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1 vealed at least 13 well segregated variants (serovars).
2 genetically similar nontyphoidal Salmonella serovars.
3 identification of closely related Salmonella serovars.
4 y both nontyphoidal and typhoidal Salmonella serovars.
5 uld likely protect against only one or a few serovars.
6 the antigenic basis of the various Listeria serovars.
7 fertility caused by different C. trachomatis serovars.
8 more robust against the most closely related serovars.
9 and a PCR was formulated that differentiated serovar 16 isolates from all 15 known serovars and other
10 used whole-genome sequencing of the proposed serovar 16 reference strain A-85/14 to confirm the prese
13 re, we demonstrate that in a highly virulent serovar 4b strain, two genes gtlB and gttB are responsib
14 nd host interaction, a capsule mutant of the serovar 5 strain HS069 was generated (HS069Deltacap) thr
18 serovar Typhimurium was the most predominant serovar, accounting for 41.8% (188/450) of NTS isolates.
19 Control and Prevention (CDC), several other serovars also contribute to clinical cases of salmonello
20 ghly lethal challenge dose of the homologous serovar and determined protection against other group B
22 tiated serovar 16 isolates from all 15 known serovars and other common respiratory pathogenic/commens
23 ive agent of leptospirosis includes multiple serovars and species of pathogenic leptospires that are
24 icity between two highly abundant Salmonella serovars and suggest that native variation in the expres
25 s is found sporadically throughout different serovars, and several inhibit activation of the innate i
26 demonstrate that a simple pathogen-defining serovar antigen, that mediates bacteriophage susceptibil
29 ighly conserved across different S. enterica serovars, but residue 161, located close to the catalyti
34 lyses were restricted to cases attributed to serovars commonly isolated from wildlife/environment (e.
35 isolation and propagation of L. interrogans serovar Copenhageni strain IC:20:001 in semi-solid media
36 of C3H/HeJ mice with Leptospira interrogans serovar Copenhageni using an enzootic mode of transmissi
37 be applied to humans, we used C. trachomatis serovar D (strain UW-3/Cx) to induce infertility in mice
38 osal and systemic routes with C. trachomatis serovar D (UW-3/Cx) rMOMP and challenged in the ovarian
39 oculations of rhesus macaques with wild-type serovar D strain D/UW-3/Cx or a plasmid-deficient deriva
40 mice were protected against challenges with serovars D (UW-3/Cx), D (UCI-96/Cx), and E (IOL-43) but
41 OMP and challenged in the ovarian bursa with serovars D (UW-3/Cx), D (UCI-96/Cx), E (IOL-43), or F (N
45 gical data show the existence of a sixteenth serovar-designated serovar 16-of A. pleuropneumoniae.
48 iming to better understand the epidemiology, serovar distribution, antimicrobial resistance (AMR), an
51 were decidualised in-vitro, infected with Ct serovar E, and changes in expression of genes of interes
52 omparative genomic analysis reveals that all serovars encode a subset of "core" effectors, suggesting
54 es of S. Typhimurium and Salmonella enterica serovar Enteritidis DeltaguaBA DeltaclpX live oral vacci
57 chick colonization with Salmonella enterica serovar Enteritidis requires a virulence-factor-dependen
59 phimurium, 10% (10) were Salmonella enterica serovar Enteritidis, and 3% (3) were Salmonella enterica
64 stinguished between all previously described serovars except 5 and 12, which were detected by the sam
66 isolation of Salmonella of a wide variety of serovars, from an array of animal feeds, food animals, a
69 sults of 2 patients, each infected only with serovar Ia strains, revealed multiple same-serovar infec
70 nce of international lineages of S. enterica serovars in food production chain is supported by conser
71 o investigate the distribution of Salmonella serovars in MCL and their products, a total of 1287 pre-
77 rovide protection against another Salmonella serovar is determined by the accessibility of shared O A
78 The host range for YSD1 across Salmonella serovars is broad, but not comprehensive, being limited
79 175) and compared with strains from the same serovars isolated from human clinical cases, livestock,
83 stics and genomes of 10 atypical S. enterica serovars linked to multistate foodborne outbreaks in the
86 show that CBA120 infects Salmonella enterica serovar Minnesota, and this host range expansion is like
89 ) in attenuating infectivity across multiple serovars of C. trachomatis without host cell toxicity.
