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1 the IgM IFA for the serological diagnosis of scrub typhus.
2 indirect immunofluorescence assay (IFA) for scrub typhus.
3 ientia tsutsugamushi, the causative agent of scrub typhus.
4 une responses and correlates of immunity for scrub typhus.
5 henotypic correlates of immune protection in scrub typhus.
6 ntia tsutsugamushi is the causative agent of scrub typhus.
7 as a vaccine candidate for the prevention of scrub typhus.
8 gens, Orientia spp., which cause a zoonosis, scrub typhus.
9 m that causes the vector-borne human disease scrub typhus.
10 or a combination of both in treating severe scrub typhus.
11 18 and CCL23 and the receptor CCR3 in severe scrub typhus.
12 s to cause the emerging global health threat scrub typhus.
13 could influence morbidity and mortality from scrub typhus.
14 singly, we identified leptospirosis (10.6%), scrub typhus (4.1%), dengue (3.7%), and Kyasanur forest
15 st commonly identified aetiologies were JEV, scrub typhus (645 [18.5%] of 3489), and dengue virus (16
16 appropriate antibiotic treatment for severe scrub typhus, a neglected but widespread reemerging zoon
18 e aromatic amino acids and histidine, causes scrub typhus, a potentially deadly infection that threat
21 JEV alone, and highlighted the importance of scrub typhus and dengue virus as important infectious ae
22 eeded for the treatment of murine typhus and scrub typhus, are not routinely advised for empirical tr
23 en a common problem with actual incidence of scrub typhus cases in Northeast India that were reported
24 Here, we report three autochthonous cases of scrub typhus caused by O. tsutsugamushi acquired on Chil
25 es (SFGR), typhus group rickettsioses (TGR), scrub typhus (caused by Orientia tsutsugamushi), ehrlich
28 ve specimens were additionally evaluated for scrub typhus, dengue virus, and West Nile virus by serum
29 8 (7.3%) met the clinical case definition of scrub typhus (detection of IgM against O. tsutsugamushi
34 pidemiologic and clinical characteristics of scrub typhus in 37 villages in Tamil Nadu, India, where
36 admission and convalescent-phase samples for scrub typhus indirect immunofluorescence assay using Bay
41 hain reaction (receptors) from patients with scrub typhus (n = 129), patients with similar febrile il
42 1.56-3.75), malaria (OR 2.00, CI 1.46-2.73), scrub typhus (OR 2.37, CI 1.41-3.96) and spotted fever g
43 itional 120 serum samples from tuberculosis, scrub typhus, or leptospirosis patients were evaluated i
44 y-acquired infections (LAIs) associated with scrub typhus (Orientia tsutsugamushi) and murine typhus
45 ombiculid species as vector and reservoir of scrub typhus outside the tsutsugamushi triangle, providi
47 autoimmunity is a concern, the reactivity of scrub typhus patient sera with purified recombinant 47-k
48 hailand raised a troubling observation; some scrub typhus patients responded poorly to doxycycline, w
51 d genetic element), previously identified in scrub typhus rickettsiae (Orientia tsutsugamushi) genome
52 erapeutic option for the treatment of severe scrub typhus than monotherapy with either drug alone.
53 olgus macaque (Macaca fascicularis) model of scrub typhus, the leading cause of treatable undifferent
54 s were performed for Orientia tsutsugamushi (scrub typhus), typhus group, and spotted fever group RD.
55 study of the immunogenicity of a recombinant scrub typhus vaccine candidate in a nonhuman primate mod
56 ulosis, leptospirosis, melioidosis, typhoid, scrub typhus), viral infections (hepatitis A, B, C, D, a
58 ho were 15 years of age or older with severe scrub typhus with at least one organ involvement were en