ライフサイエンスコーパス: renal blood flow

1 extracellular volume depletion or decreased renal blood flow.

2 re rapid dynamic alterations in differential renal blood flow.

3 ) during angiotensin II-induced reduction in renal blood flow.

4 cant change in glomerular filtration rate or renal blood flow.

5 closely related to discharge energy than is renal blood flow.

6 nal perfusion pressure but not of changes in renal blood flow.

7 mortality, impaired ventilation, and reduced renal blood flow.

8 erfusion pressure but poorly with changes in renal blood flow.

9 otection is partly related to maintenance of renal blood flow.

10 ns in methods for measuring rapid changes in renal blood flow.

11 istance blood vessels and failed to increase renal blood flow.

12 ery narrowness and can result to decrease in renal blood flow.

13 y and have been implicated in the control of renal blood flow.

14 renal pelvic pressure or acute reduction in renal blood flow.

15 ynamic PET image data were used to calculate renal blood flow.

16 ohippuric acid infusion for determination of renal blood flow.

17 ables simultaneous evaluation of cardiac and renal blood flow.

18 ge in systemic vascular resistance index and renal blood flow.

19 renal pelvic pressure or acute reduction in renal blood flow.

20 only seen with decreased cardiac output and renal blood flow.

21 n in the CD that may contribute to decreased renal blood flow.

22 c oxide synthase expressions correlated with renal blood flow.

23 y injury occurs in the presence of increased renal blood flow.

24 d flow, without changing portal pressure and renal blood flow.

25 largely because of a substantial increase in renal blood flow.

26 evosimendan induced significant increases in renal blood flow (12%, p<0.05) and glomerular filtration

27 t (2.1+/-0.2 to 2.3+/-0.2 L/min; P<0.05) and renal blood flow (131+/-17 to 162+/-18 mL/min; P<0.05) i

28 differ, there was a significant decrease of renal blood flow 2 hr after the intake of CsA compared w

29 nterval] 0.51 [0.28-0.92]) despite increased renal blood flow (241 to 343 mL/min, difference [95% con

30 Second, graded reductions in renal blood flow (25%, 50% and 75%, 90 s) produced flow-

31 7 versus 57 +/- 11 mL/min, P < or = 0.0001), renal blood flow (3.4 +/-0.7 versus 8.4 +/- 1.9 mL/min L

32 e clearance and increased plasma creatinine, renal blood flow (+46% +/- 6%) and cortical perfusion (+

33 d animals, ANG II produced a 40% decrease in renal blood flow, a level between untreated dietary grou

34 gonist of the dopamine-1 receptor, preserves renal blood flow after iodinated contrast administration

35 ericytes in vitro Initial studies monitoring renal blood flow after IRI did not find significant effe

36 There was no significant change in mean renal blood flow after PTA (P=.44).

37 of renal hypoxia and partial restoration of renal blood flow after revascularization, inflammatory c

38 After UNX, renal blood flow and amino acid delivery to the remainin

39 for high-resolution laser speckle imaging of renal blood flow and apply it to estimate the frequency

40 st magnetic resonance imaging measurement of renal blood flow and cardiac output.

41 Renal blood flow and cortical oxygen tension increased i

42 in mild CHF in association with increases in renal blood flow and decreases in renal vascular resista

43 usion preserves spinal cord, mesenteric, and renal blood flow and eliminates the potential catastroph

44 Acute pharmacological blockade increases renal blood flow and filtration rate, suggesting that re

45 fusion, HBOC-201 treated kidneys had similar renal blood flow and function compared with blood-treate

46 y allows for enhanced resolution of regional renal blood flow and functional evaluations in patients.

