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1 tions, in the urine of nonsmokers exposed to secondhand smoke.
2 e--is exquisitely sensitive to the toxins in secondhand smoke.
3 tion of the public from unwanted exposure to secondhand smoke.
4  .001) lower than for those with exposure to secondhand smoke.
5 aged dwellings after adjusting age, sex, and secondhand smoke.
6 valence of cigarette smoking and exposure to secondhand smoke.
7 ble to tobacco use in adults, including from secondhand smoke.
8  smoking, bidi smoking, chewing tobacco, and secondhand smoke.
9 sation treatment, and studies on exposure to secondhand smoke.
10 okers, and vulnerable populations exposed to secondhand smoke.
11 o stop smoking or are chronically exposed to secondhand smoke.
12 ciated with traffic-related air pollution or secondhand smoke.
13 urportedly has fewer traditional toxins than secondhand smoke.
14 conomic status and those exposed to familial secondhand smoking.
15 , was highest among children only exposed to secondhand smoke (0.494 mug/L mug/L; 95% CI, 0.386-0.633
16 o stop were reviewed: industry manipulation, secondhand smoke, addiction, cessation, youth access, sh
17                              Any exposure to secondhand smoke adversely affects both cross-sectional
18 ntal factors including exposure to active or secondhand smoke, air pollution, poor nutrition and soci
19  from increasing awareness of the dangers of secondhand smoke among nonsmokers and smokers.
20 n active smoking (AS) or passive exposure to secondhand smoke and allergic conditions.
21 ion between objectively measured exposure to secondhand smoke and biomarkers of heart disease risk ha
22                           The combination of secondhand smoke and hypercholesterolemia resulted in in
23                                         Both secondhand smoke and hypercholesterolemia were associate
24 e stress, we hypothesized that risk factors (secondhand smoke and hypercholesterolemia) are associate
25 No significant association was found between secondhand smoke and lung cancer death.
26 uit smoking to reduce children's exposure to secondhand smoke and their risk of ADHD.
27 asthma risk and higher rates of exposures to secondhand smoke and traffic-related air pollution.
28 al smoking during the in utero period and to secondhand smoke are associated with occurrence of child
29                               The effects of secondhand smoke are substantial and rapid, explaining t
30 nts indicated that industry manipulation and secondhand smoke are the most effective strategies for d
31 ntial carcinogen for malignancies induced by secondhand smoking are not fully understood yet.
32 likely than current smokers to be exposed to secondhand smoke at home (prevalence 39.1% [95% CI 36.6-
33 o smoking among non-smokers, and exposure to secondhand smoke at home and in public places.
34 spectively on smoking status and exposure to secondhand smoke based on questionnaires and biochemical
35        They not only protect bystanders from secondhand smoke but also contribute to less smoking amo
36 dren who were exposed to secondhand vapor vs secondhand smoke, but higher than in those exposed to ne
37                                              Secondhand smoke can cause adverse pregnancy outcomes, y
38 eveloping fetuses, and postnatal exposure to secondhand smoke causes adverse health effects during ch
39                                              Secondhand smoke causes cardiovascular and respiratory d
40 e to asbestos, high systolic blood pressure, secondhand smoke, child wasting, and alcohol use had a c
41 e to asbestos, high systolic blood pressure, secondhand smoke, child wasting, and alcohol use should
42 ronmental tobacco smoke (ETS), also known as secondhand smoking, contains human carcinogens associate
43 % vs 78%; OR = 4.5; 95% CI, 2.8 to 7.2), and secondhand smoke counseling (80% vs 63%; OR = 2.6; 95% C
44 line gaming (OR, 1.56; 95% CI, 1.0-2.3), and secondhand smoke exposure (OR, 1.7; 95% CI, 1.1-2.6).
45 , vaping (OR, 1.99; 95% CI, 1.1-3.5), and/or secondhand smoke exposure (OR, 1.83; 95% CI, 1.2-2.7).
46 rease in blood nitromethane by 150 ng/L, and secondhand smoke exposure (serum cotinine >0.05 ng/mL an
47                                              Secondhand smoke exposure (SHSe) is a significant modifi
48      We examined trends in and predictors of secondhand smoke exposure among nonsmoking adults to det
49                      Analyses using reported secondhand smoke exposure and cotinine-measured exposure
50  the relationship between active smoking and secondhand smoke exposure and critical illness.
