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1 in particular those of patients with chronic rhinosinusitis.
2 was a risk factor for noninfectious rhinitis/rhinosinusitis.
3 can arbitrarily be divided into rhinitis and rhinosinusitis.
4 characteristics very similar to eosinophilic rhinosinusitis.
5 the relationship between work exposures and rhinosinusitis.
6 eased incidences of sinonasal infections and rhinosinusitis.
7 onization, fungus balls, and allergic fungal rhinosinusitis.
8 seases ranging from colonization to invasive rhinosinusitis.
9 fungal sinusitis, and acute fulminant fungal rhinosinusitis.
10 review the diagnosis and treatment of acute rhinosinusitis.
11 r of spontaneously resolving acute bacterial rhinosinusitis.
12 childhood asthma, food allergy, and allergic rhinosinusitis.
13 sinusitis who present for treatment of acute rhinosinusitis.
14 rhinitis, nonallergic rhinitis, and chronic rhinosinusitis.
15 epithelial cells from patients with chronic rhinosinusitis.
16 but is impaired during chronic inflammatory rhinosinusitis.
17 gnificantly impaired in chronic inflammatory rhinosinusitis.
18 ergy, eosinophilic esophagitis, and allergic rhinosinusitis.
19 iseases, such as cystic fibrosis and chronic rhinosinusitis.
20 reating patients with upper airway bacterial rhinosinusitis.
21 ntly been associated with asthma and chronic rhinosinusitis.
24 e report herein two cases of allergic fungal rhinosinusitis accompanied by bone destruction of the ad
29 urpose was to classify acute invasive fungal rhinosinusitis (AIFR) caused by Mucor versus Aspergillus
30 ts with CRS had a higher prevalence of acute rhinosinusitis, allergic rhinitis, chronic rhinitis, ast
33 or the clinical diagnosis of acute bacterial rhinosinusitis and for those with severe rhinosinusitis
34 e (N-ERD) asthma is characterized by chronic rhinosinusitis and intolerance of aspirin and other COX1
35 nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role in type
36 nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role in type
38 n accordance with the European guidelines on rhinosinusitis and nasal polyps (EPOS 2012), CT is used
39 according to the European Position Paper on Rhinosinusitis and Nasal Polyps (EPOS) together with sin
40 A was associated with a higher prevalence of rhinosinusitis and nasal polyps as well as eosinophilia
43 exact role of microbial biofilms in chronic rhinosinusitis and orbital cellulitis were not elucidate
44 ability of the clinical isolates in chronic rhinosinusitis and orbital cellulitis, and to look for t
51 act diseases including otitis media, chronic rhinosinusitis, and exacerbations of both cystic fibrosi
52 tissue homogenates in patients with chronic rhinosinusitis, and this effect was most prominent in CR
53 espiratory tract infection," "pharyngitis," "rhinosinusitis," and "the common cold." HIGH-VALUE CARE
54 opriate antibiotic use for adults with acute rhinosinusitis apply to the diagnosis and treatment of a
56 features mimic essential aspects of chronic rhinosinusitis-associated olfactory loss, and illuminate
57 bservations establish a mechanism of chronic rhinosinusitis-associated olfactory loss, caused by a fu
60 usitis recommend that the diagnosis of acute rhinosinusitis be based on the presence of "cardinal sym
62 ad prevalence of allergic, viral and chronic rhinosinusitis, but how the brain encodes and maintains
63 n mediate corticosteroid efficacy in chronic rhinosinusitis, but the role of MUC1 in uncontrolled sev
65 lling to travel longer distances for chronic rhinosinusitis care, results suggest that distance trave
66 l reflux disease, postnasal drip syndrome or rhinosinusitis, chronic obstructive pulmonary disease, p
80 It is increasingly recognized that chronic rhinosinusitis (CRS) comprises a spectrum of different d
81 ovide composite criteria to evaluate chronic rhinosinusitis (CRS) control, taking into consideration
90 e of asthma and its association with chronic rhinosinusitis (CRS) have not been widely studied in pop
91 Current clinical classifications of chronic rhinosinusitis (CRS) have weak prognostic utility regard
93 robial interactions in patients with chronic rhinosinusitis (CRS) in hopes of elucidating mechanisms
126 ranasal sinuses for diseases such as chronic rhinosinusitis (CRS) is particularly challenging because
129 us, age-related cytokine profiles in chronic rhinosinusitis (CRS) need to be investigated for precisi
132 of the underlying cause or causes of chronic rhinosinusitis (CRS) over the past 20 or more years have
134 ently, ILC2s enrichment was noted in chronic rhinosinusitis (CRS) patients; however, the role of ILC2
139 s, were collected from patients with chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP) or witho
