ライフサイエンスコーパス: serum triglyceride

1 or metolazone increased the concentration of serum triglyceride.

2 tein, fasting glucose-insulin metabolism, or serum triglycerides.

3 f glucokinase regulatory protein (GCKR) with serum triglycerides.

4 correlated with a postprandial elevation of serum triglycerides.

5 correlated with a postprandial elevation in serum triglycerides.

6 tabolizing enzymes and resultant decrease of serum triglycerides.

7 atitis even in the absence of a reduction in serum triglycerides.

8 hanged by adjustment for body mass index and serum triglycerides.

9 ctive in cancer chemoprevention but elevates serum triglycerides.

10 1, of which three rexinoids did not elevate serum triglycerides.

11 , and IL-1beta) compared to mice with normal serum triglycerides.

12 rance, which resulted in decreased levels of serum triglycerides.

13 inefficiency, and improved fetal weights and serum triglycerides.

14 g from rheumatologic conditions and reducing serum triglycerides.

15 Os) have anti-inflammatory effects and lower serum triglycerides.

16 gh blood pressure (1.24; 1.04-1.48) and high serum triglycerides (1.18; 1.00-1.39), with a trend of i

17 into 5 groups according to strata of fasting serum triglycerides: (1) low-normal triglycerides (<100

18 Significant reductions were seen in mean serum triglycerides (1206-->226 mg/dL, P = .002), glucos

19 ed by dyslipidemic (>30% elevated, P < 0.05) serum triglycerides (139 mg/dl), very-LDLs (27.8 mg/dl),

20 ined as 3 or more of the following: elevated serum triglycerides ( 150 mg/dL), low HDL cholesterol (<

21 Changes in serum triglycerides (+2.1 +/- 63.0 vs. +38.9 +/- 37.5 mg

22 y was conducted in 114 patients with fasting serum triglycerides 200-500 mg/dL (2.26-5.65 mmol/L).

23 elihood ratio test suggested interactions on serum triglycerides (4 SNP - SNP pairs), LDL cholesterol

24 detected by magnetic resonance imaging), and serum triglycerides (-51%), improved glucose tolerance,

25 se, except for a dose-dependent elevation in serum triglycerides (a known consequence of hepatic acet

26 r small VLDL), which produced an increase in serum triglycerides; a decrease in LDL size as a result

27 MA-IR, body mass index, waist circumference, serum triglycerides, aminotransferase level, and histolo

28 nuated triglyceride secretion, and decreased serum triglyceride and alanine aminotransaminase levels.

29 Serum SLs correlated significantly with serum triglyceride and cholesterol levels as well as wit

30 Argatroban reduced serum triglyceride and cholesterol levels in mice fed a

31 ding induced hepatic steatosis and increased serum triglyceride and cholesterol levels in the KO mice

32 Corresponding with decreased serum triglyceride and cholesterol levels, DKO mice exhi

33 abolic parameters, UDCA-LPE reduced elevated serum triglyceride and cholesterol values in HFD mice.

34 causes insulin-resistant diabetes, elevated serum triglyceride and fatty acid levels, and massive tr

35 oses of WY14,643 that were tested normalized serum triglyceride and fatty acid levels.

36 ation gene expression in liver and increased serum triglyceride and FFA levels.

37 The fish diet resulted in decreased serum triglyceride and increased HDL-cholesterol concent

38 Serum triglyceride and LDL-cholesterol concentrations we

39 Elevated serum triglyceride and low HDL-cholesterol concentration

40 Guggulipid treatment in rats lowered serum triglyceride and raised serum high density lipopro

41 the gene ANGPTL4 for association with trait serum triglyceride and used ethnicity as a covariate.

42 as associated with a dramatic improvement in serum triglyceride and VLDL concentrations, a significan

43 We concurrently tested serum triglycerides and aminotransferases and estimation

44 was observed between postprandial changes in serum triglycerides and FMD (r = -0.47, p < 0.05).

