ライフサイエンスコーパス: reticular

1 ) and both neocortex and brainstem ascending reticular activating system (ARAS) nuclei.

2 ctivating structures including the ascending reticular activating system (ARAS), contributing to neoc

3 Multiple areas within the reticular activating system (RAS) can hasten awakening f

4 insults to the hypothalamus, brain stem, and reticular activating system are some of the proposed the

5 ive regions, with its peak in the brain stem reticular activating system.

6 bservations that behavioral state influences reticular activity.

7 ith OLP from saliva samples of two subtypes (reticular and erosive) of OLP patients and healthy contr

8 say enables the investigation of the role of reticular and extracellular Ca(2+) pools in the regulati

9 lexes at discrete plasmalemmal, sarcoplasmic reticular and myofilament sites, reveals differential ki

10 opodia to spread along the cell base and are reticular and/or fibrous in character.

11 such as anterior and ventromedial, midline, reticular, and posterior thalamic nuclei were also activ

12 tunity to explore its application in leading reticular architectures.

13 ways compartment: epithelium (basal region), reticular basement membrane (Rbm) and underlying lamina

14 ter RAC with regard to epithelial integrity, reticular basement membrane thickness, glandular area, e

15 modeling using image analysis, together with reticular basement membrane thickness, mucus gland area,

16 In situ, the papillary reticular boundary was indistinguishable in the young sc

17 Six episodes of reticular bullous epithelial corneal edema were identifi

18 In all cases, the reticular bullous epithelial edema improved or resolved

19 d natural history of a particular pattern of reticular bullous epithelial edema in a series of patien

20 investigate the respective contributions of reticular Ca(2+) and extracellular Ca(2+) to mPTP openin

21 n many cell types by sensing low endoplasmic reticular Ca(2+) levels and then coupling to plasma memb

22 standard CRC assay by specifically inducing reticular Ca(2+) release to investigate the respective c

23 sitive HCM, whereas sarcoplasmic endoplasmic reticular calcium ATPase 2 abundance and sarcoplasmic re

24 of genotype, as was sarcoplasmic endoplasmic reticular calcium ATPase 2/phospholamban protein ratio (

25 (45)Ca sarcoplasmic endoplasmic reticular calcium ATPaseuptake assay showed reduced upta

26 Down regulation of dStim, the endoplasmic reticular calcium sensor and a principal component of SO

27 Our data demonstrate that Cxcl12-abundant-reticular (CAR) cell subsets (Adipo-CAR and Osteo-CAR) d

28 rteriolar endothelial cells, CXCL12-abundant reticular (CAR) cells, and cells of the osteoblastic lin

29 tworks of CD45(-)gp38(+)CD31(-) fibroblastic reticular cell (FRC)-like cells.

30 However, the mechanisms that regulate reticular cell function are not well understood.

31 and pathological alterations of fibroblastic reticular cell networks in the draining lymph nodes.

32 lymph nodes, dendritic cells (DCs) maintain reticular cell survival in multiple compartments.

33 nderpinned by specialized B cell-interacting reticular cells (BRCs).

34 Fibroblastic reticular cells (FRCs) and their specialized collagen fi

35 Fibroblastic reticular cells (FRCs) form a cellular network that serv

36 Fibroblastic reticular cells (FRCs) form the cellular scaffold of lym

37 Fibroblastic reticular cells (FRCs) in the T cell zone of lymph nodes

38 At the initiation of GVHD, LN fibroblastic reticular cells (FRCs) rapidly reduced expression of gen

39 Lymph node fibroblastic reticular cells (FRCs) respond to signals from activated

40 nic stromal cells, particularly fibroblastic reticular cells (FRCs), during experimental autoimmune e

41 LN) stromal cells, particularly fibroblastic reticular cells (FRCs), provide critical structural supp

42 ttributable at least in part to fibroblastic reticular cells (FRCs), which are a major population of

43 xpressing host cells, including fibroblastic reticular cells and follicular dendritic cells.

44 romal organizers give rise to adult marginal reticular cells and form a dedicated stromal niche for i

45 Specifically, CXCL13(+) follicular reticular cells form a small-world network of guidance s

46 s support lymphocyte function, and targeting reticular cells is a potential strategy for controlling

47 component of the CHT niche, and mature into reticular cells lining and interconnecting sinusoids.

