflexes.

18 cadian photoentrainment, and pupillary light reflexes.

19 ximal leg muscles, and decreased deep tendon reflexes.

20 d motor responses, often called long-latency reflexes.

21 processes rather than immediate compensatory reflexes.

22 minae III/IV INs modulate dynamic corrective reflexes.

23 features, including impaired pupillary light reflexes.

24 had little effect on AITC-evoked respiratory reflexes.

25 disease alters nociceptive pulmonary-cardiac reflexes.

26 contribution to both short- and long-latency reflexes.

27 der-stretch sensing and urethral micturition reflexes.

28 thout altering life-saving autonomic hypoxic reflexes.

29 vascular occlusion or altered neuromuscular reflexes.

30 roportion of respondents used absent corneal reflexes (33.5%) and absent pupillary reflexes (36.2%) a

31 orneal reflexes (33.5%) and absent pupillary reflexes (36.2%) at 24 hours, which is earlier than the

32 ohol on locomotor activity, loss-of-righting reflex (a measure of alcohol sedative actions), and on b

33 It makes use of the startle reflex, a defensive response elicited by an immediate, u

34 taneous nociceptive neurons leads to a nerve-reflex action that is sufficient to provide a danger sig

35 of healthy human adults, we examined whether reflex airway defense mechanisms, specifically swallowin

36 he summation of the responses evoked by each reflex alone.

37 Vagus nerve-mediated reflexes also control immune system responses to infecti

38 tality (+27.2%), impaired neurodevelopmental reflexes, altered blood pH, and reduced bodyweight.

39 visual processes such as the pupillary light reflex and circadian entrainment but also contribute to

40 visual light responses (e.g. pupillary light reflex and circadian entrainment).

41 the sensitivity of both the pupillary light reflex and circadian photoentrainment, thereby shifting

42 effect of whole-body vibration (WBV) on the reflex and non-reflex components of spastic hypertonia a

43 Following visual motion exposure, both reflex and perceptual thresholds were significantly furt

44 ibition in vitro as well as acoustic startle reflex and social interaction in vivo of the Fmr1-KO mic

45 t for the relative weakness of the human MOC reflex and the difficulty in demonstrating a robust func

46 (DCNs) activates a nociceptive sensorimotor reflex and the same afferent stimulation also evokes blo

47 ar patients, could disrupt vestibular ocular reflex and vestibular-perceptual thresholds of self-moti

48 b) leads to partial restoration of the pupil reflex and visual function.

49 estibular input, but normal vestibulo-ocular reflexes and apparently normal motor performance during

50 ucial role in inflammation through avoidance reflexes and behaviors, but can also regulate sterile cu

51 rcadian photoentrainment and pupillary light reflexes and contrast detection for image formation.

52 ant dual role in nociception and sympathetic reflexes and could provide a therapeutic target for trea

53 ke behaviors and increased exercise-mediated reflexes and group III/IV muscle afferent sensitization.

54 presents progressive spasticity, exaggerated reflexes and muscular weakness.

55 t central neurons mediating vestibulo-spinal reflexes and self-motion perception optimally encode nat

56 ical stimulation, (2) test Hoffman reflex (H-reflex) and (3) conditioned H-reflex.

57 he summation of the responses evoked by each reflex) and hypo-additive for peripheral haemodynamics (

58 sensitivity threshold, the middle-ear muscle reflex, and the auditory-brainstem response to clicks in

59 rease in rate-dependent depression of spinal reflexes, and ground and skill locomotion were improved

60 ncreased rate-dependent depression in spinal reflexes, and improved ground and skill locomotion.

61 s, including light aversion, pupillary light reflexes, and photoentrainment of circadian rhythms.

62 ing noises activate the medial olivocochlear reflex; and (3) adaptation occurs even for highly fluctu

63 the detection of noxious stimuli, withdrawal reflexes, anxiety, or reward.

64 s contributing to the previously established reflex arc resulting in efferent vagal activity and asth

65 adjacent, unstimulated skin through a nerve reflex arc.

66 associated with bladder filling and voiding reflex arcs.

