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1 reduced neuromuscular strength, dystonia and resting tremor.
2  including gait abnormalities, rigidity, and resting tremor.
3           Usp14-deficient axJ mice display a resting tremor, a reduction in muscle mass, and notable
4 symptoms of Parkinson's disease (PD) include resting tremor, akinesia, bradykinesia, and rigidity, an
5  can achieve almost 50% average reduction in resting tremor amplitude and in so doing form the basis
6           Moreover, severe motor impairment, resting tremor and abnormal gait and posture, phenotypes
7 ensors to continuously track fluctuations in resting tremor and dyskinesia.
8  Two mutant mice were identified with marked resting tremor and further characterized using the SHIRP
9 t deficits in WT alphaS mice and the PD-like resting tremor and progressive motor decline of 3K alpha
10  gait deficits in WT aS mice and the PD-like resting tremor and progressive motor decline of 3K aS mi
11 frequency of perinatal mortality, scoliosis, resting tremors and ptosis.
12 ve parkinsonism with bradykinesia, rigidity, resting tremor, and impaired postural reflexes was seen
13 , these animals have severe limb weakness, a resting tremor, and notable neuroanatomical malformation
14  is characterized by rigidity, bradykinesia, resting tremor, and postural instability.
15 r symptoms including bradykinesia, rigidity, resting tremor, and postural instability.
16 n of clinical symptoms such as bradykinesia, resting tremor, and rigidity.
17 , decreased overall activity, acquisition of resting tremors, and increased susceptibility to pentile
18 Egr3-/- mice develop gait ataxia, scoliosis, resting tremors, and ptosis, suggesting a defect in prop
19  disease include motor impairments involving resting tremor, bradykinesia, postural instability, gait
20 ilability is associated with the severity of resting tremor but not non-motor symptoms.
21                         In a rodent model of resting tremor, CVN417 attenuated this behavioral phenot
22                     P0-GGFbeta3 mice develop resting tremors, gait abnormalities, decreased hindlimb
23                                  Parkinson's resting tremor has been linked to pathophysiological cha
24 e GPe-STN loop, which may be relevant to the resting tremor in Parkinson's disease.
25 p-brain stimulation of the STN can alleviate resting tremor in Parkinson's disease.
26 characterized by bradykinesia, rigidity, and resting tremor, is the most common neurodegenerative mov
27 tion of postural and kinetic components with resting tremors less frequently seen.
28 tive disorder characterized by bradykinesia, resting tremor, muscular rigidity, and postural instabil
29    Physiological evidence indicates that the resting tremor of Parkinson's disease originates in osci
30  compared to akinetic-rigid patients with no resting tremor (P < 0.05).
31  falls than patients with Lewy body disease; resting tremor, pill-rolling tremor and hallucinations w
32 nical features of bradykinesia, rigidity and resting tremor resulting from the deficiency of dopamine
33 ve disease with movement disorders including resting tremor, rigidity, bradykinesia and postural inst
34 ) is the most common movement disorder, with resting tremor, rigidity, bradykinesia and postural inst
35                       It is characterized by resting tremor, rigidity, bradykinesia or slowness, gait
36 eurodegenerative disorders, characterized by resting tremor, rigidity, bradykinesia, and postural ins
37 minating in severe motor symptoms, including resting tremor, rigidity, bradykinesia, and postural ins
38                Furthermore, patients exhibit resting tremor, unstable gait, and impaired balance, whi