90 primary isolation of two diverse species and serovars of pathogenic leptospires directly from host ki
91 sing standard microbiological techniques and serovars of S. enterica were determined by PCR and/or ag
97 ultures) mice challenged with C. trachomatis serovars of the same complex were protected but not thos
98 ca serovar Typhi, whereas 86% (131/152) were serovars other than Typhi (nontyphoidal Salmonella).
101 a focused minireview on Salmonella enterica serovar Panama, a serovar responsible for invasive salmo
104 i or Salmonella enterica subspecies enterica serovar Paratyphi A or C were only isolated in 14 (0.03%
106 and the expression of SPI-1 in the typhoidal serovarS Paratyphi A compared to that of the nontyphoida
107 e chronic shedding of Leptospira interrogans serovar Pomona in California sea lions (Zalophus califor
110 , whether infection or immunization with one serovar provides protection against other serovars has n
111 abolically with most, if not all, Salmonella serovars, representing a novel approach to control of th
112 iew on Salmonella enterica serovar Panama, a serovar responsible for invasive salmonellosis worldwide
115 CVD 1944 protected mice against the group D serovar Salmonella enterica serovar Dublin (85% vaccine
116 CVD 1931 protected mice against the group B serovar Salmonella enterica serovar Stanleyville (91% va
117 sistance against challenge with nontyphoidal serovar Salmonella Enteritidis than with another nontyph
119 sing infectious bacteria Salmonella enterica serovar (Salmonella typhi) in 10 muL of sample volume.
120 hat immunization of mice with live typhoidal serovar, Salmonella Typhi, generates cross-reactive immu
124 with Salmonella enterica subspecies enterica serovar Senftenberg are often associated with exposure t
128 onstruct promoted strong immune responses to serovar-specific epitopes, the conserved LNPTIAG epitope
130 inst the group B serovar Salmonella enterica serovar Stanleyville (91% vaccine efficacy), and S. Ente
131 rica serovar Paratyphi A is a human-specific serovar that, together with Salmonella enterica serovar
134 found that, in comparison with a noninvasive serovar, the invasive Salmonella strains Ty2 and D23580
135 this new niche support a bloom of Salmonella serovars, thereby ensuring transmission of the pathogen
136 protective Abs elicited with one Salmonella serovar to engage with and consequently provide protecti
137 ion of typhoidal and nontyphoidal Salmonella serovars to invasive disease varies considerably in plac
138 sia, multidrug-resistant Salmonella enterica serovar Typhi (S Typhi) has been the main cause of enter
141 d by Salmonella enterica subspecies enterica serovar Typhi (S. Typhi) and can lead to systemic illnes
143 d subsequent invasion of Salmonella enterica serovar Typhi (S. Typhi), a human-restricted pathogen.
146 Salmonella enterica subspecies enterica serovar Typhi (Salmonella Typhi) is the cause of typhoid
147 ovar that, together with Salmonella enterica serovar Typhi and Salmonella enterica serovar Sendai, ca
150 Multiyear epidemics of Salmonella enterica serovar Typhi have been reported from countries across e
151 h the causative pathogen Salmonella enterica serovar Typhi implicated in many outbreaks through histo
154 yphoid fever case with a Salmonella enterica serovar Typhi isolate showing extended spectrum beta-lac
156 Salmonella enterica subspecies enterica serovar Typhi or Salmonella enterica subspecies enterica
158 e used a live attenuated Salmonella enterica serovar Typhi strain to create a bivalent mucosal plague
159 ed a Salmonella enterica subspecies enterica serovar Typhi strain with resistance against beta-lactam
160 ctamase (ESBL)-producing Salmonella enterica serovar Typhi was identified, whole-genome sequence type
161 Salmonella enterica and Salmonella enterica serovar Typhi, and Yersinia pestis), and 3 protozoa (Lei
162 agent of typhoid fever, Salmonella enterica serovar Typhi, can partially subvert this critical innat
163 threat has increased in Salmonella enterica serovar Typhi, driven in part by the emergence of succes
164 Enteric fever, caused by Salmonella enterica serovar Typhi, is an important public health problem in
166 is a virulence factor of Salmonella enterica serovar Typhi, the causative agent of typhoid fever, and