48 critical mediators, and LTbetaR signaling on reticular cells mediated cell survival by modulating pod

49 Our data thus suggest that reticular cells of the B cell zone generate microenviron

50 Fibroblastic reticular cells responded rapidly to DST by transcribing

51 Within secondary lymphoid tissues, stromal reticular cells support lymphocyte function, and targeti

52 chment to CD4(-) lymphoid organ fibroblastic reticular cells that mediate transinfection of CD4(+) T

53 rents in the postsynaptic thalamic relay and reticular cells were dramatically elevated, favoring reb

54 Fibroblastic reticular cells were flow-sorted at different timepoints

55 ls and T-B cell border-enriched fibroblastic reticular cells, is developmentally required for T(FR) c

56 types of LN SC subsets, namely fibroblastic reticular cells, lymphatic endothelial cells, and blood

57 ells, a profile associated with fibroblastic reticular cells.

58 hat gives rise exclusively to adult marginal reticular cells.

59 Reticular chemistry approach was successfully employed t

60 Here, we demonstrate the successful use of reticular chemistry as an appropriate strategy for the d

61 The extension of reticular chemistry concepts to electrically conductive

62 ults serve as a case study demonstrating how reticular chemistry design principles can be extended to

63 We report the use of reticular chemistry for the fabrication of a chemically

64 Reticular chemistry has boosted the design of thousands

65 Here we report the use of reticular chemistry to control the binding of CO(2)RR in

66 This work expands the scope of reticular chemistry to include, for the first time, crys

67 Successful implementation of reticular chemistry using a judiciously designed rigid o

68 le blueprints for the successful practice of reticular chemistry, and par excellence ideal for the de

69 ritical tool enabling this progress has been reticular chemistry, through which researchers can desig

70 This work illustrates two principles of reticular chemistry: first, selectivity for helical over

71 m the periphery enter the draining LNs via a reticular conduit system.

72 However, when Hh signalling is inhibited the reticular dermis does not respond to epidermal beta-cate

73 e is a unique layer of adipocytes within the reticular dermis of the skin.

74 adipogenic differentiation in the developing reticular dermis.

75 to the transition between the papillary and reticular dermis.

76 basaloid cells present in the papillary and reticular dermis.

77 This work is paving the way for reticular design of highly efficient and highly active D

78 e art of surface-confined sCOFs, emphasizing reticular design, synthesis approaches, and key challeng

79 ndrial intermembrane space and is mutated in reticular dysgenesis (RD), a rare form of severe combine

80 Reticular dysgenesis is an autosomal recessive form of s

81 ity of the AK2 mutation and demonstrate that reticular dysgenesis should be considered in Amish indiv

82 Human variants in AK2 cause reticular dysgenesis, a severe combined immunodeficiency

83 D and 32 with leaky SCID, Omenn syndrome, or reticular dysgenesis.

84 Both statins induced endoplasmic reticular (ER) stress, but only atorvastatin inhibited E

85 reported the mechanochemical synthesis of a reticular family of crystalline heterobimetallic metal-o

86 ot affect TGF-beta target gene expression in reticular fibroblasts, and TGF-beta inhibition does not

87 ation, differentiation and ECM production by reticular fibroblasts.

88 ive cell cluster is centered on a tegmental (reticular) field traversed by fibers of the superior cer

89 ulomotor area from the central mesencephalic reticular formation (cMRF), a region implicated in horiz

90 ies suggested that the central mesencephalic reticular formation (cMRF), located lateral to the oculo

91 The mesencephalic reticular formation (MRF) is formed by the pedunculopont

92 he two major components of the mesencephalic reticular formation (MRF), namely the pedunculopontine a

93 lycinergic fibers ascending from the pontine reticular formation (PRF) of the brainstem evoked fast a

94 characteristic locations within the pontine reticular formation (PRF).

95 F(L) ), nucleus of the solitary tract (NTS), reticular formation (RF), pontine and midbrain vestibula

96 vibrissa-related region of the intermediate reticular formation (vIRt).

97 of Darkschewitsch, mesencephalic and pontine reticular formation and pontine nuclei.

98 he PCC to the ventral tegmental area/pontine reticular formation and thalamus, in addition to the LC,

99 ated by a central amygdala projection to the reticular formation in the brainstem.

100 racellular and extracellular recordings from reticular formation neurons, including identified reticu

101 cingulate cortex (1.6-fold increase) and the reticular formation of the medulla (6.5-fold increase).

