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1 blished papers (GPX > SeCA > selenocystine > selenomethionine).
2 bserved, indicating the previous presence of selenomethionine.
3 sothiourea, and the peroxynitrite scavenger, selenomethionine.
4 onine and substituting leucine residues with selenomethionine.
5 , and Met261 were only partially replaced by selenomethionine.
6  with methylmercury and/or Se as selenite or selenomethionine.
7 them to mayflies exposed to elevated aqueous selenomethionine.
8 at is associated with impaired metabolism of selenomethionine.
9 ially incorporated into organic Se mainly as selenomethionine.
10 ied from the sam1(-) sam2(-) strain grown in selenomethionine.
11  conducted a randomized, controlled trial of selenomethionine 200 microg daily and/or celecoxib 200 m
12 elenium as selenate (200 or 400 mug/d) or as selenomethionine (200 mug/d) for 4 wk.
13 bioaccessible selenium was mainly present as selenomethionine, a good bioavailable source of selenium
14 e accumulated in an algal-wetland system are selenomethionine, a precursor of volatile Se formation,
15                                  Toxicity of selenomethionine, an organic derivative of selenium wide
16 avelength anomalous diffraction phasing with selenomethionine analogs that retain the biophysical cha
17 ubstituting the nine methionines in hTF with selenomethionine and 2) the structure of glycosylated ap
18 d in Linxian, China to assess the effects of selenomethionine and celecoxib on the natural history of
19 y growth-arrested, toxic amino acids such as selenomethionine and fluorophenylalanine were efficientl
20  chromatography mass spectrometry to contain selenomethionine and methyl-selenocysteine (1:1-3 ratio)
21 wo fractions were identified specifically as selenomethionine and selenocystamine, estimated to be pr
22 ated by devising a simple synthesis for Fmoc-selenomethionine and substituting leucine residues with
23 enium and tellurium analogues, Se-adenosyl-L-selenomethionine and Te-adenosyl-L-telluromethionine (Se
24           Oral selenium (200 microg/d from L-selenomethionine) and matched vitamin E placebo, vitamin
25 plements of selenium (200 mug per day from L-selenomethionine) and vitamin E (400 IU per day of all r
26 eased production of protein during growth in selenomethionine, and efficient replacement of methionin
27 ength anomalous diffraction (MAD) phasing on selenomethionine, and refined to an R(cryst) = 0.24 and
28 8.63, and 9.64 ug kg(-1) for selenocysteine, selenomethionine, and seleno-methyl-selenocysteine, resp
29 rate the free amino acids selenocysteine and selenomethionine, and these are incorporated nonspecific
30 waterborne dissolved selenite and to dietary selenomethionine as selenized algae.
31 g using selenium substitution in the form of selenomethionine as the anomalous scatterer.
32 urrogate, ProSeAM (propargylic Se-adenosyl-l-selenomethionine), as a reporter of methyltransferases.
33 , and efficient replacement of methionine by selenomethionine, based on quantitative mass spectrometr
34                                     With the selenomethionine being the major form of selenium presen
35 f the species produced by the plant, such as selenomethionine, can be identified at ppb levels by RP-
36 ely to show an improvement when treated with selenomethionine compared with placebo (P = 0.02).
37 sequencing and mass spectrometry analysis of selenomethionine-containing peptides suggests that Met30
38 e the structures of the native protein and a selenomethionine-containing variant, solved to 2.8 A.
39                       The total selenium and selenomethionine contents of SEEY-1 were determined to b
40 sed to different concentrations of dietary l-selenomethionine (control, 2.3, 9.7, 32.5, or 57.7 mug S
41                              Cytotoxicity of selenomethionine decreased when the extracellular concen
42 length anomalous diffraction experiment on a selenomethionine derivative of the truncated MAO-N-D5 en
43 -wavelength anomalous dispersion (SAD) using selenomethionine-derivative protein.
44 ctional difference between the wild-type and selenomethionine derivatives.
45 d esophageal squamous dysplasia at baseline, selenomethionine did have a protective effect.