167 ellae, 14% (21/152) were Salmonella enterica serovar Typhi, whereas 86% (131/152) were serovars other
171 enteric fever caused by Salmonella enterica serovars Typhi and Paratyphi is substantial and has high
172 degrees C-42 degrees C) Salmonella enterica serovars Typhi, Paratyphi A, and Sendai significantly at
173 terial pathogens such as Salmonella enterica serovar Typhimurium (7.8%), Listeria monocytogenes (3.88
175 sinia enterocolitica and Salmonella enterica serovar Typhimurium (all gram-negative bacteria) and Sta
177 araginase II produced by Salmonella enterica serovar Typhimurium (S Typhimurium) inhibits T cell resp
183 tory system PhoP/PhoQ of Salmonella enterica serovar Typhimurium (S. Typhimurium) in mildly acidic pH
184 f human macrophages with Salmonella enterica serovar Typhimurium (S. Typhimurium) leads to inflammaso
185 model of infection with Salmonella enterica serovar Typhimurium (STM) to identify changes in intesti
187 tection of low levels of Salmonella enterica serovar typhimurium and enteritidis in blood samples wit
191 pathogens that use T3SS, Salmonella enterica serovar Typhimurium and Yersinia pseudotuberculosis.
192 es, Escherichia coli and Salmonella enterica serovar Typhimurium as model microbes, a common redox ac
193 d the vT3SS and fT3SS of Salmonella enterica serovar Typhimurium at ~5 and ~4 nm resolution using ele
194 The food-borne pathogen Salmonella enterica serovar Typhimurium benefits from acute inflammation in
197 intracellular bacterium Salmonella enterica serovar Typhimurium causes persistent systemic inflammat
198 reduced ability to kill Salmonella enterica serovar Typhimurium compared to that of macrophages isol
199 loodstream infections by Salmonella enterica serovar Typhimurium constitute a major health burden in
200 The ST313 pathovar of Salmonella enterica serovar Typhimurium contributes to a high burden of inva
201 lso demonstrate that the Salmonella enterica serovar Typhimurium core promoter is more active than pr
202 inflammatory response induced by Salmonella serovar Typhimurium creates a favorable niche for this g
203 e intracellular pathogen Salmonella enterica serovar Typhimurium decreases H-NS amounts 16-fold when
204 ane vesicles (OMVs) from Salmonella enterica serovar Typhimurium displaying the variable N terminus o
205 t colonization niche for Salmonella enterica serovar Typhimurium during gastrointestinal infections.
206 that Salmonella enterica subspecies enterica serovar Typhimurium employs a dedicated mechanism, drive
207 recent study showed that Salmonella enterica serovar Typhimurium exhibits sliding motility under magn
209 IS) to screen mutants of Salmonella enterica serovar Typhimurium for their ability to infect and grow
210 sis of core genes within Salmonella enterica serovar Typhimurium genomes reveals a high degree of all
211 bilities to infection by Salmonella enterica serovar Typhimurium has just been published in Nature Mi
212 ase and a chitinase from Salmonella enterica serovar Typhimurium hydrolyze LacNAc from Galbeta1-4GlcN
213 ed to restrict growth of Salmonella enterica serovar Typhimurium in host tissues by causing magnesium
217 found that intracellular Salmonella enterica serovar Typhimurium induced the binucleation of a large
218 pathogen and host during Salmonella enterica serovar Typhimurium infection and reveal the molecular i
219 ells more susceptible to Salmonella enterica serovar Typhimurium infection in a NOD1-dependent manner
221 RD9 is suppressed during Salmonella enterica serovar Typhimurium infection, facilitating increased IL
223 e intracellular pathogen Salmonella enterica serovar Typhimurium initiates an anti-inflammatory trans
227 ate immune resistance of Salmonella enterica serovar Typhimurium is attributed to the high-molecular-
228 c mechanism of FrmR from Salmonella enterica serovar Typhimurium is triggered by metals in vitro, and
229 vention for inactivating Salmonella enterica serovar Typhimurium LT2 (ST2) in tender coconut water (T
230 re absent in the closely related S. enterica serovar Typhimurium LT2 and from a mutant of S Montevide
231 al microcompartment from Salmonella enterica serovar Typhimurium LT2, one of the most widely studied
