ライフサイエンスコーパス: reprogram

1 eral adipogenic genes during transcriptional reprograming.

2 venile hormone (JH), a hormone elevated upon reprogramming.

3 g-term epigenetic, metabolic, and functional reprogramming.

4 ry layer underlying MET to control efficient reprogramming.

5 s and unveils mechanisms of phospholipidomic reprogramming.

6 hways, but did not induce T(RM) cell lineage reprogramming.

7 function of TET proteins during somatic cell reprogramming.

8 derstanding of the role of TETs in chromatin reprogramming.

9 and licences T cell receptor driven proteome reprogramming.

10 aspects and the limitation sides of the OSKM reprogramming.

11 ssential characteristics of cancer metabolic reprogramming.

12 ate Oct4 in fibroblasts, leading to enhanced reprogramming.

13 verted in response to injury or experimental reprogramming.

14 nabling tumour progression through metabolic reprogramming.

15 e tumours to targeted therapy via epigenetic reprogramming.

16 influence how NF-kappaB impacts Muller glia reprogramming.

17 al cells undergo substantial transcriptional reprogramming.

18 hanisms of hPGC specification and epigenetic reprogramming.

19 d increased chromatin accessibility later in reprogramming.

20 positioning, differentiation, and metabolic reprogramming.

21 fer into an oocyte, a process termed nuclear reprogramming.

22 8) is expressed on iPSCs and is important in reprogramming, (2) sGRP78 promotes cellular functions in

23 Somatic cell nuclear transfer (SCNT) can reprogram a somatic nucleus to a totipotent state.

24 s miR-9/9(*) and miR-124 (miR-9/9(*)-124) as reprogramming agents that orchestrate direct conversion

25 ssion of constitutive heterochromatin during reprogramming and a non-repressive role for H3K9me3.

26 KLF10 as a component of this transcriptional reprogramming and a novel hepato-protective factor.

27 BCL6, leading to pre-memory transcriptional reprogramming and cell-fate bias.

28 me reorganization, which leads to epigenetic reprogramming and derepression of developmentally silenc

29 he YAP/TEAD pathway, which drives epigenomic reprogramming and EMT to counteract apoptosis.

30 ion during DNA replication promotes germline reprogramming and epigenetic variation in plants propaga

31 Muller glia attractive targets for cellular reprogramming and highlighted the potential for curing d

32 In parallel, cellular reprogramming and organoid engineering are expanding the

33 cription factor (CoREST) is upregulated upon reprogramming and required for the epigenetic switch to

34 a valuable model for investigating cellular reprogramming and retinal regeneration.

35 ogic targeting of ILK reversed mitochondrial reprogramming and suppressed sEV-induced cell movements.

36 1A5 as an FTO target that promotes metabolic reprogramming and survival of VHL-deficient ccRCC cells.

37 on epigenetic trajectories, which implicates reprogramming and the pluripotency network as a central

38 human retinal epithelial cells leads to the reprogramming and transformation of these cells to neuro

39 ver of pancreatic cancer, promotes metabolic reprogramming and upregulates NRF2, a master regulator o

40 potential, DNA replication was incompletely reprogrammed, and genomic instability increased during r

41 modifications, codon-dependent translational reprogramming, and induction of the unfolded protein res

42 autophagy, generation of hiPSCs via cellular reprogramming, and neuronal differentiation.

43 the absence of individual enzymes can affect reprogramming, and thus the impact of mutations in cell

44 nvironments to promote cell infiltration and reprogramming are discussed.

45 vation leads to mitochondrial and glycolytic reprogramming are unknown.

46 nant of this process, establishing metabolic reprogramming as limiting for tumor initiation.

47 Indeed, near-complete DNA methylation reprogramming, as occurs during mammalian embryogenesis,

48 an important tool for inducing somatic cell reprogramming, as well as for dissecting the other biolo

49 Third, metabolic reprogramming assessed through real-time bioenergetic me

50 trate that rapid and extensive transcription reprogramming associated with hematopoietic differentiat

51 ar the most comprehensive dataset for kinome reprogramming associated with melanoma progression, whic

52 ccessibility profiling uncovering epigenetic reprogramming at >1400 sites in neurons after prolonged

53 l epigenetic reprogramming windows: maternal reprogramming at fertilization, embryonic stem cell (ESC

54 romatin remodelling is essential for natural reprogramming at fertilization.