102 ost likely monosynaptic, from the MLR to the reticular formation that activates reticulospinal stop c

103 cMRF input by injecting this portion of the reticular formation with anterograde tracers in combinat

104 ncerta (ZI), anterior pretectum, and pontine reticular formation) provides temporally precise and foc

105 's nucleus, Kolliker-Fuse nucleus, hindbrain reticular formation, and rostral NTS.

106 mesencephali, the cerebellar corpus C1, the reticular formation, and the Raphe nuclei.

107 AG, the cuneiform nucleus, the mesencephalic reticular formation, and the superior colliculus.

108 al midbrain tegmentum, posterior tuberculum, reticular formation, and viscerosensory lobe.

109 to a broad network of regions including the reticular formation, basal ganglia, thalamus, posterior

110 d tegmentum, laterodorsal tegmental nucleus, reticular formation, spinal cord, and retina.

111 ensory afferents and premotor neurons of the reticular formation, where central pattern generator cir

112 e serotonergic raphe nuclei of the brainstem reticular formation, with three discrete subregions in t

113 h electrical activation of the mesencephalic reticular formation.

114 ed in the nucleus lateralis valvulae and the reticular formation.

115 s and nanoplatelets, and metal plasmonic and reticular framework nanoparticles.

116 ing units to assemble an unlimited number of reticular frameworks.

117 lung occupied by ground glass, ground glass-reticular (GGR), honeycombing, emphysema, and normal lun

118 Postmortem computed tomography revealed reticular infiltration of the lungs with severe bilatera

119 tic input to thalamocortical relay cells and reticular interneurons and activate intrathalamic circui

120 Notably, DPP4+ progenitors reside in the reticular interstitium, a recently appreciated fluid-fil

121 a subpopulation of Dbx1-derived intermediate reticular (IRt) neurons are rhythmically active during i

122 he apical surface of the organ of Corti, the reticular lamina (RL), are amplified over a much broader

123 The magnitude and phase differences between reticular lamina and basilar membrane vibrations are abs

124 ell into a narrow fluid-filled space between reticular lamina and tectorial membrane.

125 r flow that arises from shearing between the reticular lamina and the tectorial membrane.

126 ng subnanometer vibrations directly from the reticular lamina at the apical ends of outer hair cells

127 s travelling wave vibrates in phase with the reticular lamina at the best frequency, and results in m

128 to the basal end of the cochlea, even though reticular lamina motion is amplified in this region, whi

129 plified in this region, which indicates that reticular lamina motion is not directly coupled to basil

130 expected; instead, they actively vibrate the reticular lamina over a broad frequency range.

131 s can generate sufficient force to drive the reticular lamina over all audible frequencies in living

132 The phase relation of reticular lamina to basilar membrane vibration changes w

133 The outer hair cell-driven reticular lamina vibration collaboratively interacts wit

134 living mouse cochleae that the sound-induced reticular lamina vibration is substantially larger than

135 ial vibrations of the tectorial membrane and reticular lamina were tuned.

136 band and slow sharply tuned responses of the reticular lamina, but only a slow tuned response of the

137 t the OHC bundle, the tectorial membrane and reticular lamina, to the transverse motion of the basila

138 cell stereocilia, the tectorial membrane and reticular lamina, were sharply tuned in the radial direc

139 This reticular-limbic pathway may thus function in processing

140 ities in acute/subacute disease to increased reticular markings and honeycombing fibrosis, which typi

141 and generate osteoblasts, chondrocytes, and reticular marrow stromal cells, but not adipocytes.

142 Reticular materials are of high interest for diverse app

143 se two approaches will facilitate the use of reticular materials in addressing major needs of society

144 The use of reticular materials in the electrochemical reduction of

145 s externally controlled tools to manufacture reticular materials over all the length scales.

146 ncompasses emerging research domains such as reticular materials, surfactants, surface functionalizat

147 CM/adhesion coupling, whereas highly pliable reticular matrices promote adhesion retraction.

148 the trans side of the plasma or endoplasmic reticular membrane, respectively.

149 The DNA liberated from neutrophils forms a reticular mesh that resembles morphologically a net, ren

150 ructures associated with a novel cytoplasmic reticular meshwork of intermediate filaments.

