ライフサイエンスコーパス: retinal pigment

1 soft indistinct drusen (23.0% versus 2.1%), retinal pigment abnormalities (26.6% versus 7.3%), exuda

2 These abnormalities included retinal pigment abnormalities (n = 6 [5.8%]), increased

3 teral risk factors for CNV (large drusen and retinal pigment abnormalities) incurs $907 (95% CI, -$63

4 They are associated with retinal pigment and diffuse retinal thinning.

5 Retinal pigment and dipping venules were present in 100%

6 We recently developed a retinal pigment ephithelium (RPE)-choroid preparation to

7 ling mechanisms have both been implicated in retinal pigment epithelial (RPE) cell differentiation.

8 ding of the immune response, we infected the retinal pigment epithelial (RPE) cell line, ARPE-19, wit

9 S179C mutant-TIMP3 in retinal pigment epithelial (RPE) cells showed increased

10 s of lipid mediators biosynthesized in human retinal pigment epithelial (RPE) cells that are oxygenat

11 is capable of transdifferentiating cultured retinal pigment epithelial (RPE) cells towards a neurona

12 gated the effects of treating differentiated retinal pigment epithelial (RPE) cells with didanosine (

13 (1% O2) to a low glucose condition by using retinal pigment epithelial (RPE) cells, which are a cruc

14 ptors, which precedes the loss of underlying retinal pigment epithelial (RPE) cells.

15 opsin is present in the apical microvilli of retinal pigment epithelial (RPE) cells.

16 nkers to release conjugated cargo within the retinal pigment epithelial (RPE) cells.

17 sociated with choroidal vascular atrophy and retinal pigment epithelial (RPE) changes including struc

18 at the ARMS2/HTRA1 locus with subretinal/sub-retinal pigment epithelial (RPE) hemorrhage related to n

19 drial phosphatase PGAM5 leads to accelerated retinal pigment epithelial (RPE) senescence in vitro and

20 Bullous serous retinal detachment (RD) with retinal pigment epithelial (RPE) tear is a rare and seve

21 e of subretinal fluid and few focal spots of retinal pigment epithelial alterations.

22 Fundus examination revealed midperipheral retinal pigment epithelial atrophy and intraretinal pigm

23 variate analysis, visual acuity at referral, retinal pigment epithelial atrophy, and macular scarring

24 drusen, retinal pigment epithelial fibrosis, retinal pigment epithelial atrophy, visual symptoms, and

25 igated the effects of UVA radiation on Human retinal pigment epithelial cell (ARPE-19) growth and pro

26 ofluorescence (NIR-AF) provided evidence for retinal pigment epithelial cell (RPE) involvement.

27 proximal tubular cell line (TEC) and a human retinal pigment epithelial cell line (ARPE-19).

28 Knockdown of Tgifs in a human retinal pigment epithelial cell line also increased EVI5

29 In addition, the retinal pigment epithelial cell line, RPE-Neo was used a

30 s and human telomerase reverse transcriptase-retinal pigment epithelial cell line, we show that RanGT

31 g and nAbs against BADrUL131-Y4 CMV in adult retinal pigment epithelial cell line-19 human epithelial

32 (alphaB) is exported out of the adult human retinal pigment epithelial cells (ARPE19) packaged in ex

33 -grade induced pluripotent stem cell-derived retinal pigment epithelial cells (iPSC-RPE).

34 rods (RodDeltaBsg), cones (ConeDeltaBsg), or retinal pigment epithelial cells (RPEDeltaBsg).

35 ar degeneration depend on a loss of ClC-2 in retinal pigment epithelial cells and Sertoli cells, resp

36 ic towards human primary blood leukocytes or retinal pigment epithelial cells at effective concentrat

37 hyde is shepherded within photoreceptors and retinal pigment epithelial cells to facilitate retinoid

38 screens in wild-type and TP53 knockout human retinal pigment epithelial cells using a focused dual gu

39 Human retinal pigment epithelial cells were treated with vario

40 lity of dysbiotic Pg-strains to invade human-retinal pigment epithelial cells(ARPE-19), their surviva

41 to individual cells, such as photoreceptors, retinal pigment epithelial cells, and blood cells in the

42 In human fetal retinal pigment epithelial cells, there is an early indu

43 iole number at the population level in human retinal pigment epithelial cells.

