戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 oded within the human pseudogenes are called retrocyclins.
2 n, contribute to the antitoxic properties of retrocyclins.
3 nd diprovirins, whose design was inspired by retrocyclins.
4 he retro, enantio, and retroenantio forms of retrocyclin 1.
5                                              Retrocyclins 1 and 2 and RTD 3 protected cervical epithe
6 esus macaque leukocytes, and three peptides (retrocyclins 1 to 3) whose sequences were inferred from
7  the ability of six theta-defensins (hominid retrocyclins 1-3 and rhesus theta-defensins 1-3) and fou
8        We tested rhesus theta-defensins 1-3, retrocyclins 1-3, and several analogues of RC-1.
9                                              Retrocyclin-1 (4 microm) completely blocked fusion media
10                                              Retrocyclin-1 acted late in the HIV-1 Env fusion cascade
11                                              Retrocyclin-1 bound with high affinity to gp120 (K(d), 3
12                                              Retrocyclin-1 inhibited HIV-1 Env-mediated fusion withou
13                                              Retrocyclin-1 inhibits the cellular entry of HIV-1, HSV,
14  CD spectroscopic analyses all revealed that retrocyclin-1 prevented 6-helix bundle formation.
15 piratory syndrome coronavirus infection, and retrocyclin-1 protects mice from infection by Bacillus a
16                                              Retrocyclin-1, a -defensin, protects target cells from h
17                                      Because retrocyclin-1, an ancestral hominid theta-defensin, can
18 d in human and FBS bound exogenous HNP-2 and retrocyclin-1, and competed with their ability to bind g
19 e inhibitory effects of the novel o-defensin retrocyclin-101 (RC-101) against flavivirus infection an
20                                    Moreover, retrocyclin-101 bound to the DE loop of the E protein of
21                 In this study, we found that retrocyclin-101 inhibited flavivirus (ZIKV and JEV) infe
22                                              Retrocyclin-101 inhibited NS2B-NS3 serine protease activ
23         Here we show that the theta-defensin retrocyclin 2 (RC2) inhibited influenza virus infection
24 Temperature shift experiments indicated that retrocyclin 2 and human alpha defensins human neutrophil
25                                              Retrocyclin 2 blocked viral attachment, and its addition
26 face plasmon resonance studies revealed that retrocyclin 2 bound to immobilized HSV-2 glycoprotein B
27 om infection by both HSV serotypes, but only retrocyclin 2 did so without causing cytotoxicity or req
28  effectiveness against both HSV-2 and HIV-1, retrocyclin 2 provides an intriguing prototype for futur
29                                              Retrocyclin-2 adopts a transmembrane orientation in dila
30 ne the orientation of multiply (15)N-labeled retrocyclin-2 in uniaxially aligned phosphocholine bilay
31                                              Retrocyclin-2 is a disulfide-stabilized cyclic beta-hair
32 rs with different compositions indicate that retrocyclin-2 selectively disrupts the orientational ord
33              The most potent theta-defensin, retrocyclin-2, bound with exceptionally high affinity to
34                                              Retrocyclin also bound fetuin, an extensively glycosylat
35                            We also tested 14 retrocyclin analogues, including the retro, enantio, and
36  plasmon resonance experiments revealed that retrocyclin bound the ectodomain of gp41 with high affin
37 urface plasmon resonance studies showed that retrocyclins bound the lethal factor rapidly and with hi
38 ported that synthetic theta-defensins called retrocyclins can neutralize and aggregate various strain
39            Thus, membrane binding orders the retrocyclin conformation by reducing the beta-sheet curv
40                                     However, retrocyclin did bind to a neoglycoprotein, BSA, with cov
41 -phase synthesis and named it "retrocyclin." Retrocyclin did not cause direct inactivation of HIV-1,
42                                Nevertheless, retrocyclin had a remarkable ability to inhibit proviral
43                  These findings suggest that retrocyclin interferes with an early stage of HIV-1 infe
44                                   IMPORTANCE Retrocyclin is an artificially humanized circular o-defe
45 h an early stage of HIV-1 infection and that retrocyclin-like agents might be useful topical agents t
46                                              Retrocyclin-mediated inhibition of the enzymatic activit
47       As such, HNP1, as well as a o-defensin retrocyclin RC-101, both interfere with Spike-mediated m
48   As such, HNP1, as well as a theta-defensin retrocyclin RC-101, both interfere with Spike-mediated m
49                                              Retrocyclin (RC)-101 is a cationic theta-defensin that i
50  the activity of a synthetic theta-defensin, retrocyclin (RC)-2, in a murine herpes simplex virus (HS
51                   In this study, we examined retrocyclin (RC)1 and RC2, humanized versions of the ant
52 ptide by solid-phase synthesis and named it "retrocyclin." Retrocyclin did not cause direct inactivat
53                                              Retrocyclin's ability to recognize and bind carbohydrate
54  BODIPY-FL-labeled RC-101, a close analog of retrocyclin, showed that the peptide formed patch-like a
55                                In a panel of retrocyclin variants, binding to immobilized gp120 and C
56 ptides that retain the antiviral activity of retrocyclins, while being easier to synthesize.
57                   Neither a circular form of retrocyclin without its tridisulfide ladder nor its beta