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1 gnosed as having TINU syndrome (confirmed by renal biopsy).
2 R potentially avoiding the need for invasive renal biopsy.
3 IGN in patients >/=65 years old diagnosed by renal biopsy.
4 sed or untreated BKN, which was confirmed by renal biopsy.
5 ith viremia without evidence of nephritis on renal biopsy.
6 propriate treatment and typically requires a renal biopsy.
7 tored by urine output, serum creatinine, and renal biopsy.
8  postprocedural hemorrhage after hepatic and renal biopsy.
9 ecipients, and definitive diagnosis requires renal biopsy.
10 urrent and de novo disease were diagnosed by renal biopsy.
11 rare finding of glomerular CMV inclusions on renal biopsy.
12 inal differentiation may be possible only by renal biopsy.
13 nt laboratory and US analysis at the time of renal biopsy.
14 ent and degree of glomerulonephritis seen on renal biopsy.
15  creatinine values obtained near the time of renal biopsy.
16 ated rejection) as compared to a concomitant renal biopsy.
17 acute rejection in kidney transplants is the renal biopsy.
18 ormalin-fixed, paraffin-embedded tissue from renal biopsies.
19 and flare are defined and the role of repeat renal biopsies.
20 ithelial cells is commonly observed in human renal biopsies.
21 nally, we evaluated AREG expression in human renal biopsies.
22 s found histologically in seven (9%) of open renal biopsies.
23 elated to the histopathologic changes in the renal biopsies.
24                    All patients had baseline renal biopsies.
25 of six urinary cell samples, and two of four renal biopsies.
26 of six urinary cell samples, and two of four renal biopsies.
27 nction was monitored by serum creatinine and renal biopsies.
28  in most centers in the evaluation of native renal biopsies.
29 gnostic information in nearly half of native renal biopsies.
30 s, and renal function was performed, as were renal biopsies.
31                    Twelve patients underwent renal biopsies.
32 r had a diagnosis of lupus nephritis made by renal biopsy 5 years before donation.
33 verity of glomerulosclerosis was assessed by renal biopsy 8 wk later, and rats were divided into four
34           Glomerulosclerosis was assessed by renal biopsy 8 wk later, and rats were divided into grou
35        There were 21 concurrent pancreas and renal biopsies, all from simultaneous pancreas-kidney al
36 routine use of electron microscopy in native renal biopsies also examined by immunofluorescence and r
37                                              Renal biopsy, an invasive method, is the main approach t
38                                              Renal biopsies and biomarker analysis from a large and d
39 was correlated with pathology in concomitant renal biopsies and BK viruria (decoy cell shedding and v
40 ere conducted on CD3 + T cells isolated from renal biopsies and blood of patients with ANCA-associate
41  using the invaluable repository of archival renal biopsies and discovered two urinary biomarkers tha
42  the largest study evaluated 213 consecutive renal biopsies and found that electron microscopy was ne
43 at ADPKD mouse and rat models, ADPKD patient renal biopsies and PKD1-/- cells exhibited hyperphosphor
44                       RNA was extracted from renal biopsies and reverse transcribed to cDNA which was
45 n the primary clinical diagnosis, motivating renal biopsies and the use of proteinuria-lowering treat
46  patients with lupus nephritis documented by renal biopsy and 26 with a history of lupus nephritis.
47 nce of peritubular capillary C4d staining on renal biopsy and donor-specific anti-human leukocyte ant
48 of polyomavirus infection was established by renal biopsy and EM of urine in five patients, by biopsy
49                                         Both renal biopsy and EM of urine samples are useful in the d
50                              Indications for renal biopsy and histological diagnosis were analyzed to
51 flammation; 9% subsequent renal referral; 1% renal biopsy and immunosuppression) was determined.
52 uences of light chain GLA extracted from the renal biopsy and light chain CHO from myocardial tissue
53 se seven patients were reviewed, as were all renal biopsy and nephrectomy specimens.