44 on microscopy for IFT88-mEOS4b in live human retinal pigment epithelial cells.

45 ORF45 tegument protein were tested in human retinal pigment epithelial cells.

46 tion (AMD) attributed to anteriorly migrated retinal pigment epithelial cells.

47 atrophy showed encompassed foveal thinning, retinal pigment epithelial clumping, and the loss of ext

48 miting toxicities included grade 3 bilateral retinal pigment epithelial detachment in one patient who

49 tachment, orange lipofuscin pigment, drusen, retinal pigment epithelial fibrosis, retinal pigment epi

50 etinal fluid compared to intraretinal or sub-retinal pigment epithelial fluid.

51 enter point thickness was 209 (175-274) mum, retinal pigment epithelial lesion complex was present in

52 ry-choroidal melanoma, and melanocytoma) and retinal pigment epithelial neoplasms showed negative res

53 herence tomography (SD-OCT) demonstrated sub-retinal pigment epithelial nodular deposits, some of whi

54 eration, and (4) absence of other signs of a retinal pigment epithelial tear.

55 r uptake and delivery of MPO to lysosomes of retinal pigmented epithelial (RPE) cells acts to clear t

56 Retinal pigmented epithelial (RPE) cells are essential f

57 d modify proangiogenic signaling produced by retinal pigmented epithelial (RPE) cells under different

58 s now using iPSCs to generate photoreceptor, retinal pigmented epithelial (RPE), and-more recently-ch

59 fects the secretion of angiogenic factors by retinal pigmented epithelial cells under normoxic, hypox

60 Here we show that when retinal-pigmented epithelial (RPE1) cells experience mit

61 ich ferritin in the outer retina in-vivo and retinal-pigment-epithelial (RPE) cells in-vitro.

62 is expressed on the basolateral membrane of retinal-pigment-epithelial (RPE) cells, where it mediate

63 To evaluate the features of acute retinal pigment epitheliitis (ARPE) at onset and in the

64 Acute retinal pigment epitheliitis resolved in a sequence of (

65 iar retinal disease, the Martinique crinkled retinal pigment epitheliopathy that begins around the ag

66 orders, resembling congenital hypertrophy of retinal pigment epithelium (CHRPE) lesions.

67 tics of combined hamartoma of the retina and retinal pigment epithelium (CHRRPE) involving the macula

68 , we use human induced pluripotent stem cell-retinal pigment epithelium (hiPSC-RPE) derived from pati

69 An in vitro model of human retinal pigment epithelium (HRPEsv) cells was treated wi

70 eceptor layer thickness: inner nuclear layer-retinal pigment epithelium (INL-RPE) and the specific su

71 ilized induced pluripotent stem cell-derived retinal pigment epithelium (iPSC-RPE) to test the potent

72 (ELM-ISOS); and inner segment outer segment-retinal pigment epithelium (ISOS-RPE).

73 usen phenotypes, including the occurrence of retinal pigment epithelium (RPE) abnormalities, choroida

74 The epigenetic plasticity of amphibian retinal pigment epithelium (RPE) allows them to regenera

75 Frank atrophy of the retinal pigment epithelium (RPE) and a neovascular compl

76 opathy that begins around the age of 30 with retinal pigment epithelium (RPE) and Bruch's membrane ch

77 pear as hyperreflective deposits between the retinal pigment epithelium (RPE) and Bruch's membrane on

78 The human retinal pigment epithelium (RPE) and choroid are complex

79 se cause remains elusive, dysfunction of the retinal pigment epithelium (RPE) and dysregulation of co

80 ter segments is an important function of the retinal pigment epithelium (RPE) and it is essential for

81 Bestrophin1 (BEST1) is expressed in human retinal pigment epithelium (RPE) and mutations in the BE

82 depend on support functions performed by the retinal pigment epithelium (RPE) and on oxygen and nutri

83 oid zone disruption), components of complete retinal pigment epithelium (RPE) and outer retinal atrop