55 b.D mutants, suggesting that transcriptional reprogramming at this time point is not required for imm

56 tion of multicellular regulatory networks by reprogramming autoantigen-experienced CD4+ T cells into

57 In summary, pioglitazone reprogrammed BM macrophages and suppressed OSM signaling

58 ellular metabolic, epigenetic and functional reprogramming, but how broadly trained immunity protects

59 ic acid metabolites and subject to metabolic reprogramming, but they have been neglected in N-ERD.

60 gnetic field can be digitally and repeatedly reprogrammed by a facile method of direct laser writing.

61 ivation and further promote cancer metabolic reprogramming by blocking the expression of the AKT inhi

62 nhibits mTOR activation and alters metabolic reprogramming by shifting glycolytic pathways and inhibi

63 Recent evidence shows that transient reprogramming can ameliorate age-associated hallmarks an

64 ministic components in the microRNA-mediated reprogramming cascade.

65 a new era of precision molecular medicine to reprogram cellular physiology.

66 regulates known drug resistance pathways to reprogram cellular responses to MEKi.

67 Accordingly, blocking the ability of IFN to reprogram cholesterol metabolism abrogates cellular prot

68 ment of H2A.Z patterns, providing a means to reprogram chromatin state and to hone gene expression le

69 recently showed that IL-1-induced glycolytic reprogramming contributes to allergic airway disease usi

70 This glycolytic reprogramming depends on Akt kinases, independent of mTO

71 that earlier errors in germ-cell epigenetic reprogramming derail differentiation in cellular progeny

72 study not only unveils genome-wide plant AS reprogramming during infection but also establishes a no

73 d targeted proteomic method to assess kinome reprogramming during melanoma metastasis in three pairs

74 that AS is a distinct layer of transcriptome reprogramming during plant-pathogen interactions.

75 t HIV reactivation, indicating an epigenetic reprogramming effect of ZL0580 on HIV long terminal repe

76 giogenesis, support invasion and metastasis, reprogram energy metabolism, induce genomic instability

77 t correlated with rapid DNA demethylation at reprogramming enhancers and increased chromatin accessib

78 the deposition of H3.3 are paramount during reprogramming events that drive zygotic activation and t

79 also expanded, revealing the crucial role of reprogramming factors and chromatin remodelling.

80 we show that transient expression of nuclear reprogramming factors, mediated by expression of mRNAs,

81 OSKM reprogramming were also observed in the reprogramming fibroblasts from a different individual.

82 ing evidence suggests that the failing heart reprograms fuel metabolism toward increased utilization

83 Metabolic reprogramming fulfils increased nutrient demands and reg

84 at prophase cohesin removal is a key step in reprogramming gene expression as cells transition from G

85 ts long after the initial exposure, but many reprogrammed genes remain transcriptionally silent with

86 n the dark, primed for rapid accumulation to reprogram growth upon light exposure.

87 patic MPhis and that HIF-2alpha subsequently reprograms hepatic MPhis to produce the hepatoprotective

88 ions include metabolic manipulation, partial reprogramming, heterochronic parabiosis, pharmaceutical

89 w, we discuss the consequences of failure to reprogram histone methylation during three crucial epige

90 auses metabolic dysfunction in adulthood and reprograms histone marks in the developing liver to acce

91 Bacille Calmette-Guerin (BCG) or beta-glucan reprograms HSCs in the bone marrow (BM) via a type II in

92 nstrate that, unlike BCG or beta-glucan, Mtb reprograms HSCs via an IFN-I response that suppresses my

93 signaling, which in turn regulates metabolic reprogramming, immune suppression, resistance to apoptos

94 -MDSC functionality and unveil strategies to reprogram immunosuppressive myelopoiesis in tumors to bo

95 H3K9me2 by inhibiting EHMT1/2 during nuclear reprogramming impacts the levels of H3K9me3, 5mC, and 5h

96 Cancer cells require extensive metabolic reprograming in order to provide the bioenergetics and m

97 Epigenetic reprogramming in Acute Myeloid Leukemia (AML) leads to t

98 The beneficial effects of OSK-induced reprogramming in axon regeneration and vision require th

99 In female mice, we observed DNA methylation reprogramming in bone marrow-derived (BMD) monocytes as

100 Metabolic reprogramming in cancer cells, vs. non-cancer cells, ele

101 ) family enzymes is essential for epigenetic reprogramming in cell state transitions.