45 diabetes and FCHL, both predisposing to high serum triglycerides and glucose intolerance, we tested t

46 rol in type 2 diabetics results in increased serum triglycerides and has a negative influence on all

47 Elevated serum triglycerides and low high-density lipoprotein (HD

48 n-3 (omega-3) fatty acid supplementation on serum triglycerides and markers of insulin sensitivity w

49 anied by increased glucose metabolism, lower serum triglycerides and reduced hepatic lipid content in

50 ctivation but displayed significantly higher serum triglycerides and transaminases compared to mice o

51 SB-induced effects were mainly on serum triglycerides and very-low-density lipoprotein (VL

52 ped a complication other than an increase in serum triglycerides and/or cholesterol.

53 (-/-) mice had reduced weight gain, lowered serum triglyceride, and increased serum cholesterol leve

54 erences in the concentrations of hemoglobin, serum triglyceride, and serum cholesterol were found bet

55 ight ratio, alanine transaminase, uric acid, serum triglycerides, and blood pressure.

56 ificant after adjustment for exercise level, serum triglycerides, and BMI (P = 0.02) but was no longe

57 These mice display elevated fat mass, serum triglycerides, and free fatty acids, but blood glu

58 y, obesity, type 2 diabetes mellitus, raised serum triglycerides, and metabolic syndrome.

59 are frequently used strategies for reducing serum triglycerides, and, yet, there is no information r

60 ive correlation of antibody levels and total serum triglycerides, apolipoprotein B, and apolipoprotei

61 serum HDL-cholesterol and a 71% reduction in serum triglycerides at the highest dose administered (10

62 glyceride increases manyfold but circulating serum triglyceride barely fluctuates.

63 VAT was an independent predictor of post-LT serum triglycerides (beta-coefficient 5.49 +/- 2.78, P =

64 2.03]; I(2)=78%), and when the difference in serum triglycerides between the two interventions at fol

65 e to Wy-14,643 effects on beta-oxidation and serum triglycerides but resistant to hepatocellular prol

66 iation between serum PCSK9 concentration and serum triglyceride, but care has to be taken in interpre

67 All five meals significantly raised serum triglycerides, but did not change other lipoprotei

68 Serum triglycerides, but not blood glucose were lower in

69 l/liter (0.258, 0.355) [mean (95% C.I.)] and serum triglyceride by 0.164 mmol/liter (0.12, 0.209) alt

70 Serum triglycerides, cholesterol, and low-density lipopr

71 Concentration of serum triglycerides, cholesterol, insulin and leptin wer

72 ion of hepatic apolipoprotein C-III mRNA and serum triglycerides compared with untreated controls.

73 HFD due to elevated hepatic lipid secretion, serum triglyceride concentration, and lipid droplet accu

74 tudies have reported on associations between serum triglyceride concentrations and the risk of corona

75 sterol concentrations increased slightly and serum triglyceride concentrations decreased slightly in

76 eated men had significantly higher follow-up serum triglyceride concentrations over baseline than did

77 data on lipidomics profiles associated with serum triglycerides concentrations, although these could

78 Liver injury markers were measured in serum, triglyceride content and endocytosis (binding and

79 There was a significant decrease in serum triglycerides (decrease of 19%) in the OI group.

80 ty acid supplementation dramatically reduced serum triglycerides, decreased arachidonic acid in the p

81 entrations (four patients), and increases in serum triglycerides (eight patients) and aspartate amino

82 star controls, HHTg rats exhibited increased serum triglycerides, elevated NEFA and impaired glucose

83 liver steatosis reduction, inflammation and serum triglyceride elevation with ACC inhibition.

84 in clozapine-treated patients; screening for serum triglyceride elevations may be warranted before tr

85 CSWD was associated with improved serum triglycerides (evaluated by mean and median change

86 dominal adiposity; hepatosteatosis; elevated serum triglycerides, FFAs, and LDL-cholesterol; and dimi

87 eased beta-oxidation and lowered hepatic and serum triglycerides, findings consistent with reduced li

88 n dosed chronically in DIO mice and depleted serum triglycerides following a lipid challenge in a dos

89 A significant decrease in serum triglycerides (from 103 to 75, 69 and 72 mg/dL), t

90 ial in treatment-naive T2DM individuals with serum triglyceride >150 mg/dL.

