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1 ants die at the transition from the first to second instar.
2 ficacy, causing 96.5% and 97.5% mortality in second instar Ae. aegypti and Cx. quinquefasciatus, sequ
3  the first instar and across the molt to the second instar and quantified the time spiderlings spent
4           Reduction of subdued expression in second instar C4da neurons not only increases thermal no
5                                       In the second-instar Drosophila larvae (L2), like in human infa
6 le (otd) are expressed throughout the entire second instar eye-antennal disc, conferring a default fa
7 e lymph glands of third-instar, but never of second-instar hosts, are almost always accompanied by di
8  instar, reduced MMP was already apparent at second instar in the cell bodies, axons and neuromuscula
9  Ae. aegypti eDNA deposited by as few as one second instar larva in 1L of water was detectable.
10 ism of insect ecdysis between the first- and second-instar larva, while enclosed in the bag.
11 ozygous for the insert arrest development as second instar larvae and die without pupating.
12 fficient to promote nociceptive responses in second instar larvae and suppress expression of subdued
13 emizygous for our emb alleles can develop to second instar larvae but persist at this stage and consi
14                 Remarkably, some E75A mutant second instar larvae display a heterochronic phenotype i
15 netic screen utilizing transgenic Drosophila second instar larvae expressing an actin, green fluoresc
16                            The transition to second instar larvae was rescued with importin alpha1, a
17 in gene targeting, die as small, immobilized second instar larvae with severely deformed musculature.
18         Hemizygous dm(4)/Y mutants arrest as second instar larvae, and fat body nuclei of dm(4)/Y mut
19 lies die around the transition from first to second instar larvae, and homozygous importin alpha3 mut
20                                   First- and second-instar larvae become infected when they ingest fo
21 ity of irradiated combination formulation on second-instar larvae Ephestia Kuehniella was 68.89%, whi
22 2)(amorphous) and free spore formulations on second-instar larvae of Ephestia kuehniella were 73.76%,
23                           In field-collected second-instar larvae, Klebsiella (70.3%) was the most ab
24 arval growth, and arrest during the first to second instar larval molt.
25 hila germline cystocytes generated either in second instar larval ovaries or in adults over-producing
26 lt stages or 20 Drosophila larvae during the second instar life stage at a spatial resolution of 10 o
27 loping larvae leaves the timing of first and second instar molts largely unchanged, but triples durat
28  to layers that are already present early in second instar nymphs.
29  and L mutually antagonize each other during second instar of larval development to restrict their fu
30                      dSREBP(-) larvae die at second instar owing to loss of dSREBP-mediated transcrip
31 (ex) driven by a heat pulse during the early second instar resulted in a severe phenotype that includ
32                               Both LS and FS second instars showed higher susceptibility to Pp-Fe (95
33                The wasps bit and carried off second instars whole, whereas third and fourth instar ki
34 e its expression is specifically elevated in second-instar wing discs during wing margin formation.
35                                       In the second instar, Wingless activates genes involved in prox