ライフサイエンスコーパス: sciatic

1 and SB216763) in Schwann cells as well as in sciatic and facial nerves.

2 En bloc sciatic and femoral nerve resection can be performed dur

3 ber diameter and myelin thickness within the sciatic and phrenic nerves.

4 rmalities are likely to reflect pathology in sciatic and tibial nerve fibers.

5 athy, demonstrating strong associations with sciatic and tibial nerve findings.

6 FA of the sciatic and tibial nerves was lowest in the sDPN group.

7 anti-inflammatory mechanism mediated by the sciatic and vagus nerves that modulates the production o

8 Complete sciatic, partial sciatic, and complete femoral nerve resection was perfor

9 second from the spinal cord; and (2) cortico-sciatic associative (CSA) protocol, in which the first s

10 associative protocols were tested: (1) spino-sciatic associative (SSA) protocol, in which the first s

11 rn identical to those of triplet proteins in sciatic axons and colocalizes with NFL on single neurofi

12 e dramatically reduces peripherin content in sciatic axons.

13 morphological measures of motor axons after sciatic crush in all tested conditions.

14 nal cord injuries with spasticity, spinal-to-sciatic DCS reduced transit and steady stretch-induced n

15 from spinal cord to sciatic nerve [spinal-to-sciatic direct current stimulation (DCS)] would inhibit

16 Mice were followed for 28 days for sciatic function index (SFI), and sciatic nerves and hin

17 Mechanistically, intra-sciatic MBP(84-104) induced phospholipase C (PLC)-driven

18 lleviated hind limb digital sensory, but not sciatic motor, nerve conduction slowing and thermal and

19 after unilateral constriction injury to the sciatic nerve (DMG) and in the contralateral control leg

20 d genes in non-neuronal cells located in the sciatic nerve (potentially representing Schwann cells (S

21 rsisted in the dorsal root ganglia (DRG) and sciatic nerve (SN) for up to 72 hours.

22 The sciatic nerve (SN) showed increased nonuniform, internod

23 egrated proteomics and metabolomics from the sciatic nerve (SN), the lumbar 4/5 dorsal root ganglia (

24 ) direct current flowing from spinal cord to sciatic nerve [spinal-to-sciatic direct current stimulat

25 Here, we report that sciatic nerve activation with electroacupuncture control

26 treatment also promoted remyelination of the sciatic nerve after crush.

27 A conditioning lesion of the sciatic nerve allows the central processes of dorsal roo

28 egeneration in dorsal root ganglia (DRG) and sciatic nerve and abundance of Schwann cells.

29 ere found in increased concentrations in the sciatic nerve and dorsal root ganglia of oxaliplatin tre

30 lso displayed a unique prolonged exposure in sciatic nerve and DRG.

31 ory components was evaluated in axons of the sciatic nerve and in spinal nerve axons after in vivo el

32 ssociated with a persistent M1 milieu in the sciatic nerve and in the regional and systemic lymphatic

33 having reliable signal acquisitions from the sciatic nerve and its branches such as the peroneal nerv

34 a small proportion of fibers that constitute sciatic nerve and its branches, but importantly breaks t

35 regeneration of growth-competent axons after sciatic nerve and spinal cord injury.

36 nction, intraepidermal nerve fiber loss, and sciatic nerve and spinal cord oxidative-nitrative stress

37 nduced by chronic constriction injury of the sciatic nerve and suppressed nerve injury-induced activa

38 e found an up-regulation of the HCAR2 in the sciatic nerve and the dorsal root ganglia in neuropathic

39 lly well in the lymph node, infection of the sciatic nerve and the rest of the nervous system was imp

40 which the first stimulus originated from the sciatic nerve and the second from the motor cortex.