84 d pigmented Abca4(-/-) mouse model presented retinal pigment epithelium (RPE) and photoreceptor degen

85 roduced by neighboring epithelial cells, the retinal pigment epithelium (RPE) and podocytes, respecti

86 ration (AMD) is a progressive disease of the retinal pigment epithelium (RPE) and the retina leading

87 ration of cone photopigments may require the retinal pigment epithelium (RPE) and/or retinal Muller g

88 nner and outer boundaries of the choroid and retinal pigment epithelium (RPE) as well as the inner re

89 outer retinal disruption and atrophy of the retinal pigment epithelium (RPE) associated with ORT on

90 that photoreceptor atrophy can occur without retinal pigment epithelium (RPE) atrophy and that atroph

91 ed on multimodal imaging and ocular history: retinal pigment epithelium (RPE) atrophy with treatment-

92 hannel is localized to the apical aspects of retinal pigment epithelium (RPE) cells and contributes t

93 polar cells, mitochondria, Muller cells, and retinal pigment epithelium (RPE) cells and were visualiz

94 Retinal pigment epithelium (RPE) cells are cultured on t

95 o evaluate the intraretinal migration of the retinal pigment epithelium (RPE) cells in age-related ma

96 Cholesterol accumulation beneath the retinal pigment epithelium (RPE) cells is supposed to co

97 In human retinal pigment epithelium (RPE) cells, the primary site

98 in cell culture media was incorporated into retinal pigment epithelium (RPE) cells.

99 status, method of subretinal fluid drainage, retinal pigment epithelium (RPE) changes, and choroidal

100 ound TRPV4 expression in the endothelium and retinal pigment epithelium (RPE) components of the BRB,

101 tory milieu in wild-type mouse, we triggered retinal pigment epithelium (RPE) damage/PRC death by sub

102 ed macular degeneration and atypical central retinal pigment epithelium (RPE) defects not attributabl

103 al dystrophy, characterised by extensive sub-retinal pigment epithelium (RPE) deposits, RPE atrophy,

104 discovery cohort of 4 eyes revealed that in retinal pigment epithelium (RPE) elevations with a great

105 d induced pluripotent stem cells to generate retinal pigment epithelium (RPE) from an individual suff

106 profiled mRNA levels in both neuroretina and retinal pigment epithelium (RPE) from mouse and baboon o

107 atrix (ECM) microenvironment surrounding the retinal pigment epithelium (RPE) has been implicated in

108 To report on the presence of retinal pigment epithelium (RPE) humps in high myopia, a

109 ed using spectral-domain OCT with respect to retinal pigment epithelium (RPE) in 836 spectral-domain

110 lipid profiles specifically localized to the retinal pigment epithelium (RPE) in Abca4 (-/-) Stargard

111 s, the vascular/neural network in DR and the retinal pigment epithelium (RPE) in AMD.

112 o assess growth and maturation phases of the Retinal Pigment Epithelium (RPE) in-vitro at the cell la

113 Daily, the retinal pigment epithelium (RPE) ingests a bolus of lipi

114 While AMD histopathology involves retinal pigment epithelium (RPE) injury associated with

115 The retinal pigment epithelium (RPE) is a highly polarized e

116 The retinal pigment epithelium (RPE) is a monolayer of cobbl

117 The retinal pigment epithelium (RPE) is a particularly vulne

118 interface between the neural retina and the retinal pigment epithelium (RPE) is critical for several

119 Lipofuscin in the retinal pigment epithelium (RPE) is the major source of

120 he long-term natural history of the residual retinal pigment epithelium (RPE) is unclear, with report

121 < 0.001-0.03) were: hyperreflective foci and retinal pigment epithelium (RPE) layer atrophy or absenc

122 is study was to evaluate focal damage in the retinal pigment epithelium (RPE) layer in serous retinal

123 s calculated from the SD-OCT and the area of retinal pigment epithelium (RPE) loss from the FAF.