102 hat acute administration of cocaine triggers reprogramming in circadian gene expression in the striat

103 etylase (HDAC) inhibitors elicited metabolic reprogramming in concert with disruption of several Warb

104 Metabolic reprogramming in endothelial cells (ECs) thereby is cruc

105 Repression of cellular reprogramming in germ cells is critical to maintaining c

106 sphorylation independent eIF4E translational reprogramming in governing the protein synthesis of ERal

107 provide a rationale for exploiting metabolic reprogramming in pancreatic cancer cells to confer thera

108 life cycle during which the genome undergoes reprogramming in preparation for embryogenesis.

109 are separable from the preceding epigenetic reprogramming in primordial germ cells.

110 ew studies have directly addressed metabolic reprogramming in the context of drug-resistant Mtb infec

111 y milieu in the breast as well as epigenetic reprogramming in the mammary gland, which can affect cel

112 Remarkably, transcriptional reprogramming includes a positive autoregulatory feedbac

113 Efferocytosis triggers cellular reprogramming, including the induction of mRNA transcrip

114 Thus, we hypothesize that the metabolic reprograming induced by Poldip2 deficiency controls VSMC

115 Overall, these data indicate that GH reprograms inflammatory macrophages to an anti-inflammat

116 pecified by the endothelium is essential for reprogramming interstitial macrophages and dampening inf

117 f zebrafish, Muller glia have the ability to reprogram into stem cells capable of regenerating all cl

118 ailable mesenchymal stromal cells (MSC) were reprogrammed into induced endothelial cells (iEC).

119 fied a key role for smooth muscle cell (SMC) reprogramming into a mesenchymal stem cell (MSC)-like st

120 This revealed that reprogramming into primed and naive pluripotency follows

121 on, we show that MEKi leads to CD8(+) T cell reprogramming into T(SCM) that acts as a reservoir for e

122 cells react to their environment by flexibly reprogramming intracellular metabolic pathways that subs

123 Metabolic reprogramming is a hallmark of malignancy.

124 Oncogene-induced metabolic reprogramming is a hallmark of pancreatic cancer (PDAC),

125 Reprogramming is accomplished through the interplay of t

126 PPARgamma reprogramming is altered in mice with cell-specific abla

127 Metabolism reprogramming is critical for both cancer progression an

128 Epigenetic reprogramming is involved in the response to heat acclim

129 Metabolic reprogramming is known to occur during the epithelial-me

130 te transport processes, indicating that host reprogramming is probably a common strategy in the NCLDV

131 Here we summarize how BCAA metabolic reprogramming is regulated in cancer cells and how it in

132 rm effects of EHS are mediated by epigenetic reprogramming is unknown.

133 CoV-2 must (1) enter a target/host cell, (2) reprogram it to ensure its replication, (3) exit the hos

134 ate intracellular parasite Toxoplasma gondii reprograms its metabolism.

135 and that induced by MYCN-mediated metabolic reprogramming, leads to synthetic lethality.

136 nduced metabolic regulator, is sufficient to reprogram lipid metabolism and drive progression of RAS

137 l enhancers were driven by AP-1 and EGR that reprogrammed LXR functions required for Kupffer cell ide

138 onclude that GPR35 activation by pamoic acid reprograms Ly-6C(Lo) monocytes/macrophages to relay a ne

139 istone fraction resisting post-fertilisation reprogramming marks developmental genes whose expression

140 y, omega-6 PUFA-enriched 'Western' diets can reprogram maternal bodily metabolism with maternal nutri

141 This suggests that epigenomic reprogramming may be a fundamental driver of glioma.

142 This transcriptional reprogramming may contribute to the beneficial effects o

143 y applicable strategy across tumor types for reprogramming MDSCs and TAMs into antitumorigenic immune

144 ogenitor cells (HSPCs) undergo translational reprogramming mediated by previously uncharacterized mec

145 t parental metabolic syndrome epigenetically reprograms members of the TGF-beta family, including neu

146 These data suggest that reprogramming metabolism via activation of PKM2 is a nov

147 e 1 (SnRK1) is a major metabolic sensor that reprograms metabolism upon carbon deprivation.

148 in cancer including angiogenesis, metabolic reprogramming, metastasis and DNA repair.

149 ferences in ASCL1 binding in progenitors and reprogrammed MG.

150 -1R-MyD88-IRAK2-PHB/OPA1 signaling axis that reprograms mitochondrial metabolism in adipocytes to exa

151 Elevated MYCN levels promote metabolic reprogramming, mitochondrial dysfunction, reactive-oxyge

152 atment limit interpretation, we believe that reprogramming monocyte or macrophages by targeting immun

153 nowledge to research efforts directed toward reprogramming Muller glia in mammals.