41 which the first stimulus originated from the sciatic nerve and the second from the spinal cord; and (

42 ing required to prevent axonal damage of the sciatic nerve and to terminate the P0106-125-specific im

43 We used a chronic constriction injury of sciatic nerve as a model of neuropathic pain in male Spr

44 tivity, and morphological alterations in the sciatic nerve as evidenced by decrease in g-ratio; it al

45 Analysis of the sciatic nerve at 11 d after nerve crush showed that the

46 cord, and, during their apoptosis induced by sciatic nerve avulsion, nuclear and cytoplasmic 5-methyl

47 otyping after selectively silencing TRPV1(+) sciatic nerve axons by perineural injections of QX-314 a

48 , degenerating) nerve stumps on day 1 in the sciatic nerve axotomy model in rats.

49 l enrichment (EE) followed by a conditioning sciatic nerve axotomy that precedes a spinal cord injury

50 We evaluated the utility and efficiency of sciatic nerve block as an alternative method to relieve

51 nts who received ultrasound-guided popliteal sciatic nerve block for the relief of severe rest pain d

52 Ultrasound-guided popliteal sciatic nerve block provides effective pain control, whi

53 reveal major structural changes at proximal sciatic nerve branches or distal toe nerve fascicles at

54 Two-site stimulation of sciatic nerve bundles by MOSD induces precise extension

55 r was generated and delivered into the mouse sciatic nerve by a single injection immediately distal t

56 Here, we validate EIT imaging in rat sciatic nerve by comparison to micro-computed tomography

57 Infiltration of cytotoxic NK cells into the sciatic nerve by extravasation occurs within 3 days foll

58 vivo chromatin immunoprecipitation (ChIP) of sciatic nerve cells revealed a Sox10 binding site upstre

59 ng techniques, we have used a rodent partial sciatic nerve chronic constriction injury model (n = 5-8

60 Sciatic nerve conditioning at A-delta fiber strength, kn

61 elinated nerve fibers, specifically sural or sciatic nerve conduction velocities, but significantly i

62 Furthermore, treated mice showed increased sciatic nerve conduction velocities, improvement of myel

63 rmance, quadriceps muscle contractility, and sciatic nerve conduction velocities.

64 athy remarkably ameliorated DPN by improving sciatic nerve conduction velocity and increasing thermal

65 mechanical hyperalgesia, cold allodynia, and sciatic nerve conduction velocity.

66 phagic flux in skeletal muscle 14 days after sciatic nerve constriction.

67 Electroacupuncture at the sciatic nerve controls systemic inflammation by inducing

68 O mice were subjected to neuropathic pain by sciatic nerve crash injury (SNI).

69 tion was impaired in CLU(-/-) mice following sciatic nerve crush and impaired regeneration nerve fibe

70 upregulated via MEK/ERK signaling and after sciatic nerve crush and Neto2(-/-) neurons from adult mi

71 nd conduction velocities consequent to acute sciatic nerve crush compared with wild-type control anim

72 Regeneration of axons was examined after sciatic nerve crush in pre- and symptomatic SOD1(G93A) m

73 kine IL-10 in terminating inflammation after sciatic nerve crush injury and promoting regeneration.

74 ution of inflammation and regeneration after sciatic nerve crush injury in mice.

75 One day sciatic nerve crush injury triggered a robust increase i

76 Sciatic nerve crush injury triggers sterile inflammation

77 Male Sprague Dawley rats were subjected to a sciatic nerve crush under anesthesia and mechanical thre

78 Adult rats with sciatic nerve crush were immediately and systemically in

79 genic mice enhanced locomotor recovery after sciatic nerve crush, associated to an improvement in key

80 After sciatic nerve crush, functional recovery in vivo was ret

81 The resulting mice were challenged with sciatic nerve crush.

82 changes in wild-type and fat-1 mice after a sciatic nerve crush.