54 -associated systemic vasculitis confirmed by renal biopsy and serum creatinine >500 micromol/L (5.8 m
55 mputed tomography (CT)-guided native medical renal biopsy and to evaluate its efficacy and safety com
56 ined as >/=10,000 viral copies/mL) underwent renal biopsy and treated with 30% to 50% reduction in do
57 otal of 90% and 82% of patients had complete renal-biopsy and retinopathy data, respectively.
58  were analyzed to describe the prevalence of renal biopsy, and changing prevalence between period 1 (
59 e levels, increased interstitial fibrosis on renal biopsy, and increased fractional excretion of immu
60          No death was related to transvenous renal biopsy, and median biopsy-to-death interval was 38
61 osis of "osmotic nephrosis" was confirmed by renal biopsy, and the condition was reversed by cessatio
62                                     Although renal biopsies are not routinely performed as part of th
63 tion of atypical retinal features and timely renal biopsy are essential to distinguish PICGN from Goo
64 undant tubular calcium phosphate deposits on renal biopsy are referred to as nephrocalcinosis, a cond
65 ial fibrosis and tubular atrophy (IFTA) on a renal biopsy are strong indicators of disease chronicity
66 e presence of tubulointerstitial fibrosis on renal biopsy as independent predictors of ESRD.
67                                 We performed renal biopsies at baseline and after 5 years of enzyme r
68 antly prolonged graft survival (P=0.02), and renal biopsy at 1 month showed significantly reduced ant
69 dividuals with IgA nephropathy who underwent renal biopsy at our institution between 1973 and 1995.
70 lyzing microscopic- and WSI-level changes in renal biopsies attempts to mimic the pathologist and pro
71  From the total of patients, 151 underwent a renal biopsy because of renal dysfunction, whereas the 2
72                                 We performed renal biopsies before, during, and after surgically indu
73          All children underwent percutaneous renal biopsy before the institution of therapy.
74 ,630 hospitalized patients undergoing native renal biopsy between January 1, 2009 and December 31, 20
75 ommon with older age and is characterized on renal biopsy by global glomerulosclerosis, tubular atrop
76                                              Renal biopsy can be relatively safe in this population,
77        Patients with active lupus nephritis (renal biopsy class III, IV, or V) were recruited for the
78 get genes were upregulated in fibrotic human renal biopsies compared with controls.
79  indicated cortical hyperechogenicity, and a renal biopsy confirmed IgAN with mesangial IgA depositio
80 ulointerstitial transcriptomes from protocol renal biopsy cores were analyzed for differential and co
81                                              Renal biopsy data further suggest that renal tubular cel
82 s of podocyte death and put experimental and renal biopsy data in a unified perspective.
83 us nephritis were identified from 5 sources: renal biopsy databases, dialysis/transplant databases, n
84                                              Renal biopsy demonstrated a florid, diffuse, proliferati
85                                              Renal biopsy demonstrated a pauci-immune crescentic glom
86  A consultation through the Internet after a renal biopsy demonstrated crescentic, necrotizing glomer
87                                              Renal biopsy demonstrated focal and segmental glomerulos
88 re detectable at a median of 274 days before renal biopsy diagnosis (interquartile range, 71-821 days
89 cted before the use of immunofluorescence in renal biopsy diagnosis became widespread and before seve
90                                              Renal biopsy diagnosis preceded clinical evidence of dys
91                                              Renal biopsy elucidated the cause of acute deterioration
92                                              Renal biopsy established the diagnosis of non-Hodgkin's
93                                           In renal biopsies, expression of Mig was detected in glomer
94                                      Whether renal biopsy findings add to the clinical assessment in
95 nal mesangial expansion score (MES) based on renal biopsy findings and diabetes duration.
