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1 xposed to visible light and photosensitizer (rose bengal).
2 nglet oxygen sensitizer ([Ru(bpy)(3)](2+) or Rose Bengal).
3 nglet oxygen generation efficiency over free rose bengal.
4 ere varied to alter the fraction of adsorbed rose bengal.
5 of efficient photodynamic therapies based on rose bengal.
6 drazines in visible light, photocatalyzed by rose bengal.
7 ated photodynamically by the photosensitizer Rose Bengal.
8 s, which do not express mucins, stained with rose bengal.
9 of islands of stratified cells that excluded rose bengal.
10 PDAT was performed by applying a solution of rose bengal (0.1% or 0.2% RB in balanced salt solution)
13 we show the in vitro photodynamic effect of rose bengal activated by intracellular generation of lig
14 d time to carotid artery occlusion following Rose Bengal administration and laser-induced arterial in
17 groups: Group 1, no treatment; Group 2, 0.1% rose bengal alone; Group 3, 518 nm irradiation alone; Gr
18 illators are conjugated with photosensitizer rose bengal and arginylglycylaspartic acid (RGD) peptide
20 ico characterization of interactions between rose bengal and cationic poly(amidoamine) (PAMAM) and po
21 etal-free multicatalytic system comprised of rose bengal and cesium carbonate allowed the efficient f
23 ts show that the strong affinity of PPI-5 to Rose Bengal and erythrosine B is attributed to the good
25 ation of the mechanism of the ocular surface rose bengal and fluorescein staining that occurs in this
26 l retinal artery by intravenous injection of rose bengal and green laser irradiation of the artery.
28 Retinal vascular occlusion was introduced by rose Bengal and laser photocoagulation on chimeric mice
31 noparticles was developed, which showed that Rose Bengal and Rapa have high non-specific encapsulatio
32 ravenous injection of the photosensitive dye rose bengal and skull irradiation with a beam of focused
33 43 nm were prepared and functionalized with rose bengal and sulforhodamine B by a ligand-exchange pr
35 e-doped upconversion nanoparticle-conjugated rose bengal and triphenylphosphonium (LD-UCNP@CS-Rb-TPP)
36 the presence of a sensitizer (riboflavin or rose Bengal) and O(2), then incubated with a Cys-contain
37 et oxygen yield of a common photosensitizer (Rose Bengal) and the theoretical electric field enhancem
38 itive to peroxide but not methyl viologen or Rose Bengal, and GPXs, APX, and MSRA2 genes (encoding gl
40 ein mouse models with the photosensitive dye Rose bengal, and monitored plaque formation in real time
41 synthetic photosensitizers (methylene blue, rose bengal, and nitrite) and two model natural photosen
43 n the DG, the VGLUT inhibitors Congo Red and Rose Bengal, and the mGlu2/3 agonist LY354740, also redu
44 sis in wild-type (WT) and Apoh-/- mice using rose bengal- and FeCl3-induced carotid thrombosis, laser
46 0.1% rose bengal; Group III, MRSA with 0.03% rose bengal; and Group IV, MRSA with 0.1% riboflavin.
47 on products of SQ incubated in solution with Rose Bengal as a photooxidizer were isolated by semiprep
48 of imidazo[1,2-a]pyridine heterocycles using rose bengal as a photoredox catalyst at room temperature
49 o-2,3-dihydrobenzofuran-2-carboxylates using rose bengal as a triplet photosensitizer at ambient temp
51 idines with alcohols has been achieved using rose bengal as an organic photoredox catalyst at room te
54 as been achieved using a catalytic amount of rose bengal as an organophotoredox-catalyst and tert-but
55 presence of the photooxidants riboflavin and Rose Bengal as well as the diffusible one-electron oxida
59 rmula: see text] inhibitor sulfasalazine and rose bengal, but not by system L inhibitor 2-aminobicycl
60 n the targeted artery, we photoactivated the rose bengal by illuminating the longitudinal hippocampal
61 tion include the use of metal-free, low-cost Rose Bengal catalyst and practical operation (ambient te
62 h MRSA strains was demonstrated (1) for both rose bengal concentrations under ambient and green LED i
64 aggregation inhibition by ERB analogs except rose bengal correlated well to the inhibition of Abeta c
69 ed manner using photodynamic thrombosis with rose bengal dye and thermal burns from an argon laser wi
71 neas were treated on the incision walls with rose bengal dye followed by exposure to 514-nm laser rad
73 N ischemia was generated using laser-coupled rose Bengal dye photoactivation, and the infarct localiz
74 ity laser illumination of mice injected with Rose Bengal dye to induce photochemical injury in the re
76 ed in adult rats by intravenous injection of Rose Bengal dye, followed by argon green laser treatment
78 up I, MRSA control; Group II, MRSA with 0.1% rose bengal; Group III, MRSA with 0.03% rose bengal; and
79 olerance against stress induced by Paraquat, Rose Bengal, heavy metal, and the synthetic auxins 1-nap
82 othiocyanate, and rose Bengal), the CsPbI(3)-rose Bengal hybrid with the strongest binding showed the
83 ls at ambient temperature in the presence of rose bengal in acetonitrile under an oxygen atmosphere.