151 roscopy revealed that RPMs are embedded in a reticular meshwork of red pulp fibroblasts characterized

152 s of Parkin did produce abnormal tubular and reticular mitochondria restricted to the motor cell bodi

153 s and T cells, which resulted in an expanded reticular network and enhanced immunity.

154 e vasculature, expansion of the fibroblastic reticular network and maintenance of lymphoid stromal ph

155 r adventitial compartment and its associated reticular network form a niche for lymphocytes and appea

156 he size-restrictive nature of the lymph node reticular network, delivering cargo to specific cells in

157 s partially resemble the cells that form the reticular networks in organized lymphoid tissues, potent

158 e, we explore spatial reconfiguration in the reticular networks of the medulla that generate breathin

159 Thalamic reticular neurons are inhibitory cells interconnected by

160 thors show that the receptive field sizes of reticular neurons are small enough to provide localized

161 biophysical properties interconnecting some reticular neurons in mice lacking Cx36.

162 Our results show that reticular neurons in the cat operate over discrete spati

163 ence revealed spinal-projecting Galphat-S-ir reticular neurons in the caudal hindbrain.

164 e hypothalamus and in some hindbrain lateral reticular neurons, and PSST5 in cells of the region of t

165 rks are engaged through specialized thalamic reticular neurons, including antagonistic subpopulations

166 rents within postsynaptic thalamic relay and reticular neurons.

167 innervate reticulospinal neurons in the four reticular nuclei of lampreys.

168 inergic neurons release dopamine in the four reticular nuclei where reticulospinal neurons are locate

169 mulation evoked dopamine release in all four reticular nuclei, but not in the spinal cord.

170 interpeduncular nucleus, superior and middle reticular nuclei, magnocellular vestibular nucleus, soli

171 paminergic source using tracer injections in reticular nuclei, which retrogradely labeled dopaminergi

172 d the rostral parvicellular and intermediate reticular nuclei.

173 us (mRN, rostral hindbrain) and the inferior reticular nucleus (iRN, caudal hindbrain).

174 hether neurons in the medullary intermediate reticular nucleus (IRt) are components of a central patt

175 estigate whether neurons in the intermediate reticular nucleus (IRt) form the central pattern generat

176 LR sends bilateral projections to the middle reticular nucleus (mRN, rostral hindbrain) and the infer

177 of them originating from the gigantocellular reticular nucleus (NRG), have been observed.

178 y two major inhibitory systems: the thalamic reticular nucleus (TRN) and extrathalamic inhibitory (ET

179 s for a specific involvement of the thalamic reticular nucleus (TRN) come from its unique neuronal ch

180 nerally thought that neurons in the thalamic reticular nucleus (TRN) form GABAergic synapses with oth

181 It is known that the thalamic reticular nucleus (TRN) gates sensory information en rou

182 The inhibitory thalamic reticular nucleus (TRN) is a hub of the attentional syst

183 The thalamic reticular nucleus (TRN) is a unique brain structure at t

184 The thalamic reticular nucleus (TRN) is implicated in schizophrenia p

185 agation in thalamocortical (TC) and thalamic reticular nucleus (TRN) neurons remains unknown.

186 liminates rebound bursting in model thalamic reticular nucleus (TRN) neurons.

187 During sleep, neurons in the thalamic reticular nucleus (TRN) participate in distinct types of

188 Neurons within the mature thalamic reticular nucleus (TRN) powerfully inhibit ventrobasal (

189 activity through modality-specific thalamic reticular nucleus (TRN) subnetworks.

190 ortex and inhibitory neurons of the thalamic reticular nucleus (TRN) that regulate the flow of those

191 hd1 is selectively expressed in the thalamic reticular nucleus (TRN), a group of GABAergic neurons th

192 The thalamic reticular nucleus (TRN), the major source of thalamic in

193 ion, we found neurons of the visual thalamic reticular nucleus (visTRN) to exhibit PFC-dependent chan

194 icity at electrical synapses in the thalamic reticular nucleus - paired burst spiking in coupled neur

195 on evoked responses from inhibitory thalamic reticular nucleus and excitatory tectothalamic terminals

196 apses (type S1), which likely arise from the reticular nucleus and GABAergic interneurons, and (c) GA

197 formed by GABAergic neurons in the thalamic reticular nucleus and glutamatergic relay neurons in the

198 s been proposed that neurons in the thalamic reticular nucleus are interconnected through GABAergic s

