ライフサイエンスコーパス: repair protein

1 amage is mediated by FANCD2, a DNA crosslink repair protein.

2 otein homolog 2 (MSH2) is a key DNA mismatch repair protein.

3 is required for loading of the Ku70/Ku80 DSB repair protein.

4 s end joining (NHEJ) DNA double strand break repair protein.

5 calized accumulations of ectopic 53BP1-a DNA repair protein.

6 hosphate-ribose) polymerase], a critical DNA repair protein.

7 by aiding the catalytic turnover of excision repair proteins.

8 ing the importance of the putative AddAB DNA repair proteins.

9 via hydrolysis of polyADP-ribose on related repair proteins.

10 r against toxic side effects of abundant DNA repair proteins.

11 nteractions between the MR complex and other repair proteins.

12 id men without defects in genes encoding DNA-repair proteins.

13 depends on the overexpression of alkylation repair proteins.

14 on of the versatility of these base excision repair proteins.

15 ne dependent on the Sir, as well as on other repair proteins.

16 nick provides an entry point for downstream repair proteins.

17 EIL1's interactions with other base excision repair proteins.

18 nts in transporter proteins and DNA mismatch repair proteins.

19 pathway using conserved repair and some non-repair proteins.

20 further causes degradation of these modified repair proteins.

21 ant trigger for activating at least some DNA repair proteins.

22 tical roles in the assembly of signaling and repair proteins.

23 interact with several well-characterized DNA repair proteins.

24 point proteins, and reduce expression of DNA repair proteins.

25 is known to interact with many cellular DNA repair proteins.

26 d immunoblot analysis of DNA replication and repair proteins.

27 mmon amongst replication, recombination, and repair proteins.

28 a broader role in epigenetics for these DNA repair proteins.

29 communication between MCM and its auxiliary repair proteins.

30 s leading to synthesis of malfunctioning DNA repair proteins.

31 litates long-range signaling between [4Fe4S] repair proteins.

32 ns that promote efficient recruitment of DNA repair proteins.

33 ay, which can lead to dependence on specific repair proteins(2).

34 changes substantially in the absence of DNA repair protein 53BP1.

35 alleles based on the accumulation of the DNA-repair protein 53BP1.

36 e ubiquitin in the recruitment of DNA damage repair proteins 53BP1 and BRCA1.

37 for understanding the success and failure of repair protein activity associated with such oxidatively

38 s a sensitive, selective, and rapid assay of repair protein activity, enabling a biological interroga

39 molecular level for how this network of DNA repair proteins acts to maintain genome stability and su

40 h TNKS1 concomitant recruitment of TNKS1 and repair proteins after damage.

41 The DNA and RNA repair protein AlkB removes alkyl groups from nucleic ac

42 Rad23 was identified as a DNA repair protein, although a role in protein degradation h

43 tion of the Ada protein, which acts as a DNA repair protein and damage sensor.

44 asthma, we observed increased levels of DNA repair proteins and apoptosis, as shown by caspase-3 cle

45 with distinct groups of DNA replication and repair proteins and by post-translational modifications.

46 ion of several DNA double-strand break (DSB) repair proteins and formation of repair complexes, and r

47 not only damages DNA but can also modify DNA repair proteins and further causes degradation of these

48 date ClpS1 substrates, including plastid DNA repair proteins and Glu tRNA reductase, which is a contr

49 rovides an alternative method to monitor DNA repair proteins and histone dynamics at the DNA damage s

50 activity, XRCC1 interacts with multiple DNA repair proteins and is a subunit of distinct DNA repair

51 erns of histone modification, recruiting DNA repair proteins and modulating chromatin packing during

52 strategy to prevent clashes with engaged DNA repair proteins and preserve the integrity of the replic

53 of DNA breaks facilitates recruitment of DNA repair proteins and promotes chromatin relaxation in par

54 However, how H4 acetylation (H4Ac) recruits repair proteins and reorganizes chromatin during DNA rep

55 eals previously unknown interactions between repair proteins and silencing proteins and suggests insi

56 genetically competent they also activate DNA repair proteins and stop dividing.