46 ethionine residues in the native enzyme with selenomethionine does not affect the structure of the Ni
47 ltiwavelength anomalous dispersion data from selenomethionine-enriched protein and refined to an R fa
48 elenomethionine indicating that selenide and selenomethionine exert their toxicity via distinct mecha
49            In unplanned stratified analyses, selenomethionine favorably affected a change in dysplasi
50 ever, extensive replacement of methionine by selenomethionine for anomalous dispersion phasing has pr
51 placement using a uranium derivative and the selenomethionine form of the enzyme (SeMAT).
52 rity of which was in the highly bioavailable selenomethionine fraction.
53 s a matrix-matched spike for the analysis of selenomethionine in a sample of selenized yeast.
54 oprotein pool, shown by others to be largely selenomethionine in albumin, declined.
55 etry (GC-MS) determination of methionine and selenomethionine in food samples is described.
56 s, the scientific community has investigated selenomethionine in most nut matrices.
57 beled tyrosine was also co-incorporated with selenomethionine in order to confirm these assignments.
58  readily incorporated forms of selenium (ie, selenomethionine) in HIV-infected breastfeeding women.
59 creased accumulation of organoselenium (e.g. selenomethionine) in selenate-treated roots.
60  forms that probably differ in the degree of selenomethionine incorporation and the extent of selenom
61 two regions near 1,284 and 900 cm(-1) due to selenomethionine incorporation.
62 pe growth rate properties in the presence of selenomethionine indicating that selenide and selenometh
63         We show that selenium in the form of selenomethionine induces a DNA repair response in normal
64  provided by selenium after incorporation of selenomethionine instead of natural methionine by geneti
65    We report a general method to incorporate selenomethionine into proteins expressed in yeast based
66 corporation of selenium as selenocysteine or selenomethionine into proteins that lack canonical encod
67 rally, combining cell-free incorporations of selenomethionine into proteins with FTIR difference spec
68 ifference spectra recorded for unlabeled and selenomethionine-labeled cell-free expressed BR closely
69 ngth anomalous diffraction technique using a selenomethionine-labeled crystal containing 88 selenium
70 e of glypican-1 was solved using crystals of selenomethionine-labeled glypican-1 core protein lacking
71                                              Selenomethionine-labeled RF1 crystallized in space group
72 X-ray absorption spectroscopy approach using selenomethionine labeling to distinguish the metal sites
73  exposures; larvae predominantly accumulated selenomethionine-like species regardless of uptake route
74 ithin the eye lens of the intact larva was a selenomethionine-like species.
75 cture of hiNadR complexed with NAD using the selenomethionine MAD phasing method.
76 terococcus faecalis (MVAS) was determined by selenomethionine MAD phasing to 2.4 A and the enzyme com
77  protein, C.AhdI, to 1.69 A resolution using selenomethionine MAD.
78                                              Selenomethionine markers clearly indicate an antiparalle
79  deficiency is associated with impairment of selenomethionine metabolism.
80 ommon dietary selenium compounds - selenite, selenomethionine, methylselenocysteine and selenocystine
81 eceive a daily supplement of 100 mug Se as l-selenomethionine (n = 129) or placebo (n = 126) for 12 w
82       After a 10-month intervention, neither selenomethionine nor celecoxib inhibited esophageal squa
83              By comparing the sensitivity to selenomethionine of mutants impaired in the sulfur amino
84 ious stages of catalysis, in the presence of selenomethionine or Se-adenosyl-L-selenomethionine, reve
85 f massive annual algal blooms showed that no selenomethionine or selenomethionine oxide was present.
86 tation were given daily selenium (200 mug as selenomethionine) or placebo as supplements from recruit
87 treated with DHEA, DFMO, tocopherol acetate, selenomethionine, or 9-cis-retinoic acid, although the e
88 nomethionine incorporation and the extent of selenomethionine oxidation.
89 al blooms showed that no selenomethionine or selenomethionine oxide was present.
90 Production and emission of selenomethionine, selenomethionine oxide, and other discrete organic selen
91 own to produce and release selenomethionine, selenomethionine oxide, and several other organic seleni
92 ia progression (14% vs 19%) compared with no selenomethionine (P = .08).
