ライフサイエンスコーパス: respiratory control

1 licits ventilatory LTF (reflecting automatic respiratory control).

2 reflect different physiologic influences on respiratory control.

3 orm to diagnose alterations in mitochondrial respiratory control.

4 dullary nucleus of the brain responsible for respiratory control.

5 nters, and thus they serve as a key nexus of respiratory control.

6 ystematically study sleep-related changes in respiratory control.

7 dulate many vital brain functions, including respiratory control.

8 tion that leads to appropriate modulation of respiratory control.

9 neural function, focusing on NG neurons and respiratory control.

10 are associated with marked changes in cardio-respiratory control.

11 es, and in the carotid body it is crucial to respiratory control.

12 hondrial bioenergetics, Ca(2+) dynamics, and respiratory control.

13 l outer membrane permeability contributes to respiratory control.

14 or (ANT), which provides a possible site for respiratory control.

15 ly behavioural to predominantly chemosensory respiratory control.

16 le electrical circuit model of mitochondrial respiratory control.

17 e phase of the circadian cycle in studies of respiratory control.

18 ured (ELISA) in three regions of interest to respiratory control: (1) ventral cervical spinal segment

19 In this study, we showed that ArcA (aerobic respiratory control), a global regulator important for E

20 have a relatively high incidence of central respiratory control abnormalities.

21 is sufficient to disrupt the development of respiratory control and augment the occurrence of apneas

22 rther the role of ATP-mediated signalling in respiratory control and central chemoreception by charac

23 Until recently, the respiratory control and function of intrinsic tongue mus

24 ATP is involved in central respiratory control and may mediate changes in the activ

25 Preterm infants have immature respiratory control and resulting intermittent hypoxia (

26 nic neurotransmission (reflecting volitional respiratory control); and (2) elicits ventilatory LTF (r

27 ways mediating physiologic behaviors such as respiratory control, and discuss how electrophysiologica

28 Additionally, cardiac arrhythmia, respiratory control, and epilepsy genes were screened fo

29 leep apnoea in adults, but the mechanisms of respiratory control are not clearly understood.

30 Respiratory control as well as modern RT techniques and

31 fects that include CA neurones have abnormal respiratory control at birth.

32 ults show loss of Scn1a function can disrupt respiratory control at the cellular and whole animal lev

33 ue CO(2)/H(+) and function as a key locus of respiratory control by integrating information from seve

34 gical studies of the brainstem pre-Botzinger respiratory control center demonstrated an abnormal rhyt

35 ough currently not generally recognized as a respiratory control center, the cerebellum is well known

36 that Mecp2 is critical within autonomic and respiratory control centers for survival.

37 +) chemoreflex, suggesting plasticity within respiratory control centers.

38 of hypoventilation by stimulation of central respiratory control centers.

39 r) were assessed by comparisons with various respiratory control conditions.

40 d be expected to inhibit the many aspects of respiratory control dependent on 5-HT, including baselin

41 se during gestation is sufficient to disrupt respiratory control development and promote pathological

42 Respiratory control disorders such as apnea of prematuri

43 ial therapeutic targets for the treatment of respiratory control disorders.

44 ful therapeutic targets for the treatment of respiratory control disorders.

45 ify therapeutic targets for the treatment of respiratory control disorders.

46 the physiological mechanisms responsible for respiratory control during hypoxia at altitude, by linki

47 We examined the role of respiratory control during O2-induced hypercarbia in pat

48 al characteristics and carotid body-mediated respiratory control during sleep with EMG (EMG+) or with

49 the development of brain regions underlying respiratory control functions.

50 ed in structures important for autonomic and respiratory control, functions that are severely affecte

51 However, its influence upon respiratory control has hardly been studied.

52 re little influenced by central chemosensory respiratory control in awake humans even when at rest un

53 e of circadian variations in respiration and respiratory control in awake humans for the first time u

54 These results provide unique insights into respiratory control in awake humans, and highlight the i

55 tributions to the literature on disorders of respiratory control in infancy and childhood are reviewe

56 ntal nicotine exposure (DNE) impacts central respiratory control in neonates born to smoking mothers.

57 ingle bout of neonatal inflammation on adult respiratory control in the form of respiratory motor pla

58 urons from many brainstem nuclei involved in respiratory control increase their firing rate in respon

59 Respiratory control index values on rat liver mitochondr

60 l respiratory function (postischemic percent respiratory control index; NAD(+)-linked: 81.3+/-3.8 ver

61 Postischemic mitochondrial respiratory control indices (RCIs) were significantly be

62 Current theory of respiratory control invokes a role of myoglobin (Mb)-fac

63 The mechanism by which CCHS impact respiratory control is incompletely understood, and even

64 he maturational shift away from ADP-mediated respiratory control is regulated by thyroid hormone in v

65 erm exposure to hypoxia generates changes in respiratory control known as ventilatory acclimatization

66 nd lateral mesencephalic reticular nucleus), respiratory control (lateral nucleus of the solitary tra

67 s include small upper airway lumen, unstable respiratory control, low arousal threshold, small lung v

68 Since ageing influences respiratory control mechanisms and serotonergic function

69 d hypercarbia does not indicate a failure of respiratory control mechanisms in the maintenance of PaC

70 critical for proper development of medullary respiratory control mechanisms.