57 interactions between PHF11 and multiple DNA repair proteins and suggests that PHF11 mediates 5' end

58 ed that NS is co-enriched positively with HR repair proteins and that high NS expression correlates w

59 interaction networks derived from human DNA repair proteins and yeast chromatin remodeling complexes

60 ing DNA damage to phosphorylate specific DNA repair proteins and/or that NMD inactivation may lead to

61 n early glycation intermediates and releases repaired proteins and lactate or glycolate, respectively

62 two representative MDR proteins, MGMT (a DNA repair protein) and ABCB1 (an efflux protein), revealing

63 B is caused by loss of dysferlin, a membrane repair protein, and LGMD 2C is caused by loss of the dys

64 easured DNA double-strand breaks (DSBs), DNA repair proteins, and apoptosis in an HDM-induced allergi

65 on proteins, transcription factors, mismatch repair proteins, and DNA polymerases discriminate betwee

66 erchromatin space, aberrant spreading of DNA repair proteins, and hyper-resection of DNA ends.

67 ) forms complexes with CtIP as well as other repair proteins, and is essential for HR repair by regul

68 ribose) include PARP-1 itself, histones, DNA repair proteins, and transcription factors.

69 in APTX, which encodes the DNA strand-break repair protein aprataxin (APTX).

70 representing the CCHH zinc finger of the DNA repair protein aprataxin, but did bind to an aprataxin p

71 PTX gene, which encodes the DNA strand-break repair protein aprataxin.

72 A base excision repair protein, Apurinic/apyrimidinic (AP) endonuclease

73 The essential base excision repair protein, apurinic/apyrimidinic endonuclease 1 (AP

74 s proteins accumulated at DSB sites, how DNA repair proteins are assembled into damaged chromatin has

75 However, the DSB-initiating and repair proteins are associated with a linear proteinaceo

76 However, although mutations in DSB repair proteins are common in hereditary cancers, mechan

77 -glycoprotein complex and the known membrane repair proteins are expressed normally, membrane reseali

78 The presence of an active repair protein as an essential component of the polymera

79 mplexes containing DNA replication and other repair proteins, as shown by gel filtration.

80 SET led to loss of expression of several DNA repair proteins, as well as decreased recruitment of DNA

81 Our findings emphasize that damage to DNA repair proteins, as well as to DNA itself, is likely to

82 iquitin chain composition for downstream DNA repair protein assembly.

83 can partner with MSH2, an important mismatch repair protein associated with hereditary non-polyposis

84 quitylation events that promote retention of repair proteins at DNA lesions.

85 ine the organization and interaction of NHEJ repair proteins at DSB ends.

86 and show via immunofluorescent detection of repair proteins at ionizing radiation-induced DNA damage

87 subunits that assist in the assembly of DNA repair proteins at sites of damage.

88 tion of nucleosomes and concentration of DNA-repair proteins at the site of damage.

89 e more dispersedly occupied by the conserved repair protein, AtBRCA1, which can also co-localise with

90 ing a positive feedback loop between the DNA repair proteins ATM and VDR.

91 difficult in the case of base extrusion DNA repair proteins because of the complexity of the repair

92 of the coefficient of diffusion for any DNA repair protein between different cell types, obtained in

93 Little is known about Tpa1 as a repair protein beyond the initial observation from a hig

94 specifically requires the DNA recombination repair proteins BLM and BRCA2 as well as a non-canonical

95 uclear exploration is regulated by the Rad51 repair protein, both at the break and throughout the gen

96 re that, together with a number of other DNA repair proteins, both ATR and its associated protein, AT

97 53, alterations in genes encoding DNA damage repair proteins (BRCA and FANC) were detected in 14% of

98 ites of DNA damage along with the DNA damage repair protein, BRCA1, and FUS and TDP43 participate in

99 D4 deficiency impairs the recruitment of DNA repair proteins BRIT1, BRCA1, and replication protein A

100 Modulation of DNA repair proteins by small molecules has attracted great i

101 Genetic testing for BRCA1, a DNA repair protein, can identify carriers of pathogenic vari

102 The nucleotide excision repair protein complex ERCC1-XPF is required for incisio

103 pose that protein stability, mediated by DNA repair protein complex formation, functions as a regulat

104 e amplification when the function of the DSB repair protein complex MRN (Mre11/Rad50/Nbs1) is impaire

105 ir proteins and is a subunit of distinct DNA repair protein complexes.