154 and MC38 colorectal carcinoma mouse models, reprogramming nanoparticles in combination with checkpoi

155 alyses confirmed that locally delivered tAPC-reprogramming nanoparticles led to a significant cell-me

156 ncer illustrates the importance of metabolic reprogramming, necessary to synthesize the building bloc

157 Sox8 and Sox9 prevents ovarian-to-testicular reprogramming observed in XX Rspo1 loss-of-function mice

158 d that both osmotic adjustment and proteomic reprogramming occurred in these naturally desiccation-to

159 Extensive metabolic reprogramming occurs during tumour initiation and progre

160 cribed (IVT) CAR or TCR mRNA for transiently reprograming of circulating T cells to recognize disease

161 Collectively, our findings reveal efficient reprograming of multiple IFN receptors toward a negative

162 Reprograming of proline metabolism is critical for tumor

163 f macrophage recruitment, depletion of TAMs, reprograming of TAMs, and blocking of the CD47-SIRPalpha

164 informed characterization, manipulation, and reprogramming of activated cellular ensembles.

165 e microenvironment drives specific metabolic reprogramming of adipose tissue macrophages, contributin

166 egulated by NF-kappaB activation, leading to reprogramming of cell metabolism towards glycolysis and

167 es a profound transcriptional and epigenetic reprogramming of cells.

168 lation at T114 is associated with a striking reprogramming of cellular metabolism induced by a profou

169 tic regulation of gene transcription and the reprogramming of cellular metabolism.

170 ects the core clock mechanism but leads to a reprogramming of circadian gene expression in the liver

171 gakaryocytes consistent with CPI203-mediated reprogramming of cord blood hematopoietic stem and proge

172 We performed a non-integrative reprogramming of dermal fibroblasts obtained from two pa

173 ced upon activation and suppresses metabolic reprogramming of effector T cells.

174 Oplr16 was also able to enhance reprogramming of fibroblasts into pluripotent cells.

175 Chemical reprogramming of fibroblasts offers an opportunity to re

176 n of the stress hormone abscisic acid (ABA), reprogramming of gene expression, and altering of growth

177 Dynamic reprogramming of gene regulatory networks (GRNs) enables

178 of hyaluronic acid, which is enabled by the reprogramming of glucose metabolism.

179 Reprogramming of glycogen metabolism has recently been s

180 s) and smooth muscle cells (iSMCs) by direct reprogramming of healthy human fibroblasts from donors o

181 impact of viruses, focusing on how metabolic reprogramming of host cells during lytic viral infection

182 D1 and NOD2 agonists induce early glycolytic reprogramming of human monocyte-derived macrophages (MDM

183 criptional level through distinct glycolytic reprogramming of human myeloid immune cells.

184 nfine cells into discrete islands during the reprogramming of human somatic cells from skin biopsies

185 Reprogramming of human somatic cells to induced pluripot

186 oadmap for the transcription-factor-mediated reprogramming of human somatic cells, indicate a role fo

187 s a RAS mutant tumor subset characterized by reprogramming of lipid metabolism, invasive and metastat

188 ic gonadotropin by TSC-derived ST reflects a reprogramming of macaque TSCs to an earlier pregnancy ph

189 r a proinflammatory metabolic and epigenetic reprogramming of macrophages in N-ERD.

190 Reprogramming of Majors upregulates Minor-biased genes a

191 growth and/or proliferation may require the reprogramming of metabolic pathways, whereby a switch fr

192 s of macrophage biology and demonstrate that reprogramming of metabolism should be considered a resul

193 ant metastatic sites and mediates epigenetic reprogramming of metastatic tumor cells.

194 Together, these findings show that while reprogramming of mitochondrial metabolism is a key facto

195 our current knowledge regarding spontaneous reprogramming of Muller glia in zebrafish and compares t

196 derstanding the mechanisms that suppress the reprogramming of Muller glia into neurogenic progenitors

197 itical regulator of inflammation, during the reprogramming of Muller glia into proliferating progenit

198 plasts, a process that involves a concurrent reprogramming of nuclear gene expression and plastid mor

199 We assess the impact of transcriptional reprogramming of nuclear-encoded chloroplast genes durin

200 romoted antiangiogenic and immunostimulatory reprogramming of Prox1-deficient tumors, destroyed tumor

201 ring this process, and facilitate the direct reprogramming of somatic cells into induced trophoblast

202 mate metabolism explained by transcriptional reprogramming of specific structural genes of the pathwa

203 muscle identity and requires transcriptional reprogramming of syncytial nuclei.

