ライフサイエンスコーパス: reperfusion

1 s mitochondrial structure and function after reperfusion.

2 of 5 days +/- 1.9 (standard deviation) after reperfusion.

3 e conditions, as well as during ischemia and reperfusion.

4 rom reactive oxygen species generated during reperfusion.

5 ve in preclinical stroke models of ischaemia-reperfusion.

6 GPX4 and ACSL4) expression alteration during reperfusion.

7 ntation, hearts were evaluated at 3 hours of reperfusion.

8 naling, focusing on the dynamics early after reperfusion.

9 point of BBB opening following 1 h tMCAO and reperfusion.

10 cally relevant time frame and independent of reperfusion.

11 (SMA) for 45 minutes followed by 120 minutes reperfusion.

12 cerebral artery occlusion followed by 28-day reperfusion.

13 des modest protection only in the context of reperfusion.

14 ken before, and immediately after, allograft reperfusion.

15 pore channel inhibitor Ned-19 at the time of reperfusion.

16 eriostin during the repair phase of ischemia reperfusion.

17 ice, right through to cold preservation and reperfusion.

18 king period) or the full 45-minute period of reperfusion.

19 than when administered orally shortly after reperfusion.

20 selective reduction of the ILC2 subset after reperfusion.

21 ut not in miR-34a(-/-) mice following stroke reperfusion.

22 lammatory mediators were assessed 24 h after reperfusion.

23 liver grafts in response to cold storage and reperfusion.

24 apies, typically focused on pulmonary artery reperfusion.

25 rior descending artery (10 min), followed by reperfusion (2 min), and arrhythmia incidence quantified

26 irectly after 45 minutes of ischemia without reperfusion (45I-0R), after 30 minutes of reperfusion (4

27 erfusion (45I-30R), and after 120 minutes of reperfusion (45I-120R), as well as a control sample not

28 ut reperfusion (45I-0R), after 30 minutes of reperfusion (45I-30R), and after 120 minutes of reperfus

29 significantly higher ILC1 frequencies before reperfusion, accompanied by elevated ILC2 frequencies af

30 sis was observed frequently after 6 hours of reperfusion, accompanied by upregulation of Rubicon, att

31 ive, 30-minute flow reductions and 30-minute reperfusions achieving complete occlusion after four cyc

32 in Cerebral Infarction (mTICI) grade 2b to 3 reperfusion (adjusted OR per 30 minutes increase in time

33 1 signaling to sensory neurons from ischemia/reperfusion-affected muscle directly modulated nocicepti

34 infarct cortex were evaluated at 24 hours of reperfusion after a 90-minute unilateral middle cerebral

35 n a rat model with acute myocardial ischemia-reperfusion (AMI/R) injury and myocardial necrosis, as w

36 t is also overexpressed after renal ischemia-reperfusion, an event that induces kidney injury and fib

37 ce and QKI-7 is knocked-down in vivo in ECs, reperfusion and blood flow recovery are markedly promote

38 aling is increased in a profibrotic ischemia reperfusion and cardiotoxin muscle injury model.

39 donor ILC subsets is altered after allograft reperfusion and is associated with PGD development, sugg

40 before and after cardiopulmonary bypass and reperfusion and left ventricular (LV) tissue from mice s

41 tive way to achieve fast first-pass complete reperfusion and should thus be used.

42 reter) were acquired 15 and 45 minutes after reperfusion and subsequently analyzed using specialized

43 tment to prevent muscle fibrosis in ischemia reperfusion and traumatic extremity injury.

44 sponse to nephrotoxins, sepsis, and ischemia/reperfusion, and in chronic kidney diseases.

45 of the therapy, and a second wave of oxygen reperfusion approximately 24 h thereafter.

46 nged UDCA treatment altered the incidence of reperfusion arrhythmias.

47 Here we used cerebral ischemia/reperfusion as a model to investigate the interaction be

48 onary alteplase infused early after coronary reperfusion associates with ischemic time.

49 s, chronic and acute kidney injury, ischemia/reperfusion, atherosclerosis, and inflammation, which ar

50 In this porcine ischemia-reperfusion autotransplant model, the left kidney of a +

51 ring continuous HMP in a pig kidney ischemia-reperfusion autotransplant model.

52 reduced oxidative stress with TV-P-supported reperfusion but not with IR or ECMO.