106 nalysis of endonuclease III (EndoIII), a DNA repair protein containing a [4Fe-4S] cluster known to be

107 aracterization of the redox chemistry of DNA repair proteins containing redox cofactors, and multiple

108 We have proposed a model where repair proteins containing redox-active [4Fe-4S] cluster

109 DNA repair protein counteracting oxidative promoter lesions

110 Cellular mechanisms aimed at repairing protein damage and maintaining homeostasis, wi

111 The DNA repair protein damaged DNA-binding 2 (DDB2) has been imp

112 o reliably identify tumors with DNA mismatch repair protein deficiency (MMR-D) on the basis of increa

113 in the nonhomologous end-joining (NHEJ) DNA repair protein DNA ligase IV (LIG4) lead to immunodefici

114 Inhibition of the DNA repair proteins DNA-dependent protein kinase (DNA-PK) or

115 ensive assessment of the localization of DSB repair proteins during KSHV replication, we have determi

116 leus, including a dozen that function as DNA repair proteins (e.g., BRCA1, 53BP1) or nucleases (e.g.,

117 orectal cancer cells deficient in DNA damage repair proteins, EME1 and MUS81, were significantly more

118 uction potential found experimentally in DNA repair proteins, enabling their HiPIP-like redox behavio

119 POLE and mismatch repair protein-encoding genes were mutated at lower freq

120 S] clusters regulates the ability of two DNA repair proteins, Endonuclease III and DinG, to bind pref

121 The DNA repair proteins ERCC1 and XPF are needed to remove cispl

122 ing activities of two well-characterized DNA repair proteins, Escherichia coli MutS and human p53.

123 hocytic infiltrate, such as loss of mismatch repair protein expression or expression of Epstein-Barr

124 interactions with other nucleotide excision repair protein factors of the two enzymes.

125 Intriguingly, ICL repair protein, Fanconi anemia complementation group A p

126 interaction of related double-stranded break repair proteins, finally causing the increase of unrepai

127 gesterone) on gene expression related to DNA repair, protein folding (chaperones), the ubiquitin-prot

128 of phospho-Smad2 (pSmad2) and Smad7 with DSB repair proteins following low and high linear energy tra

129 reveal how lncRNA effectively replaces a DNA repair protein for efficient NHEJ with implications for

130 nine-DNA alkyltransferase (MGMT) is the sole repair protein for O(6)-alkylguanine lesions in DNA and

131 low the sequential recruitment of downstream repair proteins for successful execution of NER.

132 e efficient DNA resection and recruitment of repair proteins; for example, inward translocation of th

133 cation compartments (RCs), whereas other DSB repair proteins form foci outside RCs.

134 e-transport signaling in E. coli between DNA repair proteins from distinct pathways.

135 nt mechanistic hypotheses regarding mismatch repair protein function in mediating triplet repeat expa

136 ut the extent of their interactions with DNA repair proteins has not been thoroughly characterized.

137 Multiple DNA repair proteins having ubiquitin ligase activity are rec

138 d similar telomere loss, suggesting that the repair proteins help to resolve similar problems in telo

139 n be detected by MutS homolog (MSH) mismatch repair protein heterodimers.

140 The mismatch repair protein hMSH2 was found to be a major ATR-binding

141 heterogeneous nuclear levels of the mismatch repair protein hMSH3.

142 show that the mammalian nucleotide excision repair protein homolog MMS19 can simultaneously bind pro

143 physiological Ku70, an essential DNA damage repair protein in neurons whose function is known to be

144 ude that Tpa1 is a hitherto unidentified DNA repair protein in yeast and that it plays a crucial role

145 (ERFSs), by genome-wide localization of DNA repair proteins in B cells subjected to replication stre

146 we examined the role of double-strand-break repair proteins in gene silencing and nuclear organizati

147 matin organization and suggest a role for HR repair proteins in genomic organization.

148 oscopy as methods to monitor kinetics of DSB repair proteins in living cells under conditions mimicki

149 ein kinase, catalytic subunit, essential DNA repair proteins in the nonhomologous end-joining pathway

150 nce for coordinated DNA CT between different repair proteins in their search for damage in the genome

151 Nol12 co-localizes with DNA repair proteins in vivo including Dhx9, as well as with

152 tion of DNA-PK, a non-homologous end joining repair protein, in Hec-108 cells.

153 o known as MG53), a muscle-enriched membrane repair protein, in IIM patient sera and in our mouse mod

154 in TONSL, which encodes the Tonsoku-like DNA repair protein, in nine subjects (from eight families) w

155 nates two substrates, and several downstream repair proteins including nucleases and homologous recom

156 e that requires the interplay of a number of repair proteins including those of the Fanconi anemia (F

157 The n-terminal tail of histone H4 recruits repair proteins, including 53BP1, to DNA double-strand b

158 Repair proteins, including dysferlin, EHD1, EHD2, MG53,

159 f BRCA1 in repair complexes; upregulation of repair proteins, including HMGN1 and RFC1; and regulatio

160 The binding of DNA repair proteins, including p53 and BRCA2, has been visua

161 l after additional knockdown of specific DNA repair proteins, including PARP1.

