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1  this polymodal area is modified after early sensory deprivation.
2 ressed in NM neurons and are not affected by sensory deprivation.
3 rge-scale changes in synaptic dynamics after sensory deprivation.
4 es in homeostatic gene expression related to sensory deprivation.
5 ss RBC terminals are additionally altered by sensory deprivation.
6  MC spiking patterns and APs also adapted to sensory deprivation.
7 anges in sensory or neural activity, such as sensory deprivation.
8  explain the deterioration of synergy due to sensory deprivation.
9 city and its genetic underpinnings following sensory deprivation.
10 nificant functional reorganisation following sensory deprivation.
11 with SHF are selectively preserved following sensory deprivation.
12 changes depending on the age and the mode of sensory deprivation.
13  development, or reinstated in adulthood, by sensory deprivation.
14 d limb range of motion, crowding, and visual sensory deprivation.
15 and does not appear to change in response to sensory deprivation.
16  targeted synaptic protein degradation under sensory deprivation.
17  cells, a previously unappreciated effect of sensory deprivation.
18 ecovery of cortical responsiveness following sensory deprivation.
19 recovery of retinal function after prolonged sensory deprivation.
20 he cortical reorganization in a rat model of sensory deprivation.
21 other common feature and results in combined sensory deprivation.
22 rrel cortex despite whisker trimming-induced sensory deprivation.
23 es in glutamatergic input synapses caused by sensory deprivation.
24  spectrum disorders, mental retardation, and sensory deprivation.
25    NMDA receptor currents were unaffected by sensory deprivation.
26 ir use to facilitate recovery from trauma or sensory deprivation.
27 ted by PV-positive basket cells is pruned by sensory deprivation.
28 mouse visual cortex in vivo with and without sensory deprivation.
29 rding how the deaf brain in humans adapts to sensory deprivation: (1) is meaning extracted and integr
30  GABA(A) receptors could underlie effects of sensory deprivation, [3H]muscimol binding was assessed i
31                                              Sensory deprivation after new GCs had differentiated ind
32                          Here we report that sensory deprivation alters short-term synaptic dynamics
33                                              Sensory deprivation alters the properties of synaptic pl
34 es across RBC terminals remains unaltered by sensory deprivation, although ribbon synapse output site
35     Furthermore, P2 asymmetry is modified by sensory deprivation and abolished by decreased BDNF leve
36 es two powerful drivers for brain plasticity-sensory deprivation and altered use.
37                                         Both sensory deprivation and blockade of gamma-aminobutyric a
38 ical neurons are bidirectionally modified by sensory deprivation and experience, but the synaptic bas
39     However, research has largely focused on sensory deprivation and maladaptive change.
40                                              Sensory deprivation and odor learning decreased and incr
41 sexes and studied their plasticity following sensory deprivation and odor learning.
42 and revealed their plasticity in response to sensory deprivation and odor learning.
43 3 (L2/3) of adult mouse barrel cortex during sensory deprivation and recovery.
44 meliorated by interventions that target both sensory deprivation and stress.
45 l blindness, the brain develops under severe sensory deprivation and undergoes remarkable plastic cha
46  Moreover, duplicate S1 showed plasticity to sensory deprivation, and duplicate V1 responded to visua
47 ructural and behavioral plasticity following sensory deprivation are functionally independent of each
48 deaf white cat represents an animal model of sensory deprivation because it mimics a form of human de
49  contacts within the IPL also decreased with sensory deprivation but required at least 6 weeks to rec
50 aptic proteins whose levels were affected by sensory deprivation but whose synaptic roles have not ye
51 endent homeostatic plasticity in response to sensory deprivation, but IB cells were capable of a much
52  restore cortical activity in vivo following sensory deprivation, but it is unclear whether this reco
53 plasticity was preserved under conditions of sensory deprivation, but was rapidly lost by sensory exp
54                                              Sensory deprivation by contralateral whisker trimming no
55                                   Congenital sensory deprivation can lead to reorganization of the de
56                   In contrast to experience, sensory deprivation caused homeostatic synaptic enhancem
57                   These results suggest that sensory deprivation causes synaptic depression by revers
58 ale and female mice and show that unilateral sensory deprivation causes system-wide adaptations in ax
59                                For instance, sensory deprivation causes the cortical area representin
60                           Deafferentation or sensory deprivation decreases TH expression but it is no
61 he global sensory-evoked responses following sensory deprivation, despite the fact that the identifie
62 gs have extra axon branches, suggesting that sensory deprivation disrupts axon outgrowth.
63 3 encompasses a critical period during which sensory deprivation disrupts central mechanisms that sup
64                                              Sensory deprivation during a critical period reduces spi
65 ic proteomes are altered in barrel cortex by sensory deprivation during synaptogenesis.
66                                     However, sensory deprivation during the critical period degraded
67                               In the retina, sensory deprivation during the critical period of develo
68  animals reared with certain types of visual sensory deprivation during their first few months of lif
69                                              Sensory deprivation during these periods permanently com
70 to mark the close of the critical period and sensory deprivation during this epoch disrupts developme
71 plasticity in the central nervous system and sensory deprivation during this period significantly imp
72 II function, such as occurred in rats during sensory deprivation, elevated the generation and propaga
73                          During development, sensory deprivation evokes powerful synaptic plasticity
74                                              Sensory deprivation experiments in adult rats also have
75                                 Classically, sensory deprivation first drives rapid Hebbian weakening
76                                              Sensory deprivation greatly influences the development o
77                                              Sensory deprivation, if initiated before P14, disrupted
78  homeostatic changes in spine size following sensory deprivation in a subset of inhibitory (layer 2/3
79 somatosensory cortex after a brief period of sensory deprivation in adulthood.