51 d beyond the underlying long-term decline in secondhand smoke exposure and demonstrates the positive
52  multivariate regression analyses to examine secondhand smoke exposure as measured by the proportion
53 and exposure to lead, children with reported secondhand smoke exposure at home were more likely to ha
54 2.5 mum or less, 4.7% (95% CI, 1.3%-9.6%) by secondhand smoke exposure at home, and 4.6% (95% CI, 2.1
55                          As the magnitude of secondhand smoke exposure declines because of proportion
56                               In this study, secondhand smoke exposure during childhood was associate
57                          We examined whether secondhand smoke exposure during childhood was associate
58                                              Secondhand smoke exposure during pregnancy increased the
59 ng history and detected a high prevalence of secondhand smoke exposure in a critically ill population
60                                  Measures of secondhand smoke exposure in childhood/adolescence consi
61                    Our findings suggest that secondhand smoke exposure in children is strongly associ
62 tion Survey data to assess the prevalence of secondhand smoke exposure in nonsmokers in the US popula
63                                              Secondhand smoke exposure in the home was associated wit
64                                              Secondhand smoke exposure increased EPCs and plasma vasc
65                                              Secondhand smoke exposure increases cardiovascular risk.
66                                              Secondhand smoke exposure increases the risk of acute my
67               Prenatal tobacco and postnatal secondhand smoke exposure is consistently associated wit
68                                              Secondhand smoke exposure may be a risk factor for incre
69 factors considered in this study, preventing secondhand smoke exposure may be the most effective way
70 inos, suggests that the important effects of secondhand smoke exposure occur acutely.
71 tatus did not confound the adverse effect of secondhand smoke exposure on lung function.
72                                    Childhood secondhand smoke exposure predicted increased risk for a
73 conclude that avoiding childhood/adolescence secondhand smoke exposure promotes adulthood cognitive f
74        Interaction between gene variants and secondhand smoke exposure resulted in significant percen
75   Participants were also more likely to rate secondhand smoke exposure to cannabis vs tobacco as comp
76 impact of England's smokefree legislation on secondhand smoke exposure was above and beyond the under
77                                              Secondhand smoke exposure was captured in 2.6% of trials
78                                              Secondhand smoke exposure was higher among those exposed
79                                              Secondhand smoke exposure was positively associated with
80 rinogen and homocysteine seen in relation to secondhand smoke exposure were equivalent to approximate
81 ed, and examine the association of postnatal secondhand smoke exposure with ADHD using both reported
82                           The association of secondhand smoke exposure with ADHD was examined by two
83 rature examines the association of postnatal secondhand smoke exposure with attention deficit hyperac
84 (86%) "seldom" or "never" ask patients about secondhand smoke exposure, 245 (84%) "always" or "period
85 he home, urine cotinine levels indicative of secondhand smoke exposure, and residence in more densely
86 marital status, education, household income, secondhand smoke exposure, and work status were examined
87     Because of the strong relationship among secondhand smoke exposure, bacterial infection, and sinu
88 tigated the cross-sectional relation between secondhand smoke exposure, measured objectively as cotin
89 for lifetime number of cigarettes smoked and secondhand smoke exposure.
90 ow that male smoking prevalence approximates secondhand smoke exposure.
91 bis vs tobacco in terms of daily smoking and secondhand smoke exposure.
92 rons at risk for adverse events triggered by secondhand smoke exposure.
93 ciation is robust using both measurements of secondhand smoke exposure.
94 HD using both reported and cotinine-measured secondhand smoke exposure.
95 e (TGFbeta1) amplify the negative effects of secondhand smoke exposure.
96                           Temporal trends of secondhand smoke exposure: nonsmoking workers in the Uni
97                                              Secondhand smoking exposure and traffic and industrial p
98          Evidence is less clear for passive (secondhand) smoke exposure.
99                           We exposed mice to secondhand smoke for 8 wk, followed by a period of smoke
100  nonexposed nonusers and nonusers exposed to secondhand smoke for either of these two aldehydes.
101 idence to suggest that low-level exposure to secondhand smoke has a clinically important effect on su
102                                  Exposure to secondhand smoke has been associated with a disproportio
103                                  Exposure to secondhand smoke has been associated with numerous healt
104                                              Secondhand smoke has detrimental effects on vascular smo
105                                  Exposure to secondhand smoke has plummeted.
106  cessation efforts Additional restriction of secondhand smoke in any places where the public may cong
107             Of 812 participants with data on secondhand smoke in the home, 188 (23.2%) were exposed.
108 the importance of investigating paternal and secondhand smoking in addition to maternal smoking in AS
109                                              Secondhand smoke increased EMPs and decreased FMD.
110                                              Secondhand smoke increases the risk of coronary heart di
111                                              Secondhand smoke increases the risk of vascular disease
112 h common urban indoor air pollution sources (secondhand smoke, indoor motorcycle emissions, and cooki
113                                              Secondhand smoke-induced endothelial dysfunction has bee
114 rmally harmless Ag, and they may explain why secondhand smoke is a major risk factor for the developm
115 ogistic regression models that differ in the secondhand smoke measurement used.