96                              Here, we report renal biopsy findings before and after eculizumab therap
97                              The predominant renal biopsy findings were membranoproliferative glomeru
98 on of renal failure, glofil measurement, and renal biopsy findings, offers a practical approach to th
99                              On the basis of renal biopsy findings, patients were stratified into thr
100            Here we describe a case with such renal biopsy findings, review previous reported cases, a
101 the iodine-125 iothalamate (Glofil) test and renal biopsy findings.
102  circumstances, it is essential to perform a renal biopsy for diagnosis and to guide treatment.
103 children than in adults, usually requiring a renal biopsy for diagnosis.
104                         MGV was estimated in renal biopsies from 16 diabetic patients and 13 normal s
105 athologists scored 12 descriptors in NEPTUNE renal biopsies from 242 patients with minimal change dis
106                  Here, re-examination of 546 renal biopsies from African-American patients with SLE i
107                                              Renal biopsies from eight proteinuric type 1 D patients
108                                              Renal biopsies from five normoalbuminuric patients, five
109 on of SGLT2 mRNA and protein is increased in renal biopsies from human subjects with diabetic nephrop
110                   Therefore, we interrogated renal biopsies from LN longitudinal and cross-sectional
111                            We also evaluated renal biopsies from our institutions for podocyte-associ
112 ies with podocyte-associated punctate IgG in renal biopsies from our institutions.
113                       This study examined 47 renal biopsies from patients with a variety of glomerula
114 mistry, we show increased podocyte SEMA3A in renal biopsies from patients with advanced DN.
115 situ hybridization, in serial sections of 23 renal biopsies from patients with cGN.
116 hropathy, an immunohistochemical analysis of renal biopsies from patients with diabetic nephropathy (
117 me-wide expression profiles of more than 200 renal biopsies from patients with different CKD stages r
118 as measured in glomeruli microdissected from renal biopsies from patients with DN and nondiabetic con
119  We developed a digital pipeline to classify renal biopsies from patients with DN.
120                                           In renal biopsies from patients with early nephropathy from
121            Formalin-fixed, paraffin-embedded renal biopsies from patients with HIV-associated nephrop
122 tes from HIV-1 transgenic mice as well as in renal biopsies from patients with HIV-associated nephrop
123 ed single-cell RNA sequencing (scRNA-seq) to renal biopsies from patients with LN and evaluated skin
124                      Immunohistochemistry of renal biopsies from patients with lupus nephritis, but n
125 ernative, classical and lectin) are found in renal biopsies from patients with MN.
126                    Sirt6 is downregulated in renal biopsies from patients with podocytopathies and it
127                                              Renal biopsies from patients with this disorder can reve
128        Elastin immunohistochemistry of human renal biopsies from patients with type 1 diabetes (n = 3
129                                              Renal biopsies from subjects with FSGS, but not those wi
130 cence and EndMT was confirmed by analysis of renal biopsies from the same AMR patients.
131 ted in situ within endothelial cells both in renal biopsies from transplantation patients with chroni
132                                            A renal biopsy from a 36-year-old man with AIDS showed a s
133 lized to the Golgi, a finding confirmed in a renal biopsy from an affected individual.
134     In both patients, immunocytochemistry of renal biopsy frozen sections with an anti-H(+)-ATPase mo
135 equire any proteinuria-lowering treatment or renal biopsy.FUNDINGATIP-Avenir program, Fondation Bette
136                                              Renal biopsies further confirmed resolution of inflammat
137                                              Renal biopsy has been proposed to determine the cause or
138 ve polymerase chain reaction (PCR) assay for renal biopsy has not been evaluated as a diagnostic test
139                                              Renal biopsies have associated morbidity and mortality a
140                                           On renal biopsy, high-risk genotype was associated with inc
141              This classification is based on renal biopsy immunofluorescence examination, making the
142  patterns of glomerulonephritis (GN) seen on renal biopsy impact upon the prognosis of these patients
143                  We evaluated the utility of renal biopsies in a cohort of 59 consecutive liver trans
144 staining that best predicts renal outcome in renal biopsies in a multicenter study in which local and