162 lin D1 interactors revealed a network of DNA repair proteins, including RAD51, a recombinase that dri

163 ctions as a scaffold protein to assemble DNA repair proteins into chromatin followed by efficient act

164 lguanine-DNA methyltransferase (MGMT), a DNA repair protein involved in chemotherapeutic resistance o

165 owever, when a CT-deficient mutant of either repair protein is combined with the CT-proficient repair

166 A feature of many base excision repair proteins is that they contain [4Fe4S] clusters th

167 A poorly understood aspect of DNA repair proteins is their ability to identify exceedingly

168 A common feature of DNA repair proteins is their mobilization in response to DNA

169 NA binding protein 2), a nucleotide excision repair protein, is upregulated by hypoxia.

170 MutL is a mismatch repair protein known to coordinate several steps in repa

171 PAXX interacts directly with the DSB-repair protein Ku and is recruited to DNA-damage sites i

172 ecognition of each broken DNA end by the DNA repair protein Ku is the first step in NHEJ, followed by

173 f1 to PARP1, a critical NAD(+)-dependent DNA repair protein, leading to inhibition of PARP1 auto-ADP-

174 miR-155 is known to target DNA-repair proteins, leading to a mutator phenotype, and we

175 man GBM cells had enhanced activation of DNA repair proteins, leading to increased DNA repair and sub

176 ing localize to sites of damage and some DNA repair proteins localize to heterochromatin, but the bio

177 All incident CRCs were analyzed for mismatch repair protein loss, supported by BRAF mutation and micr

178 A model is described where repair proteins may signal one another using DNA-mediate

179 The mammalian repair protein MBD4 (methyl-CpG-binding domain IV) excis

180 We identify two distinct mechanisms for repair protein-mediated silencing-via direct and indirec

181 ions differing only in expression of the DNA repair protein methyltransferase (MGMT), a TMZ-sensitivi

182 ylation of mitochondrial fusion and mismatch repair proteins, Mfn2 and Mlh1 respectively, was determi

183 ges and occurs independently of the membrane repair protein MG53.

184 Dysferlin interacts with a new membrane repair protein, mitsugumin 53 (MG53), an E3 ubiquitin li

185 stability from reduction of the DNA mismatch repair protein MLH1, are unaffected by CENP-E-dependent

186 sites marked by the binding of the mismatch repair protein MLH1.

187 y those with dMMR, based on loss of mismatch repair proteins MLH1, MSH2, MSH6, and/or PMS2.

188 s used to analyze the presence of 4 mismatch repair proteins (MLH1, MSH2, MSH6, and PMS2) in these tu

189 E1b and E4 proteins target the cellular DNA repair protein Mre11 for redistribution and degradation,

190 otruding nonhomologous 3' tail, the mismatch repair protein Msh2 does not discourage homeologous reco

191 tivity of DNA polymerase-delta, although the repair proteins Msh2, Mlh1 and Exo1 influence the extent

192 we found a strong repression of the mismatch repair proteins MSH2, MSH6, and EXO1 as well as the homo

193 inds cooperatively with UNG and the mismatch repair proteins Msh2-Msh6 to Ig Smu and Sgamma3 regions,

194 lusterin modulated the expression of the DNA repair proteins, MSH2, MSH6, OGG1 and BRCA1.

195 Cells lacking mismatch repair proteins Msh6 and Mlh1 form chimeric recombinants

196 DNA repair proteins must locate rare damaged sites within th

197 The mismatch repair protein MutL has an internal clamp-binding motif,

198 its weak-binding partners, the DNA mismatch repair protein MutL.

199 med cell death ligand 1 (PD-L1) and mismatch repair proteins MutL homolog 1 (MLH1), MutS homolog 2 (M

200 the long-range communication in DNA mismatch repair proteins MutS and its homologs where intramolecul

201 Here we use the postreplicative mismatch repair proteins MutSalpha and MutLalpha as model systems

202 e presence, not the absence, of the mismatch repair protein MutSbeta (Msh2-Msh3 heterodimer).

203 7, the transcriptional repressor TRIM27, DNA repair proteins NBN and MRE11A, regulators of apoptosis,

204 transmission of HSV-1 and found that the DNA repair protein Nbs1 from only some primate species is ab

205 , in part, through reduced activation of the repair protein Nijmegen Breakage Syndrome-1 but not p53.