67 matosensory processing, namely, sensorimotor reflexes are driven by the differential spatial recruitm

68 e (responses during co-activation of the two reflexes are greater than the summation of the responses

69 he lower urinary tract (LUT) and micturition reflexes are sexually dimorphic across mammals.

70 amics (responses during co-activation of the reflexes are smaller than the summated responses).

71 Image quality (sharpness/focus, reflex artifacts, contrast, and illumination), field-of-

72 In loss of righting reflex assessment, knockout mice revealed increased sens

73 These results demonstrate key reflex autonomic pathways regulating exercise heart rate

74 hanged with respect to lick behavior but not reflex behavior.

75 igating brain driven pain processes than the reflex behavior.

76 re, response similarity between the lick and reflex behaviors diverged near perceptual threshold.

77 cord direct muscle responses (M-waves) and H-reflexes, both of which are comparable to those recorded

78 p (AFN group 32 patients) with functional or reflex bradyarrhythmias or vagal AF treated with AFN abl

79 es airway nociceptive afferents resulting in reflex bradycardia in healthy animals.

80 sulfur dioxide (SO(2) ) triggers coughs and reflex bronchoconstriction, and stimulation of vagal bro

81 s elicited after the shortest latency spinal reflexes but prior to the onset of voluntary activity ca

82 The cough reflex can be triggered by nociceptive neurons innervati

83 appropriate operant conditioning of a spinal reflex can improve impaired locomotion.

84 how distinct components of the long-latency reflex can work independently and together to generate s

85 onduction delays, the earliest part of these reflexes can only arise from spinal circuits.

86 n cells (ipRGCs) mediate the pupillary light reflex, circadian entrainment, and may contribute to lum

87 eals how a dedicated laryngeal sensory motor reflex circuit protects our airways from aspirated foods

88 tem circuitry, we show in vitro evidence for reflex circuit-specific postnatal abnormalities in the j

89 These results demonstrate a novel reflex circuit-specific proprioceptive sensory abnormali

90 ive plasticity within the spinal sympathetic reflex circuit.

91 ctional subdivision correlated with aura and reflex component.

92 By contrast, long-latency reflex components are typically assumed to originate fro

93 e-body vibration (WBV) on the reflex and non-reflex components of spastic hypertonia and intramuscula

94 insight into the factors controlling spinal reflex conditioning; they suggest that the conditioning

95 The reflex-conditioning protocol uses electromyography (EMG)

96 and demonstrate its use in every step of the reflex-conditioning protocol.

97 In addition, L-EES may improve the reflex-conditioning protocol; it has potential to automa

98 The ENS exerts critical local reflex control over many essential gut functions; includ

99 respiratory homeostasis depends on autonomic reflexes controlled by neuronal circuits of the brainste

100 per se, rather than the medial olivocochlear reflex, could facilitate noise adaptation by reducing th

101 ession, rather than the medial olivocochlear reflex, could facilitate noise adaptation.

102 Stopping reflex CRC screening for LS after age 80 years may be re

103 genous opioids in cardiac sympathoexcitatory reflex (CSR) responses remain unclear.

104 core, injury severity score, pupillary light reflex, CT findings (compressed basal cistern and midlin

105 ories to adopt single-tiered testing without reflex culture.

106 microvascular function contribute to blunted reflex cutaneous vasodilatation during heat stress in he

107 Chronic statin treatment improves reflex cutaneous vasodilatation in formerly hypercholest

108 Attenuated reflex cutaneous vasodilatation in healthy human ageing

109 We demonstrate that reflex cutaneous vasodilatation is impaired in older hyp

110 The reflex decrease persisted for at least 6 months after co

111 The H-reflex decrease was accompanied by improvements in walki

112 With swing-phase down-conditioning, the H-reflex decreased much faster and farther than did the H-

113 ting showed gait-difficulties, absent tendon reflexes, decreased joint-position, positive Romberg's t

114 ess pain responsiveness and failing righting reflex) deficits that coincided with global lower neuron

115 iency causes an acceleration of sensorimotor reflex development in the first postnatal week followed

116 thesize or use dopamine lack the conditioned reflexes discussed by Pavlov or the appetitive behavior

117 eyelid height or postoperative upper margin reflex distance (MRD1) of less than 2 mm.