83 ation to augment neuroregeneration in both a sciatic nerve cut-and-repair and rat hindlimb transplant

84 Rats undergoing sciatic nerve cut-and-repair and treated with either loc

85 Sciatic nerve CuZnSOD content in SynTgSod1(-/-) mice was

86 liminary in vivo studies in a critical-sized sciatic nerve defect in Wistar rats confirmed conduit su

87 jected to unilateral partial ligation of the sciatic nerve developed significant mechanical and therm

88 d region (3'UTR) landscapes after unilateral sciatic nerve entrapment (SNE) injury in rats.

89 ng in a rat neuropathy induced by unilateral sciatic nerve entrapment (SNE).

90 ties vanished, and the ultrastructure of the sciatic nerve exhibited numerous tomacula and remyelinat

91 l proteins at the nodes of Ranvier of teased sciatic nerve fibers.

92 blished model of complete transection of the sciatic nerve for comparison, we observed similar but mo

93 ated a mouse model in which a portion of the sciatic nerve from one hind limb was transected at postn

94 and transmission electron microscopy of the sciatic nerve from one of the affected individuals revea

95 te was as effective as glucose in supporting sciatic nerve function, and was continuously released in

96 en assessed using von Frey filaments and the sciatic nerve functional index.

97 no significant long-term adverse effects on sciatic nerve functions.

98 supporting the re-innervation across a 10 mm sciatic nerve gap, with results close to that of the aut

99 through the loss of Limk1, results in faster sciatic nerve growth, and improved recovery of some sens

100 Single-cell RNA-sequencing of injured sciatic nerve identifies five macrophage subpopulations,

101 ting diodes embedded in a soft, circumneural sciatic nerve implant, powered and driven by a miniaturi

102 Ultrastructural analysis of adult sciatic nerve in GluN1- mice revealed increases in the d

103 ed low-voltage electrical stimulation of the sciatic nerve in live mice.

104 ord in females, but remains localized to the sciatic nerve in males (tier 3).

105 When injected proximal to the sciatic nerve in mice via intramuscular (i.m.) injection

106 by chronic constriction injury (CCI) of the sciatic nerve in mice, was related to both an increase i

107 eas western blotting detected PrP(Sc) in the sciatic nerve in one VV2 and one MV2K.

108 Transection of the sciatic nerve in the M83 Tg mice significantly delayed t

109 P was expressed throughout the length of the sciatic nerve in up to 50% of Schwann cells starting 2 w

110 Chronic constriction injury (CCI) of the sciatic nerve induced IL-33 production in the spinal cor

111 uency (0.2 or 3 Hz) stimulation (LFS) to the sciatic nerve induced long-term potentiation (LTP) of C-

112 ory neurons following direct intraganglionic sciatic nerve injection and intraperitoneal and intraven

113 orn would be altered by chronic constriction sciatic nerve injury (CCI).

114 peralgesia using a rat model of constriction sciatic nerve injury (CCI).

115 ys a critical role in neuropathic pain after sciatic nerve injury and bone cancer in rodents.

116 the mediodorsal thalamus (MD) to ACC, using sciatic nerve injury and chemotherapy-induced mouse mode

117 action against ER stress, in mouse models of sciatic nerve injury and found that ablation of the tran

118 pes in vitro and ameliorate a critical-sized sciatic nerve injury and its associated defects in a mur

119 of dorsal root ganglia (DRGs) neurons after sciatic nerve injury in the rat.

120 sures of allodynia in a chronic, neuropathic sciatic nerve injury model, but tolerance to morphine de

121 were protected from hypersensitivity in two sciatic nerve injury models.

122 Sciatic nerve injury triggered generation of two-chain S

123 tage-dependent anion channel 1 (VDAC1) after sciatic nerve injury triggers Schwann cell demyelination

124 Hyperalgesia gradually developed after sciatic nerve injury, and by the last day of testing, TH

125 Within hours of a sciatic nerve injury, the level of phosphorylated cofili

126 crease in ultrasonic, broadband clicks after sciatic nerve injury, which was reversed by THC, CBD, an

127 r mechanical hypersensitivity, or in partial sciatic nerve injury-induced mechanical allodynia.