53 , probably less than 10% of patients require reperfusion by thrombolysis or interventional treatments

54 emic stroke has greatly advanced, with rapid reperfusion by use of intravenous thrombolysis and endov

55 ngs in response to cold ischemia and ex vivo reperfusion (CI/EVR).

56 tructure including cardiolipin content after reperfusion compared with IR or ECMO.

57 er percent reduction from peak lactate after reperfusion compared with vehicle controls.

58 by elevated ILC2 frequencies after allograft reperfusion.Conclusions: The composition of donor ILC su

59 anical function during baseline and ischemia-reperfusion conditions.

60 n of antibody-mediated diseases and ischemia-reperfusion conditions.

61 Ischemia-reperfusion decreased LVDP in all hearts with return of

62 ention does not achieve effective myocardial reperfusion due to the occurrence of coronary microvascu

63 rrent protocols to monitor and exclude brain reperfusion during in situ NRP.

64 hether they're still efficacious in the post-reperfusion era is currently debated in the light of rec

65 age (SCS), followed by a 2 h period of graft reperfusion ex vivo.

66 ) of 30 mmHg from t = 40-70 min, followed by reperfusion/fluid resuscitation until t = 300 min.

67 vein (PV) sample] and post-(liver flush; LF) reperfusion for their ability to activate a panel of PRR

68 ardiomyopathy were enrolled after undergoing reperfusion for their first AMI.

69 Furthermore, upon reperfusion, HBOC-201 treated kidneys had similar renal

70 Acute myocardial ischaemia and reperfusion (I-R) are major causes of ventricular arrhyt

71 As the incidence of ischemia-reperfusion (I-R) injury has substantially increased, th

72 e have previously demonstrated that ischemia/reperfusion (I/R) impairs endoplasmic reticulum (ER)-bas

73 In the present study, myocardial ischemia/reperfusion (I/R) induced autosis in CMs, as evidenced b

74 ave an impaired recovery from acute ischemia/reperfusion (I/R) injury ex vivo, the role of dysferlin

75 ng adverse outcomes associated with ischemia/reperfusion (I/R) injury in multiple disease models.

76 We used a glaucoma model of retinal ischemia-reperfusion (I/R) injury in rats and found that endothel

77 usceptibility of steatotic liver to ischemia-reperfusion (I/R) injury is due to impaired recruitment

78 ors of MMPs (TIMPs) plays a role in ischemia/reperfusion (I/R) injury post-myocardial infarction (MI)

79 Cerebral ischemia/reperfusion (I/R) injury was significantly improved in t

80 ifen to activate the reporter before sham or reperfusion (I/R) MI surgeries.

81 ocytes on myocardial function after ischemic-reperfusion (I/R) MI.

82 erized by glycocalyx derangement is ischemia-reperfusion (I/R) of the whole body as well as during se

83 The latter trigger a version of ischemia/reperfusion (I/R) pathobiology that is singular in its o

84 Here, using a model of hind limb ischemia-reperfusion (I/R) remote lung injury, we present evidenc

85 e progression of TIF caused by UUO, ischemic/reperfusion (I/R), aristolochic acid, and repeated acute

86 tes of oxidative stress such as ischemia and reperfusion (I/R), remains unexplored.

87 M1 expression increased after renal ischemia/reperfusion (I/R)-induced AKI in mouse kidneys.

88 Intestinal ischemia-reperfusion (IIR) often occurs during and following majo

89 ediate reperfusion (IR), LV unloading before reperfusion improves myocardial energy substrate use and

90 ular unloading limits ischemic injury before reperfusion, improves myocardial energy substrate use, a

91 oute that would aid faster tissue repair and reperfusion in "no-option" patients suffering from perip

92 s 87 minutes (IQR, 62-116), with substantial reperfusion in 85.9% (5433/6324) of patients.

93 n was significantly increased at 24 hours of reperfusion in all groups.

94 udy testing whether LV unloading and delayed reperfusion in patients with STEMI without cardiogenic s

95 To describe patterns of reperfusion in the superficial vascular plexus (SVP), de

96 nant polycystic kidney disease, and ischemia/reperfusion-induced acute kidney injury.

