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1 ated on free-ranging otariids (fur seals and sea lions).
2 ity to assess the prevalence of ZcAV in live sea lions.
3 losely related to those adapted to seals and sea lions.
4 pi were obtained from control and chronic DA sea lions.
5 his mixing zone region have been optimal for sea lions.
6 since the last common ancestor of seals and sea lions.
7 achea, bronchi, and lungs from 11 California sea lions, 2 Northern elephant seals, and 10 rhesus maca
8 ith thick myelin sheaths (elephant seal: 9%, sea lion: 7%) and thin myelin sheaths (elephant seal: 91
10 the closely related isolates AAVrh.32.33 and sea lion AAV and (ii) selective blockade or removal of c
11 is the first description of coxiellosis in a sea lion and suggests that exposure to sea lions may be
13 understand susceptibility of wild California sea lions and Northern elephant seals to influenza A vir
14 and a naturally occurring condition in wild sea lions and simultaneously advance general knowledge o
15 limate change will likely prevent California sea lions (and other marine mammals) from attaining thes
16 ction and animal movement in sympatric seal, sea lion, and sea otter species sampled in the North Pac
18 rvational and experimental studies of seals, sea lions, and walruses reveal elements of vocal develop
23 sites in 48 healthy dolphins and 18 healthy sea lions, as well as those of adjacent seawater and oth
25 rmine if the spatial distribution of Steller sea lions at sea displayed similar scaling properties to
26 dent patterns in the distribution of Steller sea lions at sea or linkages with SST may have been appa
27 s indicate that the distributions of Steller sea lions at sea were more influenced by bathymetry than
29 rom 2000-2010 to estimate probabilities of a sea lion born in one DPS being seen within the range of
32 olphin, which evolved independently from the sea lion but displays similar feeding behavior, also has
33 d serum albumin concentration, but only in a sea lion colony exposed to anthropogenic environmental i
35 ntibody concentration during early Galapagos sea lion development were higher in a colony exposed to
37 a lions previously exposed to DA (chronic DA sea lions) display hippocampal neuropathology similar to
40 n an effort to find linkages between Steller sea lions (Eumetopias jubatus) and their environment, th
41 ion trends support the separation of Steller sea lions (Eumetopias jubatus) into a western distinct p
43 This ELISA provides a tool for testing live sea lions for ZcAV exposure and is valuable for subseque
48 at, contrary to predictions, male California sea lions increased rather than decreased their average
50 us, hippocampal neuropathology of chronic DA sea lions is similar to that of human patients with temp
52 in a sea lion and suggests that exposure to sea lions may be a risk factor for contracting Q fever.
53 Dairy cattle, farmed mink or South American sea lions may have the potential to serve as new mammali
54 re deployed on 10 groups of juvenile Steller sea lions (n=52) at eight different locations within the
56 e decline of the endangered New Zealand (NZ) sea lion (Phocarctos hookeri) is linked to latent levels
61 n the brainstem, thalamus, and cortex in one sea lion pup and the external anatomy of the brain in a
63 x, we processed brain sections from seal and sea lion pups for Nissl substance, cytochrome oxidase, a
64 ity of bacteriophages was higher in unweaned sea lion pups than in juveniles and animals in rehabilit
72 ates that give ample chance to escape from a sea lion-sized predator, but humpback whales could captu
73 th the northern elephant seal and California sea lion spend most of their lives at sea, but each also
75 to the inactivation of Tas1r2, we found that sea lion Tas1r1 and Tas1r3 are also pseudogenized, consi
76 ed serum and lung samples (n = 96) from wild sea lions that stranded along the California coast were
78 r receptor function is not restricted to the sea lion: the bottlenose dolphin, which evolved independ
80 dimension were calculated for each group of sea lions using a unit square box-counting method, where
81 nt capsid of Parkville virus, and San Miguel sea lion virus serotype 4 (SMSV4), which are representat
84 to human patients, hippocampal sclerosis in sea lions was unilateral in 79% of cases, mossy fiber sp
85 f wild marine carnivore, three seals and one sea lion, we find that Ly49 and KIR are each represented
86 ained opportunistically postmortem from wild sea lions with and without chronic clinical signs of tox
87 eal (Mirounga angustirostris) and California sea lion (Zalophus californianus) are members of a diver
88 documented energy budgets of the California sea lion (Zalophus californianus), a consumer of these s
89 s of the top marine predator, the California sea lion (Zalophus californianus), which have been stead
93 ira interrogans serovar Pomona in California sea lions (Zalophus californianus) as a case study to il
96 interneuron and bouton numbers in California sea lions (Zalophus californianus) that naturally develo