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1 loped in the upper urinary tract (ureter and renal pelvis).
2 henotype and functions of these cells in the renal pelvis.
3  ICs are a minor population of ICs in murine renal pelvis.
4 f typical smooth muscle cells (TSMCs) in the renal pelvis.
5 e medulla, where they release urine into the renal pelvis.
6 ers of the small bowel, ovaries, breast, and renal pelvis.
7 nary tract, such as the bladder, ureter, and renal pelvis.
8 adder, causing dilation of the ureter and/or renal pelvis.
9  16, 25, 30, or 35 cm above the level of the renal pelvis.
10 illae (2 vertical and 3 horizontal), and the renal pelvis.
11 l(-) channel, anoctamin-1, across the entire renal pelvis.
12 ) ICs were distinct from c-Kit(+) ICs in the renal pelvis.
13 ne (IRR 3.56 [95% CI 2.96-4.27]), kidney and renal pelvis (2.92 [2.50-3.42]), and pancreatic (2.61 [2
14 rine attenuation was lower in the obstructed renal pelvis (7.4 HU) than in the bladder (11.4 HU) (P <
15 ly removed by peristaltic contraction of the renal pelvis, a smooth muscle structure unique to placen
16 om a ring-like anastomosis under the nascent renal pelvis; a site of VEGF-C expression, to form a pat
17 mon disease characterized by dilation of the renal pelvis and calices, resulting in loss of kidney fu
18 r mL; dosed according to volume of patient's renal pelvis and calyces, maximum 60 mg per instillation
19 instillation) via retrograde catheter to the renal pelvis and calyces.
20  with highest posttransplant recognition for renal pelvis and cortex specific antigens.
21 ee transitional cell carcinomas (TCC) of the renal pelvis and five Wilms' tumors were compared with n
22 dynamic injection of a DNA solution into the renal pelvis and found that luciferase expression was st
23 inner and outer medulla, papillary tips, and renal pelvis and from glomeruli isolated by sieving.
24 a were observed in groups at the wall of the renal pelvis and in the angular space formed by the pole
25 /CD45(+) ) are more abundant in the proximal renal pelvis and pelvis-kidney junction regions whereas
26 y devoid of AT1 receptor fail to develop the renal pelvis and the ureteral peristaltic movement.
27 in humans that drinking-water arsenic causes renal pelvis and ureter cancer.
28                                          For renal pelvis and ureter cancers, the adjusted odds ratio
29 included 76 renal cell, 24 transitional cell renal pelvis and ureter, and 22 other kidney cancers.
30 iferation causes abnormal development of the renal pelvis and ureter, leading to defective pyelourete
31 5(-) ) are found predominantly in the distal renal pelvis and ureteropelvic junction.
32                   The contrast medium in the renal pelvis and ureters was virtually removed from excr
33 lionic fibers projected into the wall of the renal pelvis and/or to the interlobar arteries extending
34 er the induction of elevated pressure in the renal pelvis, and after the pressure was returned to nor
35    In severe cases dilatation of the ureter, renal pelvis, and calyces might be seen.
36 gauge needle was inserted into the opacified renal pelvis, and double-contrast pyelography was perfor
37                    Cancers of the kidney and renal pelvis are among the most prevalent types of urina
38              Resident pacemaker cells in the renal pelvis are critical to this process and spontaneou
39                   Most c-Kit(+) cells in the renal pelvis are mast cells.
40 chyma and its subsequent filtration into the renal pelvis at a temporal resolution down to 1 s.
41         In contrast, the incidence rates for renal pelvis cancer declined among white men and remaine
42                  The corresponding rates for renal pelvis cancer were 1.5, 0.7, 0.8, and 0.5 per 1000
43 en with cancer (renal-cell cancer in 759 and renal-pelvis cancer in 136) were identified by cross-lin
44 a greater risk of both renal-cell cancer and renal-pelvis cancer than men who were not smokers.
45 mass index or blood pressure and the risk of renal-pelvis cancer.
46 o outline the incidence trends of kidney and renal pelvis cancers by age, sex, race/ethnicity, and hi
47 e identification of patients with kidney and renal pelvis cancers with morphologies of renal cell car
48 84 years had the highest ASIR for kidney and renal pelvis cancers.