206 (iRFP670), with the latter fused to the DNA repair protein O(6)-methylguanine-DNA-methyltransferase

207 ses S-nitrosylation and depletion of the DNA repair protein O6-alkylguanine-DNA-alkyltransferase (AGT

208 CHD4 is recruited by the excision repair protein OGG1 for oxidative damage to interact wit

209 DDAs by interfering with trafficking of DNA repair proteins on interphase microtubules.

210 investigating the influence of specific DNA repair proteins on the biological response toward DNA da

211 ethods based on monitoring of recruitment of repair proteins or histone modifications at the damage s

212 lutionally conserved antioxidant enzyme that repairs protein oxidation by reversing the oxidative mod

213 of shot1 thermotolerance, indicating HSP101 repairs protein oxidative damage and/or reduced oxidativ

214 The DNA repair protein p53 binding protein 1 (53BP1) protects th

215 oth CCHC DNA-binding zinc fingers of the DNA repair protein PARP-1 (poly(ADP-ribose) polymerase-1).

216 zymatic cofactor for enzymes such as the DNA repair protein PARP.

217 sustaining the damage by inhibiting the DNA repair protein poly(ADP-ribose) polymerase (PARP).

218 otential synthetic lethal partner of the DNA repair protein polynucleotide kinase/phosphatase (PNKP).

219 We also examined expression of the DNA repair protein Rad18, which is regulated by MTDH at the

220 The human homolog of the yeast DNA repair protein RAD23, hHR23A, has been found previously

221 omplex [meiotic recombination 11 (Mre11)/DNA repair protein Rad50/Nijmegen breakage syndrome 1 protei

222 The DNA repair protein RAD51 and the trans-lesion synthesis DNA

223 hesis that Jab1 positively regulates the DNA repair protein Rad51 and, in turn, cellular response to

224 cancer stem-like cells via inhibition of DNA repair protein RAD51 homolog 1 (RAD51).

225 P = 4.51 x 10(-5)) between a SNP in the DNA repair protein RAD51 homolog 2 gene (RAD51L1; rs10483813

226 foci of DNA damage marker gammaH2AX and the repair protein Rad51 in damaged cells.

227 2 loss did not affect the recruitment of the repair protein RAD51 to sites of double-strand breaks (D

228 c or pharmacologic suppression of the HR DNA repair protein RAD51.

229 ous p220 aberrantly directs at least one DNA repair protein, RAD51, to damage sites, where their acti

230 es and regulates the recruitment of the core repair protein, RAD51, to hydroxyurea-induced foci.

231 The DNA repair protein RAD52 is an emerging therapeutic target o

232 protein RPA and the homologous recombination repair protein Rad52.

233 omplex structure, ABRAXAS integrates the DNA repair protein RAP80 and provides a high-affinity bindin

234 lcytosine (mC), and downstream base excision repair proteins restore a G.C pair.

235 on gene NTHL1, which encodes a base excision repair protein, revealed a mutational footprint (signatu

236 athway that included DNA double-strand break repair proteins RexAB, recombinase A, and polymerase V.

237 s active under ambient CO(2)), the metabolic repair protein RsRca (Rs-activase) was introduced via nu

238 s, likely by facilitating the recruitment of repair proteins similar, but not identical, to its propo

239 As a result, DNA repair proteins such as BRCA1, RAP80, and Rad51 cannot b

240 A adducts, is linked to their recognition by repair proteins such as HMGB1a.

241 alyzed repair outcomes in the absence of DNA repair proteins such as KU70 and LIG4 (both involved in

242 articipate in interaction with MLH1 mismatch-repair protein, suggesting that the FANCJ activity suppo

243 l loss of function of Ku80, an essential DNA repair protein, suppressed poly(GR)-induced retinal dege

244 How human DNA repair proteins survey the genome for UV-induced photopr

245 (HBD, HBB, IL36G), and decreased epithelial repair proteins (TFF3, F11R).

246 1) is a multi-functional double-strand break repair protein that is essential for class switch recomb

247 The endonuclease CtIP is a DNA repair protein that is well-known to promote genome stab

248 rG(Mtb) , forms a cytosolic complex with DNA-repair proteins that assembles independently of either D

249 inding site for p53 and other DNA damage and repair proteins that contain amphipathic alpha helical d

250 noubiquitinated-FANCD2 serves to recruit DNA repair proteins that contain ubiquitin-binding motifs.