199 gene Cacna1h in iKOp/q mice reduces thalamic reticular nucleus burst firing and promotes rather than

200 is preferentially expressed in the thalamic reticular nucleus during development, pharmacological re

201 ulvinar projections that engage the thalamic reticular nucleus enable the pulvinar to estimate decisi

202 The thalamic reticular nucleus has a critical role in modulating info

203 address the question of whether cells in the reticular nucleus have receptive fields small enough to

204 The thalamic reticular nucleus is an important structure governing th

205 alamocortical neurons and GABAergic thalamic reticular nucleus neurons and that these properties are

206 Moreover, in thalamic reticular nucleus neurons, burst firing is impaired acco

207 In thalamocortical and thalamic reticular nucleus neurons, the site of AP generation and

208 rebellar nuclei neurons onto gigantocellular reticular nucleus neurons, which might produce an action

209 accompanied by persistent firing in thalamic reticular nucleus neurons.

210 the first 2 weeks after birth, the thalamic reticular nucleus of the mouse lacks intrinsic GABAergic

211 caudate-putamen, 0.26 ug . g(-1) +/- 0.05 in reticular nucleus of the thalamus, 0.24 ug . g(-1) +/- 0

212 The visual sector of the overlying thalamic reticular nucleus receives input from relay cells and su

213 hlight hypothesis proposes that the thalamic reticular nucleus regulates thalamic relay activity thro

214 tions from the visual sector of the thalamic reticular nucleus to the lateral geniculate nucleus comp

215 antagonist within the middle rhombencephalic reticular nucleus was sufficient to decrease reticulospi

216 rt a Brn3c(+) RGC projection to the thalamic reticular nucleus, a visual nucleus that was not previou

217 al nucleus, somatosensory thalamus, thalamic reticular nucleus, and primary somatosensory cortex.

218 cially in the hypothalamus, septum, thalamic reticular nucleus, certain cortices and other limbic str

219 the cochlear nucleus, and via caudal pontine reticular nucleus, pontine central gray, and MS, reached

220 her FEF connections were with the claustrum, reticular nucleus, zona incerta, lateral posterior and m

221 wed a functional perturbation of the lateral reticular nucleus-cerebellum internal feedback pathway i

222 Interestingly, inborn deletion of thalamic reticular nucleus-enriched, human childhood absence epil

223 ypotheses about the function of the thalamic reticular nucleus.

224 uence thalamic relay nuclei via the thalamic reticular nucleus.

225 by driving inhibitory cells of the thalamic reticular nucleus.

226 beta1, and gamma2 were most abundant in the reticular nucleus.

227 m two sources, substantia nigra and thalamic reticular nucleus.

228 he retina, visual cortices, and the thalamic reticular nucleus.

229 along the interlobular septa, causing a fine reticular pattern on CT images.

230 yloid deposits were usually distributed in a reticular/pericellular pattern, whereas transthyretin am

231 Senile reticular pigmentary change was the predominant peripher

232 tary changes, reticular pseudodrusen, senile reticular pigmentary changes, cobblestone degeneration,

233 Polymorphisms in the endoplasmic reticular protein orosomucoid-like protein 3 (ORMDL3), w

234 MD to include another extracellular deposit, reticular pseudodrusen (RPD) (also termed subretinal dru

235 rrelation to visual acuity (VA) in eyes with reticular pseudodrusen (RPD) vs those with drusen withou

236 Presence of reticular pseudodrusen (RPD) was assessed by masked grad

237 color fundus photography: large soft drusen, reticular pseudodrusen (RPD), refractile drusen, hyperpi

238 zed as soft drusen, cuticular drusen, and/or reticular pseudodrusen (RPD).

239 The ability to detect reticular pseudodrusen (RPD)/subretinal drusenoid deposi

240 ect modification based on the coexistence of reticular pseudodrusen (RPD; adjusted interaction P = 0.

