ライフサイエンスコーパス: rich medium

1 ene under standard conditions (30 degrees C, rich medium).

2 er low-iron conditions and repressed in iron-rich medium.

3 ined glucose-containing medium or a nutrient-rich medium.

4 n the cytoplasm of Escherichia coli grown in rich medium.

5 expression compared to growth in a nutrient-rich medium.

6 ts involving 67 different metabolic genes in rich medium.

7 uring growth in minimal medium compared with rich medium.

8 per cell in cells grown in either minimal or rich medium.

9 f SER3 is tightly repressed during growth in rich medium.

10 or induction of phenazine gene expression in rich medium.

11 CS) was stimulated by pheromone signaling in rich medium.

12 ximately 3% of yeast genes in cells grown in rich medium.

13 dent when cells approach stationary phase in rich medium.

14 exposure to NCS, although grown in nutrient-rich medium.

15 bacillus Calmette-Guerin on minimal but not rich medium.

16 t on the in vitro growth of V. cholerae in a rich medium.

17 long as those of cells growing in phosphate-rich medium.

18 stems normally repressed in cells growing in rich medium.

19 strain is half that of the parent strain in rich medium.

20 y a slight increase in autophagy in nitrogen-rich medium.

21 ndergo a similar invasive growth response in rich medium.

22 gbuABC) allowed DeltadacA mutants to grow in rich medium.

23 , 17 percent were essential for viability in rich medium.

24 m and are unable to form colonies on complex rich medium.

25 cts of nup184 mutants when grown in nutrient-rich medium.

26 ic complete medium, but were able to grow on rich medium.

27 vasive filaments which penetrate the agar in rich medium.

28 in stationary-phase cultures after growth in rich medium.

29 a temperature-sensitive-lethal phenotype in rich medium.

30 ned in early-stationary-phase cells grown in rich medium.

31 hydroxamate to cultures growing in nutrient-rich medium.

32 m7G-46 modification as well as by growth in rich medium.

33 on of oxyR in E. coli growing aerobically in rich medium.

34 old) over a broad range of cell densities in rich medium.

35 growing in a minimal medium than those in a rich medium.

36 d in short-term maintenance in starvation or rich medium.

37 e) complex when yeast are grown in phosphate-rich medium.

38 ressed at early stationary phase in nutrient-rich medium.

39 of the mutant was even more pronounced in a rich medium.

40 toplasmic when yeast were grown in phosphate-rich medium.

41 without affecting cell viability at 30 C in rich medium.

42 their fitness relative to the wild type in a rich medium.

43 ole in germination of C. difficile spores in rich medium.

44 41 when the bacteria were grown in phosphate-rich medium.

45 s) using a chemically defined, growth factor-rich medium.

46 l growth rate (mu) and maximum growth (A) in rich medium.

47 detected for the other csp genes in defined rich medium.

48 RNA accumulation after cold-shock in defined rich medium.

49 cond messenger is essential for viability on rich medium.

50 ntial or strongly advantageous for growth in rich medium.

51 ng filamentation at 37 degrees C in nutrient-rich medium.

52 ed for survival of engineered bacterium on a rich medium.

53 uired for L. monocytogenes aerobic growth in rich medium.

54 is repressed by NilR and growth in nutrient-rich medium.

55 ld reduced for cells grown in minimal versus rich medium.

56 nes necessary for optimal growth in nutrient-rich medium.

57 Unexpectedly, the strain was not viable on rich medium.

58 thesis but replication competent in vitro in rich medium.

59 on in Clostridium difficile during growth in rich medium.

60 w about 30% slower than the parent strain in rich medium.

61 ickly and having a higher growth rate in the rich medium.

62 ble but switched at high rates when grown in rich medium.

63 re but had no detectable effect on growth in rich medium.

64 rmed on mid-logarithmic-phase cells grown in rich medium.

65 ptor and declined to a greater extent in LDL-rich medium.

66 ors not influenced during growth in nutrient-rich medium.

67 that germinate when transferred to nutrient-rich medium.

68 cpA isogenic mutant strain grown in nutrient-rich medium.

69 found in TC during rapid growth in a complex rich medium.

70 on, and during growth in the laboratory on a rich medium.

71 tion of Caco-2 cells for 3 days in a peptide-rich medium (4 mM Gly-Gln replacing 4 mM Gln).

72 With nutrient-rich medium addition at stages beyond the commitment poi

73 If cells are returned to rich medium after they have committed to meiosis, the tr

74 fluence the rate of growth or spreading on a rich medium, AldA was required for growth on a minimal m

75 le, sup70-65, induces pseudohyphal growth on rich medium, an inappropriate nitrogen starvation respon

76 ssue culture monolayers but normal growth in rich medium anaerobically.

