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1 s, and to correlate nerve fibre density with sensory abnormalities.
2 ural damage, external-sphincter weakness, or sensory abnormalities.
3  that may underlie both short- and long-term sensory abnormalities.
4 ects of movement disorders include intrinsic sensory abnormalities and the effects of external sensor
5                Many forms of dHMN have minor sensory abnormalities and/or a significant upper-motor-n
6 the phenomenology and physiological basis of sensory abnormalities, and about the role of the basal g
7           In mood disorders, psychomotor and sensory abnormalities are prevalent, disabling, and inte
8                    The data do not support a sensory abnormality as the primary cause of microstrabis
9           TRN dysfunction has been linked to sensory abnormality, attention deficit and sleep disturb
10 dysfunction and behavioral disorders and how sensory abnormalities can contribute to the etiology of
11          No clinical or electrophysiological sensory abnormality existed; however, skin biopsy result
12 r activity and how mechanisms underlying the sensory abnormalities fit in with the proposed 'network
13 ing myelin deficits to synaptic activity and sensory abnormalities in ASD.
14 novel reflex circuit-specific proprioceptive sensory abnormality in ALS.SIGNIFICANCE STATEMENT Neurod
15 parent including hyperactivity, seizures and sensory abnormalities including a characteristic increas
16  and whether its activity levels may lead to sensory abnormalities is still unclear.
17 rders (ASD), but the neural basis underlying sensory abnormality is not completely understood.
18                                   Widespread sensory abnormalities, not limited to the CRPS limb, hav
19  in the larva (mixed sexes), paralleling the sensory abnormalities observed in individuals with ASD.
20 ot be readily distinguished from the similar sensory abnormalities produced by the ensuing diabetic n
21 etitive behaviors, and comorbidities such as sensory abnormalities, sleep disturbances, and seizures.
22                                              Sensory abnormalities such as numbness and paresthesias
23     Here we demonstrate that the presence of sensory abnormalities such as tingling, correlate with t
24 eptors in FM; however, the presence of other sensory abnormalities suggests involvement of large diam
25 ynaptic reorganization may contribute to the sensory abnormalities that accompany peripheral neuropat
26 ermine whether microstrabismic subjects have sensory abnormalities that could give rise to a small an
27 ic factor (GDNF) both prevented and reversed sensory abnormalities that developed in neuropathic pain
28 ARS-CoV-2 infection has been associated with sensory abnormalities that manifest in both acute and ch
29                   Ciliopathies manifest from sensory abnormalities to syndromic disorders with multi-
30 gain insight on the molecular basis of these sensory abnormalities, we used the golden hamster model