128 ing the same population of neurons following sciatic nerve inoculation.

129 Here, we show that peripheral sciatic nerve lesion in adult mice leads to elevated lev

130 was to evaluate the functional relevance of sciatic nerve lesions in DPN, with the expectation of co

131 tic polyneuropathy (DPN) have found proximal sciatic nerve lesions.

132 e noxious stimuli, but subsequent to partial sciatic nerve ligation (PNL), KOs did not develop mechan

133 nitiation of neuropathic pain in the partial sciatic nerve ligation (PSNL) mouse model.

134 null mutant mice are protected from partial sciatic nerve ligation (PSNL)-induced neuropathic pain,

135 of behavioral hypersensitivity after partial sciatic nerve ligation (PSNL).

136 mplete Freund's adjuvant (CFA) or by partial sciatic nerve ligation (PSNL).

137 Neuropathic pain was induced by partial sciatic nerve ligation (which induces hypersensitivity t

138 mycin treatment of human SMA fibroblasts and sciatic nerve ligation in SMA mice provoked robust phosp

139 lations of murine models, we have shown that sciatic nerve ligation induces a re-emergence of immatur

140 tive properties in the capsaicin and partial sciatic nerve ligation models in mice.

141 R-neutralizing antibody to rats with partial sciatic nerve ligation resulted in a delayed onset of ne

142 We found that in mice with partial sciatic nerve ligation, TRPA1 silencing in nociceptors a

143 anscriptomic and spatial characterization of sciatic nerve macrophages (snMacs).

144 Ki 60 nM) and also completely reversed mouse sciatic nerve mechanoallodynia (in vivo, 3 mumol/kg, po)

145 in vitro findings in vivo using a transected sciatic nerve model.

146 cifically colocalize with SMIT1 and SMIT2 at sciatic nerve nodes of Ranvier and in axon initial segme

147 ed into partially denervated branches of the sciatic nerve of adult mice.

148 The sciatic nerve of Ahr knockout mice exhibited both reduce

149 in vivo during the first postnatal week, the sciatic nerve of all 3 conditional KO animals displayed

150 cuff-mounted and acutely implanted onto the sciatic nerve of anaesthetized rats, the device conferre

151 pathic pain was induced by cuffing the right sciatic nerve of C57BL/6J mice.

152 tudied mitochondrial transport in the intact sciatic nerve of living mice and analyzed axonal mitocho

153 ice after chronic constriction injury of the sciatic nerve of the left hindpaw and observed a strikin

154 ticularly in the spinal cord, but not in the sciatic nerve of the PNS.

155 nodal expression of selected isoforms in the sciatic nerve of the transgenic mouse Oct6(DeltaSCE/beta

156 ssion was upregulated in the spinal cord and sciatic nerve of WT mice.

157 were performed in dorsal root ganglia after sciatic nerve or dorsal column axotomy.

158 ic with streptozotocin displayed less severe sciatic nerve oxidative-nitrative stress and peripheral

159 n, whereas wild-type mice developed complete sciatic nerve paralysis due to massive CPNI.

160 eported that myelin clearance in the injured sciatic nerve proceeds unhindered in the Ccr2(-/-) mouse

161 tion of 6 mul of 150 mum ATP into female rat sciatic nerve quadrupled the number of axons growing int

162 ined GSK3 activity show markedly accelerated sciatic nerve regeneration.

163 e (M6P), a scar reducing agent, to a site of sciatic nerve repair.

164 ize independently after complete and partial sciatic nerve resection, respectively.

165 ieved after pelvic exenteration with en bloc sciatic nerve resection.