91 en); fasting blood glucose > or = 100 mg/dL; serum triglycerides > or = 150 mg/dL; blood pressure > o

92 7; 95% confidence interval [CI]: 1.15-9.89), serum triglycerides >/=150 mg/day (OR 4.35; 95% CI: 1.70

93 glucose level) and insulin resistance (e.g., serum triglycerides, high density lipoprotein cholestero

94 ncarriers had lower fasting and postprandial serum triglycerides, higher levels of HDL-cholesterol an

95 n assembly and allows homeostatic control of serum triglyceride in a fasted state.

96 that regulate lipid metabolism, and reduced serum triglycerides in a PPARalpha-dependent mechanism.

97 APOC3/APOA5 constitutes a major locus for serum triglycerides in Amerindians, especially the Pimas

98 The decrease in serum triglycerides in aP2/DTA mice was due to a marked

99 eptor protein was positively associated with serum triglycerides in children.

100 ound a statistically significant decrease in serum triglycerides in germ-free rats fed a high sugar d

101 ths; however, by 6 months, blood glucose and serum triglycerides in LIMP2 KO mice were modestly eleva

102 ar role for PPARbeta in regulating levels of serum triglycerides in mice on a high fat Western diet b

103 CK sensitivity best correlated with elevated serum triglycerides in normal-weight participants and wi

104 proportion to the increase in liver fat and serum triglycerides in subjects with PNPLA3-148IIbut not

105 teraction to be statistically significant on serum triglycerides in the 1958BC.

106 e, and high levels of C-reactive protein and serum triglycerides in the blood may be associated with

107 ms in cell-based assays and strikingly lower serum triglycerides in vivo.

108 Serum leptin was doubled and serum triglycerides increased by 44% after LPS administr

109 ing the SBe+MD treatment, VLDL fractions and serum triglycerides increased.

110 sitivity, lower hepatic weight and decreased serum triglycerides indicating a protective phenotype.

111 those of a pharmaceutical dose (3.4 g/d) on serum triglycerides, inflammatory markers, and endotheli

112 are positively correlated with FEV1, whereas serum triglycerides, LDL cholesterol, and apoB are assoc

113 21, P = .048), IMCL (r = 0.27, P = .02), and serum triglyceride level (r = 0.33, P = .001), independe

114 High one-year posttransplant serum triglyceride level and pretransplant body mass ind

115 Female patients experienced serum triglyceride level elevations regardless of antips

116 ssue in preventive cardiology is whether the serum triglyceride level is an independent risk factor f

117 The TG/HDL-C ratio was calculated using the serum triglyceride level over HDL cholesterol and the cu

118 analysis in nonobese patients, only elevated serum triglyceride level was independently associated wi

119 antipsychotic dose, total cholesterol level, serum triglyceride level, and concurrent medications wer

120 patic lipids (IHL), serum cholesterol level, serum triglyceride level, and measures of insulin resist

121 lood pressure, platelet sensitivity, and the serum triglyceride level.

122 um cholesterol and a significant increase in serum triglycerides level.

123 w-carbohydrate diet had greater decreases in serum triglyceride levels (change, -0.84 mmol/L vs. -0.3

124 I (OR: 0.62 per 1 SD; 95% CI: 0.40 to 0.94), serum triglyceride levels (OR: 0.66 per 1 SD; 95% CI: 0.

125 p also experienced a significant decrease in serum triglyceride levels (P = 0.04).