124 ated kinase 1/2 (ERK1/2) is increased in the retinal pigment epithelium (RPE) of age-related macular

125 shown that autophagy is dysfunctional in the retinal pigment epithelium (RPE) of the AMD donor eyes (

126 ther NLRP3 inflammasome activation mainly in retinal pigment epithelium (RPE) or rather in non-RPE ce

127 s in DPED, estimate of coverage by different retinal pigment epithelium (RPE) phenotypes in the DPED

128 nd that increased activation of Rap1a in the retinal pigment epithelium (RPE) reduces oxidative signa

129 ean (SD) height of 45.3 (36.1) mum above the retinal pigment epithelium (RPE) reference plane that wa

130 ements were obtained from en face SS-OCT sub-retinal pigment epithelium (RPE) slab images.

131 dy the impact of miR-204 loss on retinal and retinal pigment epithelium (RPE) structure and function.

132 To investigate when retinal pigment epithelium (RPE) tears occur and their a

133 ek 100, including central macular mean EZ to retinal pigment epithelium (RPE) thickness (2q4: 26.6 mu

134 ted to OCT measurement parameters, including retinal pigment epithelium (RPE) thickness, central macu

135 roduce robust gene editing in vivo in murine retinal pigment epithelium (RPE) tissue and skeletal mus

136 d efficient genome editing in vivo in murine retinal pigment epithelium (RPE) tissue via subretinal i

137 rdly rectifying potassium ion channel in the retinal pigment epithelium (RPE) to maintain ionic homeo

138 abolite transport is a major function of the retinal pigment epithelium (RPE) to support the neural r

139 Retinectomies expose the retinal pigment epithelium (RPE) to the vitreous cavity;

140 ling demonstrated no adverse perturbation of retinal pigment epithelium (RPE) transcriptional program

141 Electron microscopy of the obelix(td15) retinal pigment epithelium (RPE) uncovered reduced phago

142 rmalities in regions with normally appearing retinal pigment epithelium (RPE) were the loss of the PO

143 Proteins in the retinal pigment epithelium (RPE), a cell layer adjacent

144 lated macular degeneration (AMD) affects the retinal pigment epithelium (RPE), a cell monolayer essen

145 ted Cl(-) channel predominantly expressed in retinal pigment epithelium (RPE), and over 250 genetic m

146 en the inner and outer segments (IS/OS), the retinal pigment epithelium (RPE), and the choriocapillar

147 tina, resulting from loss of photoreceptors, retinal pigment epithelium (RPE), and underlying chorioc

148 e photoreceptor support system involving the retinal pigment epithelium (RPE), Bruch's membrane, and

149 a well-characterized pathway in cells of the retinal pigment epithelium (RPE), cone visual pigments a

150 on was found in the photoreceptor-supporting retinal pigment epithelium (RPE), especially in a zone c

151 h the coordinated terminal maturation of the retinal pigment epithelium (RPE), fenestrated choroid en

152 rusen, an accumulation of material below the retinal pigment epithelium (RPE), have long been establi

153 subretinal hyperreflective material (SHRM), retinal pigment epithelium (RPE), hyperreflective foci (

154 tinoid-containing lipofuscin pigments in the retinal pigment epithelium (RPE), increased oxidative st

155 of TGF-beta signaling in the entire eye, the retinal pigment epithelium (RPE), or the vascular endoth

156 On B-scans, the choroid, retinal pigment epithelium (RPE), photoreceptor (PR) lay

157 n (lws) in red cone photoreceptors, while in retinal pigment epithelium (RPE), TH regulates expressio

158 ning of photoreceptor layers, despite intact retinal pigment epithelium (RPE), to approximately 70% o

159 The retinal pigment epithelium (RPE), which lies between the

160 artially met by Muller glia and cells of the retinal pigment epithelium (RPE), which provide essentia

161 ciations of ocular and systemic factors with retinal pigment epithelium (RPE)-Bruch's membrane (BM) c

162 ia 9.7, P = .001; SE -2.27 D [SD 4.65]), and retinal pigment epithelium (RPE)-related dystrophies (OR

163 A nonvisual opsin known as retinal pigment epithelium (RPE)-retinal G-protein-coupl

164 teractions of matured photoreceptors and the retinal pigment epithelium (RPE).

