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1 xhibited increased serum ammonia and reduced serum urea.
2 sting status (>/=7 days) was estimated using serum urea and creatinine levels of 1,448 samples collec
3 ol diet, reflected in significantly elevated serum urea and creatinine.
4 rombosis, glomerular crescents, albuminuria, serum urea, and glomerular neutrophil infiltration when
5 highest blood lactate concentration, highest serum urea, and lowest platelet count over the first 24
6 e dose (P <.001), weight (P = .009), and the serum urea concentration (P = .003), and a significant,
7 s and resulted in a significant reduction in serum urea concentration, indicating a decrease in amino
8 ved these conditions, proven by reduction in serum urea, creatinine, cardiac creatine phosphokinase-M
9 s ratio, 4.9 [95% CI, 1.1-22.7]; p = 0.041), serum urea greater than 10 mM (odds ratio, 7.0 [95% CI,
10 rular filtration rate, serum creatinine, and serum urea identified a suggestive peak (p(min) = 1.67 x
11  biosensors to detect glucose level in blood serum, urea level in urine solution, hemoglobin, immunog
12 red with 2.29 +/- 2.18 g/d; P = 0.06), lower serum urea nitrogen (54.1 +/- 13.7 compared with 64.4 +/
13            Vadadustat treatment also lowered serum urea nitrogen and creatinine concentrations and de
14 ed mice had significantly lower increases in serum urea nitrogen and developed significantly less mor
15  the increase in kidney weight by -27.6% and serum urea nitrogen by -53.7% and decreased the incremen
16 t by -49.7%, kidney cyst area by -34.0%, and serum urea nitrogen by -72.8%; these indices were restor
17 on of matrix and four were clearly azotemic (serum urea nitrogen concentration of 40 to 112 mg/dl).
18 val, 1.9 to 6.3; P<0.001) and higher initial serum urea nitrogen concentrations (relative risk of cer
19 ressures of arterial carbon dioxide and high serum urea nitrogen concentrations at presentation and w
20 ailure, acute respiratory failure, and lower serum urea nitrogen concentrations.
21 ssures of arterial carbon dioxide and higher serum urea nitrogen concentrations.
22 rterial pressure, age, respiratory rate, and serum urea nitrogen level.
23  age, female sex, nonblack ethnicity, higher serum urea nitrogen levels, and lower serum albumin leve
24                                              Serum urea nitrogen was significantly decreased during s
25 uestionnaire symptom stability score, higher serum urea nitrogen, and male sex (all P<0.0001).
26 ors for the mortality risk model were higher serum urea nitrogen, male sex, and lower body mass index
27 ry cytokines, 3-HKA improves proteinuria and serum urea nitrogen, overall ameliorating immune-mediate
28 o-potassium ratio; slower rate of decline of serum urea nitrogen, serum creatinine, serum uric acid,
29 ine aminotransferase, lactate dehydrogenase, serum urea nitrogen, total red blood cells, white blood
30 model to predict 30-day mortality, including serum urea nitrogen, white blood cell count, body mass i
31 d the duration of T2DM, age, fibrinogen, and serum urea nitrogen.
32 e grounds (LR, 9.6; 95% CI, 4.0-23.0), and a serum urea nitrogen:creatinine ratio of more than 30 (su
33 tric lavage with blood or coffee grounds, or serum urea nitrogen:creatinine ratio of more than 30 inc