118 orced eyelid closure, upper eyelid margin-to-reflex distance (MRD1), corneal staining, static and dyn

119 Margin reflex distance 1 (MRD1), MRD2, and levator function wer

120 rovement using lower lid margin-to-pupillary reflex distance was the most common outcome measure.

121 All previous studies conditioned the reflex during steady-state maintenance of a specific pos

122 rast, the present study down-conditioned the reflex during the swing-phase of locomotion in people wi

123 ecifically, we down-conditioned the soleus H-reflex during the swing-phase of locomotion in people wi

124 ractive cardiovascular consequences of these reflexes during exercise and revealed various modes of i

125 conditioned H-reflex relative to the test H-reflex) during APAs before step initiation (functional t

126 exhibited low-pass filtered vestibulocollic reflex dynamics.

127 vertebrates, with rapidly responding neural reflexes ensuring proper blood flow despite changes in p

128 Reflex epilepsies have been demonstrated to exploit spec

129 Although the exercise pressor reflex (EPR) and the chemoreflex (CR) are recognized for

130 e interactive effect of the exercise pressor reflex (EPR) and the chemoreflex (CR) on the cardiovascu

131 The exercise pressor reflex (EPR) is defined by a rise in mean arterial press

132 novel working model of the exercise pressor reflex (EPR).

133 eptive signals and modulate exercise pressor reflexes (EPRs).

134 a (inhaled isoflurane) inhibited the cardiac reflexes evoked by inhaled AITC but not injected AITC.

135 Furthermore, anaesthesia reduced the cardiac reflexes evoked by inhaled but not injected AITC.

136 naptic circuits may underlie polarized motor reflexes evoked by local gut stimulation.

137 n is impaired in rLG, confirming the role of reflex feedback in regulating force duration in intact m

138 alation in SH rats evokes de novo adrenergic reflexes following vagal afferent activation.

139 tocol uses electromyography (EMG) to measure reflexes from specific muscles elicited using transcutan

140 osensory input to powerfully alter autonomic reflex function and other behaviors.SIGNIFICANCE STATEME

141 st that the conditioning protocols targeting reflex function in a specific movement phase provide a p

142 The results suggest that conditioning reflex function in a specific phase of a dynamic movemen

143 ly related to several clinical conditions as reflex/functional bradyarrhythmias and vagal atrial fibr

144 out electrical stimulation, (2) test Hoffman reflex (H-reflex) and (3) conditioned H-reflex.

145 Operant conditioning of Hoffmann's reflex (H-reflex) is a non-invasive and targeted therape

146 Although the medial OC (MOC) reflex has been extensively studied in humans, via contr

147 ate that habituation of the auditory startle reflex (hASR) tested at bedside constitutes a novel, sim

148 ircuitry subserving the defensive hand-blink reflex (HBR), a response elicited by intense somatosenso

149 ient navigation, and anti-HCV screening with reflex HCV RNA testing.

150 MENT Following a limb perturbation, multiple reflexes help to restore limb position.

151 addition of the individual responses to each reflex (i.e. additive interaction).

152 f proprioceptive axons mediating the stretch reflex (Ia afferents).

153 The aim was to modify the functioning of the reflex in a specific phase of a dynamic movement.

154 eased much faster and farther than did the H-reflex in all previous animal or human studies with the

155 conditioned H-reflex relative to the test H-reflex in both the tasks.

156 ing the exaggeration of the exercise pressor reflex in PAD and a reduction in the activity of the P2X

157 ing the exaggeration of the exercise pressor reflex in PAD and a reduction of the activity of the P2X

158 effects on the exaggerated exercise pressor reflex in PAD rats.

159 effects on the exaggerated exercise pressor reflex in rats with peripheral artery disease (PAD).

