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1 rs, WHO performance status of 2, or elevated serum lactate dehydrogenase).
2 nced-stage disease, B symptoms, and elevated serum lactate dehydrogenase.
3 ted closely with hemolysis, as determined by serum lactate dehydrogenase.
4 s, abnormal cytogenetics, and high levels of serum lactate dehydrogenase and beta(2)-microglobulin as
5 had t(14;20), four (4%) of 100 had elevated serum lactate dehydrogenase, and 17 (17%) had two or mor
6 actors, such as volume and sites of disease, serum lactate dehydrogenase, and BRAF mutation status, a
7 te count, disease stage, performance status, serum lactate dehydrogenase, and number of extranodal si
8 deletion or 11q deletion by FISH, increased serum lactate dehydrogenase, and unmutated IGHV mutation
9 esence/absence of liver and lung metastases, serum lactate dehydrogenase, blood neutrophil-lymphocyte
10 s (ie, age, tumor stage, performance status, serum lactate dehydrogenase concentration, and number of
11 operative Oncology Group performance status, serum lactate dehydrogenase concentration, and receipt o
13 with BL, age 40 years, performance status 2, serum lactate dehydrogenase > 3x upper limit of normal,
14 ow Karnofsky performance status (<80%), high serum lactate dehydrogenase (> 1.5 times upper limit of
15 thrombocytopenia (<20,000/microl), elevated serum lactate dehydrogenase (>1,500U/liter), schistocyto
16 oliferation and apoptosis markers as well as serum lactate dehydrogenase isoenzyme 1 (S-LD-1) may hav
17 [HR], 1.9 [95% CI, 1.2 to 2.1]; P = .0009), serum lactate dehydrogenase (LDH) above upper limit of n
18 jury was of sufficient magnitude to increase serum lactate dehydrogenase (LDH) and was oxidative in n
19 opportunistic pneumonia had a higher median serum lactate dehydrogenase (LDH) concentration than did
20 these patients were median age of 56 years; serum lactate dehydrogenase (LDH) greater than 1N, 60%;
21 data showed that a lower pre-lymphodepletion serum lactate dehydrogenase (LDH) level and a favorable
24 e of liver metastases and elevated levels of serum lactate dehydrogenase (LDH) offered superior progn
25 seen even in the elderly, in those with high serum lactate dehydrogenase (LDH) or beta2-microglobulin
26 ion after CD138 plasma cell purification and serum lactate dehydrogenase (LDH) to evaluate their prog
28 RT-PCR results were also correlated with serum lactate dehydrogenase (LDH), treatment status, and
31 uded WHO performance status (0 v 1 or 2) and serum lactate dehydrogenase (LDH; </= v > 1.5x the upper
33 riable and multivariable analysis, increased serum lactate dehydrogenase level and histology for a me
36 d normalization of platelet count, decreased serum lactate dehydrogenase level, and absence of new or
37 ent, significant nodal involvement, elevated serum lactate dehydrogenase level, and prognostic indica
38 n had poor-risk features defined as elevated serum lactate dehydrogenase level, stage IV, and bulky m
42 Patients with ES-SCLC, stratified by sex and serum lactate dehydrogenase levels, were randomly assign
45 e frequent in patients with normal levels of serum lactate dehydrogenase, no bone marrow involvement,
46 (P < .001), age >60 years (P < .001), raised serum lactate dehydrogenase (P = .005), and large-cell t
47 rular filtration rate levels, and had higher serum lactate dehydrogenase, procalcitonin, and interleu
48 btype, and correlated with disease stage and serum lactate dehydrogenase (R(s) = 0.79, P < .001).
49 , primary plasma cell leukaemia and elevated serum lactate dehydrogenase (two times the upper limit o