ライフサイエンスコーパス: safranin

1 ape of a ring of cells that stained red with Safranin.

2 We have found that safranin also inhibits hGR in a noncompetitive fashion,

3 markers: acidic toluidine blue, alcian blue/safranin, and an antiserum to gonadotropin-releasing hor

4 pared hybrid system of GC/MWCNTs-NH2/Den/GDH/Safranin as anode in a membraneless enzyme-based glucose

5 We report on safranin-based stain that uniformly stains oocysts of Cy

6 -phenazinium chloride (safranin) showed that safranin bound at this same site.

7 es in young animals to mixed alcian blue and safranin granules in older animals, and an increase in G

8 of 200 kDa) were colored with two red dyes, Safranin O and Pararosanilin, selected to block the exci

9 Cartilage damage on safranin O histologic slides was quantified with Osteoar

10 s of cartilage proteoglycan (aggrecan) using Safranin O staining and antibodies to neoepitopes genera

11 stochemistry revealed a dramatic decrease in Safranin O staining and reduced anti-aggrecan staining (

12 4(-/-)) mouse model, we found differences in Safranin O staining intensity within the articular carti

13 Moreover, the Safranin O staining of cartilage formation in the endoch

14 lied to demonstrate morphologic changes, and Safranin O staining was performed to analyze the relatio

15 radation, and loss of glycosaminoglycans (by Safranin O staining).

16 dimethylmethylene blue assay, histology with Safranin O staining, and immunohistochemistry with anti-

17 an content was determined by alcian blue and Safranin O staining, CD44 protein expression by immunohi

18 dicated by microCT results, Alcian blue, and Safranin O staining.

19 Only repetitive loading induced loss of Safranin O staining.

20 cans and profoundly reduced the intensity of Safranin O staining.

21 , samples underwent histologic analysis with safranin O staining.

22 ted with VEGF, H&E, van Gieson, Mallory, and Safranin O stains.

23 nt of glucose dehydrogenase (GDH) enzyme and safranin O to amine-derivative multiwalled carbon nanotu

24 by ATP in permeabilized cells as measured by safranin O uptake.

25 plasma membrane permeable to succinate, ADP, safranin O, and other small molecules.

26 endochondral ossification as demonstrated by Safranin O, Picrosirius red, and aniline blue staining.

27 genic histological analysis was performed by Safranin O, Picrosirius red, and aniline blue staining.

28 Histologic analysis was performed using Safranin O-fast green staining, and articular cartilage

29 ted surface layer of variable thickness with Safranin O-positive formations sometimes present, a roug

30 However, Safranin O-stained sections from the Jnk2(-/-) mice exhi

31 The loss of aggrecan in Safranin O-stained sections was quantified by morphometr

32 Safranin O-staining revealed that cellular constructs de

33 ubricin-specific monoclonal antibody 9G3 and Safranin O.

34 Safranin-O staining shows that tissue-engineered cartila

35 l time points to tissue composition found in Safranin-O-stained sections of young bovine knee cartila

36 -2,8-dimethyl-5-phenyl-phenazinium chloride (safranin) showed that safranin bound at this same site.

37 d full fluorescence, modified acid-fast- and safranin-stained smears of Cryptosporidium and Cyclospor

38 (ii) in modified acid-fast-, trichrome-, and safranin-stained smears, and (iii) with two commercial t

39 m of the patient's isolate and determined by safranin staining.

40 ocedures, such as the modified acid-fast and safranin stains, are generally employed.

41 s either the modified Kinyoun's acid-fast or safranin stains, which are not part of the standard ova-

42 hromotrope, Gram-chromotrope, acid-fast, and safranin stains.