204 umor-initiating ETS translocations result in reprogramming of the AR cistrome.

205 t metabolic disruption was largely driven by reprogramming of the Early Growth Response 1 (EGR1) tran

206 n fewer lipid droplets, as well as metabolic reprogramming of the invasive cell phenotype, effectivel

207 factor receptor 2 (HER2) inhibitors involves reprogramming of the kinome through HER2/HER3 signaling

208 reatment with gemcitabine leads to extensive reprogramming of the pancreatic tumor microenvironment a

209 ies, thus obscuring the true transcriptional reprogramming of the phagocyte.

210 LPA and S1P, suggesting an LPA/S1P-mediated reprogramming of the TSC lipidome.

211 teomic screening, and identified substantial reprogramming of tumor metabolism involving oxidative ph

212 Metabolic reprogramming of tumor-specific T cells through enforced

213 Here, we show statistical reprogramming of two-dimensional (2D), noncompact self-a

214 This study depicted the reprogramming of WJ-MSC into rEC using unique transcript

215 Recent work has demonstrated that metabolic reprogramming, often mediated by inflammation, can lead

216 ication of primordial germ cells, epigenetic reprogramming or meiosis, and demonstrate that oocyte gr

217 This epigenomic reprogramming persists long after the initial exposure,

218 at down-regulate plant defense responses and reprogram plant metabolism for colonization and survival

219 ent for oxidation to 5fC/5caC can rescue the reprogramming potential of Tet2-deficient mouse embryoni

220 an embryonal carcinoma cell resulting from a reprogrammed primordial germ cell from the thymus.

221 and are also of paramount importance for the reprogramming process.

222 The molecular mechanism that underpins these reprogramming processes remains largely unexplored, whic

223 on: what are the translatome remodelers that reprogram protein output to activate biochemical adaptat

224 standing and limits rational improvements to reprogramming protocols.

225 nd the diverse differentiation potentials of reprogrammed PSCs.

226 brain energy homeostasis and that metabolic reprogramming regulates microglial polarization, thereby

227 Thus, this mechanism of translation reprogramming regulates the cellular response to metabol

228 Reprogrammed regulatory elements commissioned in metasta

229 In prostate cancer, the AR cistrome is reprogrammed relative to normal prostate epithelium and

230 ntify, evaluate, and enrich for iPSCs during reprogramming relies on methods that fix, destroy, or si

231 Injury-induced transcriptional reprogramming requires ATF3, a transcription factor that

232 gether, unbiased analysis of transcriptional reprogramming revealed a significant degree of negative

233 Genome-wide profiling during reprogramming reveals CoREST represses expression of enz

234 We found that after reprogramming SCAP-O or subclone SCAP-O(BCOR-mut) into i

235 the complex interactions between these plant-reprogramming sedentary endoparasites and their infected

236 ith the goal of identifying the persistently reprogrammed signaling pathways driven by chronic inflam

237 fic chromatin network is a critical event in reprogramming somatic cells into pluripotent status.

238 In this study, we embedded these partially reprogrammed spheroids in collagen-I matrices of varying

239 In this present study, we aimed to reprogram stem cells from a tooth apical papilla (SCAP)

240 ature amino acid change in PmxA was found to reprogram substrate specificity and was applied to predi

241 alysis of genomic loci bound by GATA3 during reprogramming suggests a correlation of recognition moti

242 exogenously applied calcium may specifically reprogram sweet cherry development and ripening physiogn

243 ment of curative interventions, for example, reprogramming T cell defects during the period of asympt

244 Reprogramming T cells to express a gain-of-function EZH2

245 c-MET inhibition causes profound metabolic reprogramming that can be targeted by drug combination t

246 ying efficiencies depending on the method of reprogramming that created them.

247 ne interleukin-13 in orchestrating metabolic reprogramming that drives adaptation to endurance exerci

248 he tissue-of-origin in shaping the metabolic reprogramming that enables tumour growth.