97 UDCA protects against ischaemia-induced and reperfusion-induced arrhythmias in the adult myocardium,

98 idence of mitochondrial injury, specifically reperfusion-induced reactive oxygen species generation a

99 linical porcine model, we performed ischemia-reperfusion injuries using balloon occlusion for 60 minu

100 EACAM1 signaling may affect hepatic ischemia-reperfusion injury (IRI) and OLT outcomes.

101 t also contributes significantly to ischemia reperfusion injury (IRI) associated with myocardial infa

102 Ischemia reperfusion injury (IRI) during liver-metastasis resecti

103 ic aneurysm (AAA) repair results in ischemia-reperfusion injury (IRI) in local (i.e. kidney) and dist

104 Ischemia-reperfusion injury (IRI) is a complex inflammatory proce

105 Ischemia-reperfusion injury (IRI) is an important risk factor for

106 Ischemia-reperfusion injury (IRI) is believed to contribute to gr

107 Here, using a murine renal ischemia/reperfusion injury (IRI) model, we show that intercalate

108 Here, genes related to ischemia-reperfusion injury (IRI) or graft rejection may be silen

109 lammatory responses associated with ischemia/reperfusion injury (IRI) play a central role in alloimmu

110 Ischemia reperfusion injury (IRI) predisposes to the formation of

111 is a major clinical concern during ischemia-reperfusion injury (IRI) triggered by traumatic events,

112 irected pharmacomodulation of renal ischemia-reperfusion injury (IRI) without the need for systemic d

113 released from cells damaged during ischemia-reperfusion injury (IRI), in heart attack or stroke sett

114 C57BL/6 mice challenged with renal ischemia reperfusion injury (IRI), treated before or after injury

115 This phenomenon is called ischemia reperfusion injury (IRI).

116 or organ preservation which reduces ischemia-reperfusion injury (IRI).

117 repairing kidneys on day two after ischemia reperfusion injury (IRI).

118 Myocardial ischemia-reperfusion injury (MIRI) is a serious threat to the hea

119 Lung ischemia-reperfusion injury after transplantation is associated w

120 ns in vascular inflammation such as ischemia-reperfusion injury and cardiovascular diseases.

121 e as pathogenically contributing to ischemia-reperfusion injury and delayed graft function (DGF) in h

122 ose of agrin is capable of reducing ischemia-reperfusion injury and improving heart function, demonst

123 f exercise on metabolic parameters, ischemia-reperfusion injury and regeneration after hepatectomy.

124 s steatosis; however, its impact on ischemia-reperfusion injury and regeneration is unknown.

125 nct donor macrophage populations in ischemia-reperfusion injury and rejection, including their intera

126 the important role of platelets in ischemia/reperfusion injury and SEC injury.

127 The patients in the low-PF group had severe reperfusion injury and were more frequently complicated

128 ted within a few hours of bilateral ischemia-reperfusion injury at these sites and new sites of proxi

129 ture and cardiomyocyte death during ischemia-reperfusion injury by inducing mitochondrial permeabilit

130 yocytes and partial protection from ischemia-reperfusion injury by reducing mitochondrial permeabilit

131 d2 ablation protected against renal ischemia-reperfusion injury by suppressing the expression of proi

132 Cold ischemia and reversible ischemia-reperfusion injury dampened antigen presentation by rena

133 In donor kidneys subjected to ischemia-reperfusion injury during kidney transplant, phagocytes

134 renal macrophages after reversible ischemia-reperfusion injury in a mouse model of congeneic renal t

135 ant HS oligosaccharides limit liver ischemia reperfusion injury in a mouse model.

136 bolism to possibly better cope with ischemia reperfusion injury in discarded kidneys.

137 therapeutic potential in preventing ischemia-reperfusion injury in heart transplant.

138 pivotal role in the pathogenesis of ischemia-reperfusion injury in solid organ transplantation.

139 mirococept (APT070) for preventing ischaemia-reperfusion injury in the kidney allograft (EMPIRIKAL) t

140 ied and compared renal responses to ischemia-reperfusion injury in the presence and absence of GDF15.

141 nd on ferroptosis-associated renal ischaemia-reperfusion injury in vivo.

142 Ischemia reperfusion injury induces a systemic increase of cf-mt-

143 ule 1 (CEACAM1) exhibited increased ischemia-reperfusion injury inflammation and decreased function i

144 Liver ischemia reperfusion injury is associated with coagulation and in

145 und After acute myocardial infarction (AMI), reperfusion injury is associated with microvascular lesi

146 Ischemia-reperfusion injury is inevitable during intestinal trans

147 A feasible therapy targeting reperfusion injury is remote ischemic conditioning (RIC)

148 g periostin expression in the renal ischemia-reperfusion injury model, and primary cultures of isolat

149 a human-disease-relevant myocardial ischemia reperfusion injury mouse model after i.v. injection conf

150 8)Ga-DOTA-ECL1i were compared in an ischemia-reperfusion injury mouse model.