251 icing activity arose to post-translationally repair proteins that had been inactivated by deleterious

252 reatments were associated with damage to DNA repair proteins that reduced the efficiency of nucleotid

253 stance (MDR), generally caused by innate DNA repair proteins that reverse the DNA modification by ant

254 allow the virus to evade localization of DSB repair proteins that would otherwise have a detrimental

255 are recognized and removed by base excision repair proteins, the 5hmC base accumulates to substantia

256 irus (HCMV) selectively relocalizes many DNA repair proteins, thereby avoiding a potentially detrimen

257 f biological macromolecules such as mismatch repair proteins through biotinylated DNA substrates.

258 cate a general link between the ability of a repair protein to carry out DNA CT and its ability to re

259 ic strength influence the binding of two DNA repair proteins to a variety of DNA structures.

260 d to modulate the DDR by affecting access of repair proteins to chromatin.

261 trand breaks (DSB), promoting recruitment of repair proteins to chromatin.

262 ng affinity regulates the ability of [4Fe4S] repair proteins to collaborate in the lesion detection p

263 Recruitment of DNA repair proteins to DNA damage sites is a critical step f

264 histone octamers, however, impedes access of repair proteins to DNA damage.

265 hat accelerates cell recovery by helping DNA repair proteins to locate their targets, and facilitatin

266 Migration also causes multiple DNA repair proteins to segregate away from DNA, with cytopla

267 tion in which it directly interacts with DNA repair proteins to sensitize prostate cancer cells to th

268 e PARylation orchestrates the recruitment of repair proteins to sites of damage.

269 wn as crucial facilitators of recruitment of repair proteins to sites of DNA damage.

270 ins, as well as decreased recruitment of DNA repair proteins to sites of DNA double-strand breaks (DS

271 verse contributions of several recombination/repair proteins to telomere maintenance in Ustilago mayd

272 uitination and recruitment of additional DSB repair proteins to the break.

273 aces to recruit multiple postreplication DNA repair proteins to the CRL4-DCAF1 E3 ligase for ubiquiti

274 tokines, caused hMSH3, but no other mismatch repair proteins, to move from the nucleus to the cytosol

275 We conclude DNA damage-repair proteins traffic on microtubules and addition of

276 bility including cell cycle checkpoints, DNA repair, protein ubiquitination, chromatin remodelling, t

277 NR-21, NR-22 and NR-27 and loss of mismatch repair proteins using four different markers (MLH1, MSH6

278 Similar to other DNA repair proteins, USP20 is phosphorylated post DNA damage

279 ase subunit RpoB and the nucleotide excision repair protein UvrA.

280 e complex formed between the prokaryotic DNA repair protein UvrA2 and DNA.

281 umors that had normal expression of mismatch repair proteins (validation cohort).

282 reserves the pool of reduced thioredoxin for repairing proteins vital to survival.

283 only treated cells; the transcription of DNA repair proteins was also reduced.

284 cells; and significant up-regulation of DNA repair proteins was observed after co-exposure.

285 DNA lesions also interact with base excision repair proteins, we investigated whether CREB1 and repai

286 lguanine-DNA alkyltransferase (AGT) is a DNA repair protein which removes alkyl groups from the O-6 p

287 MSH2 is a key DNA mismatch repair protein, which plays an important role in genomic

288 MutL homolog 1 (MLH1) is a key DNA mismatch repair protein, which plays an important role in mainten

289 inase/phosphatase (PNKP), a DNA strand break repair protein with DNA 5'-kinase and DNA 3'-phosphatase

290 utilize DNA-modified electrodes to generate repair proteins with [4Fe4S] clusters in the 2+ and 3+ s

291 NA damage-regulated complex with the key DNA repair protein xeroderma pigmentosum A (XPA).

292 ty associated with the deficiency of the DNA repair protein xeroderma pigmentosum type A (XPA).

293 We show that loss of the DNA repair protein XPA markedly augments the synthetic letha

294 the rapid proteolytic degradation of the DNA repair protein XPB.

295 ome group B (CSB), but not the global genome repair protein XPC.

296 quitylome identified the nucleotide excision repair protein, XPC, as a critical mediator of the USP22

297 2 and is required for recruitment of the ICL repair protein, XPF, to damage-induced foci at telomeres

298 (+) levels suppressed recruitment of the DNA repair protein XRCC1 to sites of genomic DNA damage and

299 inhibition abolishes the recruitment of the repair proteins XRCC1 and polymerase beta at damaged tel

300 Here we show that the Fe-S cluster repair protein YtfE contributes to the survival of Yersi