47 Given the differences in renal blood flow and GFR in control and postischemic kid

48 -gated calcium channels can maintain overall renal blood flow and GFR stability despite severely impa

49 rom 141 +/- 3 (SE) mm Hg to 101 +/- 2 mm Hg; renal blood flow and GFR were not significantly altered

50 sodium excretion; however, the increases in renal blood flow and GFR were not sustained as systemic

51 re characterized by progressive reduction in renal blood flow and glomerular filtration rate and show

52 Reductions in renal blood flow and glomerular filtration rate are unde

53 Renal blood flow and glomerular filtration rate exhibite

54 Renal blood flow and glomerular filtration rate were mea

55 omerular resistance vessels, increasing both renal blood flow and glomerular filtration rate without

56 I/R markedly depressed renal blood flow and increased the production in O2, PGE

57 on was evaluated by parallel measurements of renal blood flow and inulin clearance.

58 Renal blood flow and mean arterial pressure responses to

59 t on F344 background, hypothesising enhanced renal blood flow and protection from angiotensin-II-indu

60 l hypertension, and a progressive decline in renal blood flow and renal function.

61 de plays an important role in the control of renal blood flow and renal function.

62 ith renal vasoconstriction, but no change in renal blood flow and substantial femoral vasodilatation

63 on during endotoxic shock actually increases renal blood flow and that this effect is not the result

64 ay be a valuable tool to further investigate renal blood flow and the effects of therapies on renal b

65 The mean renal blood flow and total urine output were 68.0 mL/min

66 AM reflect unique actions of the peptide on renal blood flow and tubular function.

67 ic volume expansions reversed the changes in renal blood flow and urine output, but impaired creatini

68 scored based on the macroscopic appearance, renal blood flow and urine output.

69 lant with good global perfusion and adequate renal blood flow and urine production.

70 We developed a technique for measuring renal blood flow and used a pig model to determine wheth

71 a role in reversing LPS-induced decreases in renal blood flow and volume, although the effects on PAR

72 en demand while lower extremity, splanchnic, renal blood flows and arteriovenous oxygen content diffe

73 ilability in renal vascular cells, augmented renal blood flow, and decreased systemic blood pressure

74 e imaged with a 1.5-T imager to estimate EF, renal blood flow, and glomerular filtration rate.

75 es systemic blood pressure, reduces regional renal blood flow, and increases platelet counts and proc

76 zed sheep, decrements in hemodynamic status, renal blood flow, and kidney function incurred following

77 on of renal microvasculature, improvement in renal blood flow, and less tissue hypoxia than TbetaRII(

78 rapidly decreases blood pressure, increases renal blood flow, and maintains or improves the glomerul

79 nsion would reduce portal pressure, increase renal blood flow, and produce insignificant changes in a

80 served glomerular filtration rate, increased renal blood flow, and promoted diuresis and natriuresis.

81 ynamic parameters, including cardiac output, renal blood flow, and vascular resistance.

82 nd resulted in lower basal GFR (16%), higher renal blood flow (approximately 22%), and lower filtrati

83 y renal nerve stimulation, the reductions in renal blood flow at each stimulation frequency were grea

84 genic response of afferent arterioles and in renal blood flow autoregulation, which were rescued in A

85 has been shown under conditions of efficient renal blood flow autoregulation.

86 ributes importantly to the early decrease in renal blood flow (basal versus handgrip, 4.2 +/- 0.2 ver

87 boreflex contributes to further decreases in renal blood flow (basal versus posthandgrip circulatory

88 Obstruction of renal blood flow before and during RF ablation resulted

89 AKI), but there were no changes over time in renal blood flow between groups (P > 0.30) or over time

90 model, NE was associated with an increase in renal blood flow both before and after endotoxin adminis

91 BQ-123 increased effective renal blood flow (BQ-123, -0.1 +/- 2.4%; BQ-123+E, 10.9

92 in normotensive F2s (n = 3) without altering renal blood flow but was inactive in hypertensive F2s (n

93 and a high dose of aminoguanidine normalized renal blood flow, but did not alter creatinine clearance

94 Sepsis increased cardiac output by 60%, renal blood flow by 35%, and arterial lactate by approxi

95 mg kg-1 bis in die (b.i.d.)) rats decreased renal blood flow by 46 and 29 % (both P < 0.001), respec