140 as obtained from subjects with AERD, chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP), CRS wit
142 g endoscopic sinus surgery (ESS) for chronic rhinosinusitis (CRS) without nasal polyposis, there are
146 ronchodilator lung function, asthma, chronic rhinosinusitis (CRS), and atopy with age using a large E
148 is a prominent feature of asthma and chronic rhinosinusitis (CRS), and the endothelium plays a key ro
149 nasal polyps (NPs) of patients with chronic rhinosinusitis (CRS), as well as in bronchoalveolar lava
150 the high prevalence and morbidity of chronic rhinosinusitis (CRS), little is known about the mechanis
151 tems, we identified respondents with chronic rhinosinusitis (CRS), migraine headache, and fatigue sym
152 ed outcomes during the management of chronic rhinosinusitis (CRS), PROMs will play an essential role
162 plays a role in the exacerbation of chronic rhinosinusitis (CRS); however, the mechanism for this is
164 -to-treat CRS was defined as noncontrollable rhinosinusitis despite successful sinus surgery and appr
166 definitive diagnosis of eosinophilic chronic rhinosinusitis (eCRS) requires invasive surgical tissue
168 affected organs in asthma, rhinitis, chronic rhinosinusitis, eosinophilic esophagitis, food allergy,
169 study to use the European Position Paper on Rhinosinusitis (EPOS) criteria to study the prevalence o
170 ifficult to distinguish from acute bacterial rhinosinusitis, especially during the first 10 days of s
171 s with CRS, which was defined as symptomatic rhinosinusitis for more than 12 weeks and documented imm
172 hould reserve antibiotic treatment for acute rhinosinusitis for patients with persistent symptoms for
173 rs may impact upon airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity
174 morbidities are discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstructive sle
175 Invasive disease includes indolent chronic rhinosinusitis, granulomatous fungal sinusitis, and acut
177 onship between allergic rhinitis and chronic rhinosinusitis has been assessed in a number of observat
178 surgery and antibiotics in managing chronic rhinosinusitis has contributed to a five-times variation
181 tial management of suspected acute bacterial rhinosinusitis in adults and children were prepared by a
182 and treatment of acute maxillary and ethmoid rhinosinusitis in adults who are not immunocompromised.
184 orticosteroids are beneficial to treat acute rhinosinusitis in patients with a history of chronic or
185 hould reserve antibiotic treatment for acute rhinosinusitis in patients with persistent symptoms for
186 robiota: Staphylococcus aureus infection and rhinosinusitis in the nasal-sinus mucosa, as well as cys
188 r 10 days, the likelihood of acute bacterial rhinosinusitis increases, and initiation of antibiotic t
189 smitted infections, cystic fibrosis, chronic rhinosinusitis, inflammatory bowel disease, and glaucoma
197 ce to support antibiotic treatment for acute rhinosinusitis is limited, yet antibiotics are commonly
199 e included acute inflammation (n=6), chronic rhinosinusitis (n=2), and allergic rhinitis (n=20) and u
203 etions in pathologic states, such as chronic rhinosinusitis or hyperglycemia, promotes tonic activati
204 e cases, given the likelihood of acute viral rhinosinusitis or of spontaneously resolving acute bacte
205 ing disease (asthma, nasal polyps or chronic rhinosinusitis, or both), as well as on the methodology
206 cough, recurrent chest infections, perennial rhinosinusitis, otitis media with effusion, and bronchie
207 es (Short-Form Health Questionnaire, 31-item Rhinosinusitis Outcome Measuring Instrument, and Asthma
208 onic obstructive pulmonary disease, acquired rhinosinusitis, pancreatitis, and lethal secretory diarr
210 s flavus which is a common cause of allergic rhinosinusitis, postoperative aspergillosis and fungal k
211 gh rarely some patients with acute bacterial rhinosinusitis present with dramatic symptoms of severe
212 Adult patients with eosinophilic chronic rhinosinusitis received 100mg mepolizumab subcutaneously
213 ve traditionally guided treatment in chronic rhinosinusitis, recent research has favored categorizati
214 s and treatment of acute viral and bacterial rhinosinusitis recommend that the diagnosis of acute rhi
217 The clinical diagnosis of acute bacterial rhinosinusitis should be reserved for patients with rhin
218 mptoms of asthma, allergic rhinitis, chronic rhinosinusitis, smoking status, and history of NSAID-ind
219 ed from malignant disease or invasive fungal rhinosinusitis, so an understanding of the clinical feat
220 level of disease control based on VAS global rhinosinusitis symptom score and adapted EPOS criteria.
