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1 ally affected in sciatic motor neurons after sciatic nerve injury.
2 ng gelatinases are thought to play a role in sciatic nerve injury.
3  activities were determined before and after sciatic nerve injury.
4 c cryoneurolysis, and a chronic constriction sciatic nerve injury.
5 roptosis, promotes functional recovery after sciatic nerve injury.
6 sensitivity (allodynia) following peripheral sciatic nerve injury.
7 s, and impairs axonal regeneration following sciatic nerve injury.
8 ys a critical role in neuropathic pain after sciatic nerve injury and bone cancer in rodents.
9  the mediodorsal thalamus (MD) to ACC, using sciatic nerve injury and chemotherapy-induced mouse mode
10 ired to mediate pain and inflammation during sciatic nerve injury and collagen antibody-induced arthr
11 action against ER stress, in mouse models of sciatic nerve injury and found that ablation of the tran
12 pes in vitro and ameliorate a critical-sized sciatic nerve injury and its associated defects in a mur
13 mice, which show increased proteolysis after sciatic nerve injury and lung fibrosis.
14                In this study, an in vivo rat sciatic nerve injury and regeneration model was combined
15 t mRNAs, Gap43, Snap25, and Fubp1, following sciatic nerve injury and these mice show accelerated ner
16       Hyperalgesia gradually developed after sciatic nerve injury, and by the last day of testing, TH
17  of genes are differentially expressed after sciatic nerve injury by microarray analysis.
18 orn would be altered by chronic constriction sciatic nerve injury (CCI).
19 peralgesia using a rat model of constriction sciatic nerve injury (CCI).
20 aviors in WKY rats with chronic constriction sciatic nerve injury (CCI).
21  model of the condition chronic constriction sciatic nerve injury (CCI).
22                                     However, sciatic nerve injury did result in significant post-axot
23 al nerve ligation (ED(50) = 47 mg/kg, i.p.), sciatic nerve injury (ED(50) = 85 mg/kg, i.p.), capsaici
24     In animals with combined spinal cord and sciatic nerve injury, folate-mediated CNS axon regenerat
25 e earliest documented molecular events after sciatic nerve injury in adult rats is the rapid, long-te
26  present studies of allodynia in response to sciatic nerve injury in male and female rats focus on th
27 ammatory neuronal degeneration, we performed sciatic nerve injury in mice.
28  of dorsal root ganglia (DRGs) neurons after sciatic nerve injury in the rat.
29  expression program at homeostasis and after sciatic nerve injury in wild-type and cohesin-deficient
30 ally at the injury site in multiple forms of sciatic nerve injury, including crush injury, chronic co
31 r mechanical hypersensitivity, or in partial sciatic nerve injury-induced mechanical allodynia.
32                                              Sciatic nerve injury instead induces axon regeneration t
33 ts the hypothesis that the c-jun response to sciatic nerve injury involves a distance-dependent signa
34 sures of allodynia in a chronic, neuropathic sciatic nerve injury model, but tolerance to morphine de
35  were protected from hypersensitivity in two sciatic nerve injury models.
36 is one explanation for the minimal effect of sciatic nerve injury on DC regeneration.
37                              No instances of sciatic nerve injury or compartment syndrome were observ
38 nsport increased 3.5-fold within 24 hr after sciatic nerve injury or dorsal rhizotomy.
39 ysis of ex vivo explants from mice following sciatic nerve injury showed that inhibition of Slit2 dec
40 plement membrane attack complex (MAC) at the sciatic nerve injury site.
41 c pain, we evaluated myelin integrity at the sciatic nerve injury site.
42  and preservation of myelin integrity at the sciatic nerve injury site.
43 nt in T cell signaling both before and after sciatic nerve injury (SNI) in mice.
44 its a transcriptional response distinct from sciatic nerve injury (SNI).
45                            Within hours of a sciatic nerve injury, the level of phosphorylated cofili
46                      When these mice undergo sciatic nerve injury to activate neurogenic inflammation
47 ut a fetal spinal implant, was combined with sciatic nerve injury to attempt to stimulate an intracel
48                                              Sciatic nerve injury triggered generation of two-chain S
49 tage-dependent anion channel 1 (VDAC1) after sciatic nerve injury triggers Schwann cell demyelination
50          NG2 expression also increased after sciatic nerve injury, wherein axons successfully regener
51 crease in ultrasonic, broadband clicks after sciatic nerve injury, which was reversed by THC, CBD, an