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1 sition of IgG-containing immune complexes in renal glomeruli.
2 ost recipient organs, notably the thymus and renal glomeruli.
3 ded 125I uptake and beta-Gal activity in the renal glomeruli.
4 nti-DNA serum titers and in Ig deposition in renal glomeruli.
5 tial novel anti-inflammatory role of IL-6 in renal glomeruli.
6 meruli unless histones have already bound to renal glomeruli.
7 ease, characterized by IgA deposition in the renal glomeruli.
8 and SHP-1 or inhibition of VEGF signaling in renal glomeruli.
9 bs and deposition of immune complexes in the renal glomeruli.
10 veral protein kinase C (PKC) isoforms in the renal glomeruli.
11 6 versus 73.4 +/- 1.6 mmHg; P < 0.05), fewer renal glomeruli (57.8 +/- 23.8 versus 64.6 +/- 19.3 x 10
12 pes buried within NC1 hexamers obtained from renal glomeruli abundant in alpha3(IV) NC1 domains.
13 ycin reduced infiltration of leukocytes into renal glomeruli, an effect which was partially reversed
14 an evolution-directed selection protocol for renal glomeruli and identified what we believe to be a n
15  an activation and up-regulation of Rap1b in renal glomeruli and in cultured MCs, which then stimulat
16 mRNA and protein expression was increased in renal glomeruli and induced in tubular epithelium.
17 other PKC isoforms are also increased in the renal glomeruli and retina.
18  transgene mRNA was widely expressed both in renal glomeruli and tubules.
19 ctor (VEGF) is expressed by the podocytes of renal glomeruli, and has profound influences on systemic
20 ression of VEGF and its receptors in retina, renal glomeruli, aorta, and myocardium in insulin-resist
21                             Thus, within the renal glomeruli, aPC preserves endothelial cells via a p
22 and immunoglobulin deposition was present in renal glomeruli at 49 d.
23 undant HGF protein specifically localized in renal glomeruli, despite an extremely low level of trans
24 lated nephrin, and miR-29a levels in primary renal glomeruli from streptozotocin-induced diabetic mic
25 herapy increased transketolase expression in renal glomeruli, increased the conversion of triosephosp
26 e cases, we show its capacity to distinguish renal glomeruli injury states, quantify morphometric cha
27                              Finally, in the renal glomeruli of diabetic rats, the acetylation of S6
28 ctivated PKC alpha and beta1 isoforms in the renal glomeruli of diabetic rats.
29 1q-CLR are deposited and concentrated in the renal glomeruli of patients with SLE.
30    In addition, Ig deposits were detected in renal glomeruli of TGF- beta 1 knockout mice.
31                                              Renal glomeruli of the G3YR mice had significantly reduc
32 ly by a modest increase in the proportion of renal glomeruli producing renin in their juxtaglomerular
33 ively using both peripheral blood smears and renal glomeruli specimens.
34 diabetes in the cultured mesangial cells and renal glomeruli that can be prevented by LY333531.
35 lective in the epidermis, exocrine pancreas, renal glomeruli, the red pulp of the spleen, and within