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1 structures during blood meal acquisition and salivation.
2 ed for blood glucose and pilocarpine-induced salivation.
3 mediated analgesia, tremor, hypothermia, and salivation.
4 nausea, anorexia, weight loss, and increased salivation.
5 P5), which are membrane proteins involved in salivation.
6 e transcriptional machinery immediately upon salivation.
7 sent a novel mechanism for the regulation of salivation.
8 ve agonist JWH133 (4 mg/kg) had an effect on salivation.
9 so prevented the odorant-induced increase in salivation.
10 d: eating and drinking, eating distress, and salivation.
11 oid anandamide and related lipids, regulates salivation.
12                    But what about stimulated salivation?
13 8/120, 73 % vs. 26/55, 47 %, pcorr = 0.024), salivation (87/120, 73 % vs. 23/55, 42 %, pcorr = 0.004)
14  3b induced a brief "serotonin syndrome" and salivation, an indication of 5-HT1A receptor activation.
15 ve motor activity, sensorimotor integration, salivation, and visceral regulation.
16  neostigmine included abdominal pain, excess salivation, and vomiting.
17 ist CP55940 (0.5 mg/kg, IP) lowered baseline salivation, as shown previously, but also prevented the
18 t at the parotid gland to inhibit stimulated salivation but also in the olfactory system, where funct
19 ore, prolonged host plant phloem exposure to salivation by RSV-infected adults should further enhance
20 mmunohistochemistry and tested regulation of salivation by THC and cannabinoid-related ligands.
21 eated with clozapine experienced more excess salivation, dizziness, and sweating and less dry mouth a
22 inicopathological factors influence PROs for salivation, eating and drinking, and eating distress.
23  oocysts to salivary glands, and from forced salivation experiments.
24                 P2 receptor agonists induced salivation in an ex vivo submandibular gland preparation
25 receptor agonist CP55940 (0.5 mg/kg) reduced salivation in both male and female mice 1 h after treatm
26 s and contributes a large portion of the net salivation in humans.
27 elivery of CA12 to salivary glands increases salivation in mice and of the human mutation CA12(E143K)
28                                              Salivation in response to pilocarpine stimulation was re
29 pe, aphids displayed a different duration of salivation in the phloem.
30 inently inhibited ductal fluid secretion and salivation in vivo.
31                                              Salivation is easily taken for granted, but without norm
32 outh prompted a study that showed that basal salivation is likely regulated by cannabinoid CB1 recept
33                              In summary, the salivation kinetics of the 3 major glands are distinct,
34 ion within the salivary glands cause reduced salivation leading to xerostomia.
35                                Consequently, salivation occurs in response to agonists that generate
36 ctivated pharyngeal swallowing (PS), profuse salivation, or physiological correlates of emesis.
37 the Na-K-2Cl cotransporter NKCC1 in hearing, salivation, pain perception, spermatogenesis, and the co
38 5 mg/kg, s.c.) or unchanged (15 mg/kg, s.c.) salivation responses, respectively, in M(1) and M(3) rec
39 for only a small portion of variation on the salivation scale (R2 = 14.0%).
40 to a muscarinic agonist, the primary in situ salivation signal.
41 easily taken for granted, but without normal salivation, simple essential tasks such as chewing and s
42 g, was characterized for pilocarpine-induced salivation, the presence of serum autoantibodies, sialoa
43 s system, activating CB1 receptors to reduce salivation, thus offering a mechanism underlying the dry
44 r episodes), dilated pupils (four episodes), salivation (two episodes), dryness of mouth (two episode
45 tigated cannabinoid regulation of stimulated salivation using functional and protein-expression studi
46                                              Salivation was reduced in FAAH knockout mice as well as
47 ntly, purinergic receptor agonist-stimulated salivation was suppressed by more than 70% in submandibu
48 ch as hypothermia, hypoactivity, tremor, and salivation were enhanced in GRK5-KO animals.
49 o-M-induced hypothermia, hypolocomotion, and salivation were markedly reduced in these animals, while