116 active smokers (n = 6, 21%) or as exposed to secondhand smoke (n = 21, 75%).
117 search describing the mechanistic effects of secondhand smoke on the cardiovascular system, emphasizi
118    Interactions between diet and exposure to secondhand smoke on the prevalence of childhood obesity:
119 ke or vapor indoors in the past 7 days (only secondhand smoke, only secondhand vapor, or neither).
120                                     Although secondhand smoke or environmental tobacco smoke (ETS) ha
121                         Reported exposure to secondhand smoke or vapor indoors in the past 7 days (on
122 crease in the weekly duration of exposure to secondhand smoke (P<0.001 by the chi-square test for tre
123 osure to sidestream cigarette smoke (SS), or secondhand smoke, promoted nicotinic acetylcholine recep
124 men who reported smoking or being exposed to secondhand smoke (rho = 0.24).
125 gated the independent effects of exposure to secondhand smoke, road vehicle traffic, and dietary frui
126 , recent smokers or significantly exposed to secondhand smoke (SHS) (0.5-13.9 ng/mL), lightly exposed
127 n developed for the detection of cotinine, a secondhand smoke (SHS) biomarker.
128 line in smoking rates over time, exposure to secondhand smoke (SHS) continues to cause harm to nonsmo
129 the association between objectively measured secondhand smoke (SHS) exposure and incident cardiovascu
130         We have previously demonstrated that secondhand smoke (SHS) exposure exacerbates bacterial in
131                                              Secondhand smoke (SHS) exposure has been linked to the w
132                                              Secondhand smoke (SHS) exposure has been related to vari
133                                              Secondhand smoke (SHS) exposure is a known risk factor f
134                                     Although secondhand smoke (SHS) exposure is predominant in Bangla
135 ing pregnancy (in utero smoking) and current secondhand smoke (SHS) exposure on asthma control is poo
136 study sought to analyze the effects of acute secondhand smoke (SHS) exposure on the number and functi
137        We sought to determine the effects of secondhand smoke (SHS) exposure on vascular reactivity i
138         Despite progress in tobacco control, secondhand smoke (SHS) exposure remains prevalent worldw
139 tandard biomarkers to differentiate THS from secondhand smoke (SHS) exposure.
140                                              Secondhand smoke (SHS) has been associated with a variet
141                       Children's exposure to secondhand smoke (SHS) has been causally linked to a num
142                The scientific consensus that secondhand smoke (SHS) increases cardiovascular disease
143                                              Secondhand smoke (SHS) is associated with a 30% increase
144                      The issue of adolescent secondhand smoke (SHS) is globally significant, given th
145                                  Exposure to secondhand smoke (SHS) may increase risk for obesity, bu
146 creased risk of dementia, but the effects of secondhand smoke (SHS) on dementia risk are not known to
147 oth active smoking and household exposure to secondhand smoke (SHS) on subsequent bladder cancer risk
148  characterized the exposure of nonsmokers to secondhand smoke (SHS) outside a restaurant and bar in A
149 uch intervention has not yet been applied to secondhand smoke (SHS) reduction programs that target sm
150 levels of individual VOCs in freshly emitted secondhand smoke (SHS) were in the range of 1-300 mug m(
151 alence have been associated with exposure to secondhand smoke (SHS), maternal smoking during pregnanc
152 nge of relatively low concentrations of aged secondhand smoke (SHS), similar to those encountered com
153 side of the home for nonsmokers' exposure to secondhand smoke (SHS).
154  effects of low levels of exposure including secondhand smoke (SHS).
155 njunction with the industry manipulation and secondhand smoke strategies.
156                             High exposure to secondhand smoke suggests a need for countries to pass s
157 t ultraviolet radiation, 4-aminobiphenyl and secondhand smoke that are known to be strong, moderate a
158 -control messages emphasizing the dangers of secondhand smoke to smokers and nonsmokers undermine the
159      A public ordinance reducing exposure to secondhand smoke was associated with a decrease in AMI h
160  increase of surface-bound TSNAs when sorbed secondhand smoke was exposed to 60 ppbv HONO for 3 hours
161                          Student exposure to secondhand smoke was high both at home (more than four i
162                                  Exposure to secondhand smoke was not associated with individual driv
163               Marital status and exposure to secondhand smoke were linked to higher stress in univari
164 nd apoE(-/-) mice exposed to filtered air or secondhand smoke were quantified.
165  nor vape and were not frequently exposed to secondhand smoke were recruited into 3 cohorts: 2 chroni
166 ged 4-15 years who were exposed to postnatal secondhand smoke with prevalence in those who were not e
167 shed since 1995, and compared the effects of secondhand smoke with the effects of active smoking.

 
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