165 ds on proper interaction with the underlying retinal pigment epithelium (RPE).

166 ls in a model of chronic degeneration of the retinal pigment epithelium (RPE).

167 the retina, the ciliary margin (CM), and the retinal pigment epithelium (RPE).

168 d epithelial mesenchymal transition (EMT) of retinal pigment epithelium (RPE).

169 may regulate the phagocytosis of OSs by the retinal pigment epithelium (RPE).

170 teristics, especially the involvement of the retinal pigment epithelium (RPE).

171 eracts with Ca(V) 1.3 Ca(2+) channels in the retinal pigment epithelium (RPE).

172 levels were higher in neural retina than in retinal pigment epithelium (RPE).

173 hotoreceptor outer segments (POS) within the retinal pigment epithelium (RPE).

174 e (ELM) and mild transient thickening of the retinal pigment epithelium (RPE)/Bruch's complex (Bc).

175 ntally regulated manner in chicken embryonic retinal pigment epithelium (RPE)/choroid in the absence

176 b readily penetrates the retina to reach the retinal pigment epithelium (RPE)/choroid with minimal su

177 fied negative regulator of the ER-associated retinal pigment epithelium (RPE)65 isomerase necessary f

178 the photoreceptor zone, ellipsoid zone, and retinal pigment epithelium (RPE, P < 0.001 and P = 0.005

179 r displacement of the temporal peripapillary retinal pigment epithelium (tRPE) from its position in c

180 le-layer sign (separation of hyperreflective retinal pigment epithelium [RPE] from Bruch's membrane,

181 Retinal pigment epithelium abnormalities, AVLs, neovascu

182 The 2 biomarkers were RPE65 for retinal pigment epithelium and CD163 for histiocytes, ea

183 s proliferation of POM cells surrounding the retinal pigment epithelium and decreases the expression

184 ipofuscin accumulate in the lysosomes of the retinal pigment epithelium and display cytotoxic effects

185 Electron microscopy showed thinning of the retinal pigment epithelium and disruption of the externa

186 Quantifying preserved retinal pigment epithelium and EZ areas on FAF and OCT i

187 tors then export the lactate as fuel for the retinal pigment epithelium and for neighboring Muller gl

188 lly if multimodal imaging supports an intact retinal pigment epithelium and inner retina but an abnor

189 s in the choriocapillaris, Bruch's membrane, retinal pigment epithelium and occasionally neurosensory

190 ns in the choriocapillaris in the absence of retinal pigment epithelium and outer retinal abnormaliti

191 area using 2 distinct criteria: (1) complete retinal pigment epithelium and outer retinal atrophy (cR

192 ts and achieved a more rapid recovery of the retinal pigment epithelium and photoreceptor cells.

193 in the choriocapillaris, which supplies the retinal pigment epithelium and photoreceptors, and the c

194 lar components of individual granules in the retinal pigment epithelium and present their analytical

195 dal gammadelta T cells in protection against retinal pigment epithelium and retinal injury.

196 The normal retinal pigment epithelium and uveal melanocytes did not

197 beneath the small irregular elevation of the retinal pigment epithelium at the site of the quiescent

198 eas of pathology revealed the following: (1) retinal pigment epithelium atrophy (with or without resi

199 Retinal pigment epithelium atrophy and photoreceptor lay

200 Retinal pigment epithelium atrophy and photoreceptor los

201 Retinal pigment epithelium atrophy was delineated on the

202 In age-related macular degeneration, the retinal pigment epithelium can be damaged by light actin

203 Suppression of glucose consumption in the retinal pigment epithelium can increase the amount of gl

204 y AMD, including Bruchs membrane thickening, retinal pigment epithelium cell loss, retinal functional

205 lesion perimeter (a measure of the number of retinal pigment epithelium cells exposed at the lesion b

206 Retinal pigment epithelium cells were in the centre, pho

207 Finally, in primary human fetal retinal pigment epithelium cells, ligand binding to TLR2

208 ine, bipolar, horizontal, photoreceptor, and retinal pigment epithelium cells, thus exposing the anat

209 disk ratio (3.8%), vessel tortuosity (2.5%), retinal pigment epithelium degeneration (2.5%), myelinat

210 Retinal pigment epithelium degeneration followed by reti

211 A; n = 9, collected from outside the zone of retinal pigment epithelium degeneration) were evaluated

212 changes correlating with different stages of retinal pigment epithelium degeneration.