160 The aberrant AITC-evoked reflex in SH rats was not reduced by acute blood pressur

161 we studied the optomotor gaze stabilization reflex in tethered flight and quantified how head moveme

162 r maintaining a functional O(2) chemosensory reflex in the adult, modulate sleep homeostasis, and are

163 ted at one minute after the loss of righting reflex in the mice, which was about two minutes after th

164 tributes to learning in defensive withdrawal reflexes in Aplysia californica, we investigated the mol

165 nal circuits also contribute to long-latency reflexes in distal and forearm muscles, alongside supras

166 long-range mutual inhibition between reflexes in distant segments, enabling overall motion of

167 In conclusion, sensory symptoms and loss of reflexes in Gerstmann-Straussler-Scheinker syndrome can

168 the sensory symptoms and loss of lower limb reflexes in Gerstmann-Straussler-Scheinker syndrome is d

169 erts descending regulation over airway vagal reflexes in male and female rats using a range of neuroa

170 t of the neural circuits that control penile reflexes in rats, circuits that are commonly referred to

171 Here we probe the role of reflexes in the rapid perturbation responses of muscle b

172 rs located in the pulmonary artery induces a reflex increase in sympathetic outflow; however, this ha

173 -15, -30 and -45 mmHg) was applied to elicit reflex increases in muscle sympathetic nerve activity (M

174 early (50-75 ms) portion of the long-latency reflex, indicating that these components of the rapid mo

175 art rate [DeltaHR], average heart rate [HR], reflexes, induction/recovery times) parameters in repeat

176 Metabolite-induced reflex influences in sympathetic outflow originating fro

177 data suggest the existence of a lesser-known reflex input involved in sympathetic activation in human

178 The vestibulocollic reflex is a compensatory response that stabilizes the he

179 ring sympathoexcitation in SH rats, and this reflex is dependent on vagal afferents but is not due to

180 This aberrant reflex is independent of steady state hypertension and i

181 While this reflex is readily tested in humans, mechanistic studies

182 or pupil dilation, modulation of the startle reflex is valence specific.

183 Operant conditioning of Hoffmann's reflex (H-reflex) is a non-invasive and targeted therapeutic inter

184 This condition involves an overactive spinal reflex loop that resists the passive lengthening of musc

185 ing.SIGNIFICANCE STATEMENT Cardiorespiratory reflexes maintain autonomic balance and ensure cardiovas

186 ponse is distinct from the negative feedback reflex mediated by aortic and carotid sinus baroreceptor

187 The activation of reflex micturition, with associated detrusor contraction

188 Gustatory reflexes modulate the amount and composition of saliva s

189 ng-phase conditioning protocol decreased the reflex much faster and farther than did the steady-state

190 wide range of clinical motor phenomena, from reflex myoclonus to myoclonic epilepsy, caused by abnorm

191 a peripheral role to modulate sensory-motor reflexes necessary for suckling and may be part of the m

192 ence of episodic, involuntary airway defense reflexes on sleep and vigilance and cardiovascular funct

193 Several disorders present reflex or persistent increase in vagal tone that may cau

194 de combined with no significant changes in H-reflex parameters suggests this increased strength is li

195 l that aims to change the functioning of the reflex pathway during a specific phase of a complex move

196 This is regulated by a brainstem reflex pathway.

197 d to change the excitability of the targeted reflex pathway; reflex size gradually changed over 8-10

198 he neurophysiology and neuroanatomy of these reflex pathways are well understood, however, the mechan

199 suggest distinct differences in nociceptive reflex pathways dependent on cardiovascular disease, adm

200 ans and macaque monkeys, suggesting that the reflex pathways that regulate pupil diameter are under s

201 in those testing positive by either RIDT or reflex PCR (69.9%; P < 0.05).

202 ve feeding behavior, the proboscis extension reflex (PER), elicited when external food cues are inter

203 ts using a range of neuroanatomical tracing, reflex physiology, and chemogenetic techniques.

204 Pupillary light reflex (PLR) is an involuntary response where the pupil

205 repulse inhibition of the acoustic startling reflex (PPI; a marker of psychotic-like behavior), memor

206 ms, such as recurrent inhibition and stretch reflex, probably play a major role in the synergic contr

207 e resting pressure and Recto Anal Inhibitory Reflex (RAIR) within 1-month.

