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1 ack of genetic interactions between gish and rutabaga.
2 hort-term memory formation, namely dunce and rutabaga.
3 -term synaptic plasticity, such as dunce and rutabaga.
4 ocess that requires a learning-related gene, rutabaga.
5                                       First, Rutabaga adenylyl cyclase (Rut-AC), a putative molecular
6         The dunce cAMP-phosphodiesterase and rutabaga adenylyl cyclase genes are necessary for two ke
7 on also does not interact genetically with a rutabaga adenylyl cyclase loss-of-function mutation.
8 hroom body lobes, which was dependent on the rutabaga adenylyl cyclase.
9                                          The rutabaga-adenylyl cyclase synthesizes cAMP in a Ca(2+)/c
10  tasks, and demonstrates that defects of the rutabaga and dunce products interact synergistically in
11 genes of the cAMP signaling pathway, such as rutabaga and NF1, suggesting that RDL works up stream of
12 sions from 183 B. napus (including rapeseed, rutabaga, and Siberian kale), 112 B. rapa, and 62 B. ole
13  conditioning, while individual heterozygous rutabaga/+ and octbeta1r/+ flies behave like the wild-ty
14              However, mutations that inhibit rutabaga Ca(2+)-stimulated adenylyl cyclase and dunce cA
15 s line was used to drive the expression of a rutabaga cDNA in otherwise rutabaga mutant flies.
16                             Induction of the rutabaga cDNA in the mushroom bodies only during adultho
17  observed additive effects when manipulating rutabaga co-expression in both structures.
18 ly, habituation was extremely rapid in dunce rutabaga double mutants.
19               Our experiments also show that rutabaga-encoded adenylate cyclase, which mediates coinc
20 of a genetic interaction between Nf1 and the rutabaga-encoded adenylyl cyclase (Rut-AC).
21              The transient expression of the rutabaga-encoded adenylyl cyclase in the mushroom bodies
22                  NF1 appears to regulate the rutabaga-encoded adenylyl cyclase rather than the Ras-Ra
23 anges linked to mechanisms controlled by the rutabaga-encoded adenylyl cyclase.
24 oom bodies, in a pattern coincident with the rutabaga-encoded adenylyl cyclase.
25              These data indicate that normal rutabaga function must be expressed in adulthood for nor
26                      These data suggest that rutabaga functions as a coincidence detector in an intac
27 hored by the adenylyl cyclase encoded by the rutabaga gene, is indispensable for olfactory memory for
28 mory to a rutabaga mutant with expression of rutabaga in different subsets of MB neurons.
29 s deficit and demonstrates an acute role for rutabaga in memory formation in these neurons.
30            Therefore, gish participates in a rutabaga-independent pathway for memory formation and ac
31       The Ca(2+)-responsive adenylyl cyclase RUTABAGA is believed to be a coincidence detector in gam
32 n was necessary and sufficient to rescue the rutabaga memory deficit, which rules out a developmental
33 e expression requirements for correcting the rutabaga memory impairment.
34 ion at least as strong as those of dunce and rutabaga, memory mutants with defective cAMP metabolism
35 e expression of a rutabaga cDNA in otherwise rutabaga mutant flies.
36  restore short-term or long-term memory to a rutabaga mutant with expression of rutabaga in different
37 some of the residual learning that occurs in rutabaga mutants.
38 cts the olfactory memory impairment found in rutabaga mutants.
39 d seizure satellites were both suppressed by rutabaga mutations that disrupt Ca(2+)/CaM-dependent ade
40 porting this notion, the double heterozygous rutabaga/+;octbeta1r/+ flies perform poorly in both aver
41               We found that flies mutant for rutabaga, period, and blistered were deficient for exper
42                                   In the MB, Rutabaga (Rut) adenylate cyclase acts as a coincidence d
43              For instance, expression of the Rutabaga (Rut) adenylyl cyclase in gamma neurons is suff
44                       Mutations of the genes rutabaga (rut) and dunce (dnc) affect the synthesis and
45 ) or long-term memory (LTM) was evaluated in rutabaga (rut) and dunce (dnc) mutants using aversive ph
46    Drosophila memory mutants dunce (dnc) and rutabaga (rut) are known to have altered intracellular c
47    Mutants of the Drosophila dunce (dnc) and rutabaga (rut) genes, which encode a cAMP-specific phosp
48                          The dunce (dnc) and rutabaga (rut) mutations of Drosophila affect a cAMP-dep
49 eport that cAMP signals misrelated in either rutabaga (rut) or dunce (dnc) mutants separate between c
50     Moreover, we find that adenylate cyclase Rutabaga (Rut) signaling via exchange protein activated
51 xpression of an adenylate cyclase encoded by rutabaga (rut), is sufficient to strengthen synaptic tra
52 s in Drosophila memory mutants revealed that rutabaga (rut)-dependent cAMP signals couple in a diverg
53 ulator of the cAMP pathway that involves the rutabaga (rut)-encoded adenylyl cyclase.
54                                 Mutations of rutabaga that diminish cAMP synthesis reduced the rate o
55 d plasticity, including the adenylyl cyclase Rutabaga, the Ig-CAM Fasciclin II, the transcription fac
56 nd that this plasticity was dependent on the Rutabaga type I adenylyl cyclase, linking cAMP-dependent
57                              Like mutants of rutabaga (which encodes a calcium/calmodulin-dependent a