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1 t this GABA/peptidergic projection modulates septohippocampal activity and hippocampal theta rhythm r
2 ssfully accounts for changes in behavior and septohippocampal activity observed in studies of the acq
3                                          Our septohippocampal and caudocentral divisions are topograp
4  life leads to a pathological cascade within septohippocampal and cortical brain circuits.
5  topographically comparable to the mammalian septohippocampal and septofimbrial nuclei, respectively,
6 d cholinergic neurons that contribute to the septohippocampal and septohabenular pathways exhibit som
7 ve studied the time course of changes in the septohippocampal and the hippocampal commissural project
8 eural circuitry of fear and anxiety, but the septohippocampal axis also plays an important role.
9    Eighteen MS-DB neurons were identified as septohippocampal by antidromic activation.
10 ne fibres were found in the vicinity of both septohippocampal cholinergic and GABAergic cell types.
11  loss would also obtund histamine effects on septohippocampal cholinergic and GABAergic functions and
12                 The relationship between the septohippocampal cholinergic and GABAergic systems and w
13 rogenesis supports memory by maintaining the septohippocampal cholinergic circuit across the lifespan
14 eficits that, in part, reflect disruption of septohippocampal cholinergic function.
15 rom a direct histamine-induced activation of septohippocampal cholinergic neurones and a subsequent i
16 rodegenerative disorders involving a loss of septohippocampal cholinergic neurones as such a loss wou
17 chanisms whereby acetylcholine released from septohippocampal cholinergic neurons acts to modulate hi
18 gonists in the MSDB involve an excitation of septohippocampal cholinergic neurons and a subsequent in
19 nation, there is age-related degeneration of septohippocampal cholinergic neurons and astrocytic hype
20                                        Adult septohippocampal cholinergic neurons are dependent on tr
21 hat are associated with a loss or atrophy of septohippocampal cholinergic neurons cannot be attribute
22 ling technique to selectively visualize live septohippocampal cholinergic neurons in rat brain slices
23 ize that epilepsy-related neuroplasticity of septohippocampal cholinergic neurons is capable of incre
24                              Degeneration of septohippocampal cholinergic neurons results in memory d
25 trophysiological effects of Hcrt peptides on septohippocampal cholinergic neurons that were identifie
26                           Hcrt activation of septohippocampal cholinergic neurons was reversible, rep
27               Both Hcrt1 and Hcrt2 activated septohippocampal cholinergic neurons with similar EC(50)
28 found that muscarinic agonists do not excite septohippocampal cholinergic neurons, instead they inhib
29  locally, presumably via axon collaterals of septohippocampal cholinergic neurons.
30                                          The septohippocampal cholinergic neurotransmission has long
31                The group with lesions of the septohippocampal cholinergic pathway displayed performan
32 lved in working/episodic memory but that the septohippocampal cholinergic pathway is either not contr
33                      The contribution of the septohippocampal cholinergic pathway to performance of a
34 e MSDB does not decrease impulse flow in the septohippocampal cholinergic pathway; instead, it decrea
35                       The septocingulate and septohippocampal cholinergic pathways were selectively d
36 roprotective role through maintenance of the septohippocampal cholinergic phenotype and preservation
37 tal, low-level lead exposure induces loss of septohippocampal cholinergic projection neurons in neona
38 e placed in the FF lesion cavity and induced septohippocampal cholinergic regeneration or sympathetic
39  details of structural reorganization of the septohippocampal cholinergic system in experimental epil
40 haracterized model paradigm that engages the septohippocampal cholinergic system.
41 in the aged mouse, possibly by acting on the septohippocampal cholinergic system.
42 tes aberrant BFCN gene expression within the septohippocampal circuit of trisomic mice through normal
43 en NGF gene expression and experience on the septohippocampal circuitry was assessed by counting retr
44 ergic neurons and influences both septal and septohippocampal circuitry.
45 etically encoded calcium indicators in mouse septohippocampal cocultures, we found interactive but in
46 term memory but suggest that the cholinergic septohippocampal component of this pathway is not requir
47 tion gene expression analysis at the time of septohippocampal deficits in a trisomic mouse model shed
48 activity of protein kinase C (PKC) following septohippocampal denervation of the rat hippocampus.
49 aced into lateral, medial, caudocentral, and septohippocampal divisions, with the lateral and medial
50                    There is also cholinergic septohippocampal dysfunction in the PTD model.
51 eling studies, 20% of parvalbumin-containing septohippocampal GABA neurons colocalized the mu recepto
52           Restoration of impulse flow in the septohippocampal GABA pathway, possibly via M(3) recepto
53 mechanism may be the concomitant decrease in septohippocampal GABA release and a subsequent disruptio
54 zes direct and indirect mechanisms to excite septohippocampal GABA-type neurones in a reversible, rep
55 led neurons, confirmed to be noncholinergic, septohippocampal GABA-type neurons using retrograde mark
56  and a subsequent indirect activation of the septohippocampal GABAergic neurones.