151 or spinal cord injury, glaucoma and ischemia-reperfusion injury of the eye.

152 ge of AMI/R and necrosis in conjunction with reperfusion injury of the heart.

153 g the effects of transplant-induced ischemia-reperfusion injury on the ability of donor-derived resid

154 were protected from fibrosis after ischemia-reperfusion injury or unilateral ureteral obstruction de

155 subjected SerpinB2 knockout mice to ischemia-reperfusion injury or unilateral ureteral obstruction.

156 se after brain death (BD) and posttransplant reperfusion injury play significant roles in the pathoge

157 ry blood flow after a heart attack can cause reperfusion injury potentially leading to impaired cardi

158 eutic trials aimed at prevention of ischemia/reperfusion injury to allografts based on animal data sh

159 kidney fibrosis induced via UUO or ischemia/reperfusion injury was ameliorated by systemic genetic k

160 Myocardial ischemia-reperfusion injury was induced via left anterior descend

161 Ischemia-reperfusion injury was modeled in vitro by placing human

162 'deactive transition' (relevant to ischemia-reperfusion injury) and their effects on the ubiquinone-

163 uromuscular diseases of childhood, ischaemia-reperfusion injury, and age-related neurodegenerative di

164 an array of human diseases including asthma, reperfusion injury, and cancer.

165 sis for cell therapy in mice after ischaemia-reperfusion injury, and find that-although heart functio

166 rmal kidney and following bilateral ischemia-reperfusion injury, and quantified and compared renal re

167 the patients in the high-PF group had milder reperfusion injury, but had lower intraoperative hepatic

168 d the effect of LV unloading on ischemia and reperfusion injury, cardiac metabolism, and mitochondria

169 or renal macrophage functions after ischemia-reperfusion injury, crucial to guiding the phenotype and

170 the pathophysiology of myocardial ischaemia-reperfusion injury, including the role of autophagy and

171 otective effects against myocardial ischemic/reperfusion injury, indicating their potential applicati

172 At 33 days after ischemia/reperfusion injury, LNA-21-treated hearts exhibited redu

173 ic vascular complications, cardiac ischaemia-reperfusion injury, myocardial infarction, heart failure

174 After ischemia-reperfusion injury, periostin-overexpressing mice exhibi

175 that promotes recovery from anoxia, reduces reperfusion injury, prevents oedema, and metabolically s

176 Similarly, in a rat model of renal ischemia/reperfusion injury, SAR247799 preserved renal structure

177 en the central importance of mitochondria in reperfusion injury, we hypothesized that compared with i

178 endothelial HIF-2 exacerbated renal ischemia-reperfusion injury, whereas inactivation of endothelial

179 on of proregenerative factors after ischemia-reperfusion injury, whereas knockout mice exhibited the

180 ns have a higher risk of developing ischemia-reperfusion injury, which can lead to posttransplant com

181 ecting both nephrotoxin-induced and ischemia-reperfusion injury-induced AKI in live mice.

182 es such as chronic inflammation and ischemia-reperfusion injury.

183 tory properties that could minimize ischemia reperfusion injury.

184 utic potential in diseases, such as ischemic reperfusion injury.

185 myoblasts from an in vitro model of ischemic reperfusion injury.

186 nd improved tissue repair following ischemia-reperfusion injury.

187 mRNA and protein are induced after ischemia-reperfusion injury.

188 n of PHD2 in a mouse model of renal ischemia-reperfusion injury.

189 al fibrosis 28 days post-myocardial ischemia-reperfusion injury.

190 of mitochondrial recovery following ischemia-reperfusion injury.

191 ning in mice before and after renal ischemia-reperfusion injury.

192 reasing liver damage resulting from ischemia reperfusion injury.

193 troke infarct volume after cerebral ischemia-reperfusion injury.

194 eutrophil extravasation and ischemia-induced reperfusion injury.

195 ay better protect these organs from ischemia-reperfusion injury.

196 tophagy is important for mitigating ischemia/reperfusion injury.

197 odels and livers of mice undergoing ischemia/reperfusion injury.

198 phagy, mPTP opening, and death with ischemia/reperfusion injury.

199 rimental animal models of acute ischemia and reperfusion injury.

200 tive and inhibits cardiac allograft ischemia-reperfusion injury.

