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1 he marked asymmetric polarity of the budding reproductive cell.
2 uding TOR and OSR2, may increase the size of reproductive cells.
3 in LHr-mediated gonococcal invasion of human reproductive cells.
4 d are critical for self-fertility in diploid reproductive cells.
5 or avoiding and repairing mutations in their reproductive cells.
6 unique process that allows the generation of reproductive cells.
7          Gcna gene expression is enriched in reproductive cells across eukarya - either just prior to
8 ll theory; understanding of the formation of reproductive cells and the process of fertilization expl
9 S1 mediates signals that control the fate of reproductive cells and their contiguous somatic cells.
10  reveal a two-way relationship between early reproductive cells and their helpers involving complex e
11           The molecular interactions between reproductive cells are critical for determining whether
12 ith this, we show that mutation rates in the reproductive cells are inversely correlated with generat
13 a few days old, whereas its gonidia (asexual reproductive cells) are nonmotile, specialized for growt
14 entric cell layers that surround and support reproductive cells as they progress through meiosis and
15  uncovering the processes that take place in reproductive cells buried deep in plants.
16                    In Volvox carteri adults, reproductive cells called gonidia are enclosed within a
17 ousand cells, but just two cell types: large reproductive cells called gonidia, and small, biflagella
18 matic cells and potentially immortal asexual reproductive cells called gonidia.
19 len grains) are generated in the anther from reproductive cells called microsporocytes.
20 ll division which generates haploid gametes (reproductive cells) carrying a single chromosome complem
21  Caenorhabditis elegans of radiation-induced reproductive cell death ("Radelegans") in isolation of a
22 romosomal instability (P < 0.1), and delayed reproductive cell death (the persistent reduction in pla
23 s of the biological end points of persistent reproductive cell death and apoptosis were consistent, s
24 g-held tenet that DNA damage is the cause of reproductive cell death and further validating this mode
25        These results suggest that persistent reproductive cell death can, in part, be explained by th
26      For example, after 9.5 Gy, which causes reproductive cell death in 99% of both types of cells, t
27 med that cell death in this model represents reproductive cell death in isolation from apoptotic cell
28 hway of EGFR is critical for protection from reproductive cell death in Radelegans.
29 eveloped a tissue model of radiation-induced reproductive cell death in the nematode Caenorhabditis e
30                                              Reproductive cell death is the primary mode of death in
31       Chromosomal instability and persistent reproductive cell death show a significant correlation a
32 re in a biological sample (e.g., DNA damage, reproductive cell death).
33 ing delayed HR showed no evidence of delayed reproductive cell death, and there was no correlation be
34 lones for sister chromatid exchange, delayed reproductive cell death, delayed mutation, mismatch repa
35 nse pathway is necessary for protection from reproductive cell death, supporting the long-held tenet
36 he EGFR signaling pathway in protection from reproductive cell death, the primary form of tumor stem
37 the possible role of apoptosis in persistent reproductive cell death, we analyzed subsets of chromoso
38 enetic pathways required for protection from reproductive cell death.
39  by UCN-01 do not necessarily correlate with reproductive cell death.
40 omosomal instability correlated with delayed reproductive cell death.
41 s undergo alternation of generation in which reproductive cells develop in the plant body ("sporophyt
42 ission, indicating TTI2's importance in male reproductive cell development.
43 ANT1) play a fundamental role in somatic and reproductive cell differentiation during early anther de
44 rue multicellularity, with plasmodesmata and reproductive cell differentiation, during the late Ordov
45 e functions in addition to their role during reproductive cell division.
46  across different tissues revealed that male reproductive cells express a phylogenetically younger tr
47  sterile mutants have defects in somatic and reproductive cell fate acquisition.
48 ein play key roles in regulating somatic and reproductive cell fate determination in Arabidopsis anth
49 study suggests that tissue geometry channels reproductive cell fate in the Arabidopsis ovule primordi
50 t one branch partitions the main body of the reproductive cell from the prosthecum and swarm cell.
51                                        Plant reproductive cells have traditionally been difficult to
52 uses abnormal differentiation of somatic and reproductive cells in anthers.
53 o obtain multigeneration cohorts, to collect reproductive cells in exposed parents, and to exclude ps
54 ial germ cells (PGCs) are the progenitors of reproductive cells in metazoans and are an important mod
55 n, polarity establishment and setting up the reproductive cell line can readily be recognized, they d
56 bolic changes in two different types of male reproductive cell lines.
57 n and the differentiation of PH-like asexual reproductive cells (metulae and phialides) are normal in
58 iophores with branching chains of reiterated reproductive cells (metulae), delayed conidial different
59                     This is possible because reproductive cells of these organisms are almost always
60 mosomes during the formation of each type of reproductive cell revealed that cells in asexual thalli
61 elf-fertility: these individuals can produce reproductive cells that are universally compatible, and
62 nduced enhanced gonococcal invasion of human reproductive cells that utilizes the lutropin receptor (
63 ed on the use of the early precursors of the reproductive cells, the primordial germ cells (PGCs).
64 e prosthecum, distal to the main body of the reproductive cell; thus, the chromosome must travel thro
65  size dimorphic-small worker cells and large reproductive cells-which forces the builders to join dif