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1  greater risk of constipation, insomnia, and Restless Legs.
2 hat has been implicated as a risk factor for restless legs display important features of the syndrome
3 reathlessness, feeling faint, dizziness, and restless legs, especially among men [for all listed symp
4                                              Restless legs patients complain about sensory and motor
5 gand binding and RLS severity (international restless legs scale, IRLS) in areas serving the medial p
6 ility, depression, difficulty concentrating, restless legs syndrome (32%-40%), pica (40%-50%), dyspne
7  depression (g = 1.10, 95% CI [1.06, 1.15]), restless legs syndrome (g = 1.41, 95% CI [1.25, 1.56]),
8 e were performed on patients with idiopathic restless legs syndrome (iRLS) who were not currently on
9 ted odds ratio =1.26; 95% CI = 1.02-1.54) or restless legs syndrome (n = 108, adjusted odds ratio = 1
10 med as the strongest genetic risk factor for restless legs syndrome (odds ratio 1.92, 95% CI 1.85-1.9
11  (odds ratio [OR], 4.49; 95% CI, 3.98-5.06), restless legs syndrome (OR, 3.73; 95% CI, 3.39-4.09), bi
12    In addition, there were associations with restless legs syndrome (OR, 4.19; 95% CI, 3.91-4.50), sl
13                                              Restless legs syndrome (RLS) affects up to 10% of older
14 ies suggested a positive association between restless legs syndrome (RLS) and coronary heart disease
15     Most research on the association between restless legs syndrome (RLS) and depression has involved
16                                              Restless legs syndrome (RLS) and periodic limb movements
17 n the urge to move and sensory discomfort of restless legs syndrome (RLS) and properties of melanocor
18 minergic medications relieve symptoms of the restless legs syndrome (RLS) but have the potential to c
19                                              Restless legs syndrome (RLS) is a CNS disorder involving
20                                              Restless legs syndrome (RLS) is a common neurologic cond
21                                              Restless legs syndrome (RLS) is a common neurological di
22                                              Restless legs syndrome (RLS) is a common neurological di
23                                              Restless legs syndrome (RLS) is a common sensorimotor di
24                                     Although restless legs syndrome (RLS) is a disorder recognized in
25                                              Restless legs syndrome (RLS) is a frequent sleep-related
26                                              Restless legs syndrome (RLS) is a neurological condition
27                                              Restless legs syndrome (RLS) is responsive to opioid, do
28 revious cross-sectional study, we found that restless legs syndrome (RLS) was associated with erectil
29 isual perceptive computing (19 patients with restless legs syndrome (RLS), 21 patients with obstructi
30                                              Restless legs syndrome (RLS), also known as Willis-Ekbom
31                                              Restless Legs Syndrome (RLS), first chronicled by Willis
32 spinal cord is implicated in the etiology of Restless Legs Syndrome (RLS), which is more prevalent in
33 1 dopaminergic systems in the development of restless legs syndrome (RLS)-like movements during sleep
34 on deficiency (BID), a pathogenetic model of restless legs syndrome (RLS).
35 uding 46 drug-naive patients with idiopathic restless legs syndrome (RLS).
36 ief from dysaesthesias and motor symptoms in restless legs syndrome (RLS).
37 ales (p = 0.007), had a higher proportion of restless legs syndrome (RLS; p < 0.001), had a higher bo
38 or to natural menopause had a higher risk of restless legs syndrome after the index date compared wit
39 oophorectomy, there was an increased risk of restless legs syndrome among women without a benign ovar
40 ls were isolated from the motor cortex of 11 restless legs syndrome and 14 control brains obtained at
41 entified and replicated 13 new risk loci for restless legs syndrome and confirmed the previously iden
42  small effects have been identified for both restless legs syndrome and narcolepsy with cataplexy.
43 and studied the genetic correlations between restless legs syndrome and traits of interest.
44  clinical characteristics of childhood-onset restless legs syndrome are described.
45 r, a significant percentage of patients with restless legs syndrome are responsive to intravenous iro
46                The responsible mechanisms of restless legs syndrome are still not known, although cur
47  [21.0%] vs 135 women [8.2%]; P < .001), and restless legs syndrome before the index date (32 women [
48  loss of iron regulatory protein activity in restless legs syndrome brain tissue further implicates t
49 derlying cause of the iron deficiency in the restless legs syndrome brain.