145              No acute rejection was noted on renal biopsy in either case.
146 on in HSCT patients that can be diagnosed by renal biopsy in patients with hematuria and adenoviruria
147 epidemiological trends and the prevalence of renal biopsy in various regions to shine new light on th
148 ., chicken thigh muscle with skin and murine renal biopsy including medulla (M) and cortex (C)) showe
149                                     However, renal biopsy interpretation is subjective and can render
150                                  Transvenous renal biopsy is an alternative way to obtain kidney samp
151 rlying glomerular lesion, and therefore, the renal biopsy is an essential clinical tool in the approa
152 may provide clues to the presence of HCV-GD, renal biopsy is essential to differentiate HCV-GD from H
153 deposition or endocapillary proliferation on renal biopsy is more likely a manifestation of SLE than
154 value less than 500 mg/L is detected, then a renal biopsy is needed.
155 IV-) formalin-fixed paraffin-embedded (FFPE) renal biopsies matched for degree of inflammation was pe
156                                            A renal biopsy may be necessary for diagnosis.
157                                              Renal biopsies (n = 16) from 10 patients with AHR who ha
158                                              Renal biopsies (n=165) in 40 paired recipients showed no
159           Tissue [NH(4) (+)] was measured in renal biopsies, NH(4) (+) excretion and titratable acid
160         RNA was extracted from archival FFPE renal biopsies of 52 IgAN patients, 22 non-IgAN and norm
161                                              Renal biopsies of 62 NRSOT recipients were evaluated for
162                                              Renal biopsies of CKD and kidney allograft patients reve
163                Histopathological features in renal biopsies of patients with antineutrophil cytoplasm
164 d to renal tubular epithelial cell nuclei in renal biopsies of patients with FSGS by in situ hybridiz
165                            In a series of 36 renal biopsies of patients with proliferative and nonpro
166                                              Renal biopsies of patients with proteinuria and kidney d
167 dy, we show glomerular C5b-9 deposits in the renal biopsy of a child with EHEC-associated hemolytic u
168 ent with autosomal dominant transmission and renal biopsy of at least one individual showed C3 glomer
169 e safety and diagnostic yield of transvenous renal biopsy of critically ill patients.
170                                   Utility of renal biopsy of these kidneys is similarly not well esta
171 stitial nephritis with either (1) a positive renal biopsy or (2) evidence of nephritis (elevated seru
172  without COPD, 32 nonsmokers who underwent a renal biopsy or nephrectomy, and in CS-exposed mice, we
173  the donor or recipient, rejection episodes, renal biopsy, or drug-induced nephrotoxicity.
174 ificance difference between cases and native renal biopsies (P = 0.05).
175                                   Those with renal biopsies performed >/=18 months PostTx were classi
176 tions were reviewed in 431 CT-guided medical renal biopsies performed between July 2007 and September
177                                              Renal biopsies performed in 12 patients after 12 to 41 m
178 nal gluconeogenic pathway from more than 200 renal biopsies performed on CKD patients and from 43 kid
179 ast 10 years of diabetes duration that had a renal biopsy performed for research purposes were studie
180 ncreased significantly (P <0.001) in a first renal biopsy performed within 3 months from transplantat
181 executor enzyme caspase-3 in preimplantation renal biopsies (PIB) as markers for delayed graft functi
182 , glycemia, and other variables, with repeat renal biopsies planned at 5 years after baseline.
183                                 Percutaneous renal biopsy (PRB) is a safe and effective tool in the d
184                                 Percutaneous renal biopsy (PRB) of kidney transplants might be preven
185 nosis system (KIDS), a noninvasive model for renal biopsy prediction using 13,144 retinal images from
186 inosis were identified among the 7349 native renal biopsies processed at Columbia University.
187                                              Renal biopsy provides useful diagnostic information to d
188 ollow-up visits, emergency hospitalizations, renal biopsies, rejection episodes, renal function, and
189 occurring with transjugular and percutaneous renal biopsies, respectively.