80 tenance of cortical responsiveness following sensory deprivation in adulthood.
81 e been thought to limit reorganisation after sensory deprivation in adults.
82 pidemiological studies, the role of auditory sensory deprivation in cognitive decline remains to be f
83 indings indicate that a restricted period of sensory deprivation in early postnatal life (1) impairs
84  for the loss of synaptic strength caused by sensory deprivation in visual cortex.
85 resent long-term depression (LTD) induced by sensory deprivation in vivo.
86 terneurons, glutamatergic JGCs survive under sensory deprivation, indicating that inhibitory and exci
87 n synaptic input caused by lesions or severe sensory deprivation induce marked sprouting or retractio
88                                              Sensory deprivation induced marked changes in the densit
89                                              Sensory deprivation, induced by unilateral naris occlusi
90 cortices of awake adult mice to quantify the sensory deprivation-induced changes in the responses of
91                   These results suggest that sensory deprivation-induced homeostatic down-regulation
92 tween these inhibitory circuits can regulate sensory deprivation-induced retinogeniculate remodeling.
93  that in the cortex, BC1 RNA is required for sensory deprivation-induced structural plasticity of den
94                These maps can be modified by sensory deprivation, injury and experience in both young
95                                Recovery from sensory deprivation is slow and incomplete in adult visu
96                                              Sensory deprivation is the known causal mechanism for ac
97  brain to rewire and recover from injury and sensory deprivation, it can lead to tinnitus as an unwan
98                                The resulting sensory deprivation jeopardizes auditory cortex (AC) mat
99  alters the spatial pattern of apoptosis and sensory deprivation leads to exacerbated amounts of apop
100           There is substantial evidence that sensory deprivation leads to important cross-modal brain
101                                              Sensory deprivation markedly (approximately 40%) reduced
102                            Following chronic sensory deprivation, microglia undergo a morphological t
103  of cell types is similar in V1 and wS1, but sensory deprivation minimally affects cell type developm
104                                              Sensory deprivation most likely results in AMPA receptor
105 ased sleep after injury is not attributed to sensory deprivation, nociception, or generalized inflamm
106 ether the synaptic reorganization induced by sensory deprivation occurred differently in mature neona
107                                              Sensory deprivation of the barrel cortex in live mice (b
108 y help to explain the unique consequences of sensory deprivation on plasticity in the developing vers
109                   According to the effect of sensory deprivation on synaptic plasticity of developing
110 he structural and functional consequences of sensory deprivation on the establishment of parallel cir
111 conditions of critical period delay by total sensory deprivation or critical period acceleration by d
112 kingly rescued independent of Mecp2 by early sensory deprivation or genetic deletion of the excitator
113    We demonstrate several scenarios, such as sensory deprivation or heightened plasticity, under whic
114 petition between thalamocortical axons using sensory deprivation or increasing the size of S1.
115 tances such as perinatal brain injury, early sensory deprivation or limb malformation may result in a
116 eductions in local neuronal activity through sensory deprivation or optogenetic inhibition increase m
117                                              Sensory deprivation or selective removal of thalamic aff
118 at analyses from prolonged periods of either sensory deprivation or stimulation during adulthood are
119 onses of cells in the mouse visual cortex to sensory deprivation or to stimulation during a developme
120 factory system is particularly vulnerable to sensory deprivation, owing to the widespread prevalence
121                                  By use of a sensory deprivation paradigm, we find that visual cues r
122 development enhances myelin thickness, while sensory deprivation prevents such radial growth during d
123                                    Following sensory deprivation, primary somatosensory and visual co
124 l cortex neurons would fail to develop after sensory deprivation produced by bilateral whisker trimmi
125                     We found that such brief sensory deprivation produced structural and functional p
126                                              Sensory deprivation reduced thalamocortical inputs on NG
127 nsory, but not in the visual cortex, whereas sensory deprivation reduces Shh activity, demonstrating
128                             The incidence of sensory-deprivation related strabismus was 3.7% (14/375)
129                                              Sensory deprivation-related strabismus may also occur.
130  cross-modal plasticity in the case of early sensory deprivation relates to the original functional s
131                                              Sensory deprivation reorganizes neurocircuits in the hum
132                  We have recently shown that sensory deprivation results in large changes of the shor
133       Moreover, fate mapping of preOLs under sensory deprivation revealed that neuronal activity infl
134                                              Sensory deprivation shows competitive interactions betwe
135                                              Sensory deprivation started a few days earlier at P10, h
136                            In contrast, when sensory deprivation started after synaptic formation was
137                                              Sensory deprivation started at P12-P13, but not at P16,
138 ggest that increased use of one sense due to sensory deprivation, such as touch in blind people, lead
139                                              Sensory deprivation, such as whisker deprivation, is one
140 process in a higher mammal model of complete sensory deprivation, the congenitally deaf cat.
141 how that in young adolescent mice, long-term sensory deprivation through whisker trimming prevents ne
142 logical account for neural changes following sensory deprivation, thus closing the gap between cellul
143 pression (LTD) of cortical synapses, but how sensory deprivation triggers LTP and LTD in vivo is unkn
144 t plasticity after early restricted neonatal sensory deprivation was analyzed in barrel field cortex
145                                              Sensory deprivation was done by whisker plucking, and sy
146                                    Selective sensory deprivation was induced by trimming two whiskers
147 urons is unchanged at P28 and P104 following sensory deprivation, whereas nrg3 expression by excitato
148 he greatest decrement in synchrony following sensory deprivation, while neurons with diverse inputs f
149 evel of Abeta-LTMR activity in rat models of sensory deprivation (whisker clipping, tail suspension,
150 somatosensory cortex remodels in response to sensory deprivation, with regions deprived of input inva

 
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