249 life span across species, leads to metabolic reprogramming that enhances utilisation of fatty acids f

250 by waves of transcriptional regulators that reprogram the enhancer landscape and change the wiring o

251 tially penetrated and surrounding cells, and reprogram the expression of immunity-associated genes by

252 ological module, however, can be designed to reprogram the host proteome, which in turn enhances the

253 argeting the CCL2/CCR2 signaling pathway may reprogram the immune angiogenic and microenvironments an

254 biosynthesis, how these metabolic activities reprogram the immunological state of target cells remain

255 ation in tumors, i.v. administered STING-NPs reprogram the TME towards a more immunogenic antitumor m

256 This allows them to locally reprogram the tumor microenvironment and activate antitu

257 our results indicate that treatment with KE reprogrammed the expression of genes involved in ketone

258 Peptide treatment reprogrammed the microbiome transcriptome, suppressed th

259 ributes in the cancer cell while dynamically reprogramming the function of innate and adaptive immune

260 otherapies, due in part to the difficulty of reprogramming the highly immunosuppressive tumor microen

261 an instructive role of tumor cell lineage in reprogramming the stromal microenvironment.

262 utic potential of targeting this pathway for reprogramming the TME.

263 cetylation blocks its nuclear translocation, reprograms the expression of immune-response-related gen

264 nstrate that adolescent cannabinoid exposure reprograms the initial behavioral, molecular, and epigen

265 tion of nuclear p50 NF-kappaB epigenetically reprograms the response of monocytic cells to IFNgamma t

266 Mechanistically, TIMP1 loss reprograms the senescence-associated secretory phenotype

267 When exposed to drought stress many plants reprogram their gene expression to activate adaptive bio

268 lls, we uncover how soil Pseudomonas species reprogram their metabolic pathways to prioritize siderop

269 overy and development of drugs for metabolic reprogramming, their associated challenges, and their ut

270 n-Barr virus (EBV) infects human B cells and reprograms them to allow virus replication and persisten

271 n mammals, paternal chromatin is extensively reprogrammed through the global erasure of DNA methylati

272 ease the uptake of glucose and metabolically reprogram to favor aerobic glycolysis over oxidative pho

273 ith constitutively high mTORC1 activity were reprogramed to be hyperactive in glucose and lipid metab

274 out mouse embryonic fibroblasts (MEFs) after reprograming to adipocytes.

275 We determined that translation is reprogrammed to enhance RNA metabolism and chromatin reg

276 defend the colony, but can be epigenetically reprogrammed to forage for food analogously to "Minor" w

277 d unlock metabolically restricted epigenetic reprogramming to drive metabolic dysfunction.

278 ire alpha-ketoglutarate-dependent epigenetic reprogramming to elicit anti-inflammatory gene expressio

279 vours mitochondrial biogenesis and metabolic reprogramming to ensure energy production and cellular a

280 We harnessed the power of reprogramming to examine GRP78, a chaperone protein gene

281 mice, revealing the sufficiency of metabolic reprogramming to influence immune cell functions during

282 Here, we utilize reprogramming to investigate natural behavioral specific

283 s important both for lineage commitment, and reprogramming to naive pluripotency.

284 ested a strategy of pharmacologic macrophage reprogramming to rescue HSPC mobilization.

285 e through oncogenic signalling and metabolic reprogramming to support cell survival and proliferation

286 cognate receptors, T cells undergo metabolic reprogramming to support their activation, differentiati

287 cer subtype, as most epithelial cells can be reprogrammed toward diverse lung cancer fates when expos

288 anism by which tumour-associated neurons are reprogrammed towards an adrenergic phenotype that can st

289 Currently, the molecular mechanisms by which reprogramming transcription factors and miRNAs coordinat

290 nt to a dedifferentiation mechanism by which reprogramming transcription factors Oct4 and Sox2 drive

291 diac crest, and demonstrate their ability to reprogram trunk into cardiac crest.

292 The results also suggest that reprogramming tumor cells toward non-pathological fates

293 d to deoxycytidine to deoxyuridine metabolic reprogramming upon ER stress induction.

294 Notably, this metabolic reprogramming was also associated with an accumulation o

295 This metabolic reprogramming was required to generate a number of proin

296 ate transcriptional responses of TFs to OSKM reprogramming were also observed in the reprogramming fi

297 ts in compromised development and epigenetic reprogramming, which demonstrates that heterochromatin r

298 last rejuvenation through laterally confined reprogramming, which has important implications in regen

299 methylation during three crucial epigenetic reprogramming windows: maternal reprogramming at fertili

300 lymphoid composition, and underwent partial reprogramming with acquisition of CD68/CD206 on MNPs and