190 iewed the posttransplant clinical course and renal biopsy results in 97 consecutive SLE patients who
191                  Key secondary outcomes were renal-biopsy results at 24 and 52 weeks, donor-specific
192 ch, when applied to CT-guided native medical renal biopsies, results in higher rates of sample adequa
193 a known or suspected infectious process, and renal biopsies revealed an immune complex glomerulonephr
194                                              Renal biopsy revealed a chronic tubulointerstitial nephr
195                                              Renal biopsy revealed a membranoproliferative pattern of
196                                              Renal biopsy revealed acute and chronic antibody-mediate
197                                              Renal biopsy revealed diffuse proliferative lupus nephri
198                                              Renal biopsy revealed FSGS in nine patients, four of who
199                                              Renal biopsy revealed the diagnosis of CN in 8 of 45 pat
200                                              Renal biopsy samples from 28 nonimmunosuppressed patient
201                                              Renal biopsy samples from 80 patients with lupus nephrit
202                       Studied were 136 human renal biopsy samples from a range of chronic renal disea
203  kidney, we performed microarray analyses of renal biopsy samples from patients with ANCA-associated
204 antibodies and glomerular target antigens in renal biopsy samples from patients with LN and determine
205                                 We evaluated renal biopsy samples from patients with various forms of
206 3.4 +/- 1.7 copies/cell) were observed in 74 renal biopsy samples from renal allograft recipients wit
207 pression and microRNA expression profiles in renal biopsy samples from tolerance-induced FCRx recipie
208 g planted antigens from laser-microdissected renal biopsy samples of 20 patients with LN.
209 ne, findings that could also be evidenced in renal biopsy samples of diabetic patients.
210 ents with BKV viruria, but 58 (50.4%) of 115 renal biopsy samples tested negative for BKV DNA, reflec
211                                All remaining renal biopsy samples then were genotyped for these two v
212                                              Renal biopsy samples were obtained 20 min after removal
213 tion of single-cell technologies to clinical renal biopsy samples, or even cells within urine, will i
214 olated by laser-capture microdissection from renal biopsy samples.
215                  JCV DNA was found in only 2 renal biopsy samples.
216  activity and thermal stability in 127 human renal biopsy samples.
217 completed by immunohistochemistry studies on renal biopsy samples.
218  in this study is evaluation of surveillance renal biopsies (SB) and clinically indicated biopsies (C
219 n GAG expression within normal and rejection renal biopsy sections.
220 rmed via the ipsilateral femoral vein with a renal biopsy set designed for transjugular renal biopsy
221 ed semiquantitative histologic evaluation of renal biopsies showed better preserved morphology in bot
222  acute deterioration in renal function whose renal biopsies showed typical viral cytopathic changes i
223                                            A renal biopsy showed massive tubular necrosis associated
224                                              Renal biopsy showed signs of chronic thrombotic microang
225 h donors had renal failure and pretransplant renal biopsies showing 100% of the glomeruli containing
226 n/day after transplantation (P=0.05) and had renal biopsies showing MPGN than did HCV- recipients (4/
227                                    Unstained renal biopsy slides were obtained from 296 patients.
228                                              Renal biopsy specimen analysis revealed amyloidosis with
229 tomic profiling of thousands of cells from a renal biopsy specimen at a single-cell resolution.
230                           Examination of the renal biopsy specimen revealed amyloidosis with predomin
231 ars) had pronounced cerebellar atrophy and a renal biopsy specimen that showed focal segmental glomer
232                       Studying the patient's renal biopsy specimen, we ascertained that obstructive t
233 ts also had segmental glomerular deposits on renal biopsy specimen.