77 the cooperative network of 204 regulators in Rich Medium and a subset of them in four different envir

78 s collected from PANC-1 cells grown in serum-rich medium and at 24 h following the removal of serum.

79 s were dispensable for Pseudomonas growth in rich medium and consumption of diverse hydrophilic nutri

80 erichia coli nucleoid during rapid growth in rich medium and following an induced amino acid starvati

81 as required for full Ribi gene expression in rich medium and for its modulation in response to glucos

82 or metabolic changes that occur in growth in rich medium and host cells but not minimal medium.

83 ed fitness defects in vitro during growth in rich medium and in gnotobiotic mice colonized with defin

84 an glyoxalase II (Glx2) was overexpressed in rich medium and in minimal medium containing zinc, iron,

85 er meiosis, in the shape of budding cells in rich medium and in the morphology of filamentous growth

86 sm when budding yeast are grown in phosphate-rich medium and is unphosphorylated and localized to the

87 letion mutant had a growth defect in protein-rich medium and mimicked the phenotype of a generated Ta

88 These mutants fail to form aerial hyphae on rich medium and most are defective in antibiotic product

89 romoter causes cell-cycle arrest in nutrient-rich medium and promotes developmental events, such as t

90 analyze the growth capacity under amino-acid-rich medium and provide evidence that amino acid prefere

91 WT actin cells under standard conditions in rich medium and rescued the mitochondrial phenotype resu

92 with salt stabilized the DeltadacA mutant in rich medium and restored cefuroxime resistance.

93 to be dispensable for cell growth at 30 C in rich medium and several genes whose functions are needed

94 e to nutrients, moving quickly through G1 in rich medium and slowly in poor medium.

95 rown microaerobically and anaerobically in a rich medium and soluble and membrane proteins of strain

96 prfA cultures grown at 30 or 37 degrees C in rich medium and that the lack of PrfA exacerbates the ch

97 The repressive effects of rich medium and the stimulatory effects of ppGpp were al

98 e when cells entered the stationary phase on rich medium and was replenished upon restoration of favo

99 hich are likely to be required for growth on rich medium and which represent potential therapeutic ta

100 rs in the absence of major growth defects in rich medium and with classical virulence-associated phen

101 tribute to selection of lasR mutants both on rich medium and within the CF airway, supporting a key r

102 e cycle that includes vegetative swarming on rich medium and, upon starvation, aggregation to form fr

103 0.1% of the tatC mutant cells were motile in rich medium, and <0.01% were motile in vir induction med

104 studies in minimal growth conditions and LB rich medium, and from clinical tests that identify V.cho

105 low temperature, glucose as a carbon source, rich medium, and high osmolarity.

106 ntly higher levels in minimal medium than in rich medium, and rpoS expression was similarly elevated.

107 in growth rate, moving quickly through G1 in rich medium, and slowly in poor medium.

108 ted by specific nutrients in the medium, but rich medium appears to suppress pzX formation.

109 maize (Zea mays) seedlings cultivated in Se rich medium are also presented.

110 growth and chromosome segregation defects in rich medium are suppressed by a reduction of replication

111 Bacteria were first grown in a nutrient-rich medium at 26 degrees C to establish a baseline of g

112 sly in sup70-65 mutants cultured in nitrogen-rich medium at 30 degrees C.

113 For Escherichia coli growing rapidly in rich medium at 37 degrees C, the doubling time can be as

114 aZ inhibited cell division and was lethal in rich medium at high induction levels and in minimal medi

115 The mutant derivative grew well in rich medium but did not grow in minimal medium supplemen

116 letion mutants grew comparably to WT 5448 in rich medium but exhibited reduced fitness during competi

117 logarithmic growth phase in cells growing in rich medium but poorly expressed in late logarithmic and

118 encodes sigma(S), does not affect growth in rich medium but severely decreases L. pneumophila intrac

119 himurium was unimpaired on aerobic growth in rich medium but showed enhanced sensitivity in vitro to

120 short in blue light when grown on a sucrose-rich medium but tall when grown on sucrose-deficient med

121 HI mutants of zevA and zevB grew normally in rich medium but were defective for colonization in a mou

122 re near-immediately detectable in a nutrient-rich medium, but delayed in nutrient-poor conditions, pr

123 n of Sis1 is dispensable for rapid growth on rich medium, but is required for [RNQ+] maintenance, dis

124 was also expressed as a single mRNA in iron-rich medium, but its expression was reduced approximatel

125 I transcription during exponential growth in rich medium, but the mechanism of this regulation has re

126 cells in the exponential phase of growth in rich medium by artificial induction of the synthesis of

127 her kinase in vivo inhibits GCN2 function in rich medium by reducing tRNA binding activity.