166 roduced by placing a plastic cuff around the sciatic nerve resolved within several days when the cuff

167 Molecular interrogation of the sciatic nerve reveals that aged Schwann cells (SCs) fail

168 iation of pain along the distribution of the sciatic nerve should always be looked for if abnormaliti

169 t this time, ultrastructural analysis of the sciatic nerve showed de- and dysmyelination of fibers, w

170 sthetised, ventilated dogs were elicited via sciatic nerve stimulation (50 Hz; 200 ms duration; 1 con

171 TP) following conditioning by high-frequency sciatic nerve stimulation (HFS) at intensities recruitin

172 oration of direct muscle responses evoked by sciatic nerve stimulation to pretransection levels over

173 ns (50 Hz; 200 ms duration) via supramaximal sciatic nerve stimulation were used to manipulate metabo

174 This study investigated the association of sciatic nerve structural damage in 3 Tesla (3T) magnetic

175 -positive vesicular clusters in axons in the sciatic nerve suggest that this denervation results from

176 titative RT-PCR and Western blot analysis of sciatic nerve tissues showed that SC-Exo treatment rever

177 thanized, and blood, urine, spinal cord, and sciatic nerve tissues were collected.

178 rve injury, we demonstrate that exposing the sciatic nerve to MBP(84-104) via endoneurial injection p

179 ent unilateral electrical stimulation of the sciatic nerve to mimic resistance exercise in the tibial

180 glycoprotein gp120 application onto the rat sciatic nerve to test the role of phosphorylated C/EBPbe

181 ontrol and injury conditions at 3 days after sciatic nerve transection (SNT).

182 d time course in a more severe injury model, sciatic nerve transection and reanastamosis.

183 Sciatic nerve transection induced upregulation of OPN an

184 ttern nor the number of neurons changed in a sciatic nerve transection model of neuropathic pain or i

185 as applied to bridge injured nerves in a rat sciatic nerve transection model.

186 ided into 5 groups, including naive control, sciatic nerve transection or repair, immediate transplan

187 randomly assigned to four different types of sciatic nerve transection: Simple Transection (ST), Simp

188 Sciatic nerve transections and repairs were performed in

189 ) via electrical stimulation (e-stim) of the sciatic nerve twice a week for 8 weeks.

190 After CCI, upregulation of CD11b in sciatic nerve was less in GFAP-IkappaBalpha-dn mice comp

191 hronic constriction injury (CCI) of the left sciatic nerve was performed on wild type (WT) and GFAP-I

192 following chronic constriction injury of the sciatic nerve was rapidly and dose-dependently reversed

193 In anaesthetised C57-Black-6 mice, the left sciatic nerve was sectioned and repaired using 4 epineur

194 ata for 24-week-old BKS db/db and db/+ mouse sciatic nerve were analyzed to define significantly diff

195 as confirmed by histological analysis of the sciatic nerve which showed predominantly axonal damage:

196 ignals were recorded simultaneously from the sciatic nerve with a 16-contact cuff electrode.

197 icipants underwent 3-T MR neurography of the sciatic nerve with a T2-weighed fat-suppressed sequence,

198 estigate this, we inoculated hamsters in the sciatic nerve with long-incubation-period strain 139H pr

199 hsTNT showed a negative correlation with the sciatic nerve's FA (r = -0.52, P < 0.001), with a closer

200 The sciatic nerve's fractional anisotropy (FA), a marker of

201 DTI-MRN covered proximal (sciatic nerve) and distal (tibial nerve) nerve segments

202 ring rate and stimulus-evoked (brush, pinch, sciatic nerve) responses were markedly enhanced as were

203 e neurons (CSNs) in DRGs contributing to the sciatic nerve, and a decrease in their cold temperature

204 al day 7 onward in motoneurons, axons in the sciatic nerve, and axon terminals of the neuromuscular j

205 assays, immunohistochemistry with teased rat sciatic nerve, and immunoabsorption experiments.

206 uff is well-tolerated, delivers light to the sciatic nerve, and optically stimulates muscle in freely

207 e points in CMT2D mouse muscles, retina, and sciatic nerve, as well as in embryonic hindbrain.