96 0 mg/kg) to untreated control mice increased renal blood flow by 55% (from 1.8+/-0.2 to 2.8+/-0.2 ml/

97 ute extracellular volume expansion increased renal blood flow by 84% and reduced renal vascular resis

98 ors in VSMCs are essential for regulation of renal blood flow by Ang II and highlight the capacity of

99 Both doses of [Pyr(1)]apelin-13 increased renal blood flow by ~15%, natriuresis by ~20% and free w

100 Renal blood flow can be reliably measured from cardiac (

101 , implying that the long-lasting decrease of renal blood flow contributing to kidney damage was gener

102 on of liver disease and portal hypertension, renal blood flow declines because of the hepatorenal ref

103 Renal blood flow decreased after reperfusion but was not

104 rotid and iliac circulations, mesenteric and renal blood flows decreased markedly.

105 sartan IC50 values for percentage changes in renal blood flow did not differ in the two groups of mic

106 njection of S1P in anesthetized rats reduced renal blood flow dose dependently.

107 of lipid peroxidation by U74389G maintained renal blood flow during acute CsA administration.

108 anced harmonic US imaging depicts changes in renal blood flow during acute obstruction.

109 hyperspectral imaging facilitates monitoring renal blood flow during animal surgery and holds conside

110 formed to clarify the mechanisms controlling renal blood flow during static exercise.

111 c, diuretic, glomerular filtration rate, and renal blood flow enhancing actions than native ANP in vi

112 uretic, with glomerular filtration rate- and renal blood flow-enhancing actions.

113 DC ACT mice also had greater reductions in renal blood flow following acute injections with Ang II

114 Sildenafil improved the renal blood flow for the first 30 min in the 2-hr group

115 ecule that participates in the regulation of renal blood flow, GFR, and mesangial matrix accumulation

116 We assessed BP, urinary protein, stenotic renal blood flow, GFR, microvascular structure, and oxyg

117 que and complex functional interplay between renal blood flow, GFR, O2 consumption, and arteriovenous

118 ach nephron is crucial for the regulation of renal blood flow, GFR, urine concentration, and other sp

119 al artery stenosis (ARAS) is known to reduce renal blood flow, glomerular filtration rate (GFR) and a

120 enous infusion of Hcys was found to decrease renal blood flow, glomerular filtration rate, and sodium

121 ore evaluated the effects of levosimendan on renal blood flow, glomerular filtration rate, renal oxyg

122 and generate paracrine signals that control renal blood flow, glomerular filtration, and release of

123 to control vital kidney functions, including renal blood flow, glomerular filtration, and renin relea

124 total kidney volume and decreases in GFR and renal blood flow greater than expected for a given age a

125 a high-salt diet, Tmem27(Y/-) mice had lower renal blood flow, higher abundance of renal sodium-hydro

126 hed nitric oxide bioavailability and reduced renal blood flow; however, the mechanisms leading to the

127 Fenoldopam increased renal blood flow in a dose-dependent manner compared wit

128 aluate rapid dynamic changes in differential renal blood flow in an experimental animal model.

129 in urinary sodium excretion, urine flow and renal blood flow in association with reductions in cardi

130 low in wild-type mice and an 8% reduction of renal blood flow in AT1A receptor-knockout mice.

131 ent, AngII (2 ng) produced 40% reductions in renal blood flow in both rat strains, without affecting

132 mpairment (3/4 nephrectomy plus amlodipine), renal blood flow in conscious animals (but not anestheti

133 utoregulation, we recorded concurrent BP and renal blood flow in conscious rats, comparing animals wi

134 cantly reduced the effects of vasopressin on renal blood flow in control but not in endotoxemic rats.

135 l blood flow and the effects of therapies on renal blood flow in critical illness.

136 pmol/kg, IV) produced increased reduction in renal blood flow in endotoxemic rats.

137 increases in oxygen demand on splanchnic and renal blood flow in hemorrhaged dogs.

138 ninvasive method for the mapping of regional renal blood flow in humans using PET and H(2)(15)O.