221 controlled based on evaluation of VAS global rhinosinusitis symptom score compared to 40.9% based on
222 AIDs was higher in participants with chronic rhinosinusitis symptoms (Odds Ratio 2.12; 95%CI 1.78-2.7
223 nusitis should be reserved for patients with rhinosinusitis symptoms lasting 7 days or more who have
225 ial rhinosinusitis and for those with severe rhinosinusitis symptoms-especially those with unilateral
227 Among patients with nasal polyps and chronic rhinosinusitis, the prevalence was 9.69% (95% CI, 2.16%
228 rflow is also a contributing factor in human rhinosinusitis, this in vivo model demonstrates for the
229 edius range in severity from bite wounds and rhinosinusitis to endocarditis; historically, these infe
230 mechanism in patients with aspirin-sensitive rhinosinusitis to the bronchial mucosa in patients with
231 pecimens from patients with AERD and chronic rhinosinusitis were analyzed by using quantitative PCR,
232 ) patients with clinical features of chronic rhinosinusitis were examined; patients with first-onset
233 ed trial of adults with uncomplicated, acute rhinosinusitis were recruited from 10 community practice
241 itis (AFRS) is a severe phenotype of chronic rhinosinusitis with nasal polyposis (CRSwNP), characteri
242 role for mast cells is suggested in chronic rhinosinusitis with nasal polyposis (CRSwNP), which is c
243 se (AERD) is a condition composed of chronic rhinosinusitis with nasal polyposis and asthma that is d
244 een observed in patients with severe chronic rhinosinusitis with nasal polyposis and chronic obstruct
246 s in atopic dermatitis (AD), asthma, chronic rhinosinusitis with nasal polyps (CRSwNP) and eosinophil
248 ncinate tissue (UT) of patients with chronic rhinosinusitis with nasal polyps (CRSwNP) and those with
266 1 and its ligands PD-L1 and PD-L2 in chronic rhinosinusitis with nasal polyps (CRSwNP) is poorly stud
267 is without nasal polyps (CRSsNP) and chronic rhinosinusitis with nasal polyps (CRSwNP) using real-wor
268 rogenitor cells (BSCs) obtained from chronic rhinosinusitis with nasal polyps (CRSwNP) when different
269 e (AERD) is characterized by asthma, chronic rhinosinusitis with nasal polyps (CRSwNP), and an intole
270 sitis without nasal polyps (CRSsNP), chronic rhinosinusitis with nasal polyps (CRSwNP), and aspirin-e
271 RD) is characterized by the triad of chronic rhinosinusitis with nasal polyps (CRSwNP), asthma, and i
275 e mucosa obtained from patients with chronic rhinosinusitis with nasal polyps (n = 7) and patients wi
276 5%] female and 333 [65%] male), with chronic rhinosinusitis with nasal polyps (n=410) or chronic rhin
278 irway inflammatory diseases, such as chronic rhinosinusitis with nasal polyps and asthma, show increa
280 disease compared with patients with chronic rhinosinusitis with nasal polyps and/or asthma alone.
282 g the trend in asthma, endotypes for chronic rhinosinusitis with nasal polyps have been established,
283 piratory disease is a severe form of chronic rhinosinusitis with nasal polyps in which nearly all pat
287 aracterized by the clinical triad of chronic rhinosinusitis with nasal polyps, asthma, and a hypersen
288 aracterized by the clinical triad of chronic rhinosinusitis with nasal polyps, asthma, and an intoler
289 atment of asthma, atopic dermatitis, chronic rhinosinusitis with nasal polyps, or chronic spontaneous
294 ed to some inflammatory disorders as asthma, rhinosinusitis with polyposis, and atopic dermatitis, he
295 of premorbid medical conditions for chronic rhinosinusitis without nasal polyps (CRSsNP) and chronic
296 9, at lower extent, in patients with chronic rhinosinusitis without nasal polyps (CRSsNP) in comparis
297 LFs) from controls and patients with chronic rhinosinusitis without nasal polyps (CRSsNP), chronic rh
298 nasal polyps (CRSwNP) and those with chronic rhinosinusitis without nasal polyps (CRSsNP; P < .01).
299 nusitis with nasal polyps (n=410) or chronic rhinosinusitis without nasal polyps (n=104), were recrui
300 from control subjects, patients with chronic rhinosinusitis without nasal polyps, and patients with c