50 erlying the decreased iron concentrations in restless legs syndrome brains is unknown.
51 f relative brain iron deficiency reported in restless legs syndrome brains may underlie the problems
52 romelanin cells from the substantia nigra of restless legs syndrome brains.
53 ns in the iron management protein profile in restless legs syndrome compared with controls at the sit
54 on in the brain is lower in individuals with restless legs syndrome compared with neurologically norm
55 ng of the molecular mechanisms that underlie restless legs syndrome could lead to new treatment optio
56                                              Restless legs syndrome is a common neurologic disorder t
57                                              Restless legs syndrome is a curious neurological disorde
58                                              Restless legs syndrome is a frequent neurological disord
59                                              Restless legs syndrome is a neurological disorder charac
60                                              Restless legs syndrome is a prevalent chronic neurologic
61                                              Restless legs syndrome is a sensorimotor network disorde
62 EAP4, VAV3) resided in the vicinity of known restless legs syndrome loci, whereas 5 (BBS7, CADM1, CRE
63         This cohort study found that risk of restless legs syndrome was increased among women who und
64                          A family history of restless legs syndrome was present in 23 of 32 (72%) sub
65 in and its receptor in the microvessels from restless legs syndrome was significantly decreased compa
66  controls and 14 individuals who had primary restless legs syndrome was subjected to histochemical st
67 mptoms of iron deficiency (fatigue, pica, or restless legs syndrome) and should be considered for tho
68 (parkinsonism, dystonia, tremor, chorea, and restless legs syndrome) were included.
69 found between frequent insomnia symptoms and restless legs syndrome, aging, and cardiometabolic, beha
70                         We conclude that, in restless legs syndrome, an increased HCN current in moto
71 on's disease, dystonia, Tourette's syndrome, restless legs syndrome, and akathisia, have traditionall
72 nized conditions (motor stereotypy disorder, restless legs syndrome, and infantile masturbation) as w
73 rcadian disorders, obstructive sleep apnoea, restless legs syndrome, and rapid eye movement (REM) sle
74 onia, chorea, tics, myoclonus, stereotypies, restless legs syndrome, and various other disorders with
75                                 Diagnosis of restless legs syndrome, as defined using Diagnostic and
76 id eye movement sleep behavior disorder, and restless legs syndrome, as well as circadian disorders,
77   This issue provides a clinical overview of restless legs syndrome, focusing on diagnosis, treatment
78  medical condition, obstructive sleep apnea, restless legs syndrome, idiopathic insomnia, and circadi
79                                          For restless legs syndrome, implicated variants are typicall
80 navian cohorts and on other sleep disorders (restless legs syndrome, insomnia) and sleep traits (dura
81 xt of another primary sleep disorder such as restless legs syndrome, or secondary to another underlyi
82  value of sex, daytime somnolence, insomnia, restless legs syndrome, sleep apnoea, urinary dysfunctio
83 f sleep are imperfect and may be affected by restless legs syndrome, these findings provide new biolo
84 including central sleep apnea, insomnia, and restless legs syndrome-in AF pathogenesis and progressio
85 N1) have not previously been associated with restless legs syndrome.
86 riants reveals putatively causative genes in restless legs syndrome.
87 burden of low-frequency and rare variants in restless legs syndrome.
88 ms associated with brain iron acquisition in restless legs syndrome.
89 n brain iron acquisition in individuals with restless legs syndrome.
90 in the epithelial cells of choroid plexus in restless legs syndrome.
91 or were upregulated in the choroid plexus in restless legs syndrome.
92 cept for variants previously associated with restless legs syndrome.
93 sleep disorders center received diagnoses of restless legs syndrome.
94 eurons contributes to the pathophysiology of restless legs syndrome.
95 istory are characteristic of childhood-onset restless legs syndrome.
96 the activity of this protein is decreased in restless legs syndrome; a finding similar to our earlier
97  particular circadian disorders, narcolepsy, restless-legs syndrome, and OSAS.