201 ne defense and highly vulnerable to ischemia-reperfusion injury.

202 nscriptional changes observed after ischemia-reperfusion injury.

203 al rejuvenation to the heart after ischaemia-reperfusion injury.

204 orted to have a detrimental role in ischemia-reperfusion injury.

205 Cerebral ischemia/reperfusion insult rapidly activates necroptosis to prom

206 ferences in responses to intestinal ischemia-reperfusion (IR) have been recognized in animal studies.

207 Lung ischemia-reperfusion (IR) injury is a common clinical pathology a

208 Ischemia reperfusion (IR) injury results in devastating skeletal

209 dependent mouse model of intestinal ischemia-reperfusion (IR) injury to investigate the underexplored

210 -17A play critical roles in hepatic ischemia reperfusion (IR) injury, we tested whether mice lacking

211 hicken embryo heart in response to ischaemia-reperfusion (IR) injury.

212 ich impacts the cardiac response to ischemia-reperfusion (IR) injury.

213 with subsequent restoration, termed ischemia reperfusion (IR), is a common cause of AKI.

214 we hypothesized that compared with immediate reperfusion (IR), LV unloading before reperfusion improv

215 enal tubular inflammation after ischemia and reperfusion (IR).

216 h the presence of CsA for the full 45-minute reperfusion is associated with impaired mitochondrial in

217 Reperfusion is considered as an optimal therapy followin

218 (LV) workload (LV unloading) before coronary reperfusion is emerging as a potential approach to reduc

219 New data demonstrate that ischemia/reperfusion is necessary and sufficient to induce CMV re

220 Reperfusion is the only existing strategy for patients w

221 ial infarction, following heart ischemia and reperfusion, is associated with profound changes in key

222 he loss of BBB integrity following ischaemic/reperfusion-like conditions was significantly worsened b

223 Portal blood immediately following allograft reperfusion (liver flush; LF) had increased total HMGB1,

224 significantly increases myocardial ischemia/reperfusion (MI/R) injury.

225 We used the renal ischemia-reperfusion model to investigate the role of SULT1E1 in

226 In rat ischemia-reperfusion model, an intramyocardial injection of Rg3-l

227 the middle carotid artery occlusion ischemia/reperfusion model.

228 so protective in renal and cerebral ischemia/reperfusion models, demonstrating its widespread utility

229 Substantial reperfusion (modified Thrombolysis in Cerebral Infarctio

230 the clinically relevant unilateral ischemia-reperfusion murine model of AKI at days 1, 2, 4, 7, 11,

231 In hearts subjected to ischemia-reperfusion, Nox4 limits infarct size through this mecha

232 In vivo, reperfusion of carotid artery thrombotic occlusion was a

233 The impact on coronary reperfusion of crushed P2Y(12) inhibitor tablets, which

234 The primary outcome was reperfusion of greater than 50% of the involved ischemic

235 %]) demonstrated localized areas of apparent reperfusion of nonperfused retina, more commonly in the

236 number of participants with greater than 50% reperfusion of the previously occluded vascular territor

237 min of warm ischemia time and either 4 h of reperfusion or 24 h of cold storage.

238 hanges in the percentage of ILC subsets with reperfusion or PGD (grade 3 within 72 h) were assessed.M

239 LT1E1 expression to bilateral renal ischemia-reperfusion or sham surgery, either in the absence or pr

240 .02) and NIR perfusion indices, 15 min after reperfusion (p < 0.01).

241 a Zeiss Plex Elite 9000 was used to evaluate reperfusion patterns within the SCP, DCP, and CC.

242 r 60 minutes followed by a 3-, 7-, or 28-day reperfusion period.

243 intestinal flow changes during the occlusion-reperfusion phases were accompanied by parallel changes

244 nd acute stress conditions, such as ischemia-reperfusion, phosphorylations are lost, leading to maxim

245 rial pneumonia promotes PGD through ischemia/reperfusion-primed donor TRAMs.

246 nous thrombolysis with tenecteplase improves reperfusion prior to endovascular thrombectomy for ische

247 plase did not significantly improve cerebral reperfusion prior to endovascular thrombectomy.

248 f harmful consequences of heart ischemia and reperfusion, re-establishing myocardial function.

249 SL111, 80 mg/kg) delivered immediately after reperfusion reduced the systemic and cardiac inflammator

250 mechanical function, or any benefit against reperfusion-related dysfunction.