71 but thought to involve depression of central respiratory control mechanisms.

72 gical functions, such as motor coordination, respiratory control, muscle tone and pain processing.

73 Thus, the respiratory control network is vulnerable to early-life

74 In addition, the respiratory control network of the brain has to organize

75 sult of disruptions at multiple sites of the respiratory control network, including sensory and motor

76 rocessing within canonical resting state and respiratory control networks (RCNs).

77 ro-inflammatory cytokines in their brainstem respiratory control nuclei, exhibit a higher respiratory

78 eparate brainstem pathways for syringeal and respiratory control of song production, both can affect

79 istent with a functional role for ENK in the respiratory control of the tongue.

80 , not only in brain areas closely related to respiratory control or olfactory coding but also in area

81 his period is a critical window during which respiratory control or regulation may be distinctly diff

82 d to be either unique to neurons involved in respiratory control, or at least very unusual for non-re

83 l area (pFL) is a crucial region involved in respiratory control, particularly in generating active e

84 To test the hypothesis that postmetamorphic respiratory control phenotypes arise through permanent d

85 ial oxidative phosphorylation ( P = 0.0008), respiratory control ratio ( P = 0.04), and coupling effi

86 reased State 4 respiration and decreased the respiratory control ratio (RCR) at much lower concentrat

87 ons (1-4%) and high degree of functionality (respiratory control ratio = 5-6).

88 ol-fed mice showed a significant decrease in respiratory control ratio and an increased sensitivity t

89 inhibition causes a dramatic increase in the respiratory control ratio from 6 to 40 for wild-type oxi

90 The respiratory control ratio of mitochondria from liver of

91 The respiratory control ratio of synaptic and nonsynaptic mi

92 nificant decrease in state 3 respiration and respiratory control ratio that was accompanied by an inc

93 ange of NADH levels, respiratory fluxes, and respiratory control ratio upon transitions elicited by s

94 cin was 13% lower, whereas the mitochondrial respiratory control ratio was 1.7-fold higher (both P <

95 A significant decrease in the respiratory control ratio was observed in mitochondria i

96 euronal counts, HVR, and brain mitochondrial respiratory control ratio were significantly reduced fol

97 ondrial efficiency as evidenced by increased respiratory control ratio, elevated cytochrome-c oxidase

98 , demonstrated by reduction of state III and respiratory control ratio, increased production of react

99 Despite no change in the respiratory control ratio, substrate control ratios of G

100 the outer membrane and uncorrelated with the respiratory control ratio.

101 3 respirations, and diminished mitochondrial respiratory control ratio.

102 a decrease in muscle oxidative capacity and respiratory control ratio.

103 rity of the outer mitochondrial membrane and respiratory control ratio.

104 Isolated mitochondria present increased respiratory control ratios (a measure of oxidative phosp

105 Respiratory control ratios (RCRs) of GGT(-/-) mice liver

106 deficient rats had lower liver mitochondrial respiratory control ratios and increased levels of oxida

107 Lowered respiratory control ratios were found in daily high-iron

108 the maintenance of NAD+/NADH-, ADP/ATP-, and respiratory control ratios.

109 sults, the very significant reduction in the respiratory control ratios.

110 l abnormalities and normalized mitochondrial respiratory control, reflecting protection against inner

111 the analytical scope to study mitochondrial respiratory control relative to specific tissue/cell typ

112 equent hypoxic episodes due to immaturity of respiratory control resulting in disturbances of cortica

113 in the subsequent cycles; and (c) models of respiratory control should depict a recurrent inhibitory

114 uscle responsiveness, arousal threshold, and respiratory control stability; loop gain) contributions

115 ith similar state 3 and 4 respiratory rates, respiratory control (state 3/state 4), and ADP/O ratios.

116 eptin in the hindbrain areas involved in the respiratory control such as the nucleus of the solitary

117 lasticity for therapeutic advantage when the respiratory control system is compromised (e.g., sleep a

118 arrest secondary to paralysis of the central respiratory control system or due to paralysis of the re

119 We present the view that the respiratory control system uses these sources of informa

120 nstrated considerable neuroplasticity in the respiratory control system, few studies have explored th

121 em controlled by feedback loops, such as the respiratory control system.

122 t variable of complicated cardiovascular and respiratory control systems.

123 have abnormalities in autonomic function and respiratory control that may contribute to premature let

124 which serve an important integrative role in respiratory control; the increased drive provided by enh

125 All three dyes suppressed mitochondrial respiratory control to some extent.

126 ed the "pure" effect of sleep deprivation on respiratory control under strictly controlled behavioral

127 is intrinsically linked to post-inspiratory respiratory control using the unanaesthetized working he

128 tion, and complement activity, whereas lower respiratory control was associated with Fc-mediated effe

129 Upper respiratory control was associated with virus-specific I

130 this region was not known to be involved in respiratory control, we combined chemical microstimulati

131 7) for one dose; estimates were similar when respiratory controls were used as the control group.

132 e sequelae entrained by disturbance of basic respiratory control whereby a process of which we are no

133 etermine if hypothalamic neurons involved in respiratory control, which were identified in cats by th