128 Fe(III) reductase activities were induced in rich medium by the addition of cAMP to aerobically growi

129 homeostasis genes are repressed in phosphate-rich medium by transcription of upstream lncRNAs that in

130 , pho84, and tgp1 are repressed in phosphate-rich medium by transcription of upstream lncRNAs.

131 When grown on a soft agar surface in a rich medium, cells of Salmonella typhimurium elongate, p

132 on for 106 transcription factors profiled in rich medium conditions data from over 500 expression exp

133 reatest in cells grown at low temperature in rich medium, conditions in which the growth defect was m

134 medium and high in cells growing rapidly in rich medium, consistent with the previously recognized r

135 rement for this pathway even when growing in rich medium containing DAP.

136 ced cells expressing the Mal61/HA protein to rich medium containing glucose produces a decrease in ma

137 wed that YmgB represses biofilm formation in rich medium containing glucose, decreases cellular motil

138 erely attenuated for aerobic growth, even in rich medium containing supplemental amino acids, and exh

139 tamate or casamino acids as well as nutrient-rich medium containing yeast extract, peptone, and aceta

140 I when P. pastoris is cultured in a nutrient-rich medium containing yeast extract, peptone, and metha

141 id induction of gadA and gadBC observed when rich-medium cultures enter stationary phase corresponds

142 age was reversible: upon a shift to nutrient-rich medium, expansion started almost immediately at a r

143 charomyces cerevisiae populations growing in rich medium for 1,000 generations.

144 acids (e.g., phenylalanine) and thus, needs rich medium for cell growth.

145 When grown in rich medium, greater than 50% of the minCD mutant cells

146 The cells in the rich medium grew faster, and expression of the majority

147 ere engaged in active rRNA synthesis under a rich medium growth condition.

148 into active transcription centers under the rich medium growth condition; its spatial arrangement at

149 sources and hyperaccumulation of glycogen in rich medium high in Pi.

150 Cultures were grown in glucose-rich medium in the presence or absence of 20 mM benzoate

151 MYO1 (the only myosin II gene) is lethal on rich medium in the W303 strain background and causes slo

152 cytosine methyltransferase, during growth in rich medium in vitro.

153 transposon-containing cells are selected on rich medium, insertions occur at both attTn7 sites with

154 Salmonella enterica, SP induction of RpoS in rich medium is >30 fold and includes effects on both tra

155 If nutrient-rich medium is added before a defined commitment point i

156 The pus7 deletion strain, although viable in rich medium, is growth-disadvantaged under certain condi

157 0%) has no obvious phenotypic consequence in rich medium, it is difficult to study their functions.

158 ucleolus than in the nucleoplasm, whereas on rich medium, it was more evenly distributed between the

159 ng GerO were defective in germination with a rich medium, KCl, L-asparagine, and a 1:1 chelate of Ca(

160 on in the UPEC strain UTI89 during growth in rich medium (LB medium) and urine and during infection o

161 adaptation to four laboratory environments (rich medium, low glucose, high salt, and a nonfermentabl

162 However, for cells in a rich medium, Lrp levels dropped 3- to 4-fold during the

163 ession of lrp during logarithmic growth in a rich medium may be due to low ppGpp levels under these c

164 plays a role in NO, as mukB mutants grown in rich medium often exhibit FtsZ rings on top of diffuse,

165 When cells are grown in a rich medium on agar instead, they elongate, produce more

166 When grown in a rich medium on agar, many bacteria elongate, produce mor

167 tative bacteria that can swim are grown in a rich medium on an agar surface, they become multinucleat

168 Upon growth in rich medium, only slight protein export defects were obs

169 lation after growth into stationary phase in rich medium or after osmotic challenge.

170 wo molybdoenzyme activities when cultured in rich medium or glucose-minimal medium.

171 ting) in cells grown in a chemically defined rich medium or in a minimal medium.

172 s into the culture supernatant when grown in rich medium or in vir induction minimal medium.

173 jected to nutritional or osmotic stress in a rich medium or to nitrogen exhaustion had large and dyna

174 of mitochondrial iron in cells grown in iron-rich medium overexpressing MFT1 or MFT2 show a 2-5-fold

175 cell populations (those incubated in a 5-HT-rich medium overnight) and in unloaded populations and w

176 sted temperature, osmolarity, carbon source, rich medium, oxygen levels, pH, amino acids, solid mediu

177 ium but that in plant oil minimal medium and rich medium, phaB3 seems to be unexpressed.