234 nd inflammation classification, with >90% of renal biopsy specimens adequately classified by FTIR ima
235 nd validated a classification model using 49 renal biopsy specimens and subsequently tested the robus
236  increased IL-36alpha expression detected in renal biopsy specimens and urine samples from patients w
237       IL-15 transcripts were detected in all renal biopsy specimens and was significantly increased i
238 with biopsy specimens from control patients, renal biopsy specimens from 44 patients with acute AAV h
239                    Transcriptome analyses of renal biopsy specimens from patients with CKD revealed m
240    In conclusion, the AGN classification for renal biopsy specimens is a practical and informative sc
241 ased level of active TGF-beta1 expression in renal biopsy specimens of patients receiving CsA may ind
242  Snail/nephrin axis were similar to those in renal biopsy specimens of Zucker diabetic fatty rats and
243                                              Renal biopsy specimens uniformly showed FSGS.
244                            Paraffin-embedded renal biopsy specimens were sectioned, dewaxed, and incu
245 nophenotyped the inflammatory infiltrates in renal biopsy specimens with BK polyomavirus-associated n
246                              By IHC of human renal biopsy specimens, glomerular SCD-1 induction was o
247  course and with the presence of BK virus in renal biopsy specimens.
248 rong biomarker for rapid diagnosis of FGN in renal biopsy specimens.
249 bed here can easily be recognized in routine renal biopsy specimens.
250                                       Normal renal biopsies stained positive for C5b-9 in glomeruli,
251                                     In human renal biopsies, staining of 4-hydroxynonenal (4-HNE), gl
252                                        Also, renal biopsies taken just before reperfusion, showed loc
253                                        Safer renal biopsy techniques have led to increased recognitio
254 ) is a global health challenge, but invasive renal biopsies, the gold standard for diagnosis and prog
255                       The presence of CMV in renal biopsy tissue from GPA patients was investigated b
256 mononuclear cells (PBMC), urinary cells, and renal biopsy tissue was performed using specific primers
257 Shc in peripheral blood monocytes (PBMs) and renal biopsy tissues from DN patients and then analysed
258 a renal biopsy set designed for transjugular renal biopsy (TJRB) of native kidneys.
259 ophysiologic characteristic which requires a renal biopsy to distinguish.
260 m archived formalin-fixed, paraffin-embedded renal biopsies, until recently considered an unsuitable
261                                  We reviewed renal biopsies using the Oxford classification criteria.
262 stinct population of cells within a standard renal biopsy using flow cytometry.
263                                      Because renal biopsy was performed selectively, 59% of recipient
264                                            A renal biopsy was performed.
265 tients with acute kidney injury, transvenous renal biopsy was safe and obtained a high diagnostic yie
266 ole-slide images (WSIs) processed from human renal biopsies, we developed a deep-learning framework t
267 sies from a matched control group and native renal biopsies were analyzed.
268 onuclear cells, serum creatinine levels, and renal biopsies were collected from 8 patients undergoing
269                              Histologically, renal biopsies were diagnosed as no rejection, acute tub
270 pients, 126 protocol, serial, posttransplant renal biopsies were examined by centralized, blinded Ban
271                                              Renal biopsies were examined by immunohistochemical stai
272                                              Renal biopsies were grouped as postreperfusion (n=10), s
273    The most common pathological diagnoses on renal biopsies were membranoproliferative glomerulonephr
274   From January 1996 to June 1996, 288 native renal biopsies were received, and all were evaluated by
275                                  Ninety-five renal biopsies were stained for claudin-1 (PEC marker),
276                                              Renal biopsies were taken every 24 hours or whenever ani
277 ipients without PVN on simultaneous protocol renal biopsy were analyzed by PCR; BKV genome was demons
278 tubular atrophy and interstitial fibrosis on renal biopsy were assessed.
279 1071 patients with primary IgAN diagnosed by renal biopsy were enrolled in multiple study centers for
280              Histologic assessments of donor renal biopsy were used with other clinical variables to
281  and mass spectrometry in further evaluating renal biopsies when routine assessment fails to reach an
282      We evaluated 10 serial sections from 15 renal biopsies with a range of fibrosis extent and diagn
283 te rejection within the first 3 mo and had a renal biopsy with available frozen tissue at acute rejec

 
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