232 li K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, Staphylococcus aureus, and Stre
233 s initially found on the Salmonella enetrica serovar Typhimurium multi-resistance plasmid pMG101 from
234 ompetent, but avirulent, Salmonella enterica serovar Typhimurium mutant for its ability to compete wi
235 escued the virulence defect of a S. enterica serovar Typhimurium mutant specifically defective in its
236 ing turtles, but S. enterica subsp. enterica serovar Typhimurium or lesions associated with Salmonell
237 ture of the prototypical Salmonella enterica serovar Typhimurium pathogenicity island 1 basal body, d
238 viA gene in nontyphoidal Salmonella enterica serovar Typhimurium reduced flagellin-induced pyroptosis
239 teropathogenic bacterium Salmonella enterica serovar Typhimurium requires a T6SS encoded within Salmo
240 ins PrgH and PrgK in the Salmonella enterica serovar Typhimurium Salmonella pathogenicity island 1 (S
241 lpha and Salmonella enterica subsp. enterica serovar Typhimurium secretome (STS)-induced outcomes in
242 se to the catalytic triad, is variable, with serovar Typhimurium SpvD having an arginine and serovar
243 t the genomes of S. enterica subsp. enterica serovar Typhimurium strain LT2 and Salmonella bongori st
244 genetically engineered a Salmonella enterica serovar Typhimurium strain of multilocus sequence type 3
246 ostatic activity against Salmonella enterica serovar Typhimurium that is not shared by the related pu
247 tence of Salmonella enterica subsp. enterica serovar Typhimurium through liver-resident immunoregulat
248 rdinates the response of Salmonella enterica serovar Typhimurium to diverse environmental challenges
249 examined the ability of Salmonella enterica serovar Typhimurium to infect the central nervous system
250 e CspA family members of Salmonella enterica serovar Typhimurium to link the constitutively expressed
251 the intestinal pathogen Salmonella enterica serovar Typhimurium uses specialized metal transporters
253 , N, O, and Q); however, Salmonella enterica serovar Typhimurium was the most predominant serovar, ac
254 ria monocytogenes V7 and Salmonella enterica serovar Typhimurium were used as model pathogens to eval
258 isolates, 40% (41) were Salmonella enterica serovar Typhimurium, 10% (10) were Salmonella enterica s
263 that both flagellins of Salmonella enterica serovar Typhimurium, FliC and FljB, are methylated at su
265 herichia coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogate murine infection m
266 aps of Escherichia coli, Salmonella enterica serovar Typhimurium, Pseudomonas aeruginosa, Bacillus su
267 itiation of phage P22 in Salmonella enterica serovar Typhimurium, revealing how a channel forms to al
269 Unlike the nontyphoidal Salmonella enterica serovar Typhimurium, the genomes of S. Typhi and S. Para
270 al functions, strains of Salmonella enterica serovar Typhimurium, the murine model of S Typhi, in whi
271 III protein secretion in Salmonella enterica serovar Typhimurium, we discovered that several TCMs can
272 in their ability to kill Salmonella enterica serovar Typhimurium, which was rescuable after experimen
273 an efficiently attenuate Salmonella enterica serovar Typhimurium-induced pyroptosis and proinflammato
274 sting the methodology on Salmonella enterica serovar Typhimurium-infected murine bone-marrow-derived
286 ella (NTS), particularly Salmonella enterica serovars Typhimurium and Enteritidis, is responsible for
287 e dominant NTS serovars, Salmonella enterica serovars Typhimurium and Enteritidis, were assessed usin
290 raction was done from Salmonella typhimurium serovars, under the optimized growth conditions for its
291 th the winning metabolic strategy Salmonella serovars use to edge out competing microbes in the infla
292 nvasive NTS from whom 1 of the 4 predominant serovars was isolated in pure culture, 448 (81.0%) were
294 istinct from the previously characterized 15 serovars were described, and a proposal was put forward
297 ons (1.2%) and nontyphoidal Salmonella (NTS) serovars were isolated 10,139 times (6.1%), of which 801
298 ontrolling virulence phenotypes in typhoidal serovars, which is likely to play a role in the distinct
301 f the large and diverse range of species and serovars within the genus Leptospira circulating within