139 The decrease in renal blood flow in patients with heart failure was simi

140 nic saline (7.5% NaCl) solution may maximize renal blood flow in prolonged pneumoperitoneum, but it d

141 ught to overcome such obstacles by measuring renal blood flow in septic patients with acute kidney in

142 on pressure of perfused kidneys in vitro and renal blood flow in situ were evaluated.

143 However, the accurate measurement of renal blood flow in such patients is problematic and inv

144 in a greater transfer of input signals into renal blood flow in the 0.1 to 1.0 Hz range.

145 more, laser Doppler assessment showed higher renal blood flow in the CD47mAb-treated kidneys.

146 renal AngII (1 ng) caused a 32% reduction of renal blood flow in wild-type mice and an 8% reduction o

147 Renal blood flow increased with F+T but this was not sig

148 reduction in glomerular filtration rate and renal blood flow, increased renal cytokine expression, a

149 st dose (i.e., 0.03 ILg/kg/min), significant renal blood flow increases occurred without changes in s

150 There was no rank correlation between renal blood flow index and creatinine clearance in patie

151 Renal blood flow indexed to body surface area was 244 mL

152 ow doses (0.004 to 0.02 units/min) increased renal blood flow (indicator-dilution technique), reduced

153 Because the renal blood flow is not affected by the increased cardia

154 Renal blood flow is often reduced in patients with chron

155 increased arterial pressure without reducing renal blood flow, leading to an improved renal function.

156 (maximal reduction 55+/- 6%) and increase in renal blood flow (maximal increase 136 +/- 54%).

157 of the renal arteries provided quantitative renal blood flow measurements.

158 roups in systemic vascular resistance index, renal blood flow, mesenteric blood flow, systemic oxygen

159 ined the cause of the sustained reduction in renal blood flow ('no-reflow'), which exacerbates kidney

160 there was a significant correlation between renal blood flow obtained before drug administration and

161 at the lowest dose, significantly increased renal blood flow occurred without changes in systemic bl

162 longed CO2 pneumoperitoneum caused decreased renal blood flow, oliguria, and impaired creatinine clea

163 ycardia, increased cardiac output, increased renal blood flow, oliguria, decreased creatinine clearan

164 nthase inhibitor did not reduce the elevated renal blood flow or improve renal function.

165 d water retention caused by abnormalities of renal blood flow, or as a hemodynamic problem associated

166 viable on EVNP with progressively increasing renal blood flow over the 3-hour period of perfusion.

167 early AKI was not associated with changes in renal blood flow, oxygen delivery, or histological appea

168 models with low cardiac output and decreased renal blood flow (p < 0.0001).

169 th high-sodium diet (16% vs. 56% decrease in renal blood flow, P < 0.001).

170 leads to relative hypovolemia and decreased renal blood flow, patients with decompensated cirrhosis

171 Pre-stent single kidney renal blood flow, perfusion, and GFR were reduced in the

172 ontrol rats, there was a 10-fold increase in renal blood flow power over the frequency range of 0.01

173 Although this did not affect the renal blood flow power spectrum in control rats, there w

174 o-/-) promotes renal tubular injury, reduces renal blood flow, promotes microvascular rarefaction, in

175 d that is fully tissue protective, increases renal blood flow, promotes sodium excretion, reduces inj

176 crobubbles and ultrasonic flow probe-derived renal blood flow (r = 0.82, p < 0.001) over a wide range

177 in significantly higher cardiac outputs and renal blood flow rates in treated animals compared with

178 therosclerotic renal artery stenosis reduces renal blood flow (RBF) and amplifies stenotic kidney hyp

179 GMPV), glomerular filtration rate (GFR), and renal blood flow (RBF) and decreased distal fractional s

180 sclerotic renovascular disease (RVD) reduces renal blood flow (RBF) and GFR and accelerates poststeno

181 oconstrictors contributes to the decrease in renal blood flow (RBF) and GFR observed during LPS-induc