49 ticeable increase in incidence of kidney and renal pelvis cancers.
50 dentification and purification amongst other renal pelvis cell types.
51 ary tract, smooth muscle cells that line the renal pelvis contract in a coordinated effort to form pe
52     It is not known whether mild-to-moderate renal pelvis dilatation (RPD) identified at 18-20 weeks
53 brosis, development of glomerular cysts, and renal pelvis dilation.
54 n of alimentary tract, retention of urine in renal pelvis, distension of bladder, and the development
55  adventitial and urothelial layers of murine renal pelvis do not express smooth muscle myosin heavy c
56 or (AT(1)R)-deficient mice exhibit a dilated renal pelvis (hydronephrosis) and a small papilla.
57 ient with transitional cell carcinoma of the renal pelvis, hydronephrosis proximal to the tumor may c
58 ity, as well as dilatation of the ureter and renal pelvis (hydroureternephrosis) at birth and at post
59 NIR perfusion with increased distance to the renal pelvis, identifying well and poor perfused segment
60 stalsis and point to the central role of the renal pelvis in urine transport.
61                                 Hydrodynamic renal pelvis injection enables transposon mediated-kidne
62                    Importantly, we show that renal pelvis injection overcomes pre-existing immunity,
63 erase expression quickly decreased following renal pelvis injection, the use of the piggyBac transpos
64     To assess renal injury, we performed the renal pelvis injections on uninephrectomised mice and fo
65                  Urothelial carcinoma of the renal pelvis is a deadly disease with an unclear tumorig
66 orectal, endocrine, gastrointestinal, kidney/renal/pelvis, liver, lung, genital, oral cavity, pharynx
67 nd bronchus, thyroid, colorectal, kidney and renal pelvis, melanoma, non-Hodgkin lymphoma, and leukem
68  cassette to drive human CAII cDNA, into the renal pelvis of CAII-deficient mice results in expressio
69 expressed in nascent urothelia in ureter and renal pelvis of human embryos, and it is suggested that
70           Transitional cell carcinoma of the renal pelvis or distal ureter has been extirpated with s
71 cluding renal cell cancer and cancers of the renal pelvis or ureter.
72  thyroid gland but not in the liver, spleen, renal pelvis, or salivary glands.
73     Formation of a lymphoid aggregate in the renal pelvis precedes the invasion of the kidney by infl
74 olon, non-Hodgkin lymphoma, pancreas, kidney/renal pelvis, rectum, and melanoma.
75 ne that localize to the cortex, medulla, and renal pelvis, respectively.
76 ensin type 1 receptor genes do not develop a renal pelvis, resulting in the buildup of urine and prog
77 s, but HB-EGF expression did not increase in renal pelvis smooth muscle cells under identical conditi
78 e smooth muscle and stromal cells lining the renal pelvis, the papilla and glomerular endothelial cel
79  the efficient removal of the urine from the renal pelvis to the bladder by means of their contractil
80 eter actively propels tubular fluid from the renal pelvis to the bladder, and this peristalsis, which
81 istal peristaltic waves, pump urine from the renal pelvis toward the urinary bladder.
82 eteropelvic junction (UPJ) region, where the renal pelvis transitions to the ureter, is the most comm
83                 Rhythmic contractions of the renal pelvis transport urine from the kidneys into the u
84 cidence for all urothelial cancers combined (renal pelvis, ureter, and bladder cancers: adjusted IRR
85 n development affecting the renal parechyma, renal pelvis, ureter, bladder and urethra; they show evi
86 /=18 years) with urothelial carcinoma of the renal pelvis, ureter, bladder, or urethra at 16 sites in
87  urothelial cancer, including cancers of the renal pelvis, ureter, bladder, or urethra, from eight ho
88 d disease (OR, 1.6; 95% CI, 0.8-3.0) and for renal pelvis/ureter cancers (OR, 1.7; 95% CI, 0.5-5.4).
89 ectal, prostate, bladder, thyroid, kidney or renal pelvis, uterus, pancreas, or oral cancer).
90  the pelvis-kidney junction (PKJ) and distal renal pelvis were evaluated.
91  Segmentations of the kidneys (excluding the renal pelvis) were generated by the automated method and
92 ptoms, the association of malrotation of the renal pelvis with calculus increases the risk of hematur