208 nd involuntary feedback control loops (i.e., reflexes), reflect computations associated with high-lev

209 ysiological states, autonomic nervous system reflexes regulate regional sympathetic nerve activity an

210 e pathogenic mechanism and the neuromuscular reflex-related phenotype (e.g. tremors accompanied by cl

211 However, the neuromuscular reflex-related symptoms of ANM have not been explained.

212 quantified by the ratio of the conditioned H-reflex relative to the test H-reflex in both the tasks.

213 PSI (i.e. higher ratio of the conditioned H-reflex relative to the test H-reflex) during APAs before

214 d enters the urethra at low bladder volumes, reflexes relax the bladder and evoke external urethral s

215 ifferent from the summated responses to each reflex response alone (P >= 0.1).

216 cles in the later portion (75-100 ms) of the reflex response.

217 require training are based on sub-conscious, reflex responses (e.g. optokinetic nystagmus) that don't

218 es neuroendocrine, behavioral, and autonomic reflex responses that ensure optimal internal organ func

219 Myocardial ischemia evokes powerful reflex responses through activation of vagal and sympath

220 acupoint inhibits excitatory cardiovascular reflex responses through modulation of the autonomic ner

221 have well-established roles in a variety of reflex responses to changes in ambient light intensity,

222 Here, we studied reflex responses to irritants in normotensive Wistar-Kyo

223 ly, inhibition of SubM potentiated laryngeal reflex responses, while prior lesions of VLO abolished t

224 f dorsal excitatory INs make to sensorimotor reflex responses.

225 The righting reflex (RR) is frequently used to assess level of arousa

226 It can easily be observed during the red reflex screening at neonatal wards.

227 eart rate variability (HRV) and baroreceptor reflex sensitivity (BRS) with ambulatory and beat-to-bea

228 irway eosinophilia result in increased cough reflex sensitivity to capsaicin associated with an incre

229 te the effects of allergen exposure on cough reflex sensitivity.

230 apnea, feeding problems, hyperactive startle reflex), severe postnatal progressive neurological abnor

231 atment groups 1 h after recovery of righting reflex: sham, TBI, sham RIC, TBI RIC.

232 We assessed the soleus H-reflex, shear modulus (ultrasound elastography) and vasc

233 olume, and cardiac output (CO) while causing reflex sinus rate (heart rate [HR]) increase.

234 excitability of the targeted reflex pathway; reflex size gradually changed over 8-10 weeks.

235 Vagal reflexes slow heart rate and can change where the heartb

236 activity for operant conditioning of spinal reflexes still use rigid metal electrodes with conductiv

237 imulation by lysophosphatidic acid elicits a reflex stimulation of vagal efferent activity sufficient

238 Electronic medical record (EMR)-based reflex strategy screened 4654 (15% [679] antibody positi

239 Reflex studies indicate the involvement of spinal struct

240 eptive neurons that participate in the cough reflex, suggesting additional cough-inducing mechanisms.

241 forms of memory in the defensive withdrawal reflex, suggesting functional coordination between excit

242 motor neurons impairs the larval optokinetic reflex, suggesting that neuronal clustering is important

243 aroreceptors in the pulmonary artery elicits reflex sympathoexcitation.

244 We suggest that the term vasodepression in reflex syncope should not be limited to reduced arterial

245 examination including the Hirschberg corneal reflex test to detect manifest strabismus.

246 -positive patients identified as a result of reflex testing accounted for 55 and 61% of all anti-HCV-

247 The implementation of viral load reflex testing in a central laboratory is feasible and s

248 otocol for HCV diagnosis based on viral load reflex testing of anti-HCV antibody-positive patients (k

249 dentified significant hepatitis C burden and reflex testing outperformed point-of-care linkage indica

250 ter (45,935 cases) the implementation of the reflex testing protocol.

251 Reflex testing-initial testing of priority genes followe

252 lity (HRV) indices, cardiovascular autonomic reflex tests (CARTs), and cardiac (123)I-metaiodobenzylg

253 formed standardized cardiovascular autonomic reflex tests and targeted fasting plasma metabolomic ana

254 s assessed using standardized cardiovascular reflex tests.