182 Basal renal blood flow (RBF) and glomerular filtration rate (G

183 mboxane A2 (TxA(2)) may mediate decreases of renal blood flow (RBF) and/or GFR associated with LPS-in

184 rmed in anesthetized dogs (n = 9) to examine renal blood flow (RBF) autoregulatory efficiency before

185 vely evaluate the feasibility of determining renal blood flow (RBF) by using a technique based on int

186 e myogenic response and the TGF mechanism in renal blood flow (RBF) control at the very earliest stag

187 mean arterial pressure (MABP), no change in renal blood flow (RBF) due to an increase in renal vascu

188 decreased by 50% after infusion of AngII and renal blood flow (RBF) fell by 3.3 ml min(-1) .

189 Renal blood flow (RBF) is often reduced in patients with

190 This rat renal blood flow (RBF) study quantified the impact of ni

191 hrectomy results in an immediate increase in renal blood flow (RBF) to the remnant kidney, followed b

192 Renal blood flow (RBF) was measured using an ultrasonic

193 reperfusion, plasma [ET-1] increased 66% and renal blood flow (RBF) was reduced by 38% compared with

194 II (ANGII) on the dynamic characteristics of renal blood flow (RBF) was studied in conscious dogs by

195 Hippuran clearance and renal blood flow (RBF) were measured twice, before and a

196 owed by decreases in mean arterial pressure, renal blood flow (RBF), and renal capillary perfusion at

197 ature, participates in the autoregulation of renal blood flow (RBF), but the underlying mechanisms ar

198 Renal blood flow (RBF), mean arterial pressure (MAP), an

199 +/-7 mmHg; the control decreases in cortical renal blood flow (RBF), measured with laser Doppler flow

200 But arterial pressure (ABP), renal blood flow (RBF), renal vascular conductance (RVC)

201 olume, glomerular filtration rate (GFR), and renal blood flow (RBF).

202 LPS on glomerular filtration rate (GFR) and renal blood flow (RBF).

203 sure (MABP, Delta = +18 to 26 mmHg), reduced renal blood flow (RBF, Delta = -1.8 to 2.9 ml min(-1)),

204 icantly improved renal function by restoring renal blood flow, reducing nicotinamide adenine dinucleo

205 kidney ischaemia and 30-60 min reperfusion, renal blood flow remained reduced, especially in the med

206 Renal blood flow remained unaffected by all treatment fo

207 Renal blood flow response to Ach was blunted in pigs tha

208 The aim of this study was to determine the renal blood flow response to static exercise in healthy

209 (by electromagnetic or ultrasonic flowmetry) renal blood flow responses to AngII in rats and mice.

210 was assessed in vivo by measuring transient renal blood flow responses to bolus injections of ANG II

211 After an ischemic insult, total renal blood flow returns toward normal, but marked, regi

212 Renal blood flow seems consistently reduced as a fractio

213 ptors elicited significant increases in GFR, renal blood flow, sodium excretion, and fractional sodiu

214 onsequently, the ability to maintain overall renal blood flow stability is not compromised in conscio

215 hase and neuronal nitric oxide synthase with renal blood flow suggest in this experimental model that

216 e of an associated low cardiac output or low renal blood flow syndrome.

217 patorenal syndrome may improve by increasing renal blood flow through the use of vasoconstrictors (va

218 and high ureteral pressure reduced cortical renal blood flow to 88% and 66%, respectively, of baseli

219 Autoregulatory restoration of renal blood flow to baseline after BP changes in conscio

220 ies and may play a role in the regulation of renal blood flow under physiological and patho-physiolog

221 Infusion of 1400W did not change renal blood flow, urine output, or creatinine clearance,

222 irst described in the 19th century, and GFR, renal blood flow, urine production, and electrolyte excr

223 aluated the feasibility of quantification of renal blood flow using data acquired during routine, cli

224 sing serum chemistries and histology and for renal blood flow using magnetic resonance imaging (MRI)

225 le renal resistance increased moderately and renal blood flow usually was maintained above control le

226 nder this normalized curve was compared with renal blood flow values.