213 nal pigment epithelium (RPE) layer in serous retinal pigment epithelium detachment (PED) with multi-c

214 l mice had thickening of Bruchs membrane and retinal pigment epithelium dysfunction.

215 howed a typical macular cherry-red spot with retinal pigment epithelium dystrophy in the middle perip

216 tion of the automatically provided boundary "retinal pigment epithelium fit" positioned at the level

217 e fellow eye, hemorrhage, and absence of sub-retinal pigment epithelium fluid at baseline were associ

218 lial detachment, intraretinal fluid, and sub-retinal pigment epithelium fluid were predictive of FS a

219 terminated at a variable distance before the retinal pigment epithelium in 56.67% of eyes.

220 ive foci were correlated with changes in the retinal pigment epithelium in eyes with age-related macu

221 TLR2 was robustly expressed by the retinal pigment epithelium in mouse and human eyes, both

222 ine the incidence of atrophic lesions of the retinal pigment epithelium in patients with Stargardt di

223 ite the several treatments available, if the retinal pigment epithelium is damaged, we have to cope w

224 A loss of the choriocapillaris/retinal pigment epithelium left a "window-defect", where

225 hypertransmission of light into the choroid, retinal pigment epithelium loss, and loss of outer retin

226 tors of peripheral monocyte infiltration and retinal pigment epithelium migration, and their depletio

227 Mild disease presented with retinal pigment epithelium mottling, a patchy pattern of

228 arget of TLR2 signaling, was detected in the retinal pigment epithelium of human eyes, particularly i

229 a, photoreceptor layer (PRL) outer segments, retinal pigment epithelium plus drusen (RPE+drusen) comp

230 physiological function of photoreceptors and retinal pigment epithelium requires precise regulation t

231 Retinal pigment epithelium tears act differently dependi

232 d on-study events (atrophy status, fibrosis, retinal pigment epithelium tears).

233 imes leading to a loss of photoreceptors and retinal pigment epithelium that manifests clinically as

234 from the choroidal blood passes through the retinal pigment epithelium to the retina where photorece

235 Retinal pigment epithelium undulations and vascular dila

236 and complete atrophy of the outer retina and retinal pigment epithelium were observed in both split-d

237 ss of external limiting membrane and loss of retinal pigment epithelium with hypertransmission of OCT

238 H35.54 (dystrophies primarily involving the retinal pigment epithelium), and H35.50 (unspecified mac

239 (1) hypertransmission, (2) disruption of the retinal pigment epithelium, (3) photoreceptor degenerati

240 Total retinal, retinal pigment epithelium, and choroid layer thickness

241 ion, likely by promoting inflammation of the retinal pigment epithelium, and validate TLR2 as a novel

242 angles, loss of pigment and thinning of the retinal pigment epithelium, choroidal thinning, undiffer

243 pment of ischemic infarction of the choroid, retinal pigment epithelium, outer part of the retina and

244 Interestingly, the retinal pigment epithelium, responsible for normal phago

245 erived monocytes, together with RPE65 in the retinal pigment epithelium, supports differentiation tow

246 Loss of retinal pigment epithelium, the presence of a thin choro

247 9 screens against 27 genotoxic agents in the retinal pigment epithelium-1 (RPE1) cell line.

248 Retinal pigment epithelium-BM thickness, as measured by

249 ight thickening of the nerve fiber layer and retinal pigment epithelium-Bruch's membrane, with thinni

250 ges of different retinal layers, such as the retinal pigment epithelium-drusen complex (RPEDC), were

251 limbal eye stroma and epithelial cells from retinal pigment epithelium.

252 ia but also throughout the retina and in the retinal pigment epithelium.

253 s well as degeneration of photoreceptors and retinal pigment epithelium.

