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1 proliferation in the presence of respective serum immunoglobulin G.
2 SCI patients also revealed constantly lower serum immunoglobulin G [-0.27 (95% CI: -0.45; -0.10)] an
3 e deficiency, characterized by low levels of serum immunoglobulin G, A, and/or M with loss of antibod
4 ly correlated with the level of prechallenge serum immunoglobulin G against the O127 lipopolysacchari
5 l aplasia, several vaccine/pathogen-specific serum immunoglobulin G and A (IgG and IgA) titers remain
10 ith the level of protection correlating with serum immunoglobulin G anti-protective antigen titers.
12 VRP resulted in high levels of DENV-specific serum immunoglobulin G antibodies and significant titers
14 ll-length block 2 antigens showed that human serum immunoglobulin G antibodies induced by infection c
15 orbent assays (ELISAs) for quantification of serum immunoglobulin G antibodies specific for N. mening
16 uced specific secretory immunoglobulin A and serum immunoglobulin G antibodies that persisted at subs
17 concentrations and neutralizing activity of serum immunoglobulin G antibodies to the RSV prefusion (
18 tion, as well as the specificity, of anti-P1 serum immunoglobulin G antibodies were demonstrated in g
20 d formation, an impaired ability to generate serum immunoglobulin G antibodies, and significant inhib
21 treated patients with LA have high levels of serum immunoglobulin G antibodies, and sometimes low lev
22 cator of inflammation), or edentulism and 2) serum immunoglobulin G antibody response to Aggregatibac
23 e also developed sustained anti-H. hepaticus serum immunoglobulin G antibody responses and elevated a
24 Less marked, but significant, anti-SREHP serum immunoglobulin G antibody responses were also indu
26 PI) owing to the presence of highly elevated serum immunoglobulin G antibody titers with a high avidi
27 cant levels of salivary immunoglobulin A and serum immunoglobulin G antibody to the respective inject
34 elivery of VXA-G1.1-NN elicited VP1-specific serum immunoglobulin G (IgG) and IgA and functional anti
35 ity to S. typhimurium severely depressed the serum immunoglobulin G (IgG) and IgA anti-FrgC response
36 -CAT construct induced significant levels of serum immunoglobulin G (IgG) and IgA antibody to both pa
38 with Invaplex was found to enhance anti-OVA serum immunoglobulin G (IgG) and IgA responses and induc
39 oximately 2-week intervals and evaluated for serum immunoglobulin G (IgG) and IgG subclass and for sa
41 throughout the experiment and evaluated for serum immunoglobulin G (IgG) and IgM and salivary IgA an
43 or no bacteria in the liver and lungs, high serum immunoglobulin G (IgG) and IgM antibody titers, an
44 lpha stimulated strong antigen (Ag)-specific serum immunoglobulin G (IgG) and IgM responses, while MI
45 ion of mice with CVLPs induced gp41-specific serum immunoglobulin G (IgG) and intestinal secretory Ig
46 lones were found to stimulate high levels of serum immunoglobulin G (IgG) and mucosal IgA antibodies
47 luenza virus showed significant increases in serum immunoglobulin G (IgG) and mucosal IgA antibodies
48 oxin (CT) A2 and B subunits (CTA2/B) develop serum immunoglobulin G (IgG) and mucosal IgA antibody re
49 hesizing either PspA fusion protein elicited serum immunoglobulin G (IgG) and mucosal IgA responses a
52 hK63 or LThR72 exhibited high titers of both serum immunoglobulin G (IgG) and mucosal secretory IgA a
53 between the concentration of vaccine-induced serum immunoglobulin G (IgG) and protection against colo
54 ed animals demonstrated significantly higher serum immunoglobulin G (IgG) and salivary IgA antibody l
58 enuation elicited higher Salmonella-specific serum immunoglobulin G (IgG) and/or mucosal secretory-Ig
59 later, significant reductions were seen for serum immunoglobulin G (IgG) antibodies at week 14 and f
61 recombinant proteins were reacted with human serum immunoglobulin G (IgG) antibodies in Western immun
65 ta provide evidence that a critical level of serum immunoglobulin G (IgG) antibodies to the surface p
66 were found to be immunogenic since elevated serum immunoglobulin G (IgG) antibodies without a specif
67 Oral and i.p. vaccines elicited O11-specific serum immunoglobulin G (IgG) antibodies, but IgA was obs
68 isms are found and the induction of specific serum immunoglobulin G (IgG) antibodies, we examined the
69 ay was used to measure the seroprevalence of serum immunoglobulin G (IgG) antibody among NHANES parti
71 ria during the study interval, and increased serum immunoglobulin G (IgG) antibody levels substantiat
72 nce of P. gingivalis in dental plaque and of serum immunoglobulin G (IgG) antibody levels to P. gingi
74 oPn produced greater titers of MoPn-specific serum immunoglobulin G (IgG) antibody than mice that rec
75 zed by enzyme-linked immunosorbent assay for serum immunoglobulin G (IgG) antibody to Aggregatibacter
78 rticular) demonstrated significantly reduced serum immunoglobulin G (IgG) antibody, salivary IgA anti
79 train RB51 (SRB51) produced small amounts of serum immunoglobulin G (IgG) but no IgM antibody to smoo
80 Ribi had the highest levels of PorB-specific serum immunoglobulin G (IgG) by enzyme-linked immunosorb
83 mean titer (GMTs) and 4-fold rise of anti-Vi serum immunoglobulin G (IgG) enzyme-linked immunosorbent
84 ntigenic MDI-albumin products, as defined by serum immunoglobulin G (IgG) from MDI-exposed workers, w
85 ale C57BL/6 mice treated daily with purified serum immunoglobulin G (IgG) from patients with longstan
87 immunosorbent assay to measure beta-specific serum immunoglobulin G (IgG) levels and used it to compa
91 haride (PS) conjugate vaccines may prime for serum immunoglobulin G (IgG) memory responses to meningo
92 transplantations indicate that low levels of serum immunoglobulin G (IgG) negatively impact outcomes.