227 nt signaling (including pain), regulation of renal blood flow, vascular endothelium, and inflammatory

228 Beat-to-beat changes in renal blood flow velocity (RBV; Duplex Ultrasound), mean

229 e 148 ([Cl] 98 mmol/L, Baxter Healthcare) on renal blood flow velocity and perfusion in humans using

230 ter Healthcare, Thetford, United Kingdom] on renal blood flow velocity and renal cortical tissue perf

231 sion of 0.9% saline results in reductions in renal blood flow velocity and renal cortical tissue perf

232 s measured with intravascular ultrasound and renal blood flow velocity with the aid of an intravascul

233 d perfusate at 37 degrees C for 75 min, mean renal blood flow was 110 ml/min/100 g and produced 85 ml

234 ell-based perfusate at 37 C for 75 min, mean renal blood flow was 110 ml/min/100 g and produced 85 ml

235 ), 289 (171-477), and 70 (51-91) mumol/L and renal blood flow was 270 +/- 42, 653 +/- 210, and 250 +/

236 Median renal blood flow was 482 mL/min (range 335-1137) in sept

237 The mean renal blood flow was 93.6 mL/min/100 g and the kidney pr

238 The increased renal blood flow was accompanied by more rapid intrarena

239 The CLP-caused reduction in renal blood flow was also ameliorated in Zdhhc21(dep/dep

240 PET-derived renal blood flow was correlated with imaging and clinica

241 Compared across vascular beds, resting renal blood flow was correlated with maximal stress myoc

242 Renal blood flow was determined with a flowprobe, and th

243 Renal blood flow was determined with a flowprobe.

244 Renal blood flow was gradually increased from 5 min to 3

245 PET quantification of renal blood flow was highly reproducible (intraclass cor

246 s 1.4 +/- 0.1 ml/min), and autoregulation of renal blood flow was maintained to a pressure level of a

247 Renal blood flow was measured by dynamic positron emissi

248 Renal blood flow was measured in all patients to calcula

249 Cerebral and renal blood flow was measured with colored microspheres.

250 Renal blood flow was measured with electron-beam compute

251 Total renal blood flow was not different between UT-A1/3(-/-)

252 At comparable levels of arterial BP, renal blood flow was not significantly different between

253 cant change in glomerular filtration rate or renal blood flow was observed.

254 In contrast, renal blood flow was reduced by infusion of a nonselecti

255 Basal renal blood flow was similar among the groups, whereas G

256 ients with kidney biopsy (n=12), resting PET renal blood flow was strongly negatively correlated with

257 Hg higher and glomerular filtration rate and renal blood flow were approximately 30% lower (P<0.001)

258 ar filtration rate, extraction fraction, and renal blood flow were assessed during PTA.

259 l perfusion, glomerular filtration rate, and renal blood flow were calculated.

260 oups, whereas glomerular filtration rate and renal blood flow were decreased less in uni-x sheep (PIn

261 med, and interlobar resistive index (RI) and renal blood flow were determined at baseline and during

262 al pressure, glomerular filtration rate, and renal blood flow were measured before and during NO inhi

263 n arterial pressure, forearm blood flow, and renal blood flow were measured during mental stress test

264 le kidney cortical, medullary perfusion, and renal blood flow were measured using multidetector compu

265 ood pressure, blood and urine chemistry, and renal blood flow were not different between e-5'NT/CD73(

266 Baseline mean arterial pressure and renal blood flow were similar in both dietary groups.

267 reflex renal vasoconstriction and decreased renal blood flow, which may implicate endogenous adenosi

268 els dilate, causing increased filtration and renal blood flow with decreased vascular resistance as a

269 Loss of renal blood flow with subsequent restoration, termed isc

270 etermine a dose of fenoldopam that increases renal blood flow without inducing hypotension in normote

271 hen determined: glomerular filtration rate = renal blood flow x (1 - hematocrit level) x EF.