126 at feeding resulted in a greater increase in serum triglyceride levels and an accelerated appearance

127 A good correlation was noted between serum triglyceride levels and blood viscosity (r=0.82).

128 that carry this T-87C polymorphism had lower serum triglyceride levels and significantly reduced risk

129 At week 16, serum triglyceride levels and total cholesterol levels w

130 Serum triglyceride levels at 6 and 12 months were 280 +/

131 eing glucose intolerant, and having elevated serum triglyceride levels compared to any other genotype

132 In atorvastatin treatment groups, total serum triglyceride levels decreased in a dose-dependent

133 During active weight loss, serum triglyceride levels decreased more and high-densit

134 roved adipose tissue lipid storage and lower serum triglyceride levels in the fed state, but do not c

135 h the intestinal epithelium and elevation of serum triglyceride levels in the IAP-deficient mice comp

136 Serum triglyceride levels increase as a result of accumu

137 Geometric mean serum triglyceride levels increased from 118 (95% CI, 11

138 An increase in serum triglyceride levels occurred in clozapine-treated

139 In our study, serum triglyceride levels of P-407 induced mice were ele

140 had increased hepatic lipid accumulation and serum triglyceride levels possibly due to the activation

141 ded otherwise healthy patients with elevated serum triglyceride levels to patients presenting with ac

142 n Old Order Amish participants whose fasting serum triglyceride levels were at the extremes of the di

143 Reductions in insulin resistance and serum triglyceride levels were greater in the combined-i

144 Serum triglyceride levels were increased by incremental

145 sis model assessment values were highest and serum triglyceride levels were lowest among African-Amer

146 iple blood samples were grossly lipemic, and serum triglyceride levels were markedly increased.

147 remental reduction in insulin resistance and serum triglyceride levels when combined with CPAP.

148 Black race, lower BMI, lower serum triglyceride levels, and longer QRS duration were

149 creased body-mass index, waist-to-hip ratio, serum triglyceride levels, and systolic blood pressure a

150 cardial blood volume changed with increasing serum triglyceride levels, indicating lack of vasomotion

151 roved glucose as well as positively affected serum triglyceride levels.

152 which 1.2% energy as n-3 fatty acids lowers serum triglyceride levels.

153 Fish oils rich in n-3 fatty acids reduce serum triglyceride levels.

154 crease in both apolipoprotein expression and serum triglyceride levels.

155 diabetic rats results in a rapid increase in serum triglyceride levels.

156 tions in CRP levels, insulin resistance, and serum triglyceride levels.

157 wo (TRG and 4-Me-UAB30) strikingly increased serum triglyceride levels.

158 lterations in metabolic traits, most notably serum triglyceride levels.

159 ficant association with GFR (P = 0.0006) and serum triglycerides levels (P = 0.003), after accounting

160 sity, as well as between body mass index and serum triglycerides levels with foveal thickness.

161 and body fat and increased hepatic, but not serum, triglyceride levels compared to control rats that

162 d to those without, had significantly higher serum triglycerides, lower PON-1 activity and a higher p

163 e studies suggest that DHA may have stronger serum triglyceride-lowering effects than EPA; however, t

164 he effects of EPA and DHA supplementation on serum triglycerides, markers of lipogenesis, and lipopro

165 al fat correlated positively (p < 0.05) with serum triglycerides, non-high-density lipid cholesterol,

166 hepatic apolipoprotein A-I, C-III mRNA, and serum triglycerides observed in wild-type mice is mediat

167 squares mean=2.9 kg) and a net reduction of serum triglycerides of 32.7 mg/dl.

168 l did not statistically significantly change serum triglycerides or very-low-density lipoprotein conc

169 Although serum triglycerides originate from the liver, the effect

170 emic cardiovascular benefits of exercise, as serum triglyceride, oxidized low density lipoprotein-cho

171 Insulin resistance (P = .01), serum triglycerides (P = .01), C-reactive protein (P = .

172 ion was negatively correlated with change of serum triglycerides (P = 0.016).

173 nts with small nerve fibre damage had higher serum triglycerides (P = 0.02), lower PON-1 activity (P

174 diastolic BP (P = 0.0462, beta = 0.0206) and serum triglycerides (P = 0.0206, beta = 0.1090).