251 Ischemia (hypoxia) and/or reperfusion (reoxygenation) induces various forms of cel

252 motor unit deactivation, and (2) blood flow reperfusion (REP) would result in muscle recovery and re

253 sed.Measurements and Main Results: Allograft reperfusion resulted in significantly decreased frequenc

254 In vitro-simulated ischemia/reperfusion showed that NGAL-deficient CD4(+) T cells ex

255 g use and success of interventional coronary reperfusion strategies, morbidity and mortality from acu

256 Despite significant advances in reperfusion strategies, mortality from cardiac rupture r

257 orary role of an evidence-based fibrinolytic reperfusion strategy as part of a pharmacoinvasive appro

258 ently diminished collateral flow and reduced reperfusion success.

259 e investigated the effect of ITx on PC after reperfusion (T0), during follow-up, and rejection.

260 and safety of nerinetide in human ischaemia-reperfusion that occurs with rapid endovascular thrombec

261 +/- 109 000 ng/L, P=0.023) at 6 hours after reperfusion, the levels of NT-proBNP (N-terminal pro-B-t

262 ls (RCTs), where all patients had to receive reperfusion therapies, either intravenous thrombolysis a

263 iched clinical studies of patients receiving reperfusion therapies, might prove more effective than i

264 Despite the success of reperfusion therapy in significantly reducing the extent

265 Strategies for reperfusion therapy include systemic fibrinolysis, surgi

266 Myocardial infarction patients receiving reperfusion therapy show magnetic susceptibility changes

267 he safety, efficacy, and treatment window of reperfusion therapy with t-PA by limiting hemorrhagic tr

268 ermia were also assessed in combination with reperfusion therapy, but in RCTs that only included feas

269 lammation in mice with or without concurrent reperfusion therapy.

270 ation acute ischaemic stroke, not undergoing reperfusion therapy.

271 facing the treatment of stroke in the era of reperfusion therapy: hemorrhagic transformation and the

272 tion was found between force and full finger reperfusion time (beta = -0.033 +/- 0.016; 95% CI, -0.06

273 temperature was associated with full finger reperfusion time (beta = -0.18 +/- 0.041; 95% CI, -0.26

274 clinician capillary refill time (full finger reperfusion time 1.14 s longer).

275 a consistent difference between full finger reperfusion time and clinician capillary refill time (fu

276 Correlation coefficient between full finger reperfusion time and clinician capillary refill time was

277 10 measurements, including five full finger reperfusion time and five clinician capillary refill tim

278 aster activation is associated with improved reperfusion time and outcomes in the American Heart Asso

279 d focus on the clinical value of full finger reperfusion time as a monitoring device for hemodynamics

280 Full finger reperfusion time demonstrated good reproducibility.

281 bility and validity of the novel full finger reperfusion time measurement using clinicians' visual ca

282 Full finger reperfusion time showed moderate correlation with clinic

283 Full finger reperfusion time was 1.14 seconds longer than capillary

284 core volume with age and expected imaging-to-reperfusion time will improve assessment of prognosis an

285 class correlation coefficient of full finger reperfusion time within each patient was 0.76 (95% CI, 0

286 analysis exists, referred to as full finger reperfusion time.

287 in door-out times (40 versus 68 minutes) and reperfusion times (98 versus 135 minutes) with 80.1% tre

288 fractionation, coupled early tumor ischemia/reperfusion to human cancer ablation.

289 rate experimental AKI triggered by ischaemia-reperfusion, toxic injury and systemic inflammation.

290 after stroke onset and might help to expand reperfusion treatment beyond the current time windows.

291 Ischemia-reperfusion triggered marked unilateral CX3CR1-CCR2 depe

292 ferroptotic death in different durations of reperfusion was evaluated by assessing the iron content,

293 The duration of LV unloading before reperfusion was inversely associated with infarct size i

294 d as the time from symptom onset to coronary reperfusion, was a pre-specified subgroup of interest.

295 Nevertheless, zones of apparent reperfusion were detected in some patients, and a dose r

296 implanted allografts during early stages of reperfusion while patient is hemodynamically supported o

297 earts with return of intrinsic activity, and reperfusion with 155 mM [Na(+)](o) further depressed mec

298 ated transplantation by ex vivo normothermic reperfusion with blood.

299 cardiac mechanical function during ischemia-reperfusion with perfusates containing 145 or 155 mM Na(

300 l infiltration to ischemic tissues following reperfusion worsens injury.