208 ncreased 24 h after oxaliplatin treatment in sciatic nerve, DRGs, or spinal cord tissue as revealed b

209 muscle with and was seen tracking along the sciatic nerve, explaining pain along the distribution of

210 more than 100 O-GlcNAcylated proteins in rat sciatic nerve, including Periaxin (PRX), a myelin protei

211 Glycogen was present in sciatic nerve, its concentration varying directly with a

212 We demonstrate that in sciatic nerve, myocilin is expressed in Schwann cells wi

213 In mouse sciatic nerve, myosin-1d is expressed along the axon and

214 ion in the leg along the distribution of the sciatic nerve, secondary to compression or irritation of

215 n response was identified in neural tissues (sciatic nerve, spinal cord) of streptozotocin diabetic r

216 Four weeks after axonal crush of sciatic nerve, TDP-43 transgenic mice remained paralyzed

217 The infection spread to the sciatic nerve, the spinal cord, and the brain, causing p

218 tokes Raman scattering (CARS) imaging of the sciatic nerve, we deciphered the spatiotemporal choreogr

219 of perineural HIV gp120 application onto the sciatic nerve, we found that pC/EBPbeta was triggered by

220 ort the concept that HFS conditioning of the sciatic nerve, which leads to spinal LTP, is associated

221 r perianal abscess causing irritation of the sciatic nerve, which was diagnosed on MRI of the lumbosa

222 ster of differentiation 45 expression in the sciatic nerve, with no difference between genotypes.

223 Surprisingly, sciatic nerve-infiltrating CD4(+) cells had an expansion

224 xplaining pain along the distribution of the sciatic nerve.

225 econdary to compression or irritation of the sciatic nerve.

226 ecorded spike activity from the regenerating sciatic nerve.

227 lammatory macrophages, was upregulated in KO sciatic nerve.

228 in response to electrical stimulation of the sciatic nerve.

229 implant is sutured between the stumps of the sciatic nerve.

230 of circulating CX3CR1(+) monocytes into the sciatic nerve.

231 s following extraction of the Xenopus laevis sciatic nerve.

232 mes greater at the spinal column than at the sciatic nerve.

233 ronic constriction injury (CCI) model of the sciatic nerve.

234 nance of myelination and nodes of Ranvier in sciatic nerve.

235 , as occurs with conditioning lesions of the sciatic nerve.

236 of a polyethylene cuff placed around in the sciatic nerve.

237 he dorsal root ganglion (DRG) and the distal sciatic nerve.

238 ll proliferation is upregulated in Chd4-null sciatic nerve.

239 following chronic constriction injury of the sciatic nerve.

240 ) meninges and in the epi/perineurium of the sciatic nerve.

241 olipid metabolites during myelination of the sciatic nerve.

242 ceptive neurons, increased CGRP release from sciatic nerves and DRGs, and a reduction in mechanical a

243 8 days for sciatic function index (SFI), and sciatic nerves and hind limb muscles were harvested for

244 howed presence of myc-tagged human SH3TC2 in sciatic nerves and lumbar roots in the perinuclear cytop

245 se reduces NMN accumulation in injured mouse sciatic nerves and preserves some axons for up to three

246 one marrow uptake and intense uptake in both sciatic nerves and right median nerve.

247 r3 knockdown attenuates myelination in mouse sciatic nerves as well as in zebrafish.

248 irmed expression of Cx32 in lumbar roots and sciatic nerves correctly localized at the paranodal myel

249 ponse to the loss of CCR2, injured Ccr2(-/-) sciatic nerves demonstrate prolonged expression of neutr

250 nt can be applied to measure excitability of sciatic nerves due to a stimulation of the footpad in co

251 Both autoradiography analysis of sciatic nerves excised from injured rats as well as whol

252 ilized to assess the morphology of optic and sciatic nerves following treatment, and the morphology a

253 electron microscopy, we show that injury of sciatic nerves from mice of either sex triggers extensiv

254 f gene expression arrays of large myelinated sciatic nerves from pioglitazone-treated animals reveale

255 Uninjured sciatic nerves in GluN1- mice did not demonstrate an inc

256 In response to crush injury, sciatic nerves in scLRP1(-/-) mice showed accelerated de

257 elow the incisures in the single mutants, in sciatic nerves of caspr(-/-)/caspr2(-/-) mice, these cha

258 oid precursor protein accumulated in ligated sciatic nerves of control and eribulin-treated mice, but

259 s found to be carbonylated and aggregated in sciatic nerves of dbdb mice.