255 agonist amisulpride, known to affect startle reflex that is correlated with addiction in humans, and

256 in anesthetized rats reduced respiration, a reflex that was potently inhibited by activation of SubM

257 segmentally localised reflexes that amplify axial compression in order to coun

258 oking C-fibre-mediated airway sensations and reflexes that are associated with airway inflammatory di

259 haviors, including protective and corrective reflexes that dynamically adapt ongoing movement and pos

260 Sensory neurons initiate defensive reflexes that ensure airway integrity.

261 In addition to central control, micturition reflexes that govern urination are all initiated by peri

262 ian photoentrainment and the pupillary light reflex, the characterization of multiple types has demon

263 us (PON), which controls the pupillary light reflex; the superior colliculus (SC), which mediates ori

264 spinal cord injury (SCI), changing a spinal reflex through an operant conditioning protocol can impr

265 kes ocular discomfort or pain and protective reflexes, thus being a unique model to study mechanotran

266 Using the optokinetic reflex to evaluate visual function, we observed robustly

267 n EIA; if discordant results, specimens were reflexed to NAAT) and classified as toxin positive or NA

268 ditional RIDT, with negative specimens being reflexed to PCR.

269 c arch and carotid sinus initiates autonomic reflexes to change heart rate and blood pressure for car

270 previously shown that protective respiratory reflexes to locally released bacterial bitter "taste" su

271 The incremental diagnostic yield of reflexing to trio after negative proband analysis was 0.

272 ethral sphincter (EUS) contraction (guarding reflex) to maintain continence.

273 rrantly shifts nociceptive pulmonary-cardiac reflexes towards sympathoexcitation.

274 e of a de novo nociceptive pulmonary-cardiac reflex triggering sympathoexcitation in SH rats, and thi

275 thesia (lower HR, smaller DeltaHR, decreased reflexes) under dexmedetomidine, compared to propofol an

276 Visual-vestibulo-ocular reflex (V-VOR) adaptation was also tested pre- vs post-t

277 Reflex vagal activity causes abrupt heart rate slowing w

278 nic statin treatment-induced improvements in reflex vasodilatation are mediated, in part, by increase

279 Reflex vasodilatation in response to a 1.0 degrees C ris

280 Reflex vasodilatation was blunted in hypercholesterolaem

281 of eye movement (saccades, vestibulo-ocular reflex, vergence) and gaze-holding.

282 < .0001) and higher odds of LTC in EMR-based reflex versus POC (OR, 1.51; P < .0001).

283 higher odds of RNA confirmation in EMR-based reflex versus POC (OR, 2.07; P < .0001) and higher odds

284 After qualitative reflex VL testing was implemented, care continuum outcom

285 d pre- and postimplementation of qualitative reflex VL testing; (2) calculate engaged-in-care HCV CVL

286 ntial functions such as the vestibulo-ocular reflex (VOR) and its adaptation.

287 s and by characterizing the vestibulo-ocular reflex (VOR) and vestibular and headache symptom severit

288 el SK2 (L7-SK2) show intact vestibulo-ocular reflex (VOR) gain adaptation but impaired eyeblink condi

289 sEP) responses and abnormal vestibulo-ocular reflex (VOR) responses demonstrated that the vestibular

290 In all previous conditioning studies, the reflex was conditioned during steady-state maintenance o

291 me of, or shortly after, symptom onset the H-reflex was lost.

292 The inhibition of H-reflex was sustained up to 4 minutes and 3 minutes on th

293 ing the low-pass dynamics of vestibulocollic reflexes, we then recorded vestibular afferent responses

294 In the step initiation task, the H-reflexes were evoked on the soleus muscle when the ampli

295 Stabilizing reflexes were modulated by the orientation of the haptic

296 oleus evoked V-waves (cortical drive), and H-reflexes were recorded in 12 chronic stroke patients, wi

297 delay similar to the human vestibulo-ocular reflex-whereas wing steering responses lagged by more th

298 The swing-phase H-reflex, which is absent or very small in neurologically

299 he neural dynamics producing vestibulocollic reflexes, which may respond to high-frequency transient

300 nificant interactions between cardiovascular reflexes, with the impact differing when the CR activati