93 s of LAM and shows selenium in Se-adenosyl-L-selenomethionine poised to ligate the unique iron in the
94 y compare the effect on protein stability of selenomethionine relative to methionine.
95  spectroscopy of multiple selenocysteine and selenomethionine residues in the sulfhydryl oxidase augm
96                            The SeMAT data (9 selenomethionine residues out of 383 amino acid residues
97 insic binuclear zinc centre and incorporated selenomethionine residues.
98                                     Overall, selenomethionine resulted in a trend toward increased dy
99  the entire digestion, whereas incubation of selenomethionine resulted in the chemical and microbial
100 resence of selenomethionine or Se-adenosyl-L-selenomethionine, reveals that the cofactor is cleaved o
101 certified reference material SELM-1, natural selenomethionine samples, and model solutions of artific
102  for methionine and grown in the presence of selenomethionine (Se(Met)), the single methionine of the
103  and linear relationship between total Se or selenomethionine (Se-Met) accumulation in grain and Se d
104 ascaris acus or not, were exposed to dietary selenomethionine (Se-Met) at an environmentally relevant
105                                              Selenomethionine (Se-Met) was found to be the major Se-c
106 d a XAD(R) resin was employed as sorbent for selenomethionine (Se-Met), selenomethylselenocysteine (S
107 he positions of Met95 have been confirmed by selenomethionine ((Se)Met) MAD.
108 is of two analogues of AdoMet, Se-adenosyl-L-selenomethionine (SeAdoMet) and Te-adenosyl-L-telluromet
109 e kinetics with the SAM analog Se-adenosyl-l-selenomethionine (SeAM) as a cofactor surrogate.
110 0, 100, or 200 microg Se ( approximately 60% selenomethionine), selenium-enriched onion meals ( appro
111 s identified in white oyster mushroom, while selenomethionine, selenocystine, and Se-methylselenocyst
112 w molecular weight organoselenium compounds (selenomethionine, selenoethionine, trimethylselenonium i
113                   Production and emission of selenomethionine, selenomethionine oxide, and other disc
114 o selenate were shown to produce and release selenomethionine, selenomethionine oxide, and several ot
115                                 Here, we use selenomethionine (SeM) active site labels in a series of
116             Guided by a careful study of the selenomethionine (SeM) benzylation, we have refined the
117               In earlier work we showed that selenomethionine (SeM) substitution of the coordinated M
118 methionine (DFM), trifluoromethionine (TFM), selenomethionine (SeM), and norleucine (Nle) using expre
119 ostructural amino acids norleucine (Nle) and selenomethionine (SeM).
120 ne (SeC), Se-methylselenocysteine (SeMC) and selenomethionine (SeM).
121                                              Selenomethionine (SeMet) and selenomethylcysteine (SeMeS
122 ay dietary exposure to food augmented with l-selenomethionine (SeMet) at measured concentrations of 0
123 rty of methylselenol, which is released from selenomethionine (SeMET) by cancer cells with the adenov
124         We show that selenium in the form of selenomethionine (SeMet) can activate the p53 tumor supp
125 xposure to hypersaline conditions and 50 muM selenomethionine (SeMet) decreased embryo hatch and depl
126                                              Selenomethionine (SeMet) is a potentially toxic amino ac
127 identification showed selenocystathionine-to-selenomethionine (SeMet) ratios of 75:25, 71:29, and 32:
128 te by ATP sulfurylase, and (b) conversion of selenomethionine (SeMet) to DMSe.
129 cystine (SeCys; detected as [SeCys]2 dimer), selenomethionine (SeMet), and methyl-selenocysteine (MeS
130 ioaccessibility of Se, and its organic forms selenomethionine (SeMet), and selenocysteine (SeCys2) wa
131 istance to the toxic effects of selenite and selenomethionine (SeMet), respectively.
