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1              A peripheral smear revealed low reticulocyte count.
2 an approximate 8- to 10-fold increase in the reticulocyte count.
3 blood cells and hemoglobin and a decrease in reticulocyte counts.
4  or near-normal hemoglobin values and normal reticulocyte counts.
5 evels, hematocrits, erythrocyte indices, and reticulocyte counts.
6  white blood cell, neutrophil, platelet, and reticulocyte counts.
7 globin level 8.5 g/dL or lower, and absolute reticulocyte count 60,000/mm3 or less.
8 nia, lympocytosis, hyperglycemia, and higher reticulocyte counts, along with the activation of pro-in
9 al red cell indices, in particular increased reticulocyte count and decreased hemoglobin concentratio
10 nd-Stage Liver Disease (MELD) to incorporate reticulocyte count and hemoglobin concentration (MELD-re
11 lobin is lowest in patients with the highest reticulocyte counts and concomitantly shortened RBC life
12 hrocyte and hemoglobin levels with increased reticulocyte counts and elevated plasma erythropoietin c
13 e antioxidant, tempol, resulted in decreased reticulocyte counts and improved erythrocyte survival.
14  low serum EPO levels, higher absolute basal reticulocyte counts and normal cytogenetics at study ent
15 Serum erythropoietin concentration, absolute reticulocyte count, and adverse events.
16 d by transfusion independence, increments in reticulocyte count, and nontransfused hemoglobin.
17 re killed on day 3 and the hematocrit (Hct), reticulocyte count, and numbers of erythroid and myeloid
18                        Host ABO blood group, reticulocyte count, and parasitemia were not correlated
19 rum erythropoietin concentrations, increased reticulocyte counts, and increased hemoglobin and hemato
20 iated with improvement in hemoglobin levels, reticulocyte counts, and iron stores.
21 ents had the highest baseline neutrophil and reticulocyte counts, and largest treatment-associated de
22 m a significant decrease in serum bilirubin, reticulocyte counts, and serum erythropoietin following
23 nd characterized by a persistent anemia, low reticulocyte counts, and the need for repeated transfusi
24              Evaluation of hemoglobin level, reticulocyte count, bilirubin, and mean corpuscular volu
25 ed lactate dehydrogenase levels and absolute reticulocyte counts but lowered fetal hemoglobin levels
26 .55L > M polymorphism had lower platelet and reticulocyte counts, C-reactive protein, and aspartate a
27 y lower erythrocyte and significantly higher reticulocyte counts compared to patients with low biliru
28                                              Reticulocyte counts decreased from days 1 to 3, peaking
29                                Leukocyte and reticulocyte counts decreased initially in all quartiles
30 od was assayed for hemoglobin concentration, reticulocyte count, erythropoietin levels, white blood c
31  mass, hematocrit, hemoglobin concentration, reticulocyte count, ferritin level, serum erythropoietin
32 % to 34% to 40%) and progressive decrease in reticulocyte count (from 60% to 30% to 13%).
33 nin/piperaquine and determined the Hb level, reticulocyte count, G6PD genotype, and Hb type.
34             Reversion, predicted by baseline reticulocyte count &gt;/=400 x 10(9)/L (P< .001), occurred
35 haemolysis (haemoglobin <=9.5 g/dL; absolute reticulocyte count &gt;=120 x 10(9)/L) on ravulizumab or ec
36 globin (Hb), lactate dehydrogenase, absolute reticulocyte count, haptoglobin, indirect bilirubin, and
37 rols increased hemoglobin levels and reduced reticulocyte counts in recipient animals.
38                                         Peak reticulocyte counts increased from a median of 10x10(9)
39                                Patients with reticulocyte counts less than 250 000/mm3 and hemoglobin
40 fined as transfusion-dependent anemia with a reticulocyte count of 60 x 10(9) cells/L or less and bon
41        The mean hemoglobin concentration and reticulocyte counts of the trauma patients were 8.6 +/-
42 rs; P<.001) and by blood counts at 3 months (reticulocyte count or platelet count of >50 x 10(3)/ mic
43                                     Abnormal reticulocyte count (P<0.001, c-statistic 0.623) and hemo
44 distribution width (P = 0.007) and decreased reticulocyte counts (P <= 0.02), whereas high-normal FT4
45                                Mean absolute reticulocyte count peaked at day 11 or 15 in each group
46            Investigations included hemogram, reticulocyte count, peripheral blood smear, serum vitami
47 ze rbcs in vivo as demonstrated by increased reticulocyte counts, plasma hemoglobin and bilirubin, an
48  markers of intravascular hemolysis, such as reticulocyte count (r = 0.44, P = .02).
49 al hemoglobin and higher white blood cell or reticulocyte counts, reinforcing the need for early diag
50 o significant reduction in hematocrit value, reticulocyte count, transferrin saturation, or ferritin
51 hropoietin in AIHA in the case of inadequate reticulocyte counts, use of the complement inhibitor sut
52                                     Abnormal reticulocyte count was also found to predict mortality i
53  versus normal RBC, but the correlation with reticulocyte count was poor, with inter-individual varia
54 ilar in both msk(-/-) and msk(+/+) mice, but reticulocyte count was significantly increased in msk(-/
55 , but neither the erythrocyte counts nor the reticulocyte counts were altered significantly (P > .1).