57  NA, via alpha 1-adrenoceptors, excites MSDB septohippocampal GABAergic neurons and influences both s
58 e speculate that Hcrt2-induced activation of septohippocampal GABAergic neurons will, by engaging dis
59 sence of Hcrt receptor-2 immunoreactivity in septohippocampal GABAergic neurons, as well as the prese
60 gesting the presence of alpha 1-receptors on septohippocampal GABAergic neurons.
61 y; instead, it decreases impulse flow in the septohippocampal GABAergic pathway via M(3) muscarinic r
62 sms, caused by increased impulse flow in the septohippocampal GABAergic pathway, may underlie the cog
63 assessing animals for cognitive function and septohippocampal integrity.
64                                              Septohippocampal interactions determine how stimuli are
65 luate NGF gene-experience interaction on the septohippocampal neural circuitry in mice.
66 ts of muscarine on antidromically identified septohippocampal neurons (SHNs).
67 A had the reverse effect, reducing GABAergic septohippocampal neurons and sparing cholinergic neurons
68                                  Cholinergic septohippocampal neurons are affected by circulating est
69 the neurons, we propose that the slow firing septohippocampal neurons are cholinergic whereas the bur
70 gic whereas the burst firing and fast firing septohippocampal neurons are GABAergic.
71                                  Slow firing septohippocampal neurons had significantly slower conduc
72 scent-labeling techniques to identify living septohippocampal neurons in rat brain slices, we now rep
73 ently increased firing in a subpopulation of septohippocampal neurons with fast conducting fibres (me
74 ies, mu-opioids inhibited a subpopulation of septohippocampal neurons with high conduction velocity f
75                                       Of the septohippocampal neurons, four were slow firing neurons,
76   Using recordings from retrogradely labeled septohippocampal neurons, we found that Hcrt2-excited MS
77 olinergic neurons although sparing GABAergic septohippocampal neurons.
78 ulum, and parasubiculum), and in the septum (septohippocampal nucleus and lateral dorsal septum).
79 rnix, septal nuclei, hippocampal commissure, septohippocampal nucleus, fimbria, anteroventral thalami
80 mygdaloid nucleus and dentate gyrus, but the septohippocampal nucleus, lateral septal nuclei, amygdal
81 , the CA1-CA3 fields of the hippocampus, the septohippocampal nucleus, the diagonal band of Broca, th
82 al pathway was only mildly affected, and the septohippocampal pathway and the striatum were both pres
83                                          The septohippocampal pathway contains cholinergic, GABAergic
84                Cholinergic mechanisms in the septohippocampal pathway contribute to several cognitive
85 se seen following surgical disruption of the septohippocampal pathway in adult animals.
86 inergic and catecholaminergic markers in the septohippocampal pathway in rats through fourth month of
87 ion to changes in cholinergic markers in the septohippocampal pathway of the rat.
88 ats in tasks believed to be modulated by the septohippocampal pathway tended to be differentially aff
89 enetically in the adult rat nigrostriatal or septohippocampal pathway using recombinant adeno-associa
90 n the basal forebrain that contribute to the septohippocampal pathway were found to express m2, m3, a
91                                       In the septohippocampal pathway, GABAergic deficits are observe
92 iagonal band (MSDB), which gives rise to the septohippocampal pathway, is a critical locus for the mn
93 septum-diagonal band (MSDB) complex, via the septohippocampal pathway, is thought to be critical for
94 lues that were unchanged in the striatum and septohippocampal pathway, significantly lower in the ant
95 evealed a graded effect of experience on the septohippocampal pathway, with the largest change occurr
96 ibly by coordinating activity in the SCN and septohippocampal pathway.
97 ing and regulating normal functioning of the septohippocampal pathway.
98                Spatial learning requires the septohippocampal pathway.
99 ty of mRNA included the medial habenula; the septohippocampal, periventricular, suprachiasmatic, and
100 loid (Abeta) toxicity and maintenance of the septohippocampal phenotype.
101  that acetylcholine release from cholinergic septohippocampal projections causes a long-lasting GABAe
102  imbalance between GABAergic and cholinergic septohippocampal projections, as produced by septal infu
103         Cultured postnatal day-of-birth (P0) septohippocampal (SH) neurons that were labeled with 1,1
104 e growth factor (NGF) affect the survival of septohippocampal (SH) neurons.
105 M is, however, reciprocally connected to the septohippocampal system and there is strong evidence tha
106                                   The rodent septohippocampal system contains "theta cells," which bu
107 Recent studies have supported a role for the septohippocampal system in the online processing of inte
108 battery of behavioral tests modulated by the septohippocampal system including two versions of the Mo
109                The disruption of cholinergic septohippocampal system may be an important factor in la
110 rmation and provide further evidence for the septohippocampal system's involvement in processing inte
111  do not have reciprocal connections with the septohippocampal system, but form separate neuronal loop
112 ns differ in reciprocal connections with the septohippocampal system, plausibly forming separate neur
113  of two neural systems, the amygdala and the septohippocampal system, to examine possible interaction
114  the subcellular localization of TrkA within septohippocampal terminal fields, two rabbit polyclonal
115  of the 3 main cholinergic pathways-the Ch1 (septohippocampal), the Ch4 (innominatocortical), and the
116                                 Reduction of septohippocampal TrkA receptor expression selectively im

 
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