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1 e BALB/cJ mice showed a fivefold increase in serum corticosterone.
2 icular nucleus with a concurrent increase in serum corticosterone.
4 ose of corticosterone, which did not elevate serum corticosterone, acted synergistically with sodium
6 nduced brain levels of 3alpha,5alpha-THP and serum corticosterone, although gender, gonadectomy and h
8 trophin-1) also potentiated the elevation of serum corticosterone and IL-6 levels induced by IL-1.
9 o stresses were observed including increased serum corticosterone and increased dopamine metabolism i
10 l stress-predictive cues exhibited increased serum corticosterone and significantly greater reinstate
11 dator of the rat, on fear-induced behaviors, serum corticosterone, and central dopamine metabolism.
12 Also, they displayed higher levels of blood serum corticosterone, as well as decreased body weight.
13 one in situ showed a significant increase in serum corticosterone associated with visceral hyperalges
15 n, both PBS and OVA-exposed dams had similar serum corticosterone concentration at the start (GD2) an
19 rleukin-6 (IL-6) potentiate the elevation of serum corticosterone induced by suboptimal doses of inte
21 raint stress and a slower descending rate in serum corticosterone level during a 30-min post-stress p
22 sults suggest that the chronically increased serum corticosterone levels contribute to the diabetes o
23 While AED did not antagonize the elevated serum corticosterone levels following acute infection, A
28 w that acute cold stress leading to elevated serum corticosterone levels neither induces LTP-like inc
31 citation across an extended trial range, and serum corticosterone levels were evaluated in response t
34 total percentage DNA methylation levels and serum corticosterone levels, whereas F3 males showed Pb-
39 social deficits are associated with elevated serum corticosterone levels; however, in the F2 and F3 g
43 fact, in IL-6 deficient mice, IL-1 increased serum corticosterone to a level comparable to that obser