90 d to these stratified cells, indicating that rose bengal is excluded from cells that lack negative ch
92 We measured the binding of one inhibitor, rose bengal lactone (RBL), to kinesin (dissociation cons
94 2 carotid artery injury models (FeCl(3) and Rose Bengal/laser), fXII-deficient mice are more resista
98 o occlusion of the carotid after exposure to rose bengal or FeCl3 and reduced platelet and fibrin acc
99 intervals up to 16 days post infection (dpi) rose bengal or lissamine green B was instilled in the le
102 n of dGuo produced 5-Lys-Sp exclusively when Rose Bengal or methylene blue was used to photochemicall
103 g antibody were the negative controls, while rose bengal or protoporphyrin IX with visible light were
104 boflavin + 375 nm irradiation); and Group 5, rose bengal PDT (rose bengal + 518 nm irradiation).
106 r apical surfaces provide protection against rose bengal penetrance in vitro and suggest a role for m
109 DNA samples were extracted from brucellosis Rose Bengal plate test (RBPT) seropositive samples from
110 cell concentrations (8%RB and 3%RB), French rose bengal plate test with 4.5% cell concentration (4.5
111 id automated presumptive test (RAP), Mexican rose bengal plate tests with 8 and 3% cell concentration
113 cetonitrile medium and a catalytic amount of Rose Bengal provided the corresponding phenyl, benzyl, o
114 agreement with a model photosensitization by rose bengal (RB(2-)) in deoxygenated aqueous solutions r
116 d a phospholipid coating functionalised with Rose Bengal (RB) and/or 5-fluorouracil (5-FU), were asse
117 In this manuscript, we covalently attached Rose Bengal (RB) to the amphipathic peptide (AMP) C(KLAK
119 rategy to boost the lymphotropic delivery of Rose Bengal (RB), a hydrosoluble chemotherapeutic, is pr
120 esence of (1) (1)O(2) and (*)OH sensitizers [rose Bengal (RB), perinaphthanone, and H(2)O(2)] and (2)
121 rcially available xanthene dyes as ATRP PCs: rose bengal (RB), rhodamine B (RD), and rhodamine 6G (RD
122 served human amniotic membrane, stained with Rose Bengal (RB), was placed over a full-thickness wound
125 the vesicular glutamate transport inhibitor, Rose Bengal, reduced astrocytic glutamate release, sugge
126 mine whether exclusion of negatively charged rose bengal requires a negative charge at the cell surfa
127 pulate singlet and triplet excited states of rose Bengal, respectively, the mixed halide perovskites
128 zed separately by six triphenylmethane dyes (rose bengal, rhodamine B, crystal violet, ethyl violet,
130 hB), rhodamine isothiocyanate (RhB-NCS), and rose Bengal (RoseB)-contain an increasing degree of pend
131 bilised microbubbles (MBs), decorated with a Rose Bengal sensitiser, for SDT-based treatment of a pan
132 pression; however, HCLE cells incubated with rose bengal showed that exclusion of the dye was signifi
134 erated epi-off CXL using both riboflavin and rose bengal significantly increases resistance to enzyma
136 nifera (testate protists), including 'live' (Rose Bengal stained) and dead tests, in 5 cores (0-1 cm
137 of the eyes treated with NGF plus DHA showed rose bengal staining 30 days after PRK, compared with 50
139 ased epithelial proliferation, and decreased rose bengal staining compared with NGF, DHA, or vehicle
140 The current study was undertaken to evaluate rose bengal staining in a human corneal-limbal epithelia
142 at mucins have a protective role, preventing rose bengal staining of normal ocular surface epithelial
145 ye diagnosis (Lactoplate, Schirmer test, and Rose Bengal staining), even when the other test measures
146 chirmer testing with and without anesthesia, rose bengal staining, central corneal sensitivity, nucle
150 covery after surgery in goblet cell density, rose bengal staining, Schirmer test values without anest
157 ure to the three zoonoses using serological (Rose Bengal test for Brucella spp., ELISA for C. burnett
159 (-2) hour(-1) bar(-1) and 94.5% rejection to Rose Bengal) that surpasses the permeability-selectivity
160 rhodamine B, rhodamine B isothiocyanate, and rose Bengal), the CsPbI(3)-rose Bengal hybrid with the s
163 itive than culture for detection of HSV-1 in rose bengal-treated eyes, in that 74% of rose bengal-tre
164 in rose bengal-treated eyes, in that 74% of rose bengal-treated samples were positive by PCR compare
171 rescein analogs (ethyleosin, phloxine B, and rose bengal) were relatively potent inhibitors of bindin
173 ia, only methylene blue with E. coli K12 and rose bengal with C. jejuni showed an enhancing effect.