132 afish (Danio rerio) after exposure to excess selenomethionine (SeMet, the dominant chemical species o
133 utant (used to obtain experimental phases by selenomethionine single-wavelength anomalous diffraction
134 A resolution crystal structure of MglC using Selenomethionine Single-wavelength anomalous diffraction
135  (gi 4981173) at 2.65 Angstrom resolution by selenomethionine single-wavelength anomalous dispersion
136                  The structure was solved by selenomethionine single-wavelength anomalous dispersion
137 bstitution of these methionine residues with selenomethionine slightly stabilizes the protein.
138 saminate methyl-L-selenocysteine (MSC) and L-selenomethionine (SM) to beta-methylselenopyruvate (MSP)
139                                    Moreover, selenomethionine (SM), a prostate cancer treatment adjuv
140 ic and organic Hg (HgCl2 and MeHgCl) and Se (selenomethionine, sodium selenite, and sodium selenate)
141 system containing an amino acid mixture with selenomethionine substituted for methionine.
142 aracteristics, the crystal structures of the selenomethionine substituted StnA (SeMet-StnA) and the c
143 d expressed at high levels as the native and selenomethionine-substituted (SeMet) proteins.
144 ength anomalous diffraction techniques and a selenomethionine-substituted analogue of the enzyme.
145      We have solved the crystal structure of selenomethionine-substituted CheY** in the presence of i
146 velength anomalous dispersion methods on a L-selenomethionine-substituted complex of LAM with [4Fe-4S
147 elength anomalous diffraction phasing from a selenomethionine-substituted crystal at 3.0 A resolution
148 gth anomalous diffraction (MAD) phasing of a selenomethionine-substituted derivative to define a new
149       This model was further confirmed using selenomethionine-substituted DGCR8 and mercury titration
150  with data obtained from the E. coli Ni-GlxI selenomethionine-substituted enzyme.
151                           The native and the selenomethionine-substituted forms of the phenylalanine-
152 th x-ray data collected from crystals of the selenomethionine-substituted L99A/M102L mutant of T4 lys
153                     The crystal structure of selenomethionine-substituted malate synthase G, an 81 kD
154  NMT from Thalictrum flavum was solved using selenomethionine-substituted protein (dmin = 2.8 A).
155                                        Using selenomethionine-substituted protein and multiple anomal
156           The structure was determined using selenomethionine-substituted protein and multiwavelength
157                              Crystals of the selenomethionine-substituted protein have space group P2
158                                          The selenomethionine-substituted protein was induced in the
159 nomalous diffraction, using a crystal of the selenomethionine-substituted protein.
160                                              Selenomethionine supplementation results in a similar, a
161       The activity was not increased more by selenomethionine supplementation than by the placebo in
162  by selenite uptake and by the conversion of selenomethionine to dimethylselenide.
163 enosylmethionine is blocked, exhibit reduced selenomethionine toxicity compared with wild-type yeast,
164        Involvement of superoxide radicals in selenomethionine toxicity in vivo is suggested by the hy
165                       Instead, we found that selenomethionine toxicity is mediated by the trans-sulfu
166 ced repair complex formation was observed in selenomethionine-treated cells.
167 namic range allows quantitative detection of selenomethionine, trimethylselenonium ion, methylselenog
168 This is, however, increasingly offset in the selenomethionine variants, ultimately resulting in a dif
169        Results were compared by agent group (selenomethionine vs placebo; celecoxib vs placebo).
170                                              Selenomethionine was efficiently incorporated into this
171                    For Se species screening, selenomethionine was identified in white oyster mushroom
172                                              Selenomethionine was incorporated into BR using a cell-f
173                                              Selenomethionine was the most abundant species in P. ost
174 lenocysteine, but not the non-thiol-reactive selenomethionine, was shown to induce Top2alpha cleavage
175  limits of 1 ng/g methionine and 10 ng/g for selenomethionine were obtained.
176 ies, including a cyclic oxidation product of selenomethionine, were observed, indicating the previous
177 d plants accumulated organic Se, most likely selenomethionine, whereas selenate-supplied plants accum
178                                              Selenomethionine, which is the principal dietary form of
179              Oral selenium (200 mug/d from L-selenomethionine) with matched vitamin E placebo, vitami

 
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