254 their induced pluripotent stem cell-derived retinal pigment epithelium.

255 eduction of bisretinoid formation within the retinal pigment epithelium.

256 d by oxidative stress in the choroid and the retinal pigment epithelium.

257 rrected P < 10(-9)), even in areas of intact retinal pigment epithelium.

258 ved were drusen, atrophy, and changes to the retinal pigment epithelium.

259 of increased ICP, and perhaps changes in the retinal pigment epithelium.

260 e retina, separating photoreceptors from the retinal pigment epithelium.

261 e can suppress consumption of glucose by the retinal pigment epithelium.

262 istent with PVR, and reactive changes in the retinal pigment epithelium.

263 on, optic nerve head pallor, and mottling of retinal pigment epithelium.

264 tween the interdigitation zone and an intact retinal pigment epithelium.

265 ning of the cone outer segment closer to the retinal pigment epithelium.

266 in pigment cells, including melanocytes and retinal pigment epithelium.

267 represent progressive migration and loss of retinal pigment epithelium.

268 sociated with perturbed melanogenesis in the retinal pigment epithelium.

269 ural crest, but only specifically around the retinal pigment epithelium.

270 acid transport between the neural retina and retinal pigment epithelium.

271 cted after semiautomatic segmentation of the retinal pigment epithelium/Bruch membrane complex.

272 the human visual system (retina, macula, and retinal pigment epithelium/choroid) reveals features of

273 terns of NIR-AF over the island of surviving retinal pigment epithelium: Group 1 (preserved NIR-AF ce

274 inal Muller cells, endothelial cells, and in retinal pigment epithelium; agonism of PPARalpha with ge

275 cific with majority of the expression in the retinal pigmented epithelium (RPE) and limited expressio

276 herapy, but delivery of viral vectors to the retinal pigmented epithelium (RPE) and retina can be cha

277 Funduscopy revealed focal alterations in the retinal pigmented epithelium (RPE) and yellow retinal do

278 elated macular degeneration characterized by retinal pigmented epithelium (RPE) death; the RPE also e

279 The retinal pigmented epithelium (RPE) forms the outer blood

280 One of the major biological functions of the retinal pigmented epithelium (RPE) is the clearance of s

281 ession, we delivered a wild-type Mfrp to the retinal pigmented epithelium (RPE) of Mfrp (rd6) /Mfrp (

282 ecific stem cells for neural retina (NR) and retinal pigmented epithelium (RPE) of the teleost medaka

283 ar, mutant eyes show substantially increased retinal pigmented epithelium (RPE) proliferation in the

284 enotype consisting of vision loss, increased retinal pigmented epithelium (RPE) stress, and increased

285 ipid metabolism involving dysfunction of the retinal pigmented epithelium (RPE) underlies the pathoge

286 chronic reactive oxygen species (ROS) in the retinal pigmented epithelium (RPE).

287 FH is expressed in the retinal pigmented epithelium (RPE).

288 iRNA)-processing enzyme DICER1 in the mature retinal pigmented epithelium (RPE).

289 pecialized phagocytes: Sertoli cells and the retinal pigmented epithelium (RPE).

290 sition was associated with protection of the retinal pigmented epithelium from damage caused by disea

291 Retinal pigmented epithelium has plentiful melanosomes,

292 erve fiber layer, outer plexiform layer, and retinal pigmented epithelium using image guidance and se

293 ge, indicative of pathological events in the retinal pigmented epithelium.

294 maintains therapeutic levels of sunitinib in retinal pigmented epithelium/choroid and retina for more

295 sing cells revealed molecular resemblance to retinal-pigment epithelium cells, and anatomic analysis

296 arance and accumulation of lipofuscin in the retinal-pigmented epithelium (RPE) and of lipoproteins a

297 and photoreceptor-specific markers in human retinal pigment epitheliums cells.

298 participation of lateral rim cells in a sub-retinal pigment shield in an insect eye.

299 Trichogramma evanescens Westwood 1833, a sub-retinal pigment shield is formed by pigment-bearing cell

300 copy, it was possible to reveal that the sub-retinal pigment shield of T. evanescens is not formed by