93 Overall, 23% of patients developed either a serum immunoglobulin G (IgG) response (9%) or sputum IgA
94 . denticola induced a significant (400-fold) serum immunoglobulin G (IgG) response compared to that i
97 ized with replication-defective HSV, durable serum immunoglobulin G (IgG) responses were elicited.
98 e the long-term course of H. pylori-specific serum immunoglobulin G (IgG) responses with respect to s
99 were effective in eliciting antigen-specific serum immunoglobulin G (IgG) responses, and these respon
101 fold increase over the prechallenge anti-SMV serum immunoglobulin G (IgG) titer, mostly subclass IgG1
102 were consistent with significantly declined serum immunoglobulin G (IgG) titers for HHV-6 of MS pati
103 ckerboard analyses of eight plaque bacteria, serum immunoglobulin G (IgG) titers to 17 bacteria, and
105 B) toxoid in saline elicited higher anti-SEB serum immunoglobulin G (IgG) titers when the toxoid was
107 colonization, 9 of 40 patients developed new serum immunoglobulin G (IgG) to OMP CD, as measured by e
108 ollowing infection and who had developed new serum immunoglobulin G (IgG) to their infecting strain w
110 eleased from bovine fetuin, polyclonal human serum immunoglobulin G (IgG), and human alpha1-acid glyc
111 three types of blood isolates: plasma, total serum immunoglobulin G (IgG), and total plasma extracell
112 he most effective means of immunization: the serum immunoglobulin G (IgG), IgA, and IgM anti-fragment
115 s resulted in the induction of FimA-specific serum (immunoglobulin G [IgG] and IgA) and salivary (IgA
116 adermal comparative tuberculin skin test and serum immunoglobulin G [IgG] responses) against a range
117 vated during primary infections (acute-phase serum immunoglobulin G [IgG] titers, <25), compared with
118 nses (HI, antibody-secreting cell [ASC], and serum immunoglobulin G [IgG]) in 15 LRs and 25 control H
119 minent specific anti-CtxB responses in vivo (serum immunoglobulin G [IgG], P </= 0.05; serum IgA, P <
121 ortal vein diameter, splenomegaly, increased serum immunoglobulin G level, and increasing number of a
125 ecific NCSTN variant and support the role of serum immunoglobulin G levels as a potential predictive
128 , with a 44% reduction in intracardiomyocyte serum immunoglobulin G localization in het-treated-8 mic
129 cination geometric mean titre of LT-specific serum immunoglobulin G of 3400.29, compared with 315.41
130 asma could not be duplicated by pooled human serum, immunoglobulin G, or fibrinogen, whether used sep
133 the naturally infected SCID/NCr mice had no serum immunoglobulin G response against H. hepaticus.
134 vaccine doses elicited a strong PA-specific serum immunoglobulin G response with a geometric mean ti
136 ufficient to decrease P. gingivalis-specific serum immunoglobulin G responses, but lower antibody tit
137 osted 4 weeks later induced higher levels of serum immunoglobulin G specific for PspA and for outer m
140 ked immunosorbent assay (ELISA) to determine serum immunoglobulin G titers against Shigella LPS.
141 he vhs(-)/ICP8(-) mutant showed prechallenge serum immunoglobulin G titers comparable to those immuni
142 a clinical oral-health examination, and had serum immunoglobulin G titers measured against 19 period
143 of variants with ChoP correlates with higher serum immunoglobulin G titers to LPS containing this str
148 ut adjuvant stimulated induction of specific serum immunoglobulin G, whereas antigen alone or admixed