241 Reticular pseudodrusen are a frequent finding in patient

242 Reticular pseudodrusen are associated more strongly with

243 Reticular pseudodrusen are highly concurrent with AMD an

244 Reticular pseudodrusen exhibits quick formation and coll

245 Reticular pseudodrusen is found frequently in patients w

246 Reticular pseudodrusen mostly were located both within a

247 Reticular pseudodrusen represent subretinal deposits tha

248 Reticular pseudodrusen was detected in 18 patients (62%)

249 Reticular pseudodrusen was not seen in patients younger

250 Reticular pseudodrusen were deposits juxtaposed to photo

251 Reticular pseudodrusen were detected in eyes without oth

252 Reticular pseudodrusen were found commonly in the macula

253 Reticular pseudodrusen were hyporeflective on NIR reflec

254 Reticular pseudodrusen were more frequent in female part

255 Reticular pseudodrusen were present in 130 eyes (14% of

256 Reticular pseudodrusen were seen in 1186 eyes (24% of ey

257 Peripheral reticular pseudodrusen were seen in 15%.

258 Reticular pseudodrusen were visualized with correspondin

259 ation levels (C3d:C3 ratio), the presence of reticular pseudodrusen, and AMD phenotype.

260 ciated with atrophy were fellow eye atrophy, reticular pseudodrusen, increased injections, and type 3

261 CT images were also assessed for presence of reticular pseudodrusen, outer-retinal tubules, and hypor

262 sen, hypopigmentary/hyperpigmentary changes, reticular pseudodrusen, senile reticular pigmentary chan

263 d abnormal autofluorescence, and presence of reticular pseudodrusen.

264 ect modification based on the coexistence of reticular pseudodrusen; interaction P = 0.065).

265 SupV) and the parvicellular and intermediate reticular regions dorsal to the facial motor nucleus.

266 dus externa (GPe), and substantia nigra pars reticular (SNr), and disrupted beta band oscillatory act

267 science and, in particular, in the realm of reticular solids where it still remains a great challeng

268 The endoplasmic reticular stress pathway and smooth muscle actin were un

269 nce indicates that oxidative and endoplasmic reticular stress, which trigger changes in ion channels

270 r), local caspase-3/7, and local endoplasmic reticular stress-related genes.

271 - common progenitors can give rise to marrow reticular stromal cells and perivascular mesenchymal pro

272 ibroblasts that show some resemblance to the reticular stromal cells in secondary lymphoid organs.

273 e found that in the absence of RPTPzeta, the reticular structure of PNNs is lost and phenocopies the

274 proteins, was also localized to cristae and reticular structures isolated in the matrix additional t

275 Recently, significant progress was made by reticular synthesis of related organic solid-state mater

276 applying the 'node and strut' principles of reticular synthesis to molecular crystals.

277 allization, and there are fewer examples of 'reticular synthesis', in which multiple building blocks

278 gely cortical in origin and suggest that the reticular system contributed, at least in part, to these

279 are largely cortical in origin and that the reticular system contributed, at least in part, to these

280 An acoustic startle cue, which engages the reticular system, suppressed MEP size during power grip

281 tic startle cue, a stimulus that engages the reticular system, suppressed MEP size during power grip

282 hibition we assessed the contribution of the reticular system, which projects to cortical neurons, an

283 naptic linkage between anterogradely labeled reticular terminals and retrogradely labeled medial rect

284 ortant regulators of [Cl(-)]i Neurons of the reticular thalamic (RT) nucleus express reduced levels o

285 r KCC2 is an important Cl(-) extruder in the reticular thalamic (RT) nucleus, despite this nucleus ha

286 pharmacological tool to modulate activity of reticular thalamic neurons in disease states.

287 One hub, the reticular thalamic nucleus (of the ventral thalamus), wa

288 The reticular thalamic nucleus was found to be primarily res

289 ons in BGMT and CbMT and with neurons in the reticular thalamic nucleus.

290 ets them apart from GABAergic neurons of the reticular thalamic nucleus.

291 of NMDA receptors, modulates the function of reticular thalamus neurons.

292 men, aged 18 to 69 years, who had at least 1 reticular vein with a minimum length of 10 cm in 1 of th

293 Reticular veins are subdermal veins located in the lower

294 ective than with 75% HG alone in eliminating reticular veins from the treated area (95.17% vs 85.40%;

295 sclerotherapy is the treatment of choice for reticular veins in the lower limbs, no consensus has bee

296 The reticular veins were measured on images obtained before

297 icacy end point was the disappearance of the reticular veins within 60 days after treatment with scle

298 G was superior to 75% HG alone in sclerosing reticular veins, with no statistical difference for comp

299 nol plus 70% HG or 75% HG alone to eliminate reticular veins.

300 cy and safety of 2 sclerosants used to treat reticular veins: 0.2% polidocanol diluted in 70% hyperto