260 Although the conduction velocity of sciatic nerves of mutant mice showed an 80% decrease, th

261 Sciatic nerves of myocilin null mice express reduced lev

262 Mtmr13 loss leads to axonal degeneration in sciatic nerves of older mice.

263 Electron microscopy of sciatic nerves of paclitaxel-treated mice showed reduced

264 Sciatic nerves of such mice showed thinner myelin of lar

265 s of demyelinated fibres in lumbar roots and sciatic nerves of treated Sh3tc2-/- mice.

266 Electron microscopic analysis of sciatic nerves showed a reduction in the number of neuro

267 ication of its target mRNAs in vivo in mouse sciatic nerves using ribonomics showed an enrichment of

268 appeared that myelin sheath thickness in the sciatic nerves was not increased in BACE1(-/-)/Akt-DD mi

269 conduction velocities of the trigeminal and sciatic nerves were decreased.

270 he cholesterol and sphingomyelin contents of sciatic nerves were greatly reduced and so was the numbe

271 turation, and functional recovery of injured sciatic nerves, and increased the ability of regeneratin

272 In sciatic nerves, application of extracellular QX-314 with

273 logy in isolated hearts, retinal tissues and sciatic nerves, as well as bioelectronic cardiac sensing

274 was injected directly into crush-injured rat sciatic nerves, ERK1/2 phosphorylation was observed in m

275 K3-mediated CRMP2 inhibition was detected in sciatic nerves, thus revealing a fundamental difference

276 n the cerebrum, cerebellum, heart, liver and sciatic nerves.

277 -N-SH cells and on axonal transport in mouse sciatic nerves.

278 b weakness and impaired axonal conduction in sciatic nerves.

279 r roots, as well as in the femoral motor and sciatic nerves.

280 n in the conduction velocity along the adult sciatic nerves.

281 ssion of the T cell chemoattractant IP-10 in sciatic nerves.

282 sed numbers of large-diameter axons in their sciatic nerves.

283 doxycycline-treated compared with untreated sciatic nerves.

284 epidermal nerve fibers and remyelination of sciatic nerves.

285 When joints were immobilized by sciatic neurectomy, regulation of selected genes was abr

286 ilized, either by prolonged anesthesia or by sciatic neurectomy.

287 that nerve conduction velocities between the sciatic notch and the gastrocnemius muscle were unchange

288 ggests that neuropathy may exist between the sciatic notch to the nerve rootlets.

289 a single injection immediately distal to the sciatic notch.

290 Advanced pelvic tumors involving the sciatic or femoral nerve have traditionally been conside

291 c resections, 68 patients (9.5%) had en bloc sciatic or femoral nerve resection.

292 pelvic surgery with en bloc resection of the sciatic or femoral nerves at a single center were includ

293 ts (n = 258) with lumbar disc herniation and sciatic pain, all European-Caucasian, were recruited fro

294 Complete sciatic, partial sciatic, and complete femoral nerve res

295 sphincter dysfunction, partially resembling sciatic symptoms.

296 Sciatic-to-spinal DCS caused opposite effects.

297 t current flowing in the opposite direction (sciatic-to-spinal DCS) would excite spinal motor neurons

298 n between the spinal cord and the periphery (sciatic transection), eliminates the capacity to learn,

299 euron output and that this effect survives a sciatic transection.

300 anglia, an increase in the peripheral nerve (sciatic) yet no change in the central root.