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1 I, 2.89-14.38) were also associated with new renal scarring.
2 ibiotics, bowel and bladder dysfunction, and renal scarring.
3 total nephrectomy (SNx) model of progressive renal scarring.
4  A total of 35 children (7.2%) developed new renal scarring.
5 tially reduced risk of recurrence but not of renal scarring.
6  1280 included participants, 199 (15.5%) had renal scarring.
7 1 identified 44.9% of patients with eventual renal scarring.
8 coli are at high risk for the development of renal scarring.
9 mune injury but also kidney regeneration and renal scarring.
10 ologic analyses to evaluate inflammation and renal scarring.
11 inary tract infection and the development of renal scarring.
12 erapy are associated with increased rates of renal scarring.
13 per versus lower urinary tract infection and renal scarring.
14 thout affecting the risk of recurrent UTI or renal scarring.
15 aphy for the diagnosis of pyelonephritis and renal scarring.
16  about its impact on recurrent infection and renal scarring.
17 designed to prevent recurrent infections and renal scarring.
18 ract infection and preventing recurrence and renal scarring.
19 aphy for the diagnosis of pyelonephritis and renal scarring.
20  pathogenesis of urinary tract infection and renal scarring.
21 intigraphy as the reference method to detect renal scarring.
22 ction and thereby prevent the development of renal scarring.
23 btained six months later identify those with renal scarring.
24 is is effective in reducing reinfections and renal scarring.
25 possible therapeutic strategy in progressive renal scarring.
26  the diseased kidney and could contribute to renal scarring.
27 endoscopic injection among the patients with renal scarring and bladder dysfunction were acceptable.
28                               In conclusion, renal scarring and bladder dysfunction were predictors o
29          In the multivariable analysis, only renal scarring and bladder dysfunction were significantl
30 age-mediated inflammatory responses promoted renal scarring and compromised renal function, as indica
31 rs that block angiotensin II protect against renal scarring and drug-induced arteriolopathy in this m
32  children is considered as a risk factor for renal scarring and each patient is treated with prudence
33 therefore play a role in the pathogenesis of renal scarring and fibrosis in patients with CRD and can
34 a growing concern because it poses a risk of renal scarring and irreversible loss of kidney function.
35  spontaneously resolve and those at risk for renal scarring and its sequelae.
36 of recurrent UTIs and minimizing the risk of renal scarring and long-term renal impairment.
37   Increased rates of breakthrough infection, renal scarring and surgical failure have been associated
38 y albuminuria (>2 g/24 h; n=15) had the most renal scarring and the lowest endothelial CD34 staining.
39 ment febrile urinary tract infections and/or renal scarring and vesicoureteral reflux persistence, re
40 both febrile urinary tract infections and/or renal scarring and vesicoureteral reflux persistence, th
41 y in treatment of febrile UTIs and permanent renal scarring are associated.
42 antimicrobial therapy and the development of renal scarring are inconsistent.
43 ureteral reflux, urinary tract infection and renal scarring, as well as the efficacy of various treat
44 ernative approaches to prevention of UTI and renal scarring based on research into host-pathogen inte
45                                  Analysis of renal scarring by Masson trichrome staining, kidney hydr
46                                          New renal scarring defined as the presence of photopenia plu
47                            The occurrence of renal scarring did not differ significantly between the
48                     Advanced UG-TB can cause renal scarring, distortion of renal calyces and pelvic,
49 onses might be a therapeutic tool to prevent renal scarring during acute pyelonephritis.
50 erapy remained significantly associated with renal scarring even after adjusting for these variables.
51  are susceptible to recurrent infections and renal scarring following urinary tract infection.
52  between UTI-vesicoureteral reflux (VUR) and renal scarring has been challenged by several studies.
53 s such as recurrent urinary tract infection, renal scarring, hypertension, and compromised renal func
54 ect of P2X(7) receptors on infection-induced renal scarring in a murine model of pyelonephritis.
55 boratory, and imaging variables in detecting renal scarring in children and adolescents with a first
56 prevent recurrence of UTI and development of renal scarring in children with vesicoureteral reflux.
57 amyl) lysine crosslink, occur in progressive renal scarring in humans independently of the original e
58 complement inhibition with Crry might affect renal scarring in lupus nephritis, gene transcript profi
59  prophylaxis in preventing recurrent UTI and renal scarring in young children with VUR.
60                                 Experimental renal scarring indicates that tissue transglutaminase (t
61 t understanding of the mechanisms underlying renal scarring leading to ESRD to inform on current and
62                                 It may cause renal scarring leading to secondary hypertension and chr
63                      Some pyelonephritis and renal scarring may be related to vesicoureteral reflux t
64  with a complete duplication anomaly, severe renal scarring, or persistent VUR associated with an ect
65 inine clearance (P<0.01) and positively with renal scarring (P<0.05) but did not correlate with MR mR
66 or acute pyelonephritis, which can result in renal scarring (reflux nephropathy), hypertension, end-s
67 nisms underlying acute pyelonephritis-driven renal scarring remain unknown.
68  or severe cases of pyelonephritis can cause renal scarring that subsequently can lead to progressive
69 ng recurrent UTI, vesicoureteral reflux, and renal scarring, the mechanisms underlying BBD have been
70 of antimicrobial therapy was associated with renal scarring; the median (25th, 75th percentiles) dura
71 eteral reflux was the strongest predictor of renal scarring, this degree of reflux was present in onl
72                      Secondary outcomes were renal scarring, treatment failure (a composite of recurr
73 cularly high-risk group in whom the risk for renal scarring was 30.7%.
74 antibiotic therapy in those with and without renal scarring was 72 (30, 120) and 48 (24, 72) hours, r
75                                              Renal scarring was defined by the presence of photopenia
76 for 89 percent of the children (275 of 309); renal scarring was noted in 9.5 percent of these childre