175 ttransplant body mass index (P < 0.001), and serum triglycerides (P = 0.047) independently predicted

176 y mass index (p = 0.01) and higher levels of serum triglycerides (p = 0.05), and they were inversely

177 g06500161 in gene ABCG1 associated both with serum triglycerides (P = 5.36 x 10(-9)) and waist circum

178 iators of pQTLs: Grk5 for fat pad weight and serum triglyceride pQTLs on Chr1, Krtcap3 for fat pad we

179 QTLs on Chr1, Krtcap3 for fat pad weight and serum triglyceride pQTLs on Chr6, Ilrun for a fat pad we

180 and obesity in mice, have elevated levels of serum triglycerides primarily associated with very low d

181 model assessment [HOMA] r = 0.62, p < 0.05), serum triglycerides (r = 0.31, p < 0.05), and inflammati

182 ations between small LDL particle number and serum triglycerides (r=0.61, P<0.0001) and HDL-C (r=-0.5

183 of 1-3 grams of nicotinic acid per day lower serum triglycerides, raise high density lipoprotein chol

184 P=0.03), cell volume (rho(G)=-0.73, P=0.04), serum triglycerides (rho(G)=-0.67, P=0.03), and between

185 dex, resting heart rate, C-reactive protein, serum triglycerides, serum creatinine, and measures of d

186 c blood pressure (SBP), 3000-m running time, serum triglycerides, serum uric acid and waist circumfer

187 t exhibited significantly lower postprandial serum triglycerides, suggestive of a role for TM6SF2 in

188 low-density lipoprotein (LDL) cholesterols, serum triglycerides, systolic and diastolic blood pressu

189 h fasting insulin, insulin sensitivity (Si), serum triglyceride (TG) concentration, or serum HDL chol

190 The increase in serum triglyceride (TG) concentrations in response to a

191 Serum triglyceride (TG) level is a well-known risk facto

192 t to assess the association, if any, between serum triglyceride (TG) levels and gemfibrozil consumpti

193 Serum triglyceride (TG), total cholesterol (TC), high-de

194 Biochemical parameters measured included serum triglyceride (TG), total cholesterol (TC), low-den

195 57; 95%CI 1.45-1.69, P = 3.84 x 10(-31)) and serum triglycerides (TG) (beta = 0.067, P = 4.5 x 10(-21

196 Serum triglycerides (TG) and cholesterol (CHOL) were ass

197 terol value (CHO) was more than 240 mg/dL or serum triglycerides (TG) were more than 200 mg/dL.

198 Sat expression correlates with steatosis and serum triglycerides (TGs) in humans.

199 Also, changes in serum triglycerides (TGs) were evaluated in a group of 5

200 l (TC), low-density lipoproteins (LDLs), and serum triglycerides (TGs) were significantly higher in p

201 o the identification and characterization of serum triglycerides (TGs), was assessed using extracted

202 induction of ApoA4 mRNA, ApoA4 protein, and serum triglycerides that were also sex-dependent.

203 stasis model assessment-insulin resistance), serum-triglyceride-to-serum-high-density lipoprotein-cho

204 Serum triglyceride, total cholesterol, LDL, and VLDL con

205 B. bifidum treatment significantly reduced serum triglycerides, total cholesterol, and LDL-C levels

206 nsplant patients were found to have elevated serum triglycerides, total cholesterol/ high-density lip

207 ciations between endotoxin/LPS and levels of serum triglycerides, troponin, and HDL.

208 e in HDL-cholesterol and an 84% reduction in serum triglycerides under the same treatment conditions.

209 gpat2 deficiency reports marked reduction in serum triglyceride upon feeding a fat-free diet, which s

210 Serum triglyceride values increased after starting indin

211 Serum triglyceride, very-low-density lipoprotein, and ap

212 Es, and the ACC inhibitor-mediated effect on serum triglycerides was mitigated, suggesting that PF-05

213 and high density lipoprotein cholesterol and serum triglyceride were measured after a 14-hour fast.

214 Serum triglycerides were also lower, but the difference