178 When cells were grown in rich medium, PhaZ1 was sufficient to account for intrace

179 pulsed-laser ablation of OMS-2 in an oxygen-rich medium produces a three-dimensional nanostructured

180 mutant strains were grown on a conventional rich medium (R2YE, containing high sucrose), inactivatio

181 aromyces cerevisiae culture during growth in rich medium rapidly decreases by 40 to 60% when the cell

182 rs normally during stationary phase entry in rich medium remains as a "memory" to maintain the cytopl

183 Rhodobacter sphaeroides grown in a Mn(II)-rich medium replaces the intrinsic Mg(II) ion with an EP

184 osmolarity, glucose as a carbon source, and rich medium repressed transcription of all three operons

185 ic induction of the PHO5 acid phosphatase in rich medium requires the transcriptional activators Pho4

186 dicating that preventing glutamine uptake in rich medium rescues the growth of the S. aureus dacA mut

187 Growth to SP in rich medium results in a 30-fold induction of RpoS, alth

188 Evolving faster migration in rich medium results in slow growth and fast swimming, wh

189 ghout the growth of the bacterial culture in rich medium revealed a dramatic increase in all four NTP

190 073 grown statically to exponential phase in rich medium, revealing the strategies this pathogen uses

191 ween ArnA and ArnB observed during growth on rich medium sequentially diminishes after nutrient limit

192 he generation of common and stereochemically rich medium-sized benzo-fused sultams via complementary

193 Such plasmids are stably maintained, even in rich medium, so optimising biomass production and yields

194 ntially in time after exposure to a nutrient-rich medium, so that the CaDPA amount inside the trapped

195 low-growth conditions, but were sensitive to rich medium, suggesting that the dense packing of initia

196 hat did not fix nitrogen, and grew slowly in rich medium, suggesting that the locus was a cyc gene en

197 binding, were expressed at higher levels in rich medium, suggesting that, upon exposure to a rich en

198 d in wild-type M. smegmatis, using selective rich medium supplemented with DAP unless there was an ex

199 E. coli cells grown in rich medium that contained all amino acids and low amoun

200 d into nitrogen-limited medium from nutrient-rich medium, the cell size increased two- to threefold,

201 As Fis levels increase in cells grown in rich medium, the positioning of its binding site, overla

202 s strongly induced simply upon switch from a rich medium to a minimal medium in the complete absence

203 se pathways when transferred from amino acid-rich medium to amino acid-deficient medium.

204 epeated cycles of single colony isolation on rich medium to generate lineages that are forced through

205 When irradiated log phase WT cells were in rich medium, two TS domains were evident: a fast-repaire

206 In nutrient-rich medium, UgtP is distributed more or less uniformly

207 ls to those grown to midexponential phase in rich medium under aerobic conditions.

208 he resulting mutants revealed that growth in rich medium under low-O(2) conditions was indistinguisha

209 icant selective disadvantage just growing on rich medium under normal conditions.

210 MP cyclase gene dacA is unable to survive in rich medium unless it acquires compensatory mutations.

211 ccumulate PHB when it is grown in a nutrient-rich medium (up to 23% of the cell dry weight in dextros

212 ved metabolites was also achieved in sulfate-rich medium upon addition of the protonophore carbonyl c

213 In rich medium, V. cholerae expresses almost exclusively th

214 dition, regulation of citB(Lm) expression in rich medium was growth phase dependent; during exponenti

215 Endospore formation in amino-acid-rich medium was significantly defective and germination

216 tationary-phase cultures in glucose-depleted rich medium was truncated by switching the cells to phos

217 es, spore germination, and optimal growth on rich medium, was predictive of the contributions of two

218 In rich medium we detected transcripts for 97% and 87% of t

219 Compared to E. coli cultivated in the rich medium, we found that E. coli colonized in tumors n

220 Genes required for viability of E. coli in rich medium were identified on a whole-genome scale usin

221 PBPs possess largely redundant functions in rich medium, when divalent cations are limiting, PBP1 is

222 lta with respect to ste11(+) derepression in rich medium, whereas the opposite relationship, gcn5Delt

223 due to indicator accumulation in the Ca(2+)-rich medium, which induces an increase in the fluorescen

224 pletely spliced during exponential growth in rich medium, which suggests that meiosis is not the only

225 ysine-supplemented defined medium or complex rich medium, while the BCG mutants grow only on lysine-s

226 C. albicans yeast cells were grown in rich medium with 2% glucose.

227 In contrast, a MC grown in nutrient rich medium with addition of SCB had a lower microbial d

228 In rich medium with high amino acid content further incubat

229 Supplementing rich medium with short, branched-chain fatty acids or de

230 ons confer sensitivity to growth in nutrient-rich medium (YES) that is accompanied by nuclear poly(A)