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1 or coexisting Bosniak IV lesion and/or solid renal neoplasm.
2 sease gene associated with this rare form of renal neoplasm.
3 otype, 24 (45%) had at least one member with renal neoplasms.
4 ed as a fusion gene in a subset of pediatric renal neoplasms.
5 .1;q13), characteristic of another subset of renal neoplasms.
6 70 for nontransplanted control patients with renal neoplasms.
7 e identification and differentiation between renal neoplasms.
8                                              Renal neoplasms amenable to partial nephrectomy can be i
9                                         Both renal neoplasms and normal renal cortex demonstrated sig
10 dividuals to an increased risk of developing renal neoplasms and spontaneous pneumothorax.
11  is kept in mind, if conditions that mimic a renal neoplasm are considered and excluded, and if there
12 s differentiation of high-density cysts from renal neoplasms by demonstrating deenhancement as a proo
13 ng parenchyma and by the formation of benign renal neoplasms called angiolipomas.
14 who had a marking coil placed at biopsy of a renal neoplasm (coil group) and 23 patients who did not
15                   Wilms' tumor, an embryonic renal neoplasm diagnosed primarily in young children, ca
16  World Health Organization classification of renal neoplasms does not support such separation.
17 eal of information regarding the genetics of renal neoplasms has also emerged from the extensive cyto
18 trategies in the evaluation and treatment of renal neoplasms have been fostered by developments in th
19 rcinoma (RCC) represents 10% to 15% of adult renal neoplasms; however, the molecular genetic events t
20                               Enhancement of renal neoplasms is time dependent and may not be evident
21 utopsy-based study was performed to evaluate renal neoplasms occurring in patients who underwent soli
22             In human angiomyolipomas, benign renal neoplasms often found in tuberous sclerosis patien
23                                           In renal neoplasms, RhCG was expressed by chromophobe renal
24 wo cases of a hitherto undescribed pediatric renal neoplasm that is distinctive at the morphological,
25 umor cells and the identification of primary renal neoplasms that are genetically related to alveolar
26                                    For small renal neoplasms that are poorly visualized at unenhanced
27 th diethylstilbestrol- and estradiol-induced